irrumator praetor
Lifting so I can crack nigger and jew skulls
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- Mar 24, 2020
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Alright so I will just post the list of all the treatments, highlight some upcoming ones that may be a de-facto cure, and highlight some very important anecdotal information from my own experience & surroundings:
Topical/non-systemic antiandrogenic alternatives to systemic antiandrogenic Finasteride & Dutasteride:
-RU58841 (mix powder with minox, apply topically, doesn't go systemic, blocks all androgens in the scalp, slightly weaker than FIN, can be stacked)
-Liposomal (Finasteride) solutions, topical or lipid or mesotherapy dutasteride (works on the account that the solution doesn't enter the bloodstream by being too large in molecule size/weight or that it is protected by liposomes)
-GT20029 (Close to completion of trials, works by deleting AR's and doesn't go systemic) (potential cure)
-Alfatradiol (weaker but a nice stack with other meds, efficacy is kinda like 2% topical minox but can be stacked and is useful & affordable)
-Fluridil (works like RU but is a complete product with more studies, weaker than FIN, can be stacked, stronger than RU, pretty strong) (Topolutamide, Eucapil) (Amazon, Ebay, other vendors)
-Breezula (Clascoterone, CB-03-01, Winlevi) (weaker local AR blocker, can be stacked) (weaker than RU, stronger than fluridil, more expensive than RU, just use RU, get breezula when its cheaper)
-Epristeride (could be a stronger fin alternative with less side effects as it isn't a competitive AR binder, in vitro study) (doesn't fuck with T and E levels) (only reason it isn't well known is because its kinda hard to get & fin is cheaper)
-Cyclosporine (better than Min & Fin) (Immuno-suppressant)(will kill you) (the topical should be safe though)
-darolutamide (stronger than Dutasteride) (lack of studies)
-Procapil (weak studies tho)
Growth stimulants:
-Minoxidil, topical & oral (oral is stronger but may be less safe)
-Nanoxidil (better absorbing minoxidil, supposedly, weak studies and way more expensive)
-Stemoxydine (does something to the hair growth cycle that makes it a good stack with minoxidil, pricey)
-TDM-105795 (thyromimetic, emulates the positive effects of the thyroid hormones, T3 & T4, on hair regrowth, without the sides) (could be risky needs more studies)
-Adenosene (stemoxydine is better) (weaker than 2% topical minox but an efficient substitute to minox) (minox is cheaper)
-Bimatoprost (Latisse) (minox is more effective & cheaper, works nonetheless)
-PRP (Platelet-rich Plasma)
-Latanoprost (Prostagladin, less effective than minox & more expensive)
-Redensyl (needs more data, stronger than 1% minox)
Topical absorption enhancers:
-Retinol (could be necessary to some people in combination with "topicals" as some people might have thicker scalps, apply and let absorb before applying any and all topicals, doesn't govern whether or not something goes systemic) (peels the top layer of the skin & allows better absorption)
-basic vehicle (P&G, Alcohol)
-
Written-off treatments:
-2dDR (2 deoxy D ribose) (weaker than 2% minox, can't be stacked) (mice studies)
-Progesterone (tranny drug, goes systemic when topical) (maybe liposomal solutions work? Weaker than finasteride)
-All tranny drugs (Spironolactone (topical is fine though, might be weak), Cyproterone Acetate, Flutamide, Bicalutamide) (androgen blockers that go systemic)
-Rapamycin (autophagy - like fasting) (Immuno-suppressant) (even the topical is harmful at high doses, lower doses topical might be ok)
-Astaxanthin (too weak, ineffective because it doesn't pass a minimum threshold for efficacy)
Other viable methods:
-Hair transplantation WITH verteporfin (restores transplanted hair - infinite hair transplants!)
-dNovo stem cell therapy (shady)
Alternative treatments (to alternative or co-related problems):
-Thyroid check & treatment
-Pituitary Gland check & treatment
-Nutritional deficiencies
-Blocking or allowing testosterone into the scalp may have negative effects on hair regrowth/growth. Depending on if your hair is sensitive to testosterone or not (according to Haircafe). Experiment. RU58841 blocks all androgens in the scalp.
