Soulrack
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Semen retention is critical for the sexually dimorphic traits of males to manifest. Without it you become an undifferentiated ungendered pawn. Sexual dimorphism is primarily expressed via differing levels of testosterone and estradiol (estrogen in its biologically active form). The final controller of ALL hormone expression is the brain, and specifically the hypothalamus. The only intervention that works to increase testosterone is changing your brain structure. Masturbation is proven to do this.
Before we can even get to semen retention, we have to understand how your body works, and the critically misunderstood roles that hormones play.
Why Testosterone? Most of you, I'm sure, are already convinced about the beneficial effects of testosterone in men, but I want to give a brief overview of things you probably don't know about it. Testosterone increases sociability. It increases connection to and participation in your milieu, this is true for both men and women. For women, this typically manifests as slutty behavior and ultimately ends up as cuckoldry, women with higher testosterone have sluttier faces. They also have greater sexual arousal levels: https://ww.ncbi.nlm.nih.gov/pubmed/10665617 For men however, testosterone decrease the incidence of lying and they are more cooperative with other men. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3468628 Men with high testosterone are fascistic ideal. They build, cooperate, and create civilization. Men with high testosterone are more driven to find not only mates, but to do all things. Testosterone receptors in the brain don't differentiate drive for sex from any other drive (that differentiation does occur, but requires other chemical receptors to be activated as well).
The Hypothalamus is King Androgen/Testosterone boosting supplements don't work unless you are supplementing androgens higher than 100% of your baseline test production. To understand why this is, you have to first understand the HP axes, and specifically the HPG axis. The hypothalamus acts as a prime regulator for almost all hormones related to growth, sex, and homeostasis. Hypothalamus notices an increased level of testosterone? It stops producing GnRH. GnRH is a signal to your pituitary gland to make FSH (follicle-stimulating hormone) and LH (luteinizing hormone); these signal the gonads in men to create sperm and testosterone respectively (in women's gonads these signal menstruation and estrogen). So at all times your hypothalamus is listening to the levels of sex hormones, and is only releasing enough GnRH to keep them at what it believes to be the proper homeostasis. This is why only highly potent anabolic steroids are really the only things which appear to have a phenotypic response in humans. They overpower even the hypothalamus's ability to regulate testosterone. So when taking anabolic steroids your level of GnRH is basically zero. There is no activity on the HPG axis and it permanently changes your hypothalamus to produce less GnRH, and hence why you hear stories of anabolic steroids shrinking your balls and causing infertility. So to maximize your GnRH you need to maximize the hormones which stimulate its production and minimize the ones that inhibit it. There are many hormones which either stimulate or inhibit the production of GnRH, we will get into why fapping is one of the worst things you can do if you want to maximize GnRH next.
There are several other axes of the hypothalamus which should also be mentioned. The hypothalamic—pituitary—prolactin axis (HPP) is responsible for lactation and mammary growth in females, but it is also present in males. It is not directly self-inhibiting like the HPG axis, but instead is inhibited by dopamine (this will seem contradictory at first, but is extremely important to understanding the dangers of masturbation). The main thing for now you should know is that excess prolactin causes typically feminine traits in men. The primary stimulating factor of the HPP axis is estrogen. Remember this is the only axis which isn't a direct feedback loop! More estrogen will always mean more prolactin. Finally, the higher your prolactin is, the lower GnRH output will be, which means the lower testosterone will be in males. The HPS axis has the ultimate byproduct of IGF-1, which is the most critical signal for muscle growth. HPS axis is stimulated by testosterone (via blunting the inhibitory effects of GH in the feedback loop).
Masturbation and Hormones Now to put it all together. Orgasm elevates prolactin massively. Prolactin causes a neurological effect of having little to no sexual desire, hence why you temporarily lose interest in the porn you watch after ejaculation. Now recall the previous section: Prolactin tells the hypothalamus to lower GnRH; GnRH stimulates production of sperm and testosterone; testosterone increases masculine traits and increases social cohesion in men; it also increases expression of muscles via the HPP axis. Dopamine which is high during the build-up of masturbation inhibits the HPP axis though, so how can masturbation be causing an increase of prolactin? Well, the hypothalamus can be very intelligent at what it does. It understands context. Not only that, but the massive acute hit of dopamine you are getting causes something called a hormetic effect. Hormesis is kind of like a biological blowback effect, the hypothalamus is massively inhibited on its HPP axis and then to compensate, it releases even more prolactin than it normally would in all that time as a huge burst after orgasm. Not only that, it appears to have "aftershocks" throughout the next few days and releases prolactin even more. But it gets worse. At least with a single mate in a monogamous sexual relationship your dopamine wouldn't be massively elevated, but with access to unlimited high speed pornography your dopamine will spike to never before seen levels due to the Coolidge effect (https://en.wikipedia.org/wiki/Coolidge_effect). What does this mean? For days after masturbation you will have low testosterone. But there's even more. The hypothalamus is a neurological structure. It learns and adapts. What does chronic masturbation and dopamine spiking do to the HPP axis? It makes it less and less sensitive to regular dopamine levels. Throughout the day its perfectly normal and healthy to have a moderate level of dopamine, and especially so for men. If HPP is no longer inhibited by this normal level of dopamine, then prolactin will be chronically elevated. Finally we get to the crux. Chronically elevated prolactin means a chronically inhibited HPG axis. A chronically inhibited HPG axis means chronically low levels of homeostatic testosterone. This is why the men of today have lower testosterone than the average 80 year old just 50 years ago. This is why we have trannies developing feminine traits due to increased prolactin.
