Proof Finasteride is a "safe" drug

Baldingman1998

Baldingman1998

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Safe for transitioning into the opposite gender that is.

Kevin man is a famous youtuber among the hair restoration circles.

He is a huge advocate for Finasteride and that Finasteride is completely safe and DHT is a "trash" hormone





Look at kevin he is literally transitioning into a woman look at his fucking face

Looks like a middle aged granny
 
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Hair system mogs hard
 
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Old teacher listening to student online lesson grey haired carefully class 39489768
Screenshot 20220308 132815
Screenshot 20220308 132806
 
stfu. just because you are bald doesnt mean I have to be
 
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Maybe he is aging?

Brootal nevertheless, he looked more muscular before!
 
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thank god i never have to take tranny drugs to keep my hair
 
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1646739153843
 
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Go on hairloss talk guys on there take legit tranny drugs
 
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I think he is also Vegan.. so yes I think his body is in transition as we speak.

I did T testing on Fin for 2 years then quit a year.

My T went from low mid ranges to high range. (Pre-fin levels).

Not a doctor but proved to me it reduced my T significantly while on it.
 
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He always looked like that
 
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stfu. just because you are bald doesnt mean I have to be
Finasteride is effective at stopping hairloss. But at a cost. Do what you want with this information
 
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Maybe he is aging?

Brootal nevertheless, he looked more muscular before!
Yes he is aging his skin still looks good tho, good collagen won't save you
 
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kevin mann is an obnoxious faggot who's never dealt with actual hardship in life because of his rich parents, has hardly ever worked at all in his life, and has an annoying obsessive personality which made him fixate on his hair

he's akin to a cult follower with his finasteride worshipping and inability to be impartial on matters concerning hair loss
idk why people look up to him when it's so obvious he's extremely biased on every word that comes out of his mouth
 
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Finasteride is effective at stopping hairloss. But at a cost. Do what you want with this information
Stfu you won't take away my beautiful blonde hair
 
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I think he is also Vegan.. so yes I think his body is in transition as we speak.

I did T testing on Fin for 2 years then quit a year.

My T went from low mid ranges to high range. (Pre-fin levels).

Not a doctor but proved to me it reduced my T significantly while on it.
Yes water. It increases testosterone temporarily because less is converted into dht. But long term it reduces it
 
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He always looked like that jfl, not fault of fin

Some men just aren't very masc, the long blonde hair make him more soft. When he had a fade he looked more masc
 
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He always looked like that jfl, not fault of fin

Some men just aren't very masc, the long blonde hair make him more soft. When he had a fade he looked more masc
fin also makes you age less and it makes your fat pads fuller

he also uses retin-a and microneedles his skin, good skin can make look feminine
 
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I’ve been on fin for 3 months and have C cups now
 
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Its a bad drug unfortunately we dont have many alternatives. And topicals suck so much and are hard to apply
 
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He always looked like that jfl, not fault of fin

Some men just aren't very masc, the long blonde hair make him more soft. When he had a fade he looked more masc
He looked normal here

 
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stfu. just because you are bald doesnt mean I have to be
This dogshit forum is the only place where full baldcels hate on finasteride, op is probably mad he didn’t take it earlier
 
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Just balance it out by blasting tren bro
 
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This dogshit forum is the only place where full baldcels hate on finasteride, op is probably mad he didn’t take it earlier
No go on any hairloss forum,.go on Reddit plenty of baldcels hate fin
 
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I think fin is a bad drug btw just think being bald is worse
 
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No go on any hairloss forum,.go on Reddit plenty of baldcels hate fin
The only situation where I’d hate it is if I got serious long term effects, which is probably cope

The long term effects of being a baldcel are worse
 
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The only situation where I’d hate it is if I got serious long term effects, which is probably cope

The long term effects of being a baldcel are worse
Worse effect is infertility probably
 
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Proof of fin makes your test decline long term?

Im aware it can fuck with androgen receptors but this only happens in subhumans that get PFS
https://pubmed.ncbi.nlm.nih.gov/212...icant,resulting in increased pregnancy losses



https://pubmed.ncbi.nlm.nih.gov/22789024/


https://www.dermatologytimes.com/view/study-examines-finasteride-risks-for-younger-males

