SERMs > Injecting

[chadmanlet04]

Clomid (Enclomimed if you can source it) paired with Anastrozole mogs injecting. Why? Enclomephine is a selective estrogen receptor modular. Testosterone is testosterone. One works by manipulating the body’s natural negative feedback mechanism to low estrogen levels, while the other is total replacement for the body’s endogenous production. SERMs like enclomephine work in those with a relatively intact HTPA axis. In order for enclomiphene to work, you must have a functioning hypothalamus, testes, and pituitary. Essentially, the SERM tricks your body into thinking it needs to secrete larger/more frequent pulses of FSH and LH, which in turn stimulate the testes to produce more testosterone and spermatogenesis, thus preserving fertility while also increasing endogenous testosterone. In men with certain types of hypogonadism, this style of treatment won’t work because they have a dysfunction somewhere in the HPTA axis, which is why they are likely hypogonadal in the first place. A lot of good TRT physicians will often start men off on clomid only or clomid + HCG to test a man’s HPTA to see if they respond correctly.Exogenous testosterone injections are the opposite approach. Instead of trying to get the body’s diesel generators working again, you just inject testosterone to replace what is either no longer being made, or isn’t being made in sufficient quantities for quality of life purposes. However, the exogenous T will completely shut down the body’s natural HPTA because the system will detect sufficient androgen levels and therefore will not need to manufacture any more.

If you can manage E2 sides with a higher dose AI, you will make your own body produce monster levels of test with your HPTA being intact. Bigger balls, bigger loads and significantly increased fertility on top of feeling and looking high T.

 

[Yahya]

Bookmarked + will read after my league game

 

[sportsmogger]

Who wants my SPERM’s

 

[Yahya]

Who wants my SPERM’s

ME SAAR DELICIOUS PROTO-ARYAN MILK

 

[elemanzelvadre]

also closes ur growth plates faster with estrogen receoptor binding

 

[Saint Casanova]

The increase in T won’t be that drastic to the point you’ll easily gain muscle or more dimorphism in general and it’s not smart to stay on it forever.

It does make you feel really good, make your balls full and give a natty plus boost overall.

 

[LooksmaxAutist]

Clomid isnt fucking enclomiphene you dummy

 

[menas]

Clomid (Enclomimed if you can source it) paired with Anastrozole mogs injecting. Why? Enclomephine is a selective estrogen receptor modular. Testosterone is testosterone. One works by manipulating the body’s natural negative feedback mechanism to low estrogen levels, while the other is total replacement for the body’s endogenous production. SERMs like enclomephine work in those with a relatively intact HTPA axis. In order for enclomiphene to work, you must have a functioning hypothalamus, testes, and pituitary. Essentially, the SERM tricks your body into thinking it needs to secrete larger/more frequent pulses of FSH and LH, which in turn stimulate the testes to produce more testosterone and spermatogenesis, thus preserving fertility while also increasing endogenous testosterone. In men with certain types of hypogonadism, this style of treatment won’t work because they have a dysfunction somewhere in the HPTA axis, which is why they are likely hypogonadal in the first place. A lot of good TRT physicians will often start men off on clomid only or clomid + HCG to test a man’s HPTA to see if they respond correctly.Exogenous testosterone injections are the opposite approach. Instead of trying to get the body’s diesel generators working again, you just inject testosterone to replace what is either no longer being made, or isn’t being made in sufficient quantities for quality of life purposes. However, the exogenous T will completely shut down the body’s natural HPTA because the system will detect sufficient androgen levels and therefore will not need to manufacture any more.

If you can manage E2 sides with a higher dose AI, you will make your own body produce monster levels of test with your HPTA being intact. Bigger balls, bigger loads and significantly increased fertility on top of feeling and looking high T.

@maarda @NZb6Air ?

 

[chadmanlet04]

Clomid isnt fucking enclomiphene you dummy

its literally enclomiphene with zuclomiphene which is why it spikes E2 due to long zuclomiphene half life and it accumulating over time

 

[chadmanlet04]

also closes ur growth plates faster with estrogen receoptor binding

youngcel problems

 

[Dyorotic]

Just admit you're afraid of needles and move on

This shit is such cope. Try tamoxifen and come back when your heart rate is in the 100s and your body is aching.

 

[Orc]

low estrogen is bad for gains, estrogen is anabolic.

 

[chadmanlet04]

low estrogen is bad for gains, estrogen is anabolic.

Absolutely. Let me put it in perspective for you. When my Estrogen is really high and i feel that high energy anxiety sweaty erectile disfunction even though i still have desire i know its high. And when i take a small amount of Anastrozole i feel so much better i feel high or euphoric. When things are dialed in you feel great. Low Estrogen is probably worse depending on how low or high. Low zero motivation no appetite i look horrible literally face muscle tone etc. Its actually very very interesting. I look like a 6 outta 10 with low then as its normal or high normal my skin glows its plump youthful its very interesting.

