meathead
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Testosterone decreases collagen synthesis, reducing the strength, flexibility and structural integrity of skin and joints. There are ways to mitigate this.
Androgens inhibit osteoclastic bone resorption with increase of bone formation through androgen receptor in bone tissue. Boldenone in particular was developed mainly for dystrophy treatment and to increase bone weight and density in osteoporosis.
Nonsteroid peptide alternatives include BPC-157 and TB-500.
Procollagen III is a primary indicator used to determine the rate of collagen synthesis, involved in bones, joints and tendons:
Androgens inhibit osteoclastic bone resorption with increase of bone formation through androgen receptor in bone tissue. Boldenone in particular was developed mainly for dystrophy treatment and to increase bone weight and density in osteoporosis.
- Effect of anabolic steroids on osteoporosis
- The action of anabolic steroids on bone in experimental animals
Nonsteroid peptide alternatives include BPC-157 and TB-500.
Collagen Synthesis
Procollagen III is a primary indicator used to determine the rate of collagen synthesis, involved in bones, joints and tendons:
- Boldenone at 3 mg/kg increases procollagen III by approximately 340% within one week.
- Nandrolone at 3 mg/kg a week increases procollagen III levels by 270%.
- Primobolan at 5 mg/kg increases collagen synthesis by ~180%.
- hGH at 6 IU per day increases the collagen deposition rate by around 250% in damaged collagen structures.
- Stanozolol boosts collagen production and mRNA. Stimulation of Collagen Synthesis by the Anabolic Steroid Stanozolol
- Anavar is prescribed post-surgery and has hundreds of scientific studies demonstrating increased collagen synthesis, accelerated wound healing and enhanced recovery.
- And of course, Estrogen plays an integral role in maintaining bone density and keeping tendons and joints strong.