https://www.intechopen.com/chapters/75942


The known side effects of finasteride include sexual dysfunction (e.g. loss of libido, impotence, ejaculatory or erectile dysfunction), gynecomastia, changes in cognition, and affect (including depression, which was added to the product package insert by Merck in 2010). Of particular interest is that there are side effects after finasteride is discontinued. For example, there are reports of persistent sexual dysfunction following discontinuation of finasteride [4, 46, 47, 48]. Additionally, former users of finasteride report sexual dysfunction and have high scores on the Beck Depression Inventory more than 3 months after cessation of finasteride use [2, 3]. In this study, 75% of men (61 total in the study) showing persistent sexual dysfunction following finasteride discontinuation had symptoms of depression (assessed by Beck Depression Inventory) over 3 months later. It is assumed in these individuals that endogenous androgen levels have normalized, so the question remains regarding potential mechanisms of these long-term changes in behavioral responses. A recent publication has shown that there seems to be a reduced capacity for neuroactive steroid production (as measured in cerebrospinal fluid or plasma) of individuals with side effects following discontinuation of finasteride for alopecia [49]. Studies in patients at risk for prostate cancer prescribed finasteride demonstrate increased testosterone and decreased DHT and 3α-diol glucuronide (mainly produced in the liver) in circulation [43], in addition to differences in cholesterol profile [49]. Whether levels coincide with symptoms is not understood. We have collected pilot data in support of this idea in male rats. In this study, male rats were GDX or gonadally-intact and were administered T or vehicle chronically in conjunction with finasteride or vehicle. Rats that were GDX, compared to intact rats, had lower circulating T and DHT levels, smaller prostates and longer latencies to initiate sexual contacts with a female (as to be expected). T-replacement increased plasma T and prostate mass. Similar to GDX, finasteride decreased prostate weight and inhibited sexual behavior (Figure 11). A preliminary finding in this study was that the finasteride treatment had variable effects to reduce the 5α-reduced metabolite, DHT, and that the levels determined do not seem to coincide with the behavioral patterns observed. As such, the present proposal is informed by the clinical literature and preliminary results in our laboratory using an animal model. Thus, of clinical relevance is consideration of long-term effects of androgen manipulations that extend beyond the treatment period. We propose, such as with post-finasteride syndrome, that there may be long-term consequences of changing androgen milieu that may be independent of specific levels of androgens at a particular time.


There are many more which I can't find atm. Search yourself. Jews made it hard to find
 
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https://pubmed.ncbi.nlm.nih.gov/21292254/#:~:text=Conclusion(s): The significant,resulting in increased pregnancy losses



https://pubmed.ncbi.nlm.nih.gov/22789024/


https://www.dermatologytimes.com/view/study-examines-finasteride-risks-for-younger-males

https://www.intechopen.com/chapters/75942


The known side effects of finasteride include sexual dysfunction (e.g. loss of libido, impotence, ejaculatory or erectile dysfunction), gynecomastia, changes in cognition, and affect (including depression, which was added to the product package insert by Merck in 2010). Of particular interest is that there are side effects after finasteride is discontinued. For example, there are reports of persistent sexual dysfunction following discontinuation of finasteride [4, 46, 47, 48]. Additionally, former users of finasteride report sexual dysfunction and have high scores on the Beck Depression Inventory more than 3 months after cessation of finasteride use [2, 3]. In this study, 75% of men (61 total in the study) showing persistent sexual dysfunction following finasteride discontinuation had symptoms of depression (assessed by Beck Depression Inventory) over 3 months later. It is assumed in these individuals that endogenous androgen levels have normalized, so the question remains regarding potential mechanisms of these long-term changes in behavioral responses. A recent publication has shown that there seems to be a reduced capacity for neuroactive steroid production (as measured in cerebrospinal fluid or plasma) of individuals with side effects following discontinuation of finasteride for alopecia [49]. Studies in patients at risk for prostate cancer prescribed finasteride demonstrate increased testosterone and decreased DHT and 3α-diol glucuronide (mainly produced in the liver) in circulation [43], in addition to differences in cholesterol profile [49]. Whether levels coincide with symptoms is not understood. We have collected pilot data in support of this idea in male rats. In this study, male rats were GDX or gonadally-intact and were administered T or vehicle chronically in conjunction with finasteride or vehicle. Rats that were GDX, compared to intact rats, had lower circulating T and DHT levels, smaller prostates and longer latencies to initiate sexual contacts with a female (as to be expected). T-replacement increased plasma T and prostate mass. Similar to GDX, finasteride decreased prostate weight and inhibited sexual behavior (Figure 11). A preliminary finding in this study was that the finasteride treatment had variable effects to reduce the 5α-reduced metabolite, DHT, and that the levels determined do not seem to coincide with the behavioral patterns observed. As such, the present proposal is informed by the clinical literature and preliminary results in our laboratory using an animal model. Thus, of clinical relevance is consideration of long-term effects of androgen manipulations that extend beyond the treatment period. We propose, such as with post-finasteride syndrome, that there may be long-term consequences of changing androgen milieu that may be independent of specific levels of androgens at a particular time.


