Micrognathic
I have a crippling nicotine addiction
- Joined
- Dec 10, 2025
- Posts
- 22
- Reputation
- 26
ok, maybe I lied a bit. I am going to rush this thread since I just got done with my jerk off shesh. I haven't gooned in quite some time since I was on vacation, my goon sesh lasted way longer than usual, and my hands hurt, so I'm just going to make this thread short and simple. I will be making a follow-up thread soon expanding on everything I mention here and going into more detail, and just more professional overall.
DISCALIMER!!!
I want to apologize for my shitty English, my main language is Arabic. I have been self-teaching myself English for almost a year now, and I'm still in the process of learning, so pls bare with me.
Introduction
We all know that steroid usage comes with a bunch of side effects, but most of you don't know how to properly mitigate these side effects in an effective way, ensuring your safety while on cycle.
In this thread, I am going to explain each major side effect that comes with certain steroids and how to mitigate them properly with the use of certain pharmaceutical compounds.
Buckle up, unzip your panties, cause this thread is gonna make u buuuuuuuuustttttt.
ESTROGENIC SIDE EFFECTS
For gynecomastia, the compound of choice would be raloxifene. What is raloxifene you may ask? it is a selective estrogen receptor modulator (SERM).
Basically, it blocks the estrogen receptors that are found in your breast tissue ( your big fat juicy titties ).
The reason why I prefer raloxifene over normal AIs is that raloxifene does not crush your estrogen systemically, unlike AIs.
Keep in mind that estrogen is very important for us, you need it for neuroprotection and libido, which is why systemically crushing estrogen is a bad move and why raloxifene is the preferred compound of choice for gynecomastia.
Also, raloxifene is more effective than tamoxifen, and studies show that raloxifene has fewer side effects compared to tamoxifen when treating pubertal/induced gynecomastia.
pubmed.ncbi.nlm.nih.gov
onto dosaging, usually for prevention, the typical dose would be around 30mg/day, and if you have existing gyno, my recommendation would be around 60mg/day until your titties become non-existent, then taper off that bitch.
ok, I know that it looks like I am contradicting myself because I literally just said that estrogen is crucial and should not be crushed, but keep in mind that excess estrogen is still brutal, and you'll be experiencing the same side effects as the ones I mentioned above.
Now the key here is to find the perfect dosage," the estrogenic balance".
One of the most important things in life is to find balance, and let me tell you something, do you ever wonder why you randomly start getting these gay thoughts in ur head, although you've been straight your whole life and never purposely had a gay thought before?
Well that my friend is your body trying to find balance, and it's totally normal don't be scared, it happens to all of us, but I think it happens to me the most, and now I'm starting to engage in weird gay shit.
ANYWAY, back to the main topic, estrogen shouldn't be nuked rather must be controlled smartly. Now I'm not going to spoonfeed you and tell you exactly what to do since there are a bunch of threads talking about this, but basically get your baseline E2 levels checked and start with the typical dosage, then from there it's just trial and error until you find the perfect range for YOU. everyone has a different range of E2 that their body responds perfectly to, so keep that in mind as well.
For estrogen control, the compound of choice would be Aromasin (exemestane). The reason why I chose this over Arimidex because it's a suicide inhibitor that permanently disables the aromatase enzyme, unlike Arimidex, which can cause estrogen rebound after you stop it. Also, it won't affect your lipids as much as Arimidex does.
For dosing, the typical dose is usually 12.5mg EOD, or even better, just use it when needed if you start noticing high E2 sides, gyno water retention, etc, and pls keep in mind, do not crush ur E2, it is very crucial and needed for your joints and brain health, and many other shit in your body.
Also, for all you heightmaxing faggots, I would recommend using Arimidex over aromasin since it's a non-steroidal AI that doesn't have any androgenic side effects and is easier to dose compared to aromasin.
btw, all of you gay heightmaxers aren't gaining an inch of height, keep coping you utter subhuman manlet nigger retarded faggot.
Keep in mind that most of you incels wouldn't even need an AI if you have an appropriate protocol. I started noticing many people on here were switching from testosterone enanthate/cypionate to testosterone propionate and going from pinning 2x a week to daily. The reason why this protocol is much more appropriate than the others is that test p is a very short ester compared to the rest, and combine that with daily pinning, you're going to have a stable release of testosterone which mimics ur body's natural shit, but obv you'll still have higher T levels therefore there would be no massive spikes in E2 which means no high E2 side effects so no man tittties for you lil nigga.
But let's be honest, most of you incels can't even manage to do basic self-care care so I highly doubt that you'd have the willpower to pin on a daily basis, so just kys lil nigga.
(Saint ER may he rest in peace )
Also, it's always beneficial to keep an AI on hand in case of an emergency, but I can assure you if you're pinning daily, you prob won't even need to use it, but just keep that in mind.
