Is Post-Puberty Height Increase Possible? Exploring Hormonal Manipulation to Reactivate “Closed” Growth Plates

[marlx]

I’ve been digging into how growth plates work and whether it’s really true that once they’re “closed,” you can’t grow any taller. Here’s the foundation I built by asking simple questions:

• Growth plates mostly close between ages 15–18, but some cartilage residue can remain into the mid-20s. (Yes)
• That leftover cartilage isn’t enough on its own to cause natural longitudinal bone growth. (Yes)
• Estrogen is the main hormone that causes growth plates to close. (Yes)
• Blocking estrogen can keep growth plates open longer—but not forever, because estrogen is also crucial for bone health. (Yes)
• Growth stops at the end of puberty mainly due to too little active cartilage combined with normalized levels of growth hormone (GH), IGF-1, and osteoblast activity. (Yes)
• So if you blocked estrogen and provided GH, IGF-1, and stimulated osteoblasts at the right levels, you could theoretically induce bone growth from that cartilage residue—even after puberty. (Yes)

Basically, this means that the term “closed” growth plates doesn’t necessarily mean zero potential to grow taller. It means natural growth is over without intervention. But with the right hormonal environment—blocking estrogen, boosting GH/IGF-1, and activating osteoblasts—there might be a way to push for longitudinal bone growth after what’s medically considered closure.


Technically, if someone combined aromatase inhibitors (like anastrozole), SERMs (like clomiphene or tamoxifen), HGH, IGF-1 LR3, and possibly PTH analogs, they could create the conditions for a growth spurt with “closed” growth plates.


This is all theoretical, and obviously comes with big safety concerns. But it challenges the dogma that height can’t increase after puberty ends.


I’m curious—what do you think about this?

(This information is certainly not new, but I haven't found one civilized thread about the topic)

 

[redeem]

TL;DR: "bla bla bla, bla bla bla bla, bla bla, bla bla bla"

 

[Gaygymmaxx]

I’ve been digging into how growth plates work and whether it’s really true that once they’re “closed,” you can’t grow any taller. Here’s the foundation I built by asking simple questions:

• Growth plates mostly close between ages 15–18, but some cartilage residue can remain into the mid-20s. (Yes)
• That leftover cartilage isn’t enough on its own to cause natural longitudinal bone growth. (Yes)
• Estrogen is the main hormone that causes growth plates to close. (Yes)
• Blocking estrogen can keep growth plates open longer—but not forever, because estrogen is also crucial for bone health. (Yes)
• Growth stops at the end of puberty mainly due to too little active cartilage combined with normalized levels of growth hormone (GH), IGF-1, and osteoblast activity. (Yes)
• So if you blocked estrogen and provided GH, IGF-1, and stimulated osteoblasts at the right levels, you could theoretically induce bone growth from that cartilage residue—even after puberty. (Yes)

Basically, this means that the term “closed” growth plates doesn’t necessarily mean zero potential to grow taller. It means natural growth is over without intervention. But with the right hormonal environment—blocking estrogen, boosting GH/IGF-1, and activating osteoblasts—there might be a way to push for longitudinal bone growth after what’s medically considered closure.


Technically, if someone combined aromatase inhibitors (like anastrozole), SERMs (like clomiphene or tamoxifen), HGH, IGF-1 LR3, and possibly PTH analogs, they could create the conditions for a growth spurt with “closed” growth plates.


This is all theoretical, and obviously comes with big safety concerns. But it challenges the dogma that height can’t increase after puberty ends.


I’m curious—what do you think about this?

(This information is certainly not new, but I haven't found one civilized thread about the topic)

I don't think it's possible tbh

 

[marlx]

TL;DR: "bla bla bla, bla bla bla bla, bla bla, bla bla bla"

niggA

 

[CordeXX]

I'm working on a thread ill keep you updated. It goes super in depth into new findings on growth plates and how to possibly reopen if they have closed in under 3 years.