There are many more which I can't find atm. Search yourself. Jews made it hard to find
ok PFS is rare tho

I agree fin isn't a good drug i wouldn't take it if I had other options
 
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ok PFS is rare tho

I agree fin isn't a good drug i wouldn't take it if I had other options
You better read all those essays. I had to read them to link
 
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https://pubmed.ncbi.nlm.nih.gov/21292254/#:~:text=Conclusion(s): The significant,resulting in increased pregnancy losses



https://pubmed.ncbi.nlm.nih.gov/22789024/


https://www.dermatologytimes.com/view/study-examines-finasteride-risks-for-younger-males

https://www.intechopen.com/chapters/75942


The known side effects of finasteride include sexual dysfunction (e.g. loss of libido, impotence, ejaculatory or erectile dysfunction), gynecomastia, changes in cognition, and affect (including depression, which was added to the product package insert by Merck in 2010). Of particular interest is that there are side effects after finasteride is discontinued. For example, there are reports of persistent sexual dysfunction following discontinuation of finasteride [4, 46, 47, 48]. Additionally, former users of finasteride report sexual dysfunction and have high scores on the Beck Depression Inventory more than 3 months after cessation of finasteride use [2, 3]. In this study, 75% of men (61 total in the study) showing persistent sexual dysfunction following finasteride discontinuation had symptoms of depression (assessed by Beck Depression Inventory) over 3 months later. It is assumed in these individuals that endogenous androgen levels have normalized, so the question remains regarding potential mechanisms of these long-term changes in behavioral responses. A recent publication has shown that there seems to be a reduced capacity for neuroactive steroid production (as measured in cerebrospinal fluid or plasma) of individuals with side effects following discontinuation of finasteride for alopecia [49]. Studies in patients at risk for prostate cancer prescribed finasteride demonstrate increased testosterone and decreased DHT and 3α-diol glucuronide (mainly produced in the liver) in circulation [43], in addition to differences in cholesterol profile [49]. Whether levels coincide with symptoms is not understood. We have collected pilot data in support of this idea in male rats. In this study, male rats were GDX or gonadally-intact and were administered T or vehicle chronically in conjunction with finasteride or vehicle. Rats that were GDX, compared to intact rats, had lower circulating T and DHT levels, smaller prostates and longer latencies to initiate sexual contacts with a female (as to be expected). T-replacement increased plasma T and prostate mass. Similar to GDX, finasteride decreased prostate weight and inhibited sexual behavior (Figure 11). A preliminary finding in this study was that the finasteride treatment had variable effects to reduce the 5α-reduced metabolite, DHT, and that the levels determined do not seem to coincide with the behavioral patterns observed. As such, the present proposal is informed by the clinical literature and preliminary results in our laboratory using an animal model. Thus, of clinical relevance is consideration of long-term effects of androgen manipulations that extend beyond the treatment period. We propose, such as with post-finasteride syndrome, that there may be long-term consequences of changing androgen milieu that may be independent of specific levels of androgens at a particular time.


There are many more which I can't find atm. Search yourself. Jews made it hard to find
fuck how often does this shit happen
 
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Looks completely different. Not just age. His skin and eyes look more feminine

by what mechanism

he looks more bloated now and his skin looks better
 
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he's fear mongering
yea thats the 2nd link this bald nigga posted:

comprehensive review of a total of 73 papers on medical therapies for BPH was conducted, with a focus on the effects of different pharmacological agents on sexual function.[6] The review revealed that finasteride is infrequently associated with problems of ejaculation (2.1-7.7%), erection (4.9-15.8%), and libido (3.1-5.4%).

The role of nocebo effect in the causation of ED due to finasteride has been investigated.[7] Nocebo effect refers to an adverse effect that results from the psychological awareness of the possibility of the side effects, but is not a direct result of the specific pharmacological action of the drug. In this study, the group informed about the sexual adverse effects of finasteride reported increased incidence of ED, when compared to the group without information.[7] The side effects were completely reversible in 5 days when the medicine was discontinued, confirming that nocebo effect has an influence in causation of side effects and suggesting the role of psychological factors.
 
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by what mechanism

he looks more bloated now and his skin looks better
You're literally trolling. Watch 30 seconds of the top video then watch this. Dude looks like he either has make up on or is transitioning
 
yea thats the 2nd link this bald nigga posted:

comprehensive review of a total of 73 papers on medical therapies for BPH was conducted, with a focus on the effects of different pharmacological agents on sexual function.[6] The review revealed that finasteride is infrequently associated with problems of ejaculation (2.1-7.7%), erection (4.9-15.8%), and libido (3.1-5.4%).

The role of nocebo effect in the causation of ED due to finasteride has been investigated.[7] Nocebo effect refers to an adverse effect that results from the psychological awareness of the possibility of the side effects, but is not a direct result of the specific pharmacological action of the drug. In this study, the group informed about the sexual adverse effects of finasteride reported increased incidence of ED, when compared to the group without information.[7] The side effects were completely reversible in 5 days when the medicine was discontinued, confirming that nocebo effect has an influence in causation of side effects and suggesting the role of psychological factors.
Ye I wouldn't take fin if you dont have to but if you need it then ye
 
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