Just took 0,25 cabergoline to nuke my prolactin and repair some dopamine receptors

Yusu

Yusu

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Just want to take it for 2-3 months, let us see how it goes

I have pain in my left eye but maybe placebo
And headache

After 5 minutes
 
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also dizzines lol

and sleepy a little bit
 
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how the fuck are the graveyard people able to take 1mg per day wtf I just took 0.25 and I am knocked out somewhat whatever
 
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how the fuck are the graveyard people able to take 1mg per day wtf I just took 0.25 and I am knocked out somewhat whatever
Take more tbh
 
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It inhibits growth hormone hope you know that

same with vitamin b6 p5p
Just want to take it for 2-3 months, let us see how it goes

I have pain in my left eye but maybe placebo
And headache

After 5 minutes
 
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It inhibits growth hormone hope you know that

same with vitamin b6 p5p

Do you have a source please?
Also just taking 0.25 every week, not daily

Pyridoxine (B6) suppresses the rise in prolactin and increases the rise in growth hormone induced by exercise.
 
It inhibits growth hormone hope you know that

same with vitamin b6 p5p
adjunctive therapy of acromegaly, cabergoline has low efficacy in suppressing growth hormone levels and is highly efficient in suppressing hyperprolactinemia that is present in 20-30% of acromegaly cases; growth hormone and prolactin are similar structurally and have similar effects in many target tissues, therefore targeting prolactin may help symptoms when growth hormone secretion can not be sufficiently controlled by other methods;
 
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adjunctive therapy of acromegaly, cabergoline has low efficacy in suppressing growth hormone levels and is highly efficient in suppressing hyperprolactinemia that is present in 20-30% of acromegaly cases; growth hormone and prolactin are similar structurally and have similar effects in many target tissues, therefore targeting prolactin may help symptoms when growth hormone secretion can not be sufficiently controlled by other methods;
Theyre from the same family and some reputable users said so

i wouldn’t risk it personally
 
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Theyre from the same family and some reputable users said so

i wouldn’t risk it personally
could you link them? it is important for me

thank you
 
could you link them? it is important for me

thank you
@Dyorotic2 did mention that about caber in his pubertymaxxing thread but he’s banned



Vitamin B6 - https://selfhacked.com/blog/pyridoxal-phosphate/ https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1513048/#idm140088550082112title
 
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I swear I am more balanced and concentrated
idk if this will stay longer
 
sleepy
but I am less in my head which is huge
 
also more open to sexual sensations
 
more self confidence lol

my depression is dopaminr based, I knew it
 
but maybe not significant and just for a short time who knows
 
Erectile function improved significantly. Sexual desire, orgasmic function, and the patient's and his partner's sexual satisfaction were also enhanced. Cabergoline may be an effective and safe alternative agent for men with psychogenic ED

 
needy as fuck and rock hard boners
 
“Nuking your prolactin” is as unhealthy as having hyperprolactinemia
 
“Nuking your prolactin” is as unhealthy as having hyperprolactinemia
yeah just takin 2 month lol I am not deleting my prolactin forever
 
“Nuking your prolactin” is as unhealthy as having hyperprolactinemia
also too much prolactine is 100% more common than too little prolactine
there is not even a term for too little prolactine in man lol
 
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cant sleep because of sex fantasies and energy looool and rock hard boners it even hurts
 
okay

so my needines become so strong I breaked my nofap streak of 30 days 2 times and went to a who***

however today I had a very bad day and I felt aggressive, emotional unstable, extreme down and I took 0,15mg ca. and now I am fucking neutral or even positive

another sign that my emotionality and depression is not SEROTONIN based but DOPAMIN based

everyone with depression: Try this or get wellbutrin!
 
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Does caber cause long term damage to dopamine receptors?
 
