Minoxidil on my penis

[yue]

I've begun putting minoxidil on my penis, I'll report back in a month or so if any changes have occurred.

 

[sub5pslathlete]

Why??? Hahahahahahaha

 

[sportsmogger]

Why would you do that?

 

[ey88]

Why??? Hahahahahahaha

Hair

 

[monecel]

For what reason?

 

[dellimogger9867]

someone respond to my thread

 

[monecel]

Hair

sounds like a great idea to put 5α-r inhibitor on your penis

 

[halloweed]

For what reason?

Why??? Hahahahahahaha

Why would you do that?

It’s a vasodilator so he predicts it’ll increase size by increasing blood flow.

 

[yue]

It’s a vasodilator so he predicts it’ll increase size by increasing blood flow.

yup

 

[sportsmogger]

It’s a vasodilator so he predicts it’ll increase size by increasing blood flow.

Vit c grows ur dick

 

[yue]

Vit c grows ur dick

I drink lots of OJ every day

 

[sportsmogger]

I drink lots of OJ every day

youd need to megadose it hard

plus @halloweed i think pulled some old study about injecting something in ur dick?

a 40 year old got a monster cock from it

i saw it posted in 2020 but havent found it since until he reposted

 

[halloweed]

youd need to megadose it hard

plus @halloweed i think pulled some old study about injecting something in ur dick?

a 40 year old got a monster cock from it

i saw it posted in 2020 but havent found it since until he reposted

Vitamin C penis growth study?

Thread 'Insane PE method. [LOW INHIBcels GTFIH]'
https://looksmax.org/threads/insane-pe-method-low-inhibcels-gtfih.1173428/

 

[lurking truecel]

It will get smaller gj

 

[sportsmogger]

Vitamin C penis growth study?

Thread 'Insane PE method. [LOW INHIBcels GTFIH]'
https://looksmax.org/threads/insane-pe-method-low-inhibcels-gtfih.1173428/

it was vitamin k+d not vit c mb

Enhanced Growth of the Adult Penis With Vitamin D 3

Fourteen adult men volunteered to participate in an experiment to evaluate the potential for penile growth as a result of taking 50,000 units of vitamin D 3 and 100 mcg of vitamin K 2 daily. Penis length increased an average of 0.46 inches and circumference increased an average of 0.523 inches...

looksmax.org

still really good growth

 

[halloweed]

it was vitamin k+d not vit c mb

Enhanced Growth of the Adult Penis With Vitamin D 3

Fourteen adult men volunteered to participate in an experiment to evaluate the potential for penile growth as a result of taking 50,000 units of vitamin D 3 and 100 mcg of vitamin K 2 daily. Penis length increased an average of 0.46 inches and circumference increased an average of 0.523 inches...

looksmax.org

still really good growth

I’ve seen that, it was a high dosage over a long duration and idk if it can be replicated.

 

[sub5pslathlete]

yup

Put it down ur japseye

 

[sportsmogger]

I’ve seen that, it was a high dosage over a long duration and idk if it can be replicated.

You could

10,000 iu 90 capsules just buy like 3 of them

 

[yue]

You could

10,000 iu 90 capsules just buy like 3 of them

I take 20k iu e/d and I haven't seen any particular growth, I wager most of those men were unhealthy in that study to start with

 

[swt]

yeah there’s no way you don’t have autism

 

[RICHCELDOM]

op is becoming a femboy

 

[sportsmogger]

I take 20k iu e/d and I haven't seen any particular growth, I wager most of those men were unhealthy in that study to start with

bump it up to like 40k atleast

 

[Kelly Oubre Jr]

sounds like a great idea to put 5α-r inhibitor on your penis

jfl minox isnt a 5ar inhibitor retard

 

[monecel]

jfl minox isnt a 5ar inhibitor retard

 

[Kelly Oubre Jr]

View attachment 3284329

probably in the same way that mushrooms are supposed to be an aromatase inhibitor jfl. also hair follicle 5ar is completely different from systemic 5ar inhibition

 

[AustrianMogger]

I've begun putting minoxidil on my penis, I'll report back in a month or so if any changes have occurred.