Now there's even more danger to all of this. We have huge amounts of xenoestrogens from overcompensation of plant based foods and hygiene products (you really should only be consuming meat). Estrogen stimulates the HPP axis even more, and we have blown our dopamine circuits to defend against them with acute chronic spiking. But the brain can heal and adapt. Stop looking at porn. Stop masturbating. Stop consuming xenoestrogens. Stay monogamous, and only have sex for the purpose of procreation.
Extra Credit: Dangers of SSRIs and Weed SSRIs increase the re-uptake of serotonin in the brain. Serotonin has an antagonist relationship with dopamine, which means if dopamine is high serotonin is low and vice-versa. SSRIs will chronically lower dopamine, not only taking away your enjoyment of novelty, but also once again activating the HPP axis.
Weed is made of cannibinoids. The body produces its own endocannibinoids which have receptors all throughout the nervous system. The hypothalamus is filled with CB1 receptors which have 2-AG (an natural endocannibinoid) as an agonist. 2-AG is released upon orgasm and is used in the adaptation process mentioned earlier to train the hypothalamus to be less responsive to dopamine. What else is an agonist of the CB1 receptor? THC. In fact THC is far more potent than 2-AG could ever be, meaning that every time you smoke weed, you adapt your hypothalamus to be even less responsive to dopamine than if you were masturbating to porn the entire time you were high. THC is bad news if you value testosterone.
Testosterone molecule is totally inactive in the bloodstream. If's only a messenger which in most cells activates synthesis of certain proteins (hormones, enzymes muscle proteins etc,) The message is carried on only if the testosterone hormone binds to its receptor (androgen receptor, located in the nucleus)
For those interested in medicine: the binding of ligand (now lipid soluble steroid hormone) to ifs receptor (now androgen receptor) is much more random than you might think, It's not like ligand and receptor couple everytime they happen to be nearby. The possibility for coupling can be calculated and is called the receptors affinity for ligand
Now I want you to understand that the effect that testosterone has depends from the amount and activity of androgen receptors as well as from the amount of testosterone in bloodstream, So how do we increase the quantity of androgen receptors? -By abstinence, it seems. You all know the theory about our dopamine receptors and what PMO does to them. There is evidence that suggests that the changes in hormones and neurotransmitters caused by orgasm or ejaculation decreases the amount and activity of androgen receptors, Here's my theory from biochemical and medical standpoint
Days 1-5 testosterone levels steadily increase,
Days 6 7 testosterone levels spike radically. Because of abstinence and higher testosterone levels (positive feedback regulation of androgen receptors) quantify and activity of androgen receptors increases.
Days 8 10 testosterone levels crash back to normal, because plasma free testosterone is bound to increased amount of androgen receptors, This is where it gets truly interesting! Next chapter is a bit filled with medical terminology so bare with me...
Here's what should happen next, if my thinking and medicine textbooks are correct: because testosterone is lowered to the "baseline", negative feedback-loop kicks in Plasma testosterone levels are regulated by HPA-axis and many negative feedback-loops. Now that testosterone levels are lowered, hypothalamus (endocrine gland and part of autonomous nervous system) produces more gonadotrophin releasing hormone that stimulates the pituitary gland to produce more luteinizing hormone (LH) which is the major regulator of testosterone synthesis in the Leydig cells of testicles. LH greatly accelerates the ratelimiting reaction of the reaction-chain that synthetizes testosterone by speeding up the transport of cholesterol derivate to right cell compartment. The cholesterol derivate is used to make pro hormone pregnenolone. That's the first major reaction of testosterone biosynthesis.
In plain language this means that if abstinence continues testosterone levels will rise again well over the baseline and the same cycle begins all over again So your testosterone levels and most importantly your androgen receptors will increase more and more by every cycle. That would explain the flatlines and highs that you experience when on longer "streaks", bottom line being that all the time the impact that testosterone has on you will magnify.
