Steroids, How do we counteract their negatives?

[trueagarthian09]

I would like to ask how people have managed/ mitigated the main health side effects of taking steroids such as testosterone, Aromasin, HCG, Anavar, Masteron.
For example: Injecting any form of testosterone causes a decrease in GnRh pulsatillity therefore the Pituitary glad produces less LH and FSH as a result ur balls shrink and spermatogenesis is fucked, HCG is only an LH receptor agonist at Leydig cells this means that GnRH is not restored as hCG bypasses the pituitary and again does not act as an FSH receptor agonist.
Are there anyways to mitigate FSH level loss? Is it as big a deal as doctors seem to think?

Testosterone Aromatization is necessary to live.- Estradiol is required for HDL maintenance ( I come back to this later), Bone remodeling, CNS stability , Nitric oxide signaling. My question is: When I am taking Aromasin to prevent early Epiphyseal closure and Conversion of Testosterone to oestradiol How do I prevent the side effects of over suppresion- HDL falls, Joints are fucked, Less endothelial protection.

Futhermore with steroids such as Anavar + Masteron ( DHT derived non aromatizing androgens) they increase hepatic lipase activity, decrease ApoA-I synthesis, Increase HDL particle clearance, Increase LDL particle density.
Anavar causes liver enzyme elevation.
Hematocrit ( Test drivern erythroppoiesis) stimulates EPO production in the kidney, suppresses hepcidin, stims bone marrow erythoid percursors, therefore increasing red blood cell mass as a result increasing blood viscosity = Increased blood pressure.

how can this all be mitigated so to have a minimal affect?

 

[Escanor1]

you dont let the steroids rape u

 

[trueagarthian09]

you dont let the steroids rape u

But supression is not only relevant to abusing steroids

 

[gurt]

I would like to ask how people have managed/ mitigated the main health side effects of taking steroids such as testosterone, Aromasin, HCG, Anavar, Masteron.
For example: Injecting any form of testosterone causes a decrease in GnRh pulsatillity therefore the Pituitary glad produces less LH and FSH as a result ur balls shrink and spermatogenesis is fucked, HCG is only an LH receptor agonist at Leydig cells this means that GnRH is not restored as hCG bypasses the pituitary and again does not act as an FSH receptor agonist.
Are there anyways to mitigate FSH level loss? Is it as big a deal as doctors seem to think?

Testosterone Aromatization is necessary to live.- Estradiol is required for HDL maintenance ( I come back to this later), Bone remodeling, CNS stability , Nitric oxide signaling. My question is: When I am taking Aromasin to prevent early Epiphyseal closure and Conversion of Testosterone to oestradiol How do I prevent the side effects of over suppresion- HDL falls, Joints are fucked, Less endothelial protection.

Futhermore with steroids such as Anavar + Masteron ( DHT derived non aromatizing androgens) they increase hepatic lipase activity, decrease ApoA-I synthesis, Increase HDL particle clearance, Increase LDL particle density.
Anavar causes liver enzyme elevation.
Hematocrit ( Test drivern erythroppoiesis) stimulates EPO production in the kidney, suppresses hepcidin, stims bone marrow erythoid percursors, therefore increasing red blood cell mass as a result increasing blood viscosity = Increased blood pressure.

how can this all be mitigated so to have a minimal affect?

because GnRh is pusatile in nature, and a constant level would cause tolerance you cant run a long esther synthetic GnRh. So technically possible if you injected every hour. But obviously thats just not practical and getting the stuff would probably be next to impossible. So hcg is the best you can get

 

[gurt]

I would like to ask how people have managed/ mitigated the main health side effects of taking steroids such as testosterone, Aromasin, HCG, Anavar, Masteron.
For example: Injecting any form of testosterone causes a decrease in GnRh pulsatillity therefore the Pituitary glad produces less LH and FSH as a result ur balls shrink and spermatogenesis is fucked, HCG is only an LH receptor agonist at Leydig cells this means that GnRH is not restored as hCG bypasses the pituitary and again does not act as an FSH receptor agonist.
Are there anyways to mitigate FSH level loss? Is it as big a deal as doctors seem to think?

Testosterone Aromatization is necessary to live.- Estradiol is required for HDL maintenance ( I come back to this later), Bone remodeling, CNS stability , Nitric oxide signaling. My question is: When I am taking Aromasin to prevent early Epiphyseal closure and Conversion of Testosterone to oestradiol How do I prevent the side effects of over suppresion- HDL falls, Joints are fucked, Less endothelial protection.

Futhermore with steroids such as Anavar + Masteron ( DHT derived non aromatizing androgens) they increase hepatic lipase activity, decrease ApoA-I synthesis, Increase HDL particle clearance, Increase LDL particle density.
Anavar causes liver enzyme elevation.
Hematocrit ( Test drivern erythroppoiesis) stimulates EPO production in the kidney, suppresses hepcidin, stims bone marrow erythoid percursors, therefore increasing red blood cell mass as a result increasing blood viscosity = Increased blood pressure.

how can this all be mitigated so to have a minimal affect?

for the e2 just get bloodwork

 

[gurt]

I would like to ask how people have managed/ mitigated the main health side effects of taking steroids such as testosterone, Aromasin, HCG, Anavar, Masteron.
For example: Injecting any form of testosterone causes a decrease in GnRh pulsatillity therefore the Pituitary glad produces less LH and FSH as a result ur balls shrink and spermatogenesis is fucked, HCG is only an LH receptor agonist at Leydig cells this means that GnRH is not restored as hCG bypasses the pituitary and again does not act as an FSH receptor agonist.
Are there anyways to mitigate FSH level loss? Is it as big a deal as doctors seem to think?