 

[pizda]

I’ve been digging into how growth plates work and whether it’s really true that once they’re “closed,” you can’t grow any taller. Here’s the foundation I built by asking simple questions:

• Growth plates mostly close between ages 15–18, but some cartilage residue can remain into the mid-20s. (Yes)
• That leftover cartilage isn’t enough on its own to cause natural longitudinal bone growth. (Yes)
• Estrogen is the main hormone that causes growth plates to close. (Yes)
• Blocking estrogen can keep growth plates open longer—but not forever, because estrogen is also crucial for bone health. (Yes)
• Growth stops at the end of puberty mainly due to too little active cartilage combined with normalized levels of growth hormone (GH), IGF-1, and osteoblast activity. (Yes)
• So if you blocked estrogen and provided GH, IGF-1, and stimulated osteoblasts at the right levels, you could theoretically induce bone growth from that cartilage residue—even after puberty. (Yes)

Basically, this means that the term “closed” growth plates doesn’t necessarily mean zero potential to grow taller. It means natural growth is over without intervention. But with the right hormonal environment—blocking estrogen, boosting GH/IGF-1, and activating osteoblasts—there might be a way to push for longitudinal bone growth after what’s medically considered closure.


Technically, if someone combined aromatase inhibitors (like anastrozole), SERMs (like clomiphene or tamoxifen), HGH, IGF-1 LR3, and possibly PTH analogs, they could create the conditions for a growth spurt with “closed” growth plates.


This is all theoretical, and obviously comes with big safety concerns. But it challenges the dogma that height can’t increase after puberty ends.


I’m curious—what do you think about this?

(This information is certainly not new, but I haven't found one civilized thread about the topic)

igf1lr3

 

[Maxillular]

So if you blocked estrogen and provided GH, IGF-1, and stimulated osteoblasts at the right levels, you could theoretically induce bone growth from that cartilage residue—even after puberty. (Yes

.

 

[fourpints]

A compound like CJC-1295 DAC is the only way I personally believe post-puberty height growth will ever be possible (i.e. Replicate the results of limb-lengthening surgery without breaking your legs)

 

[Maxillular]

A compound like CJC-1295 DAC is the only way I personally believe post-puberty height growth will ever be possible (i.e. Replicate the results of limb-lengthening surgery without breaking your legs)

 

[marlx]

A compound like CJC-1295 DAC is the only way I personally believe post-puberty height growth will ever be possible (i.e. Replicate the results of limb-lengthening surgery without breaking your legs)

Yeah, most GHRPs that don't significantly raise Cortisol or Prolactin are most likely viable for increased heightgrowth during puberty, so basically CJC-1295, even tho I would suggest no DAC here because you can keep up natural GH at its best. Also, Ipamorelin would be viable.

igf1lr3

I know IGF1 LR3 has become this TikTok compound that magically does everything, but it has been used for quite some years, especially in bodybuilding, and if you take hGH, you can very well use IGF1 LR3 to benefit from the bilateral effects of the combination of these two. Apart from that, it does have some anabolic effects, just not as strong as it's portrayed to have.

 

[rscel]

I’ve been digging into how growth plates work and whether it’s really true that once they’re “closed,” you can’t grow any taller. Here’s the foundation I built by asking simple questions:

• Growth plates mostly close between ages 15–18, but some cartilage residue can remain into the mid-20s. (Yes)
• That leftover cartilage isn’t enough on its own to cause natural longitudinal bone growth. (Yes)
• Estrogen is the main hormone that causes growth plates to close. (Yes)
• Blocking estrogen can keep growth plates open longer—but not forever, because estrogen is also crucial for bone health. (Yes)
• Growth stops at the end of puberty mainly due to too little active cartilage combined with normalized levels of growth hormone (GH), IGF-1, and osteoblast activity. (Yes)
• So if you blocked estrogen and provided GH, IGF-1, and stimulated osteoblasts at the right levels, you could theoretically induce bone growth from that cartilage residue—even after puberty. (Yes)

Basically, this means that the term “closed” growth plates doesn’t necessarily mean zero potential to grow taller. It means natural growth is over without intervention. But with the right hormonal environment—blocking estrogen, boosting GH/IGF-1, and activating osteoblasts—there might be a way to push for longitudinal bone growth after what’s medically considered closure.


Technically, if someone combined aromatase inhibitors (like anastrozole), SERMs (like clomiphene or tamoxifen), HGH, IGF-1 LR3, and possibly PTH analogs, they could create the conditions for a growth spurt with “closed” growth plates.


This is all theoretical, and obviously comes with big safety concerns. But it challenges the dogma that height can’t increase after puberty ends.


I’m curious—what do you think about this?