 

[Cares]

lol, no one feels good on pct. why would you even suggest this

 

[NZb6Air]

@maarda @NZb6Air ?

 

[chadmanlet04]

Just admit you're afraid of needles and move on

This shit is such cope. Try tamoxifen and come back when your heart rate is in the 100s and your body is aching.

this is not about needles, its about Endogenous vs exogenous test.

I have pinned HCG before too. HCG is suppressive in the long run. It imitates LH, it doesn't cause its production.

 

[i999z]

Clomid (Enclomimed if you can source it) paired with Anastrozole mogs injecting. Why? Enclomephine is a selective estrogen receptor modular. Testosterone is testosterone. One works by manipulating the body’s natural negative feedback mechanism to low estrogen levels, while the other is total replacement for the body’s endogenous production. SERMs like enclomephine work in those with a relatively intact HTPA axis. In order for enclomiphene to work, you must have a functioning hypothalamus, testes, and pituitary. Essentially, the SERM tricks your body into thinking it needs to secrete larger/more frequent pulses of FSH and LH, which in turn stimulate the testes to produce more testosterone and spermatogenesis, thus preserving fertility while also increasing endogenous testosterone. In men with certain types of hypogonadism, this style of treatment won’t work because they have a dysfunction somewhere in the HPTA axis, which is why they are likely hypogonadal in the first place. A lot of good TRT physicians will often start men off on clomid only or clomid + HCG to test a man’s HPTA to see if they respond correctly.Exogenous testosterone injections are the opposite approach. Instead of trying to get the body’s diesel generators working again, you just inject testosterone to replace what is either no longer being made, or isn’t being made in sufficient quantities for quality of life purposes. However, the exogenous T will completely shut down the body’s natural HPTA because the system will detect sufficient androgen levels and therefore will not need to manufacture any more.

If you can manage E2 sides with a higher dose AI, you will make your own body produce monster levels of test with your HPTA being intact. Bigger balls, bigger loads and significantly increased fertility on top of feeling and looking high T.

how can i know the best dose for ai to control my Estrogen

 

[milkcuck]

Clomid (Enclomimed if you can source it) paired with Anastrozole mogs injecting. Why? Enclomephine is a selective estrogen receptor modular. Testosterone is testosterone. One works by manipulating the body’s natural negative feedback mechanism to low estrogen levels, while the other is total replacement for the body’s endogenous production. SERMs like enclomephine work in those with a relatively intact HTPA axis. In order for enclomiphene to work, you must have a functioning hypothalamus, testes, and pituitary. Essentially, the SERM tricks your body into thinking it needs to secrete larger/more frequent pulses of FSH and LH, which in turn stimulate the testes to produce more testosterone and spermatogenesis, thus preserving fertility while also increasing endogenous testosterone. In men with certain types of hypogonadism, this style of treatment won’t work because they have a dysfunction somewhere in the HPTA axis, which is why they are likely hypogonadal in the first place. A lot of good TRT physicians will often start men off on clomid only or clomid + HCG to test a man’s HPTA to see if they respond correctly.Exogenous testosterone injections are the opposite approach. Instead of trying to get the body’s diesel generators working again, you just inject testosterone to replace what is either no longer being made, or isn’t being made in sufficient quantities for quality of life purposes. However, the exogenous T will completely shut down the body’s natural HPTA because the system will detect sufficient androgen levels and therefore will not need to manufacture any more.

If you can manage E2 sides with a higher dose AI, you will make your own body produce monster levels of test with your HPTA being intact. Bigger balls, bigger loads and significantly increased fertility on top of feeling and looking high T.

They also insta close growth plates JFL

 

[averagenormie]

Clomid (Enclomimed if you can source it) paired with Anastrozole mogs injecting. Why? Enclomephine is a selective estrogen receptor modular. Testosterone is testosterone. One works by manipulating the body’s natural negative feedback mechanism to low estrogen levels, while the other is total replacement for the body’s endogenous production. SERMs like enclomephine work in those with a relatively intact HTPA axis. In order for enclomiphene to work, you must have a functioning hypothalamus, testes, and pituitary. Essentially, the SERM tricks your body into thinking it needs to secrete larger/more frequent pulses of FSH and LH, which in turn stimulate the testes to produce more testosterone and spermatogenesis, thus preserving fertility while also increasing endogenous testosterone. In men with certain types of hypogonadism, this style of treatment won’t work because they have a dysfunction somewhere in the HPTA axis, which is why they are likely hypogonadal in the first place. A lot of good TRT physicians will often start men off on clomid only or clomid + HCG to test a man’s HPTA to see if they respond correctly.Exogenous testosterone injections are the opposite approach. Instead of trying to get the body’s diesel generators working again, you just inject testosterone to replace what is either no longer being made, or isn’t being made in sufficient quantities for quality of life purposes. However, the exogenous T will completely shut down the body’s natural HPTA because the system will detect sufficient androgen levels and therefore will not need to manufacture any more.