This doesn't need an explanation, we all know that DHT binds to your scalp follicles, which causes androgenic alopecia (hair loss), although androgenic alopecia is a mix of genetics and certain hormones, you can still prevent it and control it with the use of certain pharmaceutical compounds, and as a looksmaxer, that should be the first thing you should be doing since we all know that if you have no hair you have no life. over for balding manlet cells thinking they can "heightmax" and "hairmax", just get LL and hair transplant and call it a day lil nigga, but no, these little shitlets thinking that they're high iq pharmaceutical masterminds as if they can iq mog the one and only @Micrognathic, little do we know that all of us are sub 80 iq manlet gpt abusers jfl.
For hair loss, the compound of choice would be no other than dutasteride. The reason why I chose this over finasteride is that dut inhibits both type 1 and type 2 alpha reductase enzymes, whereas finasteride only inhibits type 2. Also, dut reduces serum DHT by 90% vs fin 70%.
onto dosing, the typical dose that is prescribed for dutasteride by jew doctors is 0.5 mg ED, and let me tell you that this dosage is utter dogshit and won't do shit, jk it's a good dose, but the IDEAL dosage would be 2.5 mg ED. Why is this the ideal dosage you may ask?
2.5mg provides an extra 30% scalp DHT suppression while changing serum levels by around 4% compared to 0.5mg, so there is literally 0 reason not to be using 2.5mg of dutasteride.
( I STOLE THIS PIECE OF INFO FROM SOME FAMOUS NIGGA ON HERE, BUT I HIGHLY DOUBT THAT HE'LL SEE THIS THREAD, SO I SHOULD BE IN THE CLEAR. IF YOU ARE SEEING THIS, THANKS FOR THE FREE INFO LIL NIGGA, I'M NOT GIVING YOU CREDIT. )
( NEVERMIND, I JUST FOUND OUT THIS NIGGA IS A MOD. I AM SO SORRY, PLS DON'T ATTACK ME ALL CREDITS GO TO @chadisbeingmade
IN ALL SERIOUSNESS, GENUINELY THANK YOU FOR THIS PIECE OF INFO )
Also, another good compound is RU58841, which is a topical androgen receptor antagonist that basically targets DHT on the scalp rather than systemically blocking DHT, as is the case with dutasteride. ig the reason why someone would want to use this over dut is to maybe preserve libido, but I have seen some anecdotes saying that RU58841 can go systemic, so take that with a grain of salt.
Also, most of the erectile dysfunction caused by dut is prob fake reports made by Jewish researchers trying to fearmonger you. Trust me, there's nothing to worry about, and even if you get ED, my dick is always free at your disposal you stinky cuck.
For dosing, the typical dose would be around 50-100mg of RU58841 to the scalp daily.
I ALSO STOLE THIS FROM @chadisbeingmade once again, thank you lil nigga.
Both testosterone and DHT bind to the androgen receptors that are found in your sebaceous glands, which forces them to enlarge and buuuuuuusstttt the excess oil. Once again, I am not going to explain this any further cuz im too lazy, and I'm saving most of the info for the actual thread I'm going to make.
The only compound you should be using for acne, whether it's from steroids, hormonal imbalances, or too much jerking off, is isotretinoin. No other compound will work better for acne than isotretinoin, so pls don't bother wasting ur time with the rest.
now the dose is really tricky since there is no one dose applicable to all, it varies from person to person. I would recommend that you use this website to calculate your cumulative and daily dose of Accutane.
ig im going to give you guys a baseline dosage for all the lazy fuckers out there.
for severe acne, my recommendation would be 40-80mg, and for prevention/mild acne, 10-20mg should work well. aswell u can microdose isotretinoin for antiaging and collagen-boosting effects, which many celebrities do. The typical dose for that is 5-10 mg.
CARDIOVASCULAR SUPPORT
(LEFT VENTRICULAR HYPERTROPHY, HIGH BP, FUCKED UP LIPIDS, HIGH LDL/ LOW HDL)
This is what gets most people killed from steroid usage. Do you ever wonder why your favourite gymbro died at the age of 40? Most prob it's because of this.
BP & LEFT VENTRICULAR HYPERTROPHY PROTECTION
Certain steroids stimulate the mineralocorticoid receptors that are found in your kidneys, which forces them to hold onto sodium and water, therefore increasing your blood volume and blood pressure, basically giving you hypertension.
The compound of choice to prevent this side effect would be Telmisartan. What is Telmisartan, you may ask? it is an Angiotensin 2 Receptor Blocker (ARB).
The reason why I chose Telmisartan is that it selectively reduces your visceral fat, while improving your insulin sensitivity, and it specifically prevents left ventricular hypertrophy caused by androgens.
the typical dosage of Telmisartan would be around 40 mg-80 mg, usually taken at night.
A certain study, where Telmisartan was used to treat hypertension, showed that 80mg of Telmisartan outperformed a well-known drug used to treat high bp called Ramipril, and had a significantly better 24-hour control than Losartan/Valsartan, which is basically Telmisartan's brother from another mother.
pmc.ncbi.nlm.nih.gov
Certain steroids, especially orals such as Anavar, Winstrol, and Dianabol, massively stimulate HTGL (Hepatic Triglyceride Lipase) in your liver. This particular enzyme rapes the living shit out of ur HDL, which is ur good cholesterol. It breaks down these particles so fast to a point where ur own body can't even replace them, causing ur HDL levels to drop significantly into the single-digit range, which leads to ur bad cholesterol (LDL) building up in your arteries.