Does caber cause long term damage to dopamine receptors?
maybe not a driect anwer but:

Several lines of evidence demonstrate that oxidative stress is involved in the pathogenesis of neurodegenerative diseases, including Parkinson's disease. Potent antioxidants may therefore be effective in the treatment of such diseases. Cabergoline, a dopamine D2 receptor agonist and antiparkinson drug, has been studied using several cell types including mesencephalic neurons, and is recognized as a potent radical scavenger. Here, we examined whether cabergoline exerts neuroprotective effects against oxidative stress through a receptor-mediated mechanism in cultured cortical neurons. We found that neuronal death induced by H2O2 exposure was inhibited by pretreatment with cabergoline, while this protective effect was eliminated in the presence of a dopamine D2 receptor inhibitor, spiperone. Activation of ERK1/2 by H2O2 was suppressed by cabergoline, and an ERK signaling pathway inhibitor, U0126, similarly protected cortical neurons from cell death. This suggested the ERK signaling pathway has a critical role in cabergoline-mediated neuroprotection. Furthermore, increased extracellular levels of glutamate induced by H2O2, which might contribute to ERK activation, were reduced by cabergoline, while inhibitors for NMDA receptor or L-type Ca2+ channel demonstrated a survival effect against H2O2. Interestingly, we found that cabergoline increased expression levels of glutamate transporters such as EAAC1. Taken together, these results suggest that cabergoline has a protective effect on cortical neurons via a receptor-mediated mechanism including repression of ERK1/2 activation and extracellular glutamate accumulation induced by H2O2.


Also if you take it just a short time then I don't think so. I think you are refering to down regulation? That occurs only if you take it for more months at a high dosage
 
Long-term, a low dose of the D2 receptor agonist CAB significantly reduced tumor volume and normalized serum PRL levels in a great majority of patients bearing macroprolactinoma. This treatment met with excellent patient compliance. This study suggests that CAB can be used as a first choice drug treatment in macroprolactinomas, as already shown for microprolactinomas and idiopathic hyperprolactinemia.


" No changes in the expression of the D2 receptor were demonstrated after treatment with a dopaminergic agonist. "
 
I took it last year for a while (0.5 per week) and it completely nuked my sex drive lol. Could nofap for a week and be fine, but it came back to make me mega horny. You also get super low inhib and fantasize about getting into fights.

Maybe taking it with Kratom back then wasnt good (since K nukes your libido T levels already) but there was some level of clarity, almost like your body didnt control your urges anymore
 
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Doesnt caber give ED?
Maybe while on it. Certainly not permanently like fin

I have adhd and restless leg syndrome, ill talk to my therapist about cabergoline against RLS
cabergoline and vyvanse are gonna be great for focus, low inhib and socialmaxxing
 
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Where do you even get cabergoline?
 
still makes me somewhat sleepy
Do you find any difficulty doing your everyday tasks as usual due to the sleepiness induced from the caber?
 
cabergoline doesn't "repair dopamine receptors". Long term treatment can cause a reduction in striatal D2 receptor density; i.e. you'd be worse off when you stop. Low dose naltrexone would be a better option, although you shouldn't self-medicate with prescription drugs in the first place
 
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Just want to take it for 2-3 months, let us see how it goes

I have pain in my left eye but maybe placebo
And headache

After 5 minutes
You know cabergoline bottoms out hgh and igf levels, it’s literally used to treat acromegaly
 
nah just taking really small dosages every 1 motnh or so

I just want upregulation of d2 receptors

thats the reason why cocaine can work that great
you have to take small dosages
 
did it again

I love it, it is giving you the 30 day nofap effects in 1 small dose
 
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It’s supposed to take like 2 weeks to actually start working, it doesn’t repair dopamine receptors from what I heard it will leave you worse off in the long run, on top of that it causes heart problems and another issue was something about side effects making you sleepy as fuck all the time in some people and the half life is like 2-3 weeks or something so if you fuck up the dose you are fucked for weeks with side effects.
 
However I wanted to take it but I won’t because of the side effects and depression long term I don’t want from it.
 
However I wanted to take it but I won’t because of the side effects and depression long term I don’t want from it.
low inhib
just take it
 

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