Cjc1295 with DAC or HGH for height maxing (14yo 5’5)

[Temporalcel]

I’m 14 and my plates are still open. My dad is 5’10 and my mom is 5’4 my dad was nutrient deprived as a child as he grew up in the third world and so was my mom. I want to heightmog as I am a 4.5/10 facially. I want to do hgh but there is the downside of cartilage growth with would make my ears and nose bigger. I know cjc1295 is less effective and ill atmost grow 1-2 past height potential but it is safer. Are there any alternatives?

 

[Deroga]

MK 25-50mg/ml with an Aromatase Inhibitor such as Letrozole (2.5mg) or Anastrozole (1mg)

 

[BR32]

MK 25-50mg/ml with an Aromatase Inhibitor such as Letrozole (2.5mg) or Anastrozole (1mg)

bro wants to stunt his growth

 

[Deroga]

bro wants to stunt his growth

? Are you retarded Mk with an AI encourages growth

 

[BR32]

HGH

Don't use an AI

CJC is not safer

HGH is perfectly safe

 

[BR32]

? Are you retarded Mk with an AI encourages growth

Can we drop the MK stuff already. I literally disproved your entire argument yesterday.

MK is shit, if it actually worked well it would be used clinically.

An AI at 14 at the doses you are suggesting would ruin this kids life forever.

You are extremely overconfident and uneducated

 

[Deroga]

Don't use an AI

It's an axis son, you realize estrogen encourages epiphyseal closure?

 

[Deroga]

Can we drop the MK stuff already. I literally disproved your entire argument yesterday.

MK is shit, if it actually worked well it would be used clinically.

An AI at 14 at the doses you are suggesting would ruin this kids life forever.

You are extremely overconfident and uneducated

No you quite literally didn't respond to Zagro who debunked your points.

"MK is shit" provides zero points BAHAHAHA

Yeah bro you shud inject estrogren!

 

[Temporalcel]

MK 25-50mg/ml with an Aromatase Inhibitor such as Letrozole (2.5mg) or Anastrozole (1mg)

HGH

Don't use an AI

CJC is not safer

HGH is perfectly safe

I’m also thinking of the cartilage growth as I don’t really want to have my nose grow and bigger. There is also the chance I grow to 5’11 or 6’0 naturally and I don’t need to take anything

 

[Deroga]

Can we drop the MK stuff already. I literally disproved your entire argument yesterday.

MK is shit, if it actually worked well it would be used clinically.

An AI at 14 at the doses you are suggesting would ruin this kids life forever.

You are extremely overconfident and uneducated

@Zagro brah im so done

 

[BR32]

It's an axis son, you realize estrogen encourages epiphyseal closure?

Faggot you realize you need E2 to grow bones

He is 14

Its not as simple as "just take an AI and your plates won't close"

Have you heard of tissue specific aromatizaiton?

Also you do so much more damage taking an AI right now

 

[BR32]

I’m also thinking of the cartilage growth as I don’t really want to have my nose grow and bigger. There is also the chance I grow to 5’11 or 6’0 naturally and I don’t need to take anything

Your nose won't grow big unless you're taking 10+ IU for years straight even then it won't be much

 

[BR32]

@Zagro brah im so done

You have zero response cause you don't know shit

 

[Temporalcel]

Your nose won't grow big unless you're taking 10+ IU for years straight even then it won't be much

If I take 5IUs will I grow to 6’1-6’2?

 

[Temporalcel]

If I take 5IUs will I grow to 6’1-6’2?

Sorry if I sound clueless

 

[Zagro]

You have zero response cause you don't know shit

"Faggot you realize you need E2 to grow bones"

provide one single piece of literature to back this claim up

 

[BR32]

"Faggot you realize you need E2 to grow bones"

provide one single piece of literature to back this claim up

let me rephrase

You need E2 for your bones to properly develop during puberty.

 

[Deroga]

Faggot you realize you need E2 to grow bones

He is 14

Its not as simple as "just take an AI and your plates won't close"

Have you heard of tissue specific aromatizaiton?