-Bayer prolactin receptor antibody (boosts growth phase rather unconventionally, probably safe)
Upcoming treatments (promising but awaiting studies):
-PP405
-SCUBE3
-Pyrilutamide (as an adjunctive therapy with Minoxidil, awaiting studies)
-HMI-115 (prolactin blocker, studies are being conducted, promising)
-JAK inhibitors
-SAMiRNA technology
-RCH-01 (cell therapy for hairloss)
-WAY-316606 (WNT pathway drug) as well as SM04554, KY19382 (hair growth stimulant), Cetirizine (prostagladin & WNT pathway), (and GT20029 but I seperated them cuz GT is the best & most promising)
Sources:
ChatGPT (for scanning various articles & forums)
Haircafe (for doing the same thing but with unclear titles, reuploads & more detail)
Important reference videos:
-Connecting retrograde alopecia with MPB, and with DUPA or Female pattern baldness or general hair thinning, linking it also to advanced MPB, thus connecting early onset of both MPB & Retrograde A. with internal health problems
-Oral Minoxidil goes systemic too quickly & predisposes you to the same heart complications as that of Catu syndrome (5% chance though, deadly within weeks)(if ur on year 1 ur probably fine lol? Im getting off)
-Faults of HMI-115
-Twist1 and other pathways
-Some information on liposomal/topical dutasteride
-Difference between lipid nanoparticles & liposomes
-Best way to get a hair transplantation
Now, for my own anecdote:
If you're experiencing hairloss before the age of 50-60, even if it is genetic (especially when it is genetic) there is something extremely wrong with you.
A range of possibilities include low prenatal T (on the account of low T levels throughout life upregulating AR sensitivity), improper or unhealthy upbringing (on the account of autoimmune issues & overall toxicity & inflammation), shitty diet as a developing child (on account of both things I just mentioned), some 32 or 70+ vaccines you have to take the second you pop out of the pussy (huge amounts of "forever chemicals" & heavy metals which among other things such as autism cancer etc cause balding), heavy metal & environmental toxicity (severe air pollution, severe EMF & other radiation, etc), low testosterone levels, thyroid & pituitary gland problems that cause early onset of MPB that is accompanied almost unnaturally by retrograde alopecia, diffuse thinning or DUPA (diffuse unpatterned alopecia) or FPHL (female pattern hairloss) which are essentially the same thing just renamed. The most extreme cases of balding, as they vary in severity, almost at all times, depending on degree of hairloss, include one or all of these listed things unknowingly as some sort of "norwood package bundle". Even though scientifically this shouldn't be the case and these cases should be separated. I've studied "big pharma" in other areas of health & life enough in order to be seeing some sort of pattern when it comes to hairloss & the entire industry surrounding it. The hairloss industry wouldn't be as big as it is if all men started actually balding at around the ages of 50 or close to retirement age (65) when they earn the status of elderly (which is how balding men were exclusively described & depicted in prehistory & ancient history but I digress and this is a topic for later). They're lying about balding statistics & hormonal health of younger generations. Huge hormonal changes such as testosterone drops, which are confirmed to upregulate AR sensitivity in the scalp & cause an early onset of balding, just one of many catastrophic side effects of this trend are ignored and forgotten. I can list you a handful of friends who have started balding rapidly during highschool (even 10 out of 30 people from my class is still 1/3rd of the fucking class?) are already norwood 7 when highschool came to an end. My friends who are 18, 19 have hairlines of 30 year olds & are severly insecure and are trying to hide it. The only people not effected by this that I know IRL are poor, who perhaps haven't partaken in modernity as much as my other peers, irregardless of actual skin colour or race, or phenotype, even though it does play a big part in the actual gene nonetheless (having the balding gene is normal - balding as a young or middle aged person is wrong though). Instead of linking these side effects by what they have in common, how they often come and present themselves, what their causes are & ensuring a holistic treatment, we take hormonal blockers & suppressants, cardiovascular drugs & all drugs doing different things with different pathways and methods of working that only and ever treat the symptom and not the cause. In fact, the symptom, which is the behaviour of DHT, is claimed to be THE only and actual cause to MPB, despite there being many things involved, such as inflammation, etc. There must be a reason for either the DHT or the AR to be acting the way they are. And yes, its been linked to a certain protein associated with aging (mentioned by Haircafe in one of his prostaglandin or PG2 vids) that triggers this & one medication or