Haha

 

[monecel]

probably in the same way that mushrooms are supposed to be an aromatase inhibitor jfl. also hair follicle 5ar is completely different from systemic 5ar inhibition

Tbf minoxidil on your penis probably wouldn't do anything, however the idea alone is JFL

 

[yue]

Tbf minoxidil on your penis probably wouldn't do anything, however the idea alone is JFL

I'm breaking boundaries here bhai

 

[HandsomeOrDeath]

jfl minox isnt a 5ar inhibitor retard

Yeah it is buddy boyo! There was a study published last year that showed it was. It's not significant compared to fin, but it does lower dht and raise estrogen in the scalp.

 

[Vista]

lol, i put nitroglycerin on my cock a couple of times, good fun. MInoxidil will probs be even better for erectionmaxing

 

[Daemon DeVille]

It’s a vasodilator so he predicts it’ll increase size by increasing blood flow.

asked meta ai about it (fuck chatgpt it gives nonsensical answers nowadays)
and this was the summary

Minoxidil's Mechanism of Action​

Minoxidil is a potassium channel opener that works by:

1. • Activating ATP-sensitive potassium channels: Minoxidil binds to and activates ATP-sensitive potassium channels (KATP channels) in smooth muscle cells. This increases the flow of potassium ions out of the cell.
   • Hyperpolarizing smooth muscle cells: The increased potassium ion flow causes hyperpolarization of the smooth muscle cells, making them less responsive to excitatory stimuli.
   • Relaxing smooth muscle: The hyperpolarization of smooth muscle cells leads to relaxation, reducing the contraction of smooth muscle and increasing blood flow.
   • Increasing nitric oxide production: Minoxidil may also increase the production of nitric oxide (NO) in the endothelium, which further contributes to smooth muscle relaxation and increased blood flow.

However, minoxidil's mechanism of action has some limitations in the context of ED:

• • Minoxidil's effects on potassium channels and smooth muscle relaxation are more pronounced in vascular smooth muscle cells, which may not be as relevant for erectile function.
  • Minoxidil's ability to increase nitric oxide production may be less significant compared to other mechanisms that directly target the nitric oxide-cyclic guanosine monophosphate (cGMP) pathway.

probably in the same way that mushrooms are supposed to be an aromatase inhibitor jfl. also hair follicle 5ar is completely different from systemic 5ar inhibition

the effects of minoxidil on 5-alpha-reductase (5-AR) in hair follicles versus systemic 5-AR effects do differ. Let's break it down:

Hair Follicle 5-AR Effects​

In the context of hair growth, minoxidil's 5-AR inhibition is:

1. • Localized: Minoxidil's 5-AR inhibition is primarily localized to the hair follicle, where it can help to reduce dihydrotestosterone (DHT) levels.
   • Type 1 5-AR inhibition: Minoxidil has been shown to inhibit type 1 5-AR, which is the predominant isoform found in hair follicles.
   • Increased hair growth: By reducing DHT levels in the hair follicle, minoxidil can help to increase hair growth and slow down hair loss.

Systemic 5-AR Effects​

In contrast, systemic 5-AR effects refer to the inhibition of 5-AR throughout the body, rather than just in the hair follicle. Systemic 5-AR inhibition can:

1. • Affect multiple tissues: Systemic 5-AR inhibition can affect multiple tissues, including the prostate, skin, and scalp.
   • Type 2 5-AR inhibition: Systemic 5-AR inhibitors, such as finasteride, primarily target type 2 5-AR, which is found in the prostate and other tissues.
   • Impact hormone balance: Systemic 5-AR inhibition can impact hormone balance, including changes in testosterone and DHT levels.

Implications for ED Treatment​

Now, let's consider the implications of minoxidil's 5-AR effects for ED treatment:

1. • Localized 5-AR inhibition: Minoxidil's localized 5-AR inhibition in hair follicles may not have a significant impact on systemic DHT levels or erectile function.
   • Type 1 5-AR inhibition: Minoxidil's inhibition of type 1 5-AR may not be as relevant for ED treatment, as type 2 5-AR is thought to play a more significant role in erectile function.
   • Unclear effects on erectile function: The effects of minoxidil's 5-AR inhibition on erectile function are unclear and may require further research.