Some benefits like better skin can be result from zinc that you save by not ejaculating frequently. It's a fact that the zinc concentration of seminal fluid is relatively highest of all human tissue. It's a fact too that human body does recycle sperm cells and seminal fluid at least to some extent.
@goat2x
Before we can even get to semen retention, we have to understand how your body works, and the critically misunderstood roles that hormones play.
Why Testosterone? Most of you, I'm sure, are already convinced about the beneficial effects of testosterone in men, but I want to give a brief overview of things you probably don't know about it. Testosterone increases sociability. It increases connection to and participation in your milieu, this is true for both men and women. For women, this typically manifests as slutty behavior and ultimately ends up as cuckoldry, women with higher testosterone have sluttier faces. They also have greater sexual arousal levels: https://ww.ncbi.nlm.nih.gov/pubmed/10665617 For men however, testosterone decrease the incidence of lying and they are more cooperative with other men. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3468628 Men with high testosterone are fascistic ideal. They build, cooperate, and create civilization. Men with high testosterone are more driven to find not only mates, but to do all things. Testosterone receptors in the brain don't differentiate drive for sex from any other drive (that differentiation does occur, but requires other chemical receptors to be activated as well).
The Hypothalamus is King Androgen/Testosterone boosting supplements don't work unless you are supplementing androgens higher than 100% of your baseline test production. To understand why this is, you have to first understand the HP axes, and specifically the HPG axis. The hypothalamus acts as a prime regulator for almost all hormones related to growth, sex, and homeostasis. Hypothalamus notices an increased level of testosterone? It stops producing GnRH. GnRH is a signal to your pituitary gland to make FSH (follicle-stimulating hormone) and LH (luteinizing hormone); these signal the gonads in men to create sperm and testosterone respectively (in women's gonads these signal menstruation and estrogen). So at all times your hypothalamus is listening to the levels of sex hormones, and is only releasing enough GnRH to keep them at what it believes to be the proper homeostasis. This is why only highly potent anabolic steroids are really the only things which appear to have a phenotypic response in humans. They overpower even the hypothalamus's ability to regulate testosterone. So when taking anabolic steroids your level of GnRH is basically zero. There is no activity on the HPG axis and it permanently changes your hypothalamus to produce less GnRH, and hence why you hear stories of anabolic steroids shrinking your balls and causing infertility. So to maximize your GnRH you need to maximize the hormones which stimulate its production and minimize the ones that inhibit it. There are many hormones which either stimulate or inhibit the production of GnRH, we will get into why fapping is one of the worst things you can do if you want to maximize GnRH next.
There are several other axes of the hypothalamus which should also be mentioned. The hypothalamic—pituitary—prolactin axis (HPP) is responsible for lactation and mammary growth in females, but it is also present in males. It is not directly self-inhibiting like the HPG axis, but instead is inhibited by dopamine (this will seem contradictory at first, but is extremely important to understanding the dangers of masturbation). The main thing for now you should know is that excess prolactin causes typically feminine traits in men. The primary stimulating factor of the HPP axis is estrogen. Remember this is the only axis which isn't a direct feedback loop! More estrogen will always mean more prolactin. Finally, the higher your prolactin is, the lower GnRH output will be, which means the lower testosterone will be in males. The HPS axis has the ultimate byproduct of IGF-1, which is the most critical signal for muscle growth. HPS axis is stimulated by testosterone (via blunting the inhibitory effects of GH in the feedback loop).
Masturbation and Hormones Now to put it all together. Orgasm elevates prolactin massively. Prolactin causes a neurological effect of having little to no sexual desire, hence why you temporarily lose interest in the porn you watch after ejaculation. Now recall the previous section: Prolactin tells the hypothalamus to lower GnRH; GnRH stimulates production of sperm and testosterone; testosterone increases masculine traits and increases social cohesion in men; it also increases expression of muscles via the HPP axis. Dopamine which is high during the build-up of masturbation inhibits the HPP axis though, so how can masturbation be causing an increase of prolactin? Well, the hypothalamus can be very intelligent at what it does. It understands context. Not only that, but the massive acute hit of dopamine you are getting causes something called a hormetic effect. Hormesis is kind of like a biological blowback effect, the hypothalamus is massively inhibited on its HPP axis and then to compensate, it releases even more prolactin than it normally would in all that time as a huge burst after orgasm. Not only that, it appears to have "aftershocks" throughout the next few days and releases prolactin even more. But it gets worse. At least with a single mate in a monogamous sexual relationship your dopamine wouldn't be massively elevated, but with access to unlimited high speed pornography your dopamine will spike to never before seen levels due to the Coolidge effect (https://en.wikipedia.org/wiki/Coolidge_effect). What does this mean? For days after masturbation you will have low testosterone. But there's even more. The hypothalamus is a neurological structure. It learns and adapts. What does chronic masturbation and dopamine spiking do to the HPP axis? It makes it less and less sensitive to regular dopamine levels. Throughout the day its perfectly normal and healthy to have a moderate level of dopamine, and especially so for men. If HPP is no longer inhibited by this normal level of dopamine, then prolactin will be chronically elevated. Finally we get to the crux. Chronically elevated prolactin means a chronically inhibited HPG axis. A chronically inhibited HPG axis means chronically low levels of homeostatic testosterone. This is why the men of today have lower testosterone than the average 80 year old just 50 years ago. This is why we have trannies developing feminine traits due to increased prolactin.