method of work/pathway addresses that but the drug needed more research. Anyway, historically balding has been exclusively associated with old age & loss of virility. Which is basically exactly what is going on with the drops of testosterone & mass poisoning of society little by little every generation. Things such as pituitary gland problems & thyroid problems are very common with children nowadays when entering puberty - because of all the environmental poisons that affect them during childhood. This alone is enough to cause an early onset of MPB. Notice how with each generation, as the native people of the western world are poisoned, generations of men start balding earlier. In the time of my dad, it was normal to start around 22-26 and rapidly thin until you're fully bald at around 30. Before him, it was normal to bald around 28-30 and be fully slaphead by 35. The bar - if you would notice - is constantly dropping. Ask google or chatgpt what the normal age for balding is. It's never concise and it lists pretty young ages. Whereas if you asked google some 5-10 years ago, you would've gotten a higher age range for balding. Even older statistics say that 30% of people are bald by 30, 50% by 50. Now its different, considering at least 50% of the male population is bald at all times, if the typical age for balding is around 30, then it cannot be that 30% of the population is bald by then. Maybe not fully bald anyway. Also, back in the day, it was normal that balding would occur slowly as the hairline slowly regressed over time. Today, its more aggressive than ever & instantaneous. You'd think of an average hairline of a middle aged man and think NW3. That isn't possible if, just a year or two after the first signs of MPB show, you would progress from NW2 lets say to NW5 or 6. Everything regarding balding has changed without us knowing or noticing. My own personal advice therefore is, if you have MPB that began way too early, deal with it using the list above & do bi-yearly or annual extensive health check-ups. Filter your water, avoid pollutants, filter your air, take vacations, lead a healthy life & diet. Take care of your gut & do heavy metal cleanses from time to time with zeolites. Try and revive your (by default fucked) T levels to those of our grandparents or great grandparents. And make sure your future kids have enough prenatal T by not being a coomer, etc. Cleanse your body & brain tissue from entrenched heavy metals & mercury that is contained in the 70 something government mandated vaccines you took as a child, against your own will. If you support vaccination or not, you cannot argue against the fact you must cleanse yourself of the heavy metals present in vaccines after taking them. Thats it. Try not to poison yourself while you fight the NW reaper. Hopefully by addressing your own health first, you can actually weaken or make less aggressive your current hairloss & hair shedding situation. Btw shit like fades & fringes are modern trend inventions that hide retrograde alopecia above the ears & in the back of the head while also covering for the hairline. These insecure zoomer trends are increasingly indicative & worrying because we all know the medical side of their health situation. Hopefully God helps us lol because at this rate my entire generation will be infertile by 30-40, even women. Now - my personal story. My MPB was triggered very early & I got it with the full package - DUPA/FPHL + retrograde alopecia. Im 19 & on FIN sporting NW2 as I started with minoxidil out of my own lunch money very early on into my massive hair sheds and luckily managed to maintain NW2 with strong emphasis on regenerative properties of oral minox which helped facilitate that NW2 against NW3 which was looming on the horizon last year or so. I still haven't gotten it checked out, even though I clearly should've by now, mainly due to ignorance on behalf of myself (being young) & parents (not particularly interested). Anyway, I imagine most people don't understand jack shit about their hairloss situation as they don't understand the medical background behind it & its variants & causes, as well as their own medical reality, so to speak. But most MPB I see IRL also sport retrograde alopecia, which is indicative of poor health in combination with early onset of MPB that is artificially induced, whether the subject is approaching middle age or just graduating highschool. But these youngling cases are only growing more common from where I see it & for obvious reason, despite the (((official data))). This is my complete thesis on this entire subject, literally. Btw my personal experience with DUT - lower libido & smaller sperm count/production. Weak erections, went from 15cm bonepressed (basically a 6 incher or so, pretty average & decent, rock hard erection and good girth all) to 12 cm of what I can't even call "bonepressed" but am forced to because it wouldn't get harder than approximately 50 or 60% or so of the actual potential. I, according to internet