If you can manage E2 sides with a higher dose AI, you will make your own body produce monster levels of test with your HPTA being intact. Bigger balls, bigger loads and significantly increased fertility on top of feeling and looking high T.

Why anastrozole and not letrozole or aromasin?

 

[chadmanlet04]

They also insta close growth plates JFL

im 27

 

[Tylermax]

im 27

sum up your bitch ass thread. and do i get this stuff from doctors or by myself? this is the important stuff nigga. get to the point ffs

 

[MA_ascender]

Cope lol

 

[chadmanlet04]

sum up your bitch ass thread. and do i get this stuff from doctors or by myself? this is the important stuff nigga. get to the point ffs

Yourself

Just get them from an UGL online

 

[Jonas2k7]

From my understanding enclo imitates some shit in the pituary gland which then tells the balls (LH and FH or sum shit) to produce more test

Why doesn't it have the same anabolic effect like exogenous test? Another Question: Are high T persons more likely to build more muscles and their androgen receptors more effectively responding to DHT and test than low T persons (Basically if higher T = more muscle building)

 

[6'3 ltn]

Clomid (Enclomimed if you can source it) paired with Anastrozole mogs injecting. Why? Enclomephine is a selective estrogen receptor modular. Testosterone is testosterone. One works by manipulating the body’s natural negative feedback mechanism to low estrogen levels, while the other is total replacement for the body’s endogenous production. SERMs like enclomephine work in those with a relatively intact HTPA axis. In order for enclomiphene to work, you must have a functioning hypothalamus, testes, and pituitary. Essentially, the SERM tricks your body into thinking it needs to secrete larger/more frequent pulses of FSH and LH, which in turn stimulate the testes to produce more testosterone and spermatogenesis, thus preserving fertility while also increasing endogenous testosterone. In men with certain types of hypogonadism, this style of treatment won’t work because they have a dysfunction somewhere in the HPTA axis, which is why they are likely hypogonadal in the first place. A lot of good TRT physicians will often start men off on clomid only or clomid + HCG to test a man’s HPTA to see if they respond correctly.Exogenous testosterone injections are the opposite approach. Instead of trying to get the body’s diesel generators working again, you just inject testosterone to replace what is either no longer being made, or isn’t being made in sufficient quantities for quality of life purposes. However, the exogenous T will completely shut down the body’s natural HPTA because the system will detect sufficient androgen levels and therefore will not need to manufacture any more.

If you can manage E2 sides with a higher dose AI, you will make your own body produce monster levels of test with your HPTA being intact. Bigger balls, bigger loads and significantly increased fertility on top of feeling and looking high T.

I'm 18 and been on enclomimed enclomiphene and mk677 for a bit over a month and the enclomiphene didn't really do anything, just boosted libido, apparently if you already have good test levels it's not going to boost it that much like people say to 1,300+ but If you're low on test would still be a recommend

 

[chadmanlet04]

I'm 18 and been on enclomimed enclomiphene and mk677 for a bit over a month and the enclomiphene didn't really do anything, just boosted libido, apparently if you already have good test levels it's not going to boost it that much like people say to 1,300+ but If you're low on test would still be a recommend

1300+ is supraphysiological and unhealthy… it’s a good level for short term blasts but cruising on it indefinitely would definitely cause cardiovascular issues in the long run…

hitting around 1000 or slightly up is more than enough for quality of life purposes, you will have high libido, good erections, healthy mentality and build muscle at an acceptable rate with good nutrition/regimen

I personally have 624ng/dl natty and on Clomid citrate I can hit around 1247ng/dl, albeit my free test gains are not as good compared to injecting Test E due to SHBG… you could take proviron alongside to raise your free test levels

 

[chadmanlet04]

Why doesn't it have the same anabolic effect like exogenous test? Another Question: Are high T persons more likely to build more muscles and their androgen receptors more effectively responding to DHT and test than low T persons (Basically if higher T = more muscle building)

It doesn’t increase free testosterone levels as much as SARMs which is why you do not see the same benefits you’d see in an injection based TRT protocol. You could increase your free testosterone by taking proviron alongside of SERMs to combat decreased SHBG.

Another Question: Are high T persons more likely to build more muscles and their androgen receptors more effectively responding to DHT and test than low T persons (Basically if higher T = more muscle building)

most likely yes, since their already high T levels could be attributed to more sensitive and active receptors, however a low T person could have sensitive and functional receptors but simply lack testosterone due to an issue either in the HPTA Axis or the testes and easily hit higher T levels with a TRT protocol.

If you have no deficiencies in your endocrine system yet have lower testosterone levels, it’s likely due to weaker and/or desensitized receptors.