Testosterone Aromatization is necessary to live.- Estradiol is required for HDL maintenance ( I come back to this later), Bone remodeling, CNS stability , Nitric oxide signaling. My question is: When I am taking Aromasin to prevent early Epiphyseal closure and Conversion of Testosterone to oestradiol How do I prevent the side effects of over suppresion- HDL falls, Joints are fucked, Less endothelial protection.

Futhermore with steroids such as Anavar + Masteron ( DHT derived non aromatizing androgens) they increase hepatic lipase activity, decrease ApoA-I synthesis, Increase HDL particle clearance, Increase LDL particle density.
Anavar causes liver enzyme elevation.
Hematocrit ( Test drivern erythroppoiesis) stimulates EPO production in the kidney, suppresses hepcidin, stims bone marrow erythoid percursors, therefore increasing red blood cell mass as a result increasing blood viscosity = Increased blood pressure.

how can this all be mitigated so to have a minimal affect?

also why would anyone run a dht derivitive over a testosterone or nandrolone derivitive, when you can't do anything against the balding with those

 

[trueagarthian09]

for the e2 just get bloodwork

See my thing is everyone just spouts blood work as soon as side effects are mentioned, What happens after blood work?

 

[trueagarthian09]

also why would anyone run a dht derivitive over a testosterone or nandrolone derivitive, when you can't do anything against the balding with those

Loads of people take Masteron or anavar

 

[gurt]

See my thing is everyone just spouts blood work as soon as side effects are mentioned, What happens after blood work?

readjust dosing...

 

[gurt]

Loads of people take Masteron or anavar

yeah but why would you risk balding taking those. there not even psychoactive either... thats half the reason you'd take steroids

 

[trueagarthian09]

yeah but why would you risk balding taking those. there not even psychoactive either... thats half the reason you'd take steroids

Baldings partly genetic though and you can just as easily take oral minoxidil, dutasteride, and topical minoxidil. But I partly agree with you however im more focused on the medical issues such as heart and liver damage.

 

[gurt]

Baldings partly genetic though and you can just as easily take oral minoxidil, dutasteride, and topical minoxidil. But I partly agree with you however im more focused on the medical issues such as heart and liver damage.

brodie, dutasteride blocks 5 alpha reductase; test to dht conversion. If you're administering a dht derivative dutasteride is not going to do anything. Minoxidil also doesn't stop balding, and you don't know if you have the gene before its too late

 

[trueagarthian09]

brodie, dutasteride blocks 5 alpha reductase; test to dht conversion. If you're administering a dht derivative dutasteride is not going to do anything. Minoxidil also doesn't stop balding, and you don't know if you have the gene before its too late.

Dutasteride doesnt block DHT derivatives directly you are right. But hairloss isnt only caused by DHT, its driven by total androgen receptor activation at follicle. Dutasteride still reduces endogenous scalp DHT therefore loweing overall androgenic pressure. Minoxidil prolongs folicle growth and delays maturization. Hair folicles dont go from being healthy to dead over night, The process takes months to years even under strong androgens.

 

[birthdefect]

I would like to ask how people have managed/ mitigated the main health side effects of taking steroids such as testosterone, Aromasin, HCG, Anavar, Masteron.
For example: Injecting any form of testosterone causes a decrease in GnRh pulsatillity therefore the Pituitary glad produces less LH and FSH as a result ur balls shrink and spermatogenesis is fucked, HCG is only an LH receptor agonist at Leydig cells this means that GnRH is not restored as hCG bypasses the pituitary and again does not act as an FSH receptor agonist.
Are there anyways to mitigate FSH level loss? Is it as big a deal as doctors seem to think?

Testosterone Aromatization is necessary to live.- Estradiol is required for HDL maintenance ( I come back to this later), Bone remodeling, CNS stability , Nitric oxide signaling. My question is: When I am taking Aromasin to prevent early Epiphyseal closure and Conversion of Testosterone to oestradiol How do I prevent the side effects of over suppresion- HDL falls, Joints are fucked, Less endothelial protection.

Futhermore with steroids such as Anavar + Masteron ( DHT derived non aromatizing androgens) they increase hepatic lipase activity, decrease ApoA-I synthesis, Increase HDL particle clearance, Increase LDL particle density.
Anavar causes liver enzyme elevation.
Hematocrit ( Test drivern erythroppoiesis) stimulates EPO production in the kidney, suppresses hepcidin, stims bone marrow erythoid percursors, therefore increasing red blood cell mass as a result increasing blood viscosity = Increased blood pressure.

how can this all be mitigated so to have a minimal affect?

hcg maintains spermatogenesis through the increase of intratesticular testosterone, which is usually enough but if you're still concerned hmg alongside hcg is an option.

there is no way to prevent the worsening of hdl and getting fucked joints, just dont crash your estrogen get an ultra sensitive e2 test regularly and get lipid tests done regularly. there were some hdl and ldl ancillaries in some thread here but i forgot about it now.

if your blood pressure starts creeping up you can take telmisartan or valsartan while on cycle

mark as solution saar

 

[birthdefect]

brodie, dutasteride blocks 5 alpha reductase; test to dht conversion. If you're administering a dht derivative dutasteride is not going to do anything. Minoxidil also doesn't stop balding, and you don't know if you have the gene before its too late

high dose ru and if you're really desperate estriol cream. other niche drugs do exist (including on this forum) but gatekeeping fr

 

[iceturtles]

If you want good answers you should do some research on the steroid forums