PYT
it’s easy to despise what you cannot have
- Joined
- May 21, 2020
- Posts
- 23,553
- Reputation
- 37,891
I heard it's catabolic or some shit
Follow along with the video below to see how to install our site as a web app on your home screen.
Note: this_feature_currently_requires_accessing_site_using_safari
Deadass?yeah there are lots of studies about this
Yeah you shouldn't be walking after the gym, its bad for your muscle hyperplasia. You should ideally be driving before and after the gym so you can save up muscle energy.Deadass?
I'm 15, tf is muscle hyperplasiaYeah you shouldn't be walking after the gym, its bad for your muscle hyperplasia. You should ideally be driving before and after the gym so you can save up muscle energy.
Basically to induce cell and fiber growth your muscle need total energy outpout to be higher than the muscle shock threshold your currently have. Walking to the gym increases your energy threshold considerably and you need to output more energy in the gym which is why most pro bodybuilders are wheelchair boundI'm 15, tf is muscle hyperplasia
Yeah.This is serious?
Just walk 1 second theoryYeah.
Skeletal muscle atrophy occurs when the rate of protein degradation exceeds the rate of protein synthesis. Such a shift in the equilibrium between protein synthesis and degradation can happen when there is an increase in the rate of protein degradation and/or a decrease in the rate of protein synthesis. In contrast to skeletal muscle hypertrophy, the loss of skeletal muscle mass is observed under many different conditions including disuse, various disease states (sepsis, cancer, AIDS, diabetes and renal failure) and aging. Accordingly, the proteolytic systems involved in muscle atrophy are responsive to a number of different triggers such as mechanical unloading (disuse), growth factors (myostatin), hormones (glucocorticoid), inflammatory cytokines (TNF-α and IL-6), oxidative stress (ROS and NO), metabolic stress (ATP levels) and nutrient availability (amino acids and glucose). The major catabolic pathways downstream of these triggers include the ubiquitinproteasome system, the lysosomal system, Ca2+- dependent calpains and caspases (for in depth review of each of these systems, readers are referred to (6, 22-24). Research conducted over the past few years has continued to further our understanding of these catabolic pathways by identifying new downstream targets, modulators and crosstalk between the pathways. Aerobic activities such as walking are catabolic by inducing skeletal muscle atrophy through protein degradation. Prior research indicates anything less than 15minutes of walking is majorly anabolic, as Ca2+- dependent calpains and caspase are not activated under 15minutes of walking. Prior research also suggests that walking more than 15minutes is also anabolic since aerobic pathways are signaled after 15minute mark triggering ATP and Mtor Pathways.Catabolic pathways activated with only 15minutes of walking involve the degradation of complex molecules into simpler ones, releasing the chemical energy stored in the bonds of those molecules. Some catabolic pathways can capture that energy to produce ATP, the molecule used to power all cellular processes. Other energy-storing molecules, such as lipids, are also broken down through similar catabolic reactions to release energy and make ATP.
TLDR:
Walking 15minutes exactly = no gains, but walking more or less is anabolic.