Now there's even more danger to all of this. We have huge amounts of xenoestrogens from overcompensation of plant based foods and hygiene products (you really should only be consuming meat). Estrogen stimulates the HPP axis even more, and we have blown our dopamine circuits to defend against them with acute chronic spiking. But the brain can heal and adapt. Stop looking at porn. Stop masturbating. Stop consuming xenoestrogens. Stay monogamous, and only have sex for the purpose of procreation.
Extra Credit: Dangers of SSRIs and Weed SSRIs increase the re-uptake of serotonin in the brain. Serotonin has an antagonist relationship with dopamine, which means if dopamine is high serotonin is low and vice-versa. SSRIs will chronically lower dopamine, not only taking away your enjoyment of novelty, but also once again activating the HPP axis.
Weed is made of cannibinoids. The body produces its own endocannibinoids which have receptors all throughout the nervous system. The hypothalamus is filled with CB1 receptors which have 2-AG (an natural endocannibinoid) as an agonist. 2-AG is released upon orgasm and is used in the adaptation process mentioned earlier to train the hypothalamus to be less responsive to dopamine. What else is an agonist of the CB1 receptor? THC. In fact THC is far more potent than 2-AG could ever be, meaning that every time you smoke weed, you adapt your hypothalamus to be even less responsive to dopamine than if you were masturbating to porn the entire time you were high. THC is bad news if you value testosterone.
Testosterone molecule is totally inactive in the bloodstream. If's only a messenger which in most cells activates synthesis of certain proteins (hormones, enzymes muscle proteins etc,) The message is carried on only if the testosterone hormone binds to its receptor (androgen receptor, located in the nucleus)
For those interested in medicine: the binding of ligand (now lipid soluble steroid hormone) to ifs receptor (now androgen receptor) is much more random than you might think, It's not like ligand and receptor couple everytime they happen to be nearby. The possibility for coupling can be calculated and is called the receptors affinity for ligand
Now I want you to understand that the effect that testosterone has depends from the amount and activity of androgen receptors as well as from the amount of testosterone in bloodstream, So how do we increase the quantity of androgen receptors? -By abstinence, it seems. You all know the theory about our dopamine receptors and what PMO does to them. There is evidence that suggests that the changes in hormones and neurotransmitters caused by orgasm or ejaculation decreases the amount and activity of androgen receptors, Here's my theory from biochemical and medical standpoint
Days 1-5 testosterone levels steadily increase,
Days 6 7 testosterone levels spike radically. Because of abstinence and higher testosterone levels (positive feedback regulation of androgen receptors) quantify and activity of androgen receptors increases.
Days 8 10 testosterone levels crash back to normal, because plasma free testosterone is bound to increased amount of androgen receptors, This is where it gets truly interesting! Next chapter is a bit filled with medical terminology so bare with me...
Here's what should happen next, if my thinking and medicine textbooks are correct: because testosterone is lowered to the "baseline", negative feedback-loop kicks in Plasma testosterone levels are regulated by HPA-axis and many negative feedback-loops. Now that testosterone levels are lowered, hypothalamus (endocrine gland and part of autonomous nervous system) produces more gonadotrophin releasing hormone that stimulates the pituitary gland to produce more luteinizing hormone (LH) which is the major regulator of testosterone synthesis in the Leydig cells of testicles. LH greatly accelerates the ratelimiting reaction of the reaction-chain that synthetizes testosterone by speeding up the transport of cholesterol derivate to right cell compartment. The cholesterol derivate is used to make pro hormone pregnenolone. That's the first major reaction of testosterone biosynthesis.
In plain language this means that if abstinence continues testosterone levels will rise again well over the baseline and the same cycle begins all over again So your testosterone levels and most importantly your androgen receptors will increase more and more by every cycle. That would explain the flatlines and highs that you experience when on longer "streaks", bottom line being that all the time the impact that testosterone has on you will magnify.
Some benefits like better skin can be result from zinc that you save by not ejaculating frequently. It's a fact that the zinc concentration of seminal fluid is relatively highest of all human tissue. It's a fact too that human body does recycle sperm cells and seminal fluid at least to some extent.
@goat2x
Last edited:
„ meanwhile I am an uncooperative guy and dont build empire still I am High t bruh
