godz
Iron
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bone smashing.
German anatomist and surgeon Julius Wolff formulated Wolff's Law. Wolff's Law is based on the observation that you and your bones are not like a Ken doll. Rather than being completely inanimate structures, your bones are constantly undergoing remodeling, with old or damaged bone continuously being resorbed and replaced by newly laid bone. Applying mechanical force or physical stress to your bones increases the rate at which such remodeling occurs and, in the process, results in firmer, thicker bones. Conversely, lacking such force or stress can lead to thinner and weaker bones. That's one of the reasons why physical activity, especially weight-bearing exercises, can help maintain bone strength and prevent bone loss.
SHIT, I SHOULDN'T have to say, but I can't be too careful
Bone Smashing Doesn't Work
There are several reasons why bone smashing fails as a facial enhancement technique:
Bone smashing is sporadic, undirected trauma. There's no control over where microfractures and swelling occur. You can't reshape bone predictably like working out muscles.
Smashing may microfracture facial bones, but not along defined lines matching your desired result. Such uncontrolled injuries cause haphazard areas of swelling and thickening.
Other Safer, More Effective Options Exist
You now understand why bone smashing is a dangerous DIY practice that is unlikely to improve your facial structure. But if you're seeking facial enhancement, there are effective options available:
Are you still interested in bone smashing?
Well DONT, to be blunt, what could work well? Cyclic loading could and is currently used to speed up bone fractures. Well, you would be adequately causing growth at the bones and would get results in ways you could cyclic loading. Well, loading has never been in a specific way, but it is the application of a load at a particular amplitude more than once. The load is increased from a minimum to a maximum value, then decreased back to the minimum, and repeated. This process can be used to study how load changes affect a material's strength. For example, the Comparison between the humerus cross-section in the playing arm and nonplaying arm of a professional tennis player.
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As seen here, there is growth in the bone by less than a cm, but still, in the jaw, a cm half a cm of more bone means it could make you look a lot different. Here is another example: Radiographs of tibiae from two newborns with reduced tibia loading due to (A) congenital neuromuscular disease and (B) spina-bifida. The reduced mechanical environment of the tibia results in a narrow, underdeveloped diaphysis that remains circular and small rather than triangular and large in cross-section. (A) Reprinted from Rodríguez et al.4 with permission from Springer-Verlag New York Inc. (B) Reprinted from Ralis et al.5 with permission from Cambridge University Press.
Osteocytes within the bone and lining cells on the bone surface are thought to be the primary mechanosensors in bone tissue. Their abundance and connectivity make them an antenna for detecting mechanical strains. These cells are the most abundant in bone — and outnumber osteoblasts and osteoclasts by over 20 to 1. Osteocytes have long dendritic processes that contact surrounding cells at gap junctions. Deformation of the bone matrix caused by mechanical loads is detected by osteocytes, which in turn send paracrine signals to osteoblasts and osteoclasts. Osteocytes form a functional network that passes intracellular calcium signals from cell to cell through gap junctions or by paracrine-like adenosine triphosphate (ATP).7 Lining cells cover over 90% of the surfaces of adult bone, and they contain gap junction connections to osteocytes and osteoblasts. Several genes have been expressed in osteocytes and bone lining cells in vivo after a mechanical stimulus (. These include the proto-oncogene c-fos (30 min after loading) and insulin-like growth factor I (IGF-I) (6 h after loading). Eight osteocytes provide key chemotactic signals that target osteoclasts to repair focal damage and microcracks.9
With all this knowledge and proof, it's safe to say that cyclic loading would work if we could isolate the location down to the mandible and zygomatic bone. Then, yes, it would work. The main issue is figuring out how to cyclic load those bones, which is a partial reason why I made this thread: so people who might be able to think of ideas that could isolate the loading might be able to help, and just general knowledge.
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German anatomist and surgeon Julius Wolff formulated Wolff's Law. Wolff's Law is based on the observation that you and your bones are not like a Ken doll. Rather than being completely inanimate structures, your bones are constantly undergoing remodeling, with old or damaged bone continuously being resorbed and replaced by newly laid bone. Applying mechanical force or physical stress to your bones increases the rate at which such remodeling occurs and, in the process, results in firmer, thicker bones. Conversely, lacking such force or stress can lead to thinner and weaker bones. That's one of the reasons why physical activity, especially weight-bearing exercises, can help maintain bone strength and prevent bone loss.