In conclusion, while minoxidil's 5-AR effects on hair follicles are well understood, its implications for ED treatment are less clear. As with any treatment, it's essential to consult with a healthcare professional to discuss the potential benefits and risks of using minoxidil for ED treatment.

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tl dr

minox's vasodilation mechanisms may not be relevant for ed purposes hence dickmaxxing options are somewhat indirect

likewise it's not clear how will its systemic effects on various 5-AR inhibition play out for same as effect is localised but there's still a chance of loss of dht

take it at your own risk

btw bump alert @yue
post your experience so far, it's almost a month

 

[lestoa]

what

 

[Kelly Oubre Jr]

asked meta ai about it (fuck chatgpt it gives nonsensical answers nowadays)
and this was the summary

Minoxidil's Mechanism of Action​

Minoxidil is a potassium channel opener that works by:

1. • Activating ATP-sensitive potassium channels: Minoxidil binds to and activates ATP-sensitive potassium channels (KATP channels) in smooth muscle cells. This increases the flow of potassium ions out of the cell.
   • Hyperpolarizing smooth muscle cells: The increased potassium ion flow causes hyperpolarization of the smooth muscle cells, making them less responsive to excitatory stimuli.
   • Relaxing smooth muscle: The hyperpolarization of smooth muscle cells leads to relaxation, reducing the contraction of smooth muscle and increasing blood flow.
   • Increasing nitric oxide production: Minoxidil may also increase the production of nitric oxide (NO) in the endothelium, which further contributes to smooth muscle relaxation and increased blood flow.

However, minoxidil's mechanism of action has some limitations in the context of ED:

• • Minoxidil's effects on potassium channels and smooth muscle relaxation are more pronounced in vascular smooth muscle cells, which may not be as relevant for erectile function.
  • Minoxidil's ability to increase nitric oxide production may be less significant compared to other mechanisms that directly target the nitric oxide-cyclic guanosine monophosphate (cGMP) pathway.

the effects of minoxidil on 5-alpha-reductase (5-AR) in hair follicles versus systemic 5-AR effects do differ. Let's break it down:

Hair Follicle 5-AR Effects​

In the context of hair growth, minoxidil's 5-AR inhibition is:

1. • Localized: Minoxidil's 5-AR inhibition is primarily localized to the hair follicle, where it can help to reduce dihydrotestosterone (DHT) levels.
   • Type 1 5-AR inhibition: Minoxidil has been shown to inhibit type 1 5-AR, which is the predominant isoform found in hair follicles.
   • Increased hair growth: By reducing DHT levels in the hair follicle, minoxidil can help to increase hair growth and slow down hair loss.

Systemic 5-AR Effects​

In contrast, systemic 5-AR effects refer to the inhibition of 5-AR throughout the body, rather than just in the hair follicle. Systemic 5-AR inhibition can:

1. • Affect multiple tissues: Systemic 5-AR inhibition can affect multiple tissues, including the prostate, skin, and scalp.
   • Type 2 5-AR inhibition: Systemic 5-AR inhibitors, such as finasteride, primarily target type 2 5-AR, which is found in the prostate and other tissues.
   • Impact hormone balance: Systemic 5-AR inhibition can impact hormone balance, including changes in testosterone and DHT levels.

Implications for ED Treatment​

Now, let's consider the implications of minoxidil's 5-AR effects for ED treatment:

1. • Localized 5-AR inhibition: Minoxidil's localized 5-AR inhibition in hair follicles may not have a significant impact on systemic DHT levels or erectile function.
   • Type 1 5-AR inhibition: Minoxidil's inhibition of type 1 5-AR may not be as relevant for ED treatment, as type 2 5-AR is thought to play a more significant role in erectile function.
   • Unclear effects on erectile function: The effects of minoxidil's 5-AR inhibition on erectile function are unclear and may require further research.

In conclusion, while minoxidil's 5-AR effects on hair follicles are well understood, its implications for ED treatment are less clear. As with any treatment, it's essential to consult with a healthcare professional to discuss the potential benefits and risks of using minoxidil for ED treatment.