users, am therefore a case of very early MPB offset, combined with DUPA & R.A., as well as very sensitive to FIN & DUT side effects, with aggressive hairloss. Hardest difficulty lol. Anyway my whole plan is to use liposomal DUT & minox & maybe expand the stack & get my whole goddamn health checked out, which at this point since I never had it checked out in 20 years of my life despite obvious signs of at least some hormonal/thyroid complications, I'm guaranteed to find something. I also wonder how my lungs look after a year of smoking & vapes which cause lung bubbles to form lol. + 20 years of heavily fluoridated tap water filled with heavy metals, all the unhealthy junk, etc. Just recently I had a run in with parasites & addressed by gut health & sugar consumption (now its at a flat 0 with no cravings). Most people have these things in line for them & they don't even know it, which is the worst part. WEF isn't joking when they are expecting huge population reductions tbqh. Also every year by using oral min you have a 10% chance of developing a water sack around your heart which, when it gets bad, requires immediate surgery or can cause death. Fun stuff
Topical/non-systemic antiandrogenic alternatives to systemic antiandrogenic Finasteride & Dutasteride:
-RU58841 (mix powder with minox, apply topically, doesn't go systemic, blocks all androgens in the scalp, slightly weaker than FIN, can be stacked)
-Liposomal (Finasteride) solutions, topical or lipid or mesotherapy dutasteride (works on the account that the solution doesn't enter the bloodstream by being too large in molecule size/weight or that it is protected by liposomes)
-GT20029 (Close to completion of trials, works by deleting AR's and doesn't go systemic) (potential cure)
-Alfatradiol (weaker but a nice stack with other meds, efficacy is kinda like 2% topical minox but can be stacked and is useful & affordable)
-Fluridil (works like RU but is a complete product with more studies, weaker than FIN, can be stacked, stronger than RU, pretty strong) (Topolutamide, Eucapil) (Amazon, Ebay, other vendors)
-Breezula (Clascoterone, CB-03-01, Winlevi) (weaker local AR blocker, can be stacked) (weaker than RU, stronger than fluridil, more expensive than RU, just use RU, get breezula when its cheaper)
-Epristeride (could be a stronger fin alternative with less side effects as it isn't a competitive AR binder, in vitro study) (doesn't fuck with T and E levels) (only reason it isn't well known is because its kinda hard to get & fin is cheaper)
-Cyclosporine (better than Min & Fin) (Immuno-suppressant)(will kill you) (the topical should be safe though)
-darolutamide (stronger than Dutasteride) (lack of studies)
-Procapil (weak studies tho)
Growth stimulants:
-Minoxidil, topical & oral (oral is stronger but may be less safe)
-Nanoxidil (better absorbing minoxidil, supposedly, weak studies and way more expensive)
-Stemoxydine (does something to the hair growth cycle that makes it a good stack with minoxidil, pricey)
-TDM-105795 (thyromimetic, emulates the positive effects of the thyroid hormones, T3 & T4, on hair regrowth, without the sides) (could be risky needs more studies)
-Adenosene (stemoxydine is better) (weaker than 2% topical minox but an efficient substitute to minox) (minox is cheaper)
-Bimatoprost (Latisse) (minox is more effective & cheaper, works nonetheless)
-PRP (Platelet-rich Plasma)
-Latanoprost (Prostagladin, less effective than minox & more expensive)
-Redensyl (needs more data, stronger than 1% minox)
Topical absorption enhancers:
-Retinol (could be necessary to some people in combination with "topicals" as some people might have thicker scalps, apply and let absorb before applying any and all topicals, doesn't govern whether or not something goes systemic) (peels the top layer of the skin & allows better absorption)
-basic vehicle (P&G, Alcohol)
-
Written-off treatments:
-2dDR (2 deoxy D ribose) (weaker than 2% minox, can't be stacked) (mice studies)
-Progesterone (tranny drug, goes systemic when topical) (maybe liposomal solutions work? Weaker than finasteride)
-All tranny drugs (Spironolactone (topical is fine though, might be weak), Cyproterone Acetate, Flutamide, Bicalutamide) (androgen blockers that go systemic)
-Rapamycin (autophagy - like fasting) (Immuno-suppressant) (even the topical is harmful at high doses, lower doses topical might be ok)
-Astaxanthin (too weak, ineffective because it doesn't pass a minimum threshold for efficacy)
Other viable methods:
-Hair transplantation WITH verteporfin (restores transplanted hair - infinite hair transplants!)
-dNovo stem cell therapy (shady)
Alternative treatments (to alternative or co-related problems):
-Thyroid check & treatment
-Pituitary Gland check & treatment
-Nutritional deficiencies
-Blocking or allowing testosterone into the scalp may have negative effects on hair regrowth/growth. Depending on if your hair is sensitive to testosterone or not (according to Haircafe). Experiment. RU58841 blocks all androgens in the scalp.
