Everything I know so far..

ltnburger

ltnburger

natty :0
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Bone growth:
growth plates (epiphyseal plates) = main place for height increase
Only open during puberty → close around age 16–18
Facial sutures = soft seams in skull, grow slower and stay open longer
Can grow wider jaws, bigger skull shape post-puberty
Primary growth drivers: HGH/IGF-1, Testosterone, PTH analogs, Mechanical load
Growth inhibitors: Estrogen (closes growth plates), high cortisol, poor nutrition, lack of sleep

Exogenous testosterone: effect
slow dose (100–200 mg/week): Increases IGF-1, mild muscle and bone growth
moderate dose (300–500 mg/week): strong muscle gain, increased bone density + width
helps amplify GH effects (especially on bone)
high dose (600+ mg/week): more risk (hair loss, gyno, BP, etc.), but massive anabolic effect
bone: thickens bone shafts, widens shoulders/jaw, but doesn't lengthen bones unless growth plates are still open

HDAC Inhibitors:
what they do: unlock gene expression for growth
help keep growth plates open longer
boosts cartilage and bone-forming genes
Examples: Sodium butyrate, valproic acid, trichostatin A
Best when stacked with GH or PTH analogs

PTH Analogs:
stimulate osteoblasts (bone-building cells)
increases bone length, width, and density
best ones: Teriparatide, Abaloparatide
used in short daily doses to promote anabolic bone formation
expensive but extremely powerful when used with hgh & Test

Exogenous HGH:

main hormone for bone length and muscle growth
Increases IGF-1, which stimulates growth plates and facial sutures
Works best before growth plates fuse
Dosage matters:
4 IU = average muscle/fat loss
6 IU = noticeable bone/facial changes (w/ test or PTH)
8+ IU = very noticeable bone growth
Can also grow hands, feet, jaw, forehead(brow)

Side effect mitigation:
aromasin: Control estrogen to prevent gyno & plate closure
TUDCA/milk thistle: Liver support
cabergoline or P5P: prolactin control (from GH, ghrp-2&6 or tren-like effects)
BP meds (if needed): Heart health
Clomid/Nolva/HCG: Post-cycle therapy for testosterone recovery

TIMING IS EVERYTHING ASCEND OR GET LEFT BEHIND
growth plates close ~16–18 (depending on genetics)after that, bone length = capped forever
facial bones grow longer (sutural growth), but slower
starting early = more results, more time to cycle multiple rounds
the longer you wait, the less potential gain

steady money = successful Cycle
HGH, PTH, Test, AI, support meds = expensive
missing doses = lost results or health risks
plan ahead: have entire cycle cost covered
keep extra for emergencies
don’t start unless you can finish properly (including PCT)
Have escape routes:
know where to store the drugs
know what excuse you will give your parents if they catch you (explain all the benefits and side effect mitigation and they might even pay for your cycle if they allow it, if they dont say you'll kys)

get out there brahs make it happen.
 
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Reactions: laworg
Bone growth:
growth plates (epiphyseal plates) = main place for height increase
Only open during puberty → close around age 16–18
Facial sutures = soft seams in skull, grow slower and stay open longer
Can grow wider jaws, bigger skull shape post-puberty
Primary growth drivers: HGH/IGF-1, Testosterone, PTH analogs, Mechanical load
Growth inhibitors: Estrogen (closes growth plates), high cortisol, poor nutrition, lack of sleep

Exogenous testosterone: effect
slow dose (100–200 mg/week): Increases IGF-1, mild muscle and bone growth
moderate dose (300–500 mg/week): strong muscle gain, increased bone density + width
helps amplify GH effects (especially on bone)
high dose (600+ mg/week): more risk (hair loss, gyno, BP, etc.), but massive anabolic effect
bone: thickens bone shafts, widens shoulders/jaw, but doesn't lengthen bones unless growth plates are still open

HDAC Inhibitors:
what they do: unlock gene expression for growth
help keep growth plates open longer
boosts cartilage and bone-forming genes
Examples: Sodium butyrate, valproic acid, trichostatin A
Best when stacked with GH or PTH analogs

PTH Analogs:
stimulate osteoblasts (bone-building cells)
increases bone length, width, and density
best ones: Teriparatide, Abaloparatide
used in short daily doses to promote anabolic bone formation
expensive but extremely powerful when used with hgh & Test

Exogenous HGH:
main hormone for bone length and muscle growth
Increases IGF-1, which stimulates growth plates and facial sutures
Works best before growth plates fuse
Dosage matters:
4 IU = average muscle/fat loss
6 IU = noticeable bone/facial changes (w/ test or PTH)
8+ IU = very noticeable bone growth
Can also grow hands, feet, jaw, forehead(brow)

Side effect mitigation:
aromasin: Control estrogen to prevent gyno & plate closure
TUDCA/milk thistle: Liver support
cabergoline or P5P: prolactin control (from GH, ghrp-2&6 or tren-like effects)
BP meds (if needed): Heart health
Clomid/Nolva/HCG: Post-cycle therapy for testosterone recovery

TIMING IS EVERYTHING ASCEND OR GET LEFT BEHIND
growth plates close ~16–18 (depending on genetics)after that, bone length = capped forever
facial bones grow longer (sutural growth), but slower
starting early = more results, more time to cycle multiple rounds
the longer you wait, the less potential gain

steady money = successful Cycle
HGH, PTH, Test, AI, support meds = expensive
missing doses = lost results or health risks
plan ahead: have entire cycle cost covered
keep extra for emergencies
don’t start unless you can finish properly (including PCT)
Have escape routes:
know where to store the drugs
know what excuse you will give your parents if they catch you (explain all the benefits and side effect mitigation and they might even pay for your cycle if they allow it, if they dont say you'll kys)

get out there brahs make it happen.
Water.
 
  • +1
Reactions: bruhtoobrutal and ltnburger

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