*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-

tl dr

minox's vasodilation mechanisms may not be relevant for ed purposes hence dickmaxxing options are somewhat indirect

likewise it's not clear how will its systemic effects on various 5-AR inhibition play out for same as effect is localised but there's still a chance of loss of dht

take it at your own risk

btw bump alert @yue
post your experience so far, it's almost a month

whole lotta nothing

 

[yue]

asked meta ai about it (fuck chatgpt it gives nonsensical answers nowadays)
and this was the summary

Minoxidil's Mechanism of Action​

Minoxidil is a potassium channel opener that works by:

1. • Activating ATP-sensitive potassium channels: Minoxidil binds to and activates ATP-sensitive potassium channels (KATP channels) in smooth muscle cells. This increases the flow of potassium ions out of the cell.
   • Hyperpolarizing smooth muscle cells: The increased potassium ion flow causes hyperpolarization of the smooth muscle cells, making them less responsive to excitatory stimuli.
   • Relaxing smooth muscle: The hyperpolarization of smooth muscle cells leads to relaxation, reducing the contraction of smooth muscle and increasing blood flow.
   • Increasing nitric oxide production: Minoxidil may also increase the production of nitric oxide (NO) in the endothelium, which further contributes to smooth muscle relaxation and increased blood flow.

However, minoxidil's mechanism of action has some limitations in the context of ED:

• • Minoxidil's effects on potassium channels and smooth muscle relaxation are more pronounced in vascular smooth muscle cells, which may not be as relevant for erectile function.
  • Minoxidil's ability to increase nitric oxide production may be less significant compared to other mechanisms that directly target the nitric oxide-cyclic guanosine monophosphate (cGMP) pathway.

the effects of minoxidil on 5-alpha-reductase (5-AR) in hair follicles versus systemic 5-AR effects do differ. Let's break it down:

Hair Follicle 5-AR Effects​

In the context of hair growth, minoxidil's 5-AR inhibition is:

1. • Localized: Minoxidil's 5-AR inhibition is primarily localized to the hair follicle, where it can help to reduce dihydrotestosterone (DHT) levels.
   • Type 1 5-AR inhibition: Minoxidil has been shown to inhibit type 1 5-AR, which is the predominant isoform found in hair follicles.
   • Increased hair growth: By reducing DHT levels in the hair follicle, minoxidil can help to increase hair growth and slow down hair loss.

Systemic 5-AR Effects​

In contrast, systemic 5-AR effects refer to the inhibition of 5-AR throughout the body, rather than just in the hair follicle. Systemic 5-AR inhibition can:

1. • Affect multiple tissues: Systemic 5-AR inhibition can affect multiple tissues, including the prostate, skin, and scalp.
   • Type 2 5-AR inhibition: Systemic 5-AR inhibitors, such as finasteride, primarily target type 2 5-AR, which is found in the prostate and other tissues.
   • Impact hormone balance: Systemic 5-AR inhibition can impact hormone balance, including changes in testosterone and DHT levels.

Implications for ED Treatment​

Now, let's consider the implications of minoxidil's 5-AR effects for ED treatment:

1. • Localized 5-AR inhibition: Minoxidil's localized 5-AR inhibition in hair follicles may not have a significant impact on systemic DHT levels or erectile function.
   • Type 1 5-AR inhibition: Minoxidil's inhibition of type 1 5-AR may not be as relevant for ED treatment, as type 2 5-AR is thought to play a more significant role in erectile function.
   • Unclear effects on erectile function: The effects of minoxidil's 5-AR inhibition on erectile function are unclear and may require further research.

In conclusion, while minoxidil's 5-AR effects on hair follicles are well understood, its implications for ED treatment are less clear. As with any treatment, it's essential to consult with a healthcare professional to discuss the potential benefits and risks of using minoxidil for ED treatment.

*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-*-

tl dr

minox's vasodilation mechanisms may not be relevant for ed purposes hence dickmaxxing options are somewhat indirect

likewise it's not clear how will its systemic effects on various 5-AR inhibition play out for same as effect is localised but there's still a chance of loss of dht

take it at your own risk

btw bump alert @yue
post your experience so far, it's almost a month

didn't do anything, however I've had impressive eyelash/eyebrow gains