-Bayer prolactin receptor antibody (boosts growth phase rather unconventionally, probably safe)
Upcoming treatments (promising but awaiting studies):
-PP405
-SCUBE3
-Pyrilutamide (as an adjunctive therapy with Minoxidil, awaiting studies)
-HMI-115 (prolactin blocker, studies are being conducted, promising)
-JAK inhibitors
-SAMiRNA technology
-RCH-01 (cell therapy for hairloss)
-WAY-316606 (WNT pathway drug) as well as SM04554, KY19382 (hair growth stimulant), Cetirizine (prostagladin & WNT pathway), (and GT20029 but I seperated them cuz GT is the best & most promising)
Sources:
ChatGPT (for scanning various articles & forums)
Haircafe (for doing the same thing but with unclear titles, reuploads & more detail)
Important reference videos:
-Connecting retrograde alopecia with MPB, and with DUPA or Female pattern baldness or general hair thinning, linking it also to advanced MPB, thus connecting early onset of both MPB & Retrograde A. with internal health problems
-Oral Minoxidil goes systemic too quickly & predisposes you to the same heart complications as that of Catu syndrome (5% chance though, deadly within weeks)(if ur on year 1 ur probably fine lol? Im getting off)
-Faults of HMI-115
-Twist1 and other pathways
-Some information on liposomal/topical dutasteride
-Difference between lipid nanoparticles & liposomes
Liposome vs. Lipid Nanoparticle: What’s the Difference? Liposome vs. Lipid Nanoparticle: What’s the Difference?
Learn the difference between liposome vs. lipid nanoparticle drug delivery systems. Learn the similarities of nanoparticles and liposomes at the same time.
ascendiapharma.com
Now, for my own anecdote:
If you're experiencing hairloss before the age of 50-60, even if it is genetic (especially when it is genetic) there is something extremely wrong with you.
A range of possibilities include low prenatal T (on the account of low T levels throughout life upregulating AR sensitivity), improper or unhealthy upbringing (on the account of autoimmune issues & overall toxicity & inflammation), shitty diet as a developing child (on account of both things I just mentioned), some 32 or 70+ vaccines you have to take the second you pop out of the pussy (huge amounts of "forever chemicals" & heavy metals which among other things such as autism cancer etc cause balding), heavy metal & environmental toxicity (severe air pollution, severe EMF & other radiation, etc), low testosterone levels, thyroid & pituitary gland problems that cause early onset of MPB that is accompanied almost unnaturally by retrograde alopecia, diffuse thinning or DUPA (diffuse unpatterned alopecia) or FPHL (female pattern hairloss) which are essentially the same thing just renamed. The most extreme cases of balding, as they vary in severity, almost at all times, depending on degree of hairloss, include one or all of these listed things unknowingly as some sort of "norwood package bundle". Even though scientifically this shouldn't be the case and these cases should be separated. I've studied "big pharma" in other areas of health & life enough in order to be seeing some sort of pattern when it comes to hairloss & the entire industry surrounding it. The hairloss industry wouldn't be as big as it is if all men started actually balding at around the ages of 50 or close to retirement age (65) when they earn the status of elderly (which is how balding men were exclusively described & depicted in prehistory & ancient history but I digress and this is a topic for later). They're lying about balding statistics & hormonal health of younger generations. Huge hormonal changes such as testosterone drops, which are confirmed to upregulate AR sensitivity in the scalp & cause an early onset of balding, just one of many catastrophic side effects of this trend are ignored and forgotten. I can list you a handful of friends who have started balding rapidly during highschool (even 10 out of 30 people from my class is still 1/3rd of the fucking class?) are already norwood 7 when highschool came to an end. My friends who are 18, 19 have hairlines of 30 year olds & are severly insecure and are trying to hide it. The only people not effected by this that I know IRL are poor, who perhaps haven't partaken in modernity as much as my other peers, irregardless of actual skin colour or race, or phenotype, even though it does play a big part in the actual gene nonetheless (having the balding gene is normal - balding as a young or middle aged person is wrong though). Instead of linking these side effects by what they have in common, how they often come and present themselves, what their causes are & ensuring a holistic treatment, we take hormonal blockers & suppressants, cardiovascular drugs & all drugs doing different things with different pathways and methods of working that only and ever treat the symptom and not the cause. In fact, the symptom, which is the behaviour of DHT, is claimed to be THE only and actual cause to MPB, despite there being many things involved, such as inflammation, etc. There must be a reason for either the DHT or the AR to be acting the way they are. And yes, its been linked to a certain protein associated with aging (mentioned by Haircafe in one of his prostaglandin or PG2 vids) that triggers this & one medication or method of work/pathway addresses that but the drug needed more research. Anyway, historically balding has been exclusively associated with old age & loss of virility. Which is basically exactly what is going on with the drops of testosterone & mass poisoning of society little by little every generation. Things such as pituitary gland problems & thyroid problems are very common with children nowadays when entering puberty - because of all the environmental poisons that affect them during