Also you do so much more damage taking an AI right now

Why are you assuming my reasoning is as linear as "suppress e2 -> plates stay open -> more height" yeah obviously I know e2 is required for growth velocity.

Bro ur fucking retarded, MK indirectly raises ur test u need an AI to regulate it

 

[Zagro]

You need E2 for your bones to properly develop during puberty.

Provide one piece of literature to back this claim up

 

[Zagro]

I know e2 is required for growth velocity.

It isn't

 

[Temporalcel]

Why are you assuming my reasoning is as linear as "suppress e2 -> plates stay open -> more height" yeah obviously I know e2 is required for growth velocity.

Bro ur fucking retarded, MK indirectly raises ur test u need an AI to regulate it

So I should take MK and and AI? What is MK?

 

[Deroga]

So I should take MK and and AI? What is MK?

It's an oral GH secretagogue, whereas rHGH is not oral and endogenous.

 

[BR32]

Provide one piece of literature to back this claim up

Here are five

Rochira et al. (2015) — aromatase-deficient males who can't convert testosterone to estradiol develop severe osteoporosis despite normal testosterone. Open plates and destroyed bone density simultaneously. E2 deficiency, not T deficiency, is the cause. https://pmc.ncbi.nlm.nih.gov/articles/PMC4383300/

ERα knockout mice (2012) — removing the estrogen receptor produces failure of normal bone formation entirely independent of plate closure. Molecular proof, not just correlation. https://journals.physiology.org/doi/full/10.1152/ajpendo.00646.2011

GnRHa suppression study — blocking estradiol in postpubertal animals reduced trabecular bone volume by ~50% and increased endocortical resorption. This is the mechanism at work when blockers are used without E2 replacement. https://pmc.ncbi.nlm.nih.gov/articles/PMC3721967/

Hormonal Contraception & Bone Health (2020) — nearly half of peak bone mass is acquired during puberty, with the most rapid mineral accrual occurring after peak height velocity. Estrogen deficiency during this window has permanent skeletal consequences. https://pmc.ncbi.nlm.nih.gov/articles/PMC7472551/

Cutler et al. — blocking aromatization in boys with precocious puberty (testosterone still in adult male range) normalized growth velocity, proving the pubertal growth spurt is driven by E2, not T directly. https://www.sciencedirect.com/science/article/abs/pii/S0960076097800052"

Read up buddy

 

[Zagro]

So I should take MK and and AI? What is MK?

Don't use anything bro spend your time researching these shit instead you seem absolutely clueless

 

[Sub5kang]

MK 25-50mg/ml with an Aromatase Inhibitor such as Letrozole (2.5mg) or Anastrozole (1mg)

 

[herculean]

If I take 5IUs will I grow to 6’1-6’2?

u need to assess other metrics first.
get an xray done of your hand or knee to see what stage your growth plates are at. if you live somewhere with free healthcare you can do the “pretend broke bone” method. i can explain more if u don’t know.

what are ur parents heights?
what has your height velocity been?

you are too young to use AIs they will mess up ur brain.

HGH is your friend, don’t do MK677 or GHRPs GHRHs. I did MK677 with GHRPs and GHRHs in highschool but would have gotten way better gains if I just stuck with HGH

 

[Deroga]

Explain or you're gay

 

[Sub5kang]

Don't use anything bro spend your time researching these shit instead you seem absolutely clueless

Literally
Guys talking about hopping on at 14 when all I was worried about was hopping on Fortnite with my mates

 

[herculean]

Don't use anything bro spend your time researching these shit instead you seem absolutely clueless

Yeah 100% bro, you’re 14 you need to read up on the topic more. Look into pubertal endocrinology.
good source is endotext.org if u feel up to it. Might be hard at first but sticking with reading instead of watching videos will IQmaxx u

 

[Deroga]

Here are five

Rochira et al. (2015) — aromatase-deficient males who can't convert testosterone to estradiol develop severe osteoporosis despite normal testosterone. Open plates and destroyed bone density simultaneously. E2 deficiency, not T deficiency, is the cause. https://pmc.ncbi.nlm.nih.gov/articles/PMC4383300/