SHIT, I SHOULDN'T have to say, but I can't be too careful
Bone Smashing Doesn't Work
There are several reasons why bone smashing fails as a facial enhancement technique:
- It doesn't apply controlled, directional forces. Weightlifting triggers bone growth by applying targeted mechanical stress. Bone smashing is uncontrolled blunt force trauma.
- It causes microfractures and swelling, not bone growth. The trauma leads to swelling and inflammation, which are not beneficial for remodeling.
- It can't produce symmetrical, predictable results. Injury patterns are random rather than controlled.
- It risks severe fractures and breaks. Facial bones are vulnerable to fracture and can require complex surgery for repair.
It Doesn't Follow Wolff's Law
Wolff's Law requires controlled, cyclic loading along stress lines to thicken bone. Think targeted resistance exercise.Bone smashing is sporadic, undirected trauma. There's no control over where microfractures and swelling occur. You can't reshape bone predictably like working out muscles.
Smashing may microfracture facial bones, but not along defined lines matching your desired result. Such uncontrolled injuries cause haphazard areas of swelling and thickening.
Other Safer, More Effective Options Exist
You now understand why bone smashing is a dangerous DIY practice that is unlikely to improve your facial structure. But if you're seeking facial enhancement, there are effective options available:
- Fat grafting involves strategically injecting fat cells to add subtle volume and shape.
- Facial fillers like Voluma and Radiesse can plump facial contours.
- Customized facial implants can be inserted surgically to augment structure.
- Osteoplasty surgically sculpts bone for precision reshaping, e.g., genioplasty.
- MyEllevate is an innovative, minimally invasive procedure to enhance the neck and jawline.
Are you still interested in bone smashing?
Well DONT, to be blunt, what could work well? Cyclic loading could and is currently used to speed up bone fractures. Well, you would be adequately causing growth at the bones and would get results in ways you could cyclic loading. Well, loading has never been in a specific way, but it is the application of a load at a particular amplitude more than once. The load is increased from a minimum to a maximum value, then decreased back to the minimum, and repeated. This process can be used to study how load changes affect a material's strength. For example, the Comparison between the humerus cross-section in the playing arm and nonplaying arm of a professional tennis player.
As seen here, there is growth in the bone by less than a cm, but still, in the jaw, a cm half a cm of more bone means it could make you look a lot different. Here is another example: Radiographs of tibiae from two newborns with reduced tibia loading due to (A) congenital neuromuscular disease and (B) spina-bifida. The reduced mechanical environment of the tibia results in a narrow, underdeveloped diaphysis that remains circular and small rather than triangular and large in cross-section. (A) Reprinted from Rodríguez et al.4 with permission from Springer-Verlag New York Inc. (B) Reprinted from Ralis et al.5 with permission from Cambridge University Press.
Osteocytes within the bone and lining cells on the bone surface are thought to be the primary mechanosensors in bone tissue. Their abundance and connectivity make them an antenna for detecting mechanical strains. These cells are the most abundant in bone — and outnumber osteoblasts and osteoclasts by over 20 to 1. Osteocytes have long dendritic processes that contact surrounding cells at gap junctions. Deformation of the bone matrix caused by mechanical loads is detected by osteocytes, which in turn send paracrine signals to osteoblasts and osteoclasts. Osteocytes form a functional network that passes intracellular calcium signals from cell to cell through gap junctions or by paracrine-like adenosine triphosphate (ATP).7 Lining cells cover over 90% of the surfaces of adult bone, and they contain gap junction connections to osteocytes and osteoblasts. Several genes have been expressed in osteocytes and bone lining cells in vivo after a mechanical stimulus (. These include the proto-oncogene c-fos (30 min after loading) and insulin-like growth factor I (IGF-I) (6 h after loading). Eight osteocytes provide key chemotactic signals that target osteoclasts to repair focal damage and microcracks.9
With all this knowledge and proof, it's safe to say that cyclic loading would work if we could isolate the location down to the mandible and zygomatic bone. Then, yes, it would work. The main issue is figuring out how to cyclic load those bones, which is a partial reason why I made this thread: so people who might be able to think of ideas that could isolate the loading might be able to help, and just general knowledge.
Take everything here with a grain of salt ALSO AS I SAID THIS IS JUST TO LET PEOPLE KNOW
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