S
Saint Casanova
Kraken
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From the evidence I’ve seen, it doesn’t since it doesn’t have estrogenic like effects on the growth plate receptors.
Chat GPT seems to say the same thing:
Selective Action: Enclomiphene is a more selective SERM compared to others. It primarily works by stimulating the hypothalamus and pituitary to increase the secretion of gonadotropins (LH and FSH), which in turn stimulate the testes to produce more testosterone. This mechanism does not involve direct estrogenic effects on bone tissue or growth plates. Therefore, enclomiphene is less likely to influence the estrogen receptors in the same way other SERMs might, which is why it doesn't pose a risk of prematurely closing growth plates.
Chat GPT seems to say the same thing:
Selective Action: Enclomiphene is a more selective SERM compared to others. It primarily works by stimulating the hypothalamus and pituitary to increase the secretion of gonadotropins (LH and FSH), which in turn stimulate the testes to produce more testosterone. This mechanism does not involve direct estrogenic effects on bone tissue or growth plates. Therefore, enclomiphene is less likely to influence the estrogen receptors in the same way other SERMs might, which is why it doesn't pose a risk of prematurely closing growth plates.