childhood. This alone is enough to cause an early onset of MPB. Notice how with each generation, as the native people of the western world are poisoned, generations of men start balding earlier. In the time of my dad, it was normal to start around 22-26 and rapidly thin until you're fully bald at around 30. Before him, it was normal to bald around 28-30 and be fully slaphead by 35. The bar - if you would notice - is constantly dropping. Ask google or chatgpt what the normal age for balding is. It's never concise and it lists pretty young ages. Whereas if you asked google some 5-10 years ago, you would've gotten a higher age range for balding. Even older statistics say that 30% of people are bald by 30, 50% by 50. Now its different, considering at least 50% of the male population is bald at all times, if the typical age for balding is around 30, then it cannot be that 30% of the population is bald by then. Maybe not fully bald anyway. Also, back in the day, it was normal that balding would occur slowly as the hairline slowly regressed over time. Today, its more aggressive than ever & instantaneous. You'd think of an average hairline of a middle aged man and think NW3. That isn't possible if, just a year or two after the first signs of MPB show, you would progress from NW2 lets say to NW5 or 6. Everything regarding balding has changed without us knowing or noticing. My own personal advice therefore is, if you have MPB that began way too early, deal with it using the list above & do bi-yearly or annual extensive health check-ups. Filter your water, avoid pollutants, filter your air, take vacations, lead a healthy life & diet. Take care of your gut & do heavy metal cleanses from time to time with zeolites. Try and revive your (by default fucked) T levels to those of our grandparents or great grandparents. And make sure your future kids have enough prenatal T by not being a coomer, etc. Cleanse your body & brain tissue from entrenched heavy metals & mercury that is contained in the 70 something government mandated vaccines you took as a child, against your own will. If you support vaccination or not, you cannot argue against the fact you must cleanse yourself of the heavy metals present in vaccines after taking them. Thats it. Try not to poison yourself while you fight the NW reaper. Hopefully by addressing your own health first, you can actually weaken or make less aggressive your current hairloss & hair shedding situation. Btw shit like fades & fringes are modern trend inventions that hide retrograde alopecia above the ears & in the back of the head while also covering for the hairline. These insecure zoomer trends are increasingly indicative & worrying because we all know the medical side of their health situation. Hopefully God helps us lol because at this rate my entire generation will be infertile by 30-40, even women. Now - my personal story. My MPB was triggered very early & I got it with the full package - DUPA/FPHL + retrograde alopecia. Im 19 & on FIN sporting NW2 as I started with minoxidil out of my own lunch money very early on into my massive hair sheds and luckily managed to maintain NW2 with strong emphasis on regenerative properties of oral minox which helped facilitate that NW2 against NW3 which was looming on the horizon last year or so. I still haven't gotten it checked out, even though I clearly should've by now, mainly due to ignorance on behalf of myself (being young) & parents (not particularly interested). Anyway, I imagine most people don't understand jack shit about their hairloss situation as they don't understand the medical background behind it & its variants & causes, as well as their own medical reality, so to speak. But most MPB I see IRL also sport retrograde alopecia, which is indicative of poor health in combination with early onset of MPB that is artificially induced, whether the subject is approaching middle age or just graduating highschool. But these youngling cases are only growing more common from where I see it & for obvious reason, despite the (((official data))). This is my complete thesis on this entire subject, literally. Btw my personal experience with DUT - lower libido & smaller sperm count/production. Weak erections, went from 15cm bonepressed (basically a 6 incher or so, pretty average & decent, rock hard erection and good girth all) to 12 cm of what I can't even call "bonepressed" but am forced to because it wouldn't get harder than approximately 50 or 60% or so of the actual potential. I, according to internet users, am therefore a case of very early MPB offset, combined with DUPA & R.A., as well as very sensitive to FIN & DUT side effects, with aggressive hairloss. Hardest difficulty lol. Anyway my whole plan is to use liposomal DUT & minox & maybe expand the stack & get my whole goddamn health checked out, which at this point since I never had it checked out in 20 years of my life despite obvious signs of at least some hormonal/thyroid complications, I'm guaranteed to find something. I also wonder how my lungs look after a year of smoking & vapes which cause lung bubbles to form lol. + 20 years of heavily fluoridated tap water filled with heavy metals, all the unhealthy junk, etc. Just recently I had a run in with parasites & addressed by gut health & sugar consumption (now its at a flat 0 with no cravings). Most people have these things in line for them & they don't even know it, which is the worst part. WEF isn't joking when they are expecting huge population reductions tbqh. Also every year by using oral min you have a 10% chance of developing a water sack around your heart which, when it gets bad, requires immediate surgery or can cause death. Fun stuff
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