fleurybobby
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I am trying to figure out ways to increase bone remodeling to safe levels. Bone remodeling is the process of osteoclast resorbing old bone tissue away and osteoblast forming new bone tissue in its place, and osteocytes for the lack of a better analogy responds to stress on the bone so you could say it sends the message to the bones to respond to mewing for example, osteotes will recognize the pressure and communicate to the osteoclast and blast cells.
The body remodels 10% of bone tissue per year on average. That is 0.0274% of bone per day that is remodeled. As vou can see mewing for noticeable results as an adult will take years, luckily remodeling would be accelerated in the area of focus due to the physical stress induced but it is not fast either way Expecting your ramus to grow and an adult and your gonial angle to improve will take years. Best solution I got is to speed up bone remodeling.
Remodeling can not happen without osteoclast, the boogeyman that everyone is trying to reduce to maintain bone mass in older age. You can raise osteoclast to extremes but you need to raise osteoblast to extremes as well to protect yourself from net bone less. Another thing is that without an appropriate amount of osteoblast to go with osteoclast, the bone remodeling is also stunted in that bone movement is brittle rather than pliable and flexible.
Here are the 4 easiest ways I know of to raise osteoclast
The body remodels 10% of bone tissue per year on average. That is 0.0274% of bone per day that is remodeled. As vou can see mewing for noticeable results as an adult will take years, luckily remodeling would be accelerated in the area of focus due to the physical stress induced but it is not fast either way Expecting your ramus to grow and an adult and your gonial angle to improve will take years. Best solution I got is to speed up bone remodeling.
Remodeling can not happen without osteoclast, the boogeyman that everyone is trying to reduce to maintain bone mass in older age. You can raise osteoclast to extremes but you need to raise osteoblast to extremes as well to protect yourself from net bone less. Another thing is that without an appropriate amount of osteoblast to go with osteoclast, the bone remodeling is also stunted in that bone movement is brittle rather than pliable and flexible.
Here are the 4 easiest ways I know of to raise osteoclast
- Inhibit estrogen. This is very effective at raising osteoclast to extremes, perhaps the most effective. Completely crushing estrogen to the ground is too strong though, not even heaps of testosterone for osteoblast and GH can level this perfectly. Also even if we could boost osteoblast enough to counter this we would potentially remodel towards other weak features. Check out this study for instance where an aromatase deficient male was producing 2,000 ng/di test since puberty but had shit tier bone mass and features. Click here
- GH. GH is the best method since it raises both osteoclast and blast together and at a rate where the osteoblast increase is greater for a net bone increase.
- Cortisol.... I don't think anybody wants a cortisol face. Only method I would mess with for this myself is using GHRPZ of GHRP6 over Ipramorelin since either of those nets an increase in bone mass even with the rise in bone resorbing cortisol. (Found alternative ways to increasing osteoclast)
- Prolactin.... nobody wants a lot of this either. Copy what I said about GHRP's from above, same story here. (found alternative ways to increase osteoclast)
- Parathyroid hormone. This s a viable option as well but methods of raising PTH are not so easy. I have yet to find a good solution to this
- Calcitonin. This might increase remodeling, I am finding mixed studies on this however. Salmon contains calcitonin.
- BMP2 increases resorption while also increasing formation and net bone increase
- VEG increases resorption while also increasing formation and net bone increase. This also has similar but more potent bone vascularizing functions to esrogen, might be able to remedy some side effects from too little estrogen.
lam considering both BMP2 and VEGF as novel keys to this solution along with GH.
I have been looking into this subject, I am hoping some people here might want to reserch into this with me.
1am leaning on the idea of cycling high amounts of DHT to antagonize estrogen instead of using an Al since DHT will raise free T for T related effects while DHT in its own will have its own bone anabolic benefits and running GH. I have other ideas but this appears to be the most stable route for now.