ERα knockout mice (2012) — removing the estrogen receptor produces failure of normal bone formation entirely independent of plate closure. Molecular proof, not just correlation. https://journals.physiology.org/doi/full/10.1152/ajpendo.00646.2011

GnRHa suppression study — blocking estradiol in postpubertal animals reduced trabecular bone volume by ~50% and increased endocortical resorption. This is the mechanism at work when blockers are used without E2 replacement. https://pmc.ncbi.nlm.nih.gov/articles/PMC3721967/

Hormonal Contraception & Bone Health (2020) — nearly half of peak bone mass is acquired during puberty, with the most rapid mineral accrual occurring after peak height velocity. Estrogen deficiency during this window has permanent skeletal consequences. https://pmc.ncbi.nlm.nih.gov/articles/PMC7472551/

Cutler et al. — blocking aromatization in boys with precocious puberty (testosterone still in adult male range) normalized growth velocity, proving the pubertal growth spurt is driven by E2, not T directly. https://www.sciencedirect.com/science/article/abs/pii/S0960076097800052"

Read up buddy

"YOO CHATGPT, please find me 5 studies on why E2 is required for growth"

 

[Sub5kang]

Explain or you're gay

Funnily enough today I went through my old threads and I found one I made as a grey saying Jordan Barrett kissed me turning me gay

 

[Zagro]

Here are five

Rochira et al. (2015) — aromatase-deficient males who can't convert testosterone to estradiol develop severe osteoporosis despite normal testosterone. Open plates and destroyed bone density simultaneously. E2 deficiency, not T deficiency, is the cause. https://pmc.ncbi.nlm.nih.gov/articles/PMC4383300/

ERα knockout mice (2012) — removing the estrogen receptor produces failure of normal bone formation entirely independent of plate closure. Molecular proof, not just correlation. https://journals.physiology.org/doi/full/10.1152/ajpendo.00646.2011

GnRHa suppression study — blocking estradiol in postpubertal animals reduced trabecular bone volume by ~50% and increased endocortical resorption. This is the mechanism at work when blockers are used without E2 replacement. https://pmc.ncbi.nlm.nih.gov/articles/PMC3721967/

Hormonal Contraception & Bone Health (2020) — nearly half of peak bone mass is acquired during puberty, with the most rapid mineral accrual occurring after peak height velocity. Estrogen deficiency during this window has permanent skeletal consequences. https://pmc.ncbi.nlm.nih.gov/articles/PMC7472551/

Cutler et al. — blocking aromatization in boys with precocious puberty (testosterone still in adult male range) normalized growth velocity, proving the pubertal growth spurt is driven by E2, not T directly. https://www.sciencedirect.com/science/article/abs/pii/S0960076097800052"

Read up buddy

None prove your point

Aromatase deficiencies=from birth deficiency
ERa knockout=Estrogen receptor alpha doesn't exist
GnRHa suppression=both t and e are affected

Do you want me to read the studies you tried to use to "prove" your point? You will get embarrassed though it's up to you

 

[Deroga]

Funnily enough today I went through my old threads and I found one I made as a grey saying Jordan Barrett kissed me turning me gay

To be honest I couldn't care less about you're attraction for the male sex but at least explain on ur GIF

 

[Deroga]

None prove your point

Aromatase deficiencies=from birth deficiency
ERa knockout=Estrogen receptor alpha doesn't exist
GnRHa suppression=both t and e area affected

Do you want me to read the studies you tried to use to "prove" your point? You will get embarrassed though it's up to you

He just asked ChatGPT to find him studies for what he was arguing

 

[herculean]

Why are you assuming my reasoning is as linear as "suppress e2 -> plates stay open -> more height" yeah obviously I know e2 is required for growth velocity.

Bro ur fucking retarded, MK indirectly raises ur test u need an AI to regulate it

bro “MK raises your test” lmaoo. where did u get this??? saying he needs AIs. ur acting as if he’s blasting test

 

[Zagro]

He just asked ChatGPT to find him studies for what he was arguing
View attachment 4887969

Yeah i know you don't even need a detector because one of the studies prove my point, three of them are completely different concepts and only one of them could be extrapolated from

Took him 3 minutes too

 

[Sub5kang]

To be honest I couldn't care less about you're attraction for the male sex but at least explain on ur GIF

Your point is mk indirectly increases test no?