(This information is certainly not new, but I haven't found one civilized thread about the topic)

dnr cope cope cope cope

 

[pizda]

Yeah, most GHRPs that don't significantly raise Cortisol or Prolactin are most likely viable for increased heightgrowth during puberty, so basically CJC-1295, even tho I would suggest no DAC here because you can keep up natural GH at its best. Also, Ipamorelin would be viable.


I know IGF1 LR3 has become this TikTok compound that magically does everything, but it has been used for quite some years, especially in bodybuilding, and if you take hGH, you can very well use IGF1 LR3 to benefit from the bilateral effects of the combination of these two. Apart from that, it does have some anabolic effects, just not as strong as it's portrayed to have.

Igf1lr3 for height growth is absolut dog shit, but for muscle, its good, good alternative to gear ngl

 

[childishkillah]

Technically, if someone combined aromatase inhibitors (like anastrozole), SERMs (like clomiphene or tamoxifen), HGH, IGF-1 LR3, and possibly PTH analogs, they could create the conditions for a growth spurt with “closed” growth plates.

 

[alex2334]

I’ve been digging into how growth plates work and whether it’s really true that once they’re “closed,” you can’t grow any taller. Here’s the foundation I built by asking simple questions:

• Growth plates mostly close between ages 15–18, but some cartilage residue can remain into the mid-20s. (Yes)
• That leftover cartilage isn’t enough on its own to cause natural longitudinal bone growth. (Yes)
• Estrogen is the main hormone that causes growth plates to close. (Yes)
• Blocking estrogen can keep growth plates open longer—but not forever, because estrogen is also crucial for bone health. (Yes)
• Growth stops at the end of puberty mainly due to too little active cartilage combined with normalized levels of growth hormone (GH), IGF-1, and osteoblast activity. (Yes)
• So if you blocked estrogen and provided GH, IGF-1, and stimulated osteoblasts at the right levels, you could theoretically induce bone growth from that cartilage residue—even after puberty. (Yes)

Basically, this means that the term “closed” growth plates doesn’t necessarily mean zero potential to grow taller. It means natural growth is over without intervention. But with the right hormonal environment—blocking estrogen, boosting GH/IGF-1, and activating osteoblasts—there might be a way to push for longitudinal bone growth after what’s medically considered closure.


Technically, if someone combined aromatase inhibitors (like anastrozole), SERMs (like clomiphene or tamoxifen), HGH, IGF-1 LR3, and possibly PTH analogs, they could create the conditions for a growth spurt with “closed” growth plates.


This is all theoretical, and obviously comes with big safety concerns. But it challenges the dogma that height can’t increase after puberty ends.


I’m curious—what do you think about this?

(This information is certainly not new, but I haven't found one civilized thread about the topic)

how short do you have to be to cope this much

 

[kerchmaniac]

pure cope bruh

 

[test monster]

I’ve been digging into how growth plates work and whether it’s really true that once they’re “closed,” you can’t grow any taller. Here’s the foundation I built by asking simple questions:

• Growth plates mostly close between ages 15–18, but some cartilage residue can remain into the mid-20s. (Yes)
• That leftover cartilage isn’t enough on its own to cause natural longitudinal bone growth. (Yes)
• Estrogen is the main hormone that causes growth plates to close. (Yes)
• Blocking estrogen can keep growth plates open longer—but not forever, because estrogen is also crucial for bone health. (Yes)
• Growth stops at the end of puberty mainly due to too little active cartilage combined with normalized levels of growth hormone (GH), IGF-1, and osteoblast activity. (Yes)
• So if you blocked estrogen and provided GH, IGF-1, and stimulated osteoblasts at the right levels, you could theoretically induce bone growth from that cartilage residue—even after puberty. (Yes)

Basically, this means that the term “closed” growth plates doesn’t necessarily mean zero potential to grow taller. It means natural growth is over without intervention. But with the right hormonal environment—blocking estrogen, boosting GH/IGF-1, and activating osteoblasts—there might be a way to push for longitudinal bone growth after what’s medically considered closure.


Technically, if someone combined aromatase inhibitors (like anastrozole), SERMs (like clomiphene or tamoxifen), HGH, IGF-1 LR3, and possibly PTH analogs, they could create the conditions for a growth spurt with “closed” growth plates.


This is all theoretical, and obviously comes with big safety concerns. But it challenges the dogma that height can’t increase after puberty ends.


I’m curious—what do you think about this?

(This information is certainly not new, but I haven't found one civilized thread about the topic)

Banded sleeping is being researched as of late