Biologically bonesmashing works

[Walahu]

Simple Visual Map
Impact → Tissue Strain → Capillary Rupture → Subperiosteal Hematoma
↓
Inflammation → Cytokine Release
↓
Periosteal Cell Activation → Osteoblast Differentiation
↓
Collagen Deposition → Woven Bone Formation
↓
Osteoclast Remodeling → Lamellar Bone
↓
Adaptive Remodeling (if mechanical stress persists)

Explanation

Immediate event: Mechanical force applied to bone.
-Tissues involved:
Periosteum – thin, highly vascularized membrane around the bone; contains osteogenic cells.
Bone matrix – dense tissue made of collagen and hydroxyapatite.
Surrounding soft tissue – skin, subcutaneous fat, muscles.
Observation: If the force is mild (like light pressure), no bleeding; if moderate, tiny capillaries rupture; if severe, bone can fracture.

First 0–24 Hours: Hematoma Formation & Inflammatory Response​

If a subperiosteal hematoma forms:

Blood pools under periosteum.
Inflammatory cells (neutrophils, macrophages) migrate to site.
Cytokines released: IL-6, TNF-α, VEGF.
Purpose:
Remove debris and damaged tissue.
Trigger periosteal cells to start differentiating into osteoblasts.

Days 1–7: Cellular Activation & Scaffold Formation​

Osteogenic periosteal cells are activated:

Hematoma acts as biological scaffold:
Platelets release growth factors (PDGF, TGF-β).
Fibroblasts deposit temporary collagen matrix.
Capillary ingrowth (angiogenesis) supports nutrient delivery.

Result: A soft callus begins forming — the first step toward new bone.

Weeks 1–4: Woven Bone Formation​

Osteoblasts deposit collagen type I → mineralizes into woven bone (disorganized but functional).
Micro-fractures heal via Wolff’s Law: stress guides where bone is reinforced.

Key signals:

BMPs (bone morphogenetic proteins) → drive osteoblast differentiation.
VEGF → supports blood vessel growth.
IL-6 → recruits additional osteoprogenitor cells.

Observation: Bone starts “thickening” locally, but shape may not be perfectly symmetric.

---

Weeks 4–12: Remodeling to Lamellar Bone​

Osteoclasts resorb disorganized woven bone.
Osteoblasts lay down structured lamellar bone.

Mechanical stress continues to shape the bone subtly.
Callus becomes more solid and functionally integrated with surrounding bone.

Outcome: Stronger bone in the area, sometimes slightly raised if periosteal lifting occurred.

---

Months 3+: Long-Term Adaptation​

If repeated low-intensity stress occurs (chewing, muscle tension, zygo pressing, bonesmashing)
Bone continues to respond via adaptive remodeling.
“Useful” bone growth is possible but small.
High-impact trauma risks: fibrosis, necrosis, permanent irregularities.


You need to let the tissue heal so my guess is to bonesmash however hard you can for however much time you can every 8-10 days with knuckles or even better a rubber mullet
Thoughts?

 

[rotation]

if I wanted help from ChatGPT I would have asked it myself.

 

[inversions]

You can tell chatgpt that jerking off will give you forward growth and it will believe you

 

[IrishSlayer1483]

Simple Visual Map
Impact → Tissue Strain → Capillary Rupture → Subperiosteal Hematoma
↓
Inflammation → Cytokine Release
↓
Periosteal Cell Activation → Osteoblast Differentiation
↓
Collagen Deposition → Woven Bone Formation
↓
Osteoclast Remodeling → Lamellar Bone
↓
Adaptive Remodeling (if mechanical stress persists)

Explanation

Immediate event: Mechanical force applied to bone.
-Tissues involved:
Periosteum – thin, highly vascularized membrane around the bone; contains osteogenic cells.
Bone matrix – dense tissue made of collagen and hydroxyapatite.
Surrounding soft tissue – skin, subcutaneous fat, muscles.
Observation: If the force is mild (like light pressure), no bleeding; if moderate, tiny capillaries rupture; if severe, bone can fracture.

First 0–24 Hours: Hematoma Formation & Inflammatory Response​

If a subperiosteal hematoma forms:

Blood pools under periosteum.
Inflammatory cells (neutrophils, macrophages) migrate to site.
Cytokines released: IL-6, TNF-α, VEGF.
Purpose:
Remove debris and damaged tissue.
Trigger periosteal cells to start differentiating into osteoblasts.

Days 1–7: Cellular Activation & Scaffold Formation​

Osteogenic periosteal cells are activated:

Hematoma acts as biological scaffold:
Platelets release growth factors (PDGF, TGF-β).
Fibroblasts deposit temporary collagen matrix.
Capillary ingrowth (angiogenesis) supports nutrient delivery.

Result: A soft callus begins forming — the first step toward new bone.

​

Weeks 1–4: Woven Bone Formation​

Osteoblasts deposit collagen type I → mineralizes into woven bone (disorganized but functional).
Micro-fractures heal via Wolff’s Law: stress guides where bone is reinforced.

Key signals:

BMPs (bone morphogenetic proteins) → drive osteoblast differentiation.
VEGF → supports blood vessel growth.
IL-6 → recruits additional osteoprogenitor cells.

Observation: Bone starts “thickening” locally, but shape may not be perfectly symmetric.

---

Weeks 4–12: Remodeling to Lamellar Bone​

Osteoclasts resorb disorganized woven bone.
Osteoblasts lay down structured lamellar bone.

Mechanical stress continues to shape the bone subtly.
Callus becomes more solid and functionally integrated with surrounding bone.

Outcome: Stronger bone in the area, sometimes slightly raised if periosteal lifting occurred.

---

Months 3+: Long-Term Adaptation​

If repeated low-intensity stress occurs (chewing, muscle tension, zygo pressing, bonesmashing)
Bone continues to respond via adaptive remodeling.
“Useful” bone growth is possible but small.
High-impact trauma risks: fibrosis, necrosis, permanent irregularities.


You need to let the tissue heal so my guess is to bonesmash however hard you can for however much time you can every 8-10 days with knuckles or even better a rubber mullet
Thoughts?

these niggas gotta be banned

 

[hej1377]

Dude fondled chat gpts ballsack and still provided no evidence for the contested question which is if visually noticeable levels of bone growth are possible

Get of this site