 

[Deroga]

bro “MK raises your test” lmaoo. where did u get this??? saying he needs AIs. ur acting as if he’s blasting test

yess bro user xyz who joined yesterday and is "bped from 13" must know more!

u realize the whole point of mk is igf-1 production which acts directly on the leydig cells brother

mk raises gh -> raises igf-1 -> leydig cells become more efficient -> increase in test output (espeically free t) and higher test increases e2 bhai

i mean maybe if u want to do 0.5mg ana

 

[Deroga]

Your point is mk indirectly increases test no?

obviously it does

 

[Sub5kang]

obviously it does

Then why not just pin test
Or is this guy one of those "oral onlys"

 

[Deroga]

Then why not just pin test
Or is this guy one of those "oral onlys"

what? i think u missed my whole point

mks main point is to not increase test its to increase gh and igf-1 production and height growth, but it also indirectly increases ur test (a side effect of elevated igf-1) which -> increases ur e2 -> makes ur plates close faster

so u combat this with an ai like anastrozole such as what i'm doing u, 0.5-1mg

 

[Temporalcel]

He just asked ChatGPT to find him studies for what he was arguing
View attachment 4887969

lol

 

[Temporalcel]

Yeah 100% bro, you’re 14 you need to read up on the topic more. Look into pubertal endocrinology.
good source is endotext.org if u feel up to it. Might be hard at first but sticking with reading instead of watching videos will IQmaxx u

Alr thanks

 

[Sub5kang]

what? i think u missed my whole point

mks main point is to not increase test its to increase gh and igf-1 production and height growth, but it also indirectly increases ur test (a side effect of elevated igf-1) which -> increases ur e2 -> makes ur plates close faster

so u combat this with an ai like anastrozole such as what i'm doing u, 0.5-1mg

Then why rather mk over straight up GH with no test conversion this no need for an AI

 

[Deroga]

Then why rather mk over straight up GH with no test conversion this no need for an AI

I proved this yesterday, Mk at a higher dose is sysmeticly better than anything ~7-8IU or lower of GH

 

[Temporalcel]

Should I buy from a Chinese generic or try and find a pharma grade I already found a Chinese generic that was very good prices. I just don’t want to end up finding cum in my vial.

 

[BR32]

None prove your point

Aromatase deficiencies=from birth deficiency
ERa knockout=Estrogen receptor alpha doesn't exist
GnRHa suppression=both t and e are affected

Do you want me to read the studies you tried to use to "prove" your point? You will get embarrassed though it's up to you

Fair on the first three, but you skipped the Cutler study entirely.

Boys with precocious puberty and testosterone already in the adult male range. They blocked aromatization with testolactone. Testosterone stayed high the whole time, only E2 was reduced. Growth velocity normalized and bone maturation slowed.

No congenital deficiency, no missing receptor, and no HPG suppression. Just high T, blocked conversion to E2, bones stop developing properly.

Please explain this to me because your critiques don't explain it

 

[BR32]

@Deroga

Why are you so addicted to MK

 

[BR32]

Also @Zagro

Were not even talking about all of the other reasons you want normal serum concentrations of E2 during puberty.

Even if an AI did boost growth is it worth all of the horrific side effects that will cause?

Cognitive development, libido/sexual development, Cardiovascular funciton, Joint health, mental health. basic stuff

I'm purely open to having a civil conversation, I value your input

 

[Deroga]

Fair on the first three, but you skipped the Cutler study entirely.

Boys with precocious puberty and testosterone already in the adult male range. They blocked aromatization with testolactone. Testosterone stayed high the whole time, only E2 was reduced. Growth velocity normalized and bone maturation slowed.

No congenital deficiency, no missing receptor, and no HPG suppression. Just high T, blocked conversion to E2, bones stop developing properly.

Please explain this to me because your critiques don't explain it

For the love of God stop using AI

@Zagro this dude can't even respond without AI