The compound of choice would be Ezetimibe. I will not be going into detail, but basically, it prevents the absorption of cholesterol in the intestines.
The typical dosage for Ezetimibe would be around 10mg daily.
In a certain study, Ezetimibe lowered LDL by 18% when used by itself, and when combined with a low-dose statin, it decreased LDL by around 40%.
I would recommend you to read this thread made by the user @SlayerJonas, he goes into much more detail and explains it way better than me.
Thread 'How to manage your cholesterol effectively - The last secret about longevity'
looksmax.org
The second compound of choice would be Citrus Bergamot, which basically acts like a weak statin.
For dosing, the typical dose of Citrus Bergamot would be around 1000mg daily, but pls keep in mind it must be a high-quality extract and not some random shitty one from Indiamart. Although Indiamart is a good and cheap place to buy ur shit, you can just buy Citrus Bergamot OTC.
In a certain study, Citrus bergamot dosed at 500-1000mg/day reduced LDL by around 24-40% and Triglycerides by around 30%
Also, this is safer than Rosuvastatin since it doesn't stress your liver, whereas Rosuvastatin rapes the living shit out of it, although Rosuvastatin is more potent in lowering LDL cholesterol, so keep that in mind.
pmc.ncbi.nlm.nih.gov
ORGAN SUPPORT (Liver & Kidneys)
Hepatotoxicity
We all know that orals like Dbol, Anadrol, and Winstrol fuck ur liver up, and the reason why this happens is that there is a chemical modification made to these certain steroids called 17-alpha alkylated. This chemical modification allows these compounds to pass through ur liver without getting degraded, therefore causing cholestasis, which is a condition where the bile flow stops, causing the toxic bile acids to build up in your liver cells, literally poisoning your liver cells from the inside out.
The compound of choice to prevent Hepatotoxicity while on cycle would be Tudca, which is a bile acid that prevents cholestasis.
Why I chose Tudca over NAC is that Tudca specifically treats cholestasis, whereas NAC is used for general oxidative stress. These two compounds are usually combined, but I wouldn't recommend overusing antioxidants since they can fuck u up even more. I advise you to read this thread made by the user @7evenvox22.
Thread 'Do antioxidants secretly damage your health?'
looksmax.org
Now onto dosing, my recommendation would be 500-1000mg of Tudca daily, or only on days you take your orals.
A certain study showed a 40% decrease in ALP levels ( the bile buildup marker ) and an 80% decrease in ALT levels in patients with liver cirrhosis.
KIDNEY DEFENSE
Now we all know that steroids will massively increase lean muscle mass and bp, but did you know that due to this, it will force your kidneys to filter the blood at an unsustainable rate. This stress will physically damage the delicate filters called glomeruli that are found in your kidneys.
Also, recent studies confirm that steroids are directly toxic to your kidney cells, which leads to a specific type of scarring called focal segmental glomerulosclerosis (FSGS), which is a permanent type of scarring and can lead to kidney failure, brutal.
The compound of choice to combat this would be Astragalus Membranaceus. It works by increasing your renal blood flow and reduces the protein in your urine.
Typical dosage would be around 4g-15g.
A certain study showed that Astragalus Membranaceus significantly stabilized eGFR ( your kidney filtration rate ) as well as delaying the decline compared to the other controlled groups.
pubmed.ncbi.nlm.nih.gov
HPTA & FERTILITY (preserving your stinky Dalit Jewish balllzz)
we all know that exogenous testosterone causes Testicular atrophy and makes you shoot blanks lmao.
Testicular Maintenance ( maintaining your stinky Dalit Jewish balllzz)
The compound of choice for maintaining your stinky Dalit Jewish balllzz would be HCG (Human Chorionic Gonadotropin).
Basically, it works by mimicking LH, therefore keeping your Leydig cells active, which preserves your stinky Dalit Jewish balllzz.
The recommended dose would be somewhere around 250IU to 500IU EOD.
A certain study showed that Men on TRT + 250IU HCG EOD maintained intratesticular testosterone levels only 7% below baseline, whereas TRT-only patients dropped by 94% and Men on 500IU EOD actually increased it by 26%.
pmc.ncbi.nlm.nih.gov
PROLACTIN CONTROL
(19-Nors / Deca / Tren)
If you have browsed a BB forum before, or even on here, you have probably stumbled on the term "Deca Dick." This term basically refers to sexual dysfunction caused by elevated prolactin levels, which not only fucks ur dick up but also makes ur titties lactate lmao.
Dopamine Agonist
The compound of choice to combat this is going to be cabergoline.
How does it work? it works by agonizing your dopamine receptors, which directly inhibits prolactin secretion from the pituitary.
For dosing, my recommendation would be 0.25mg to 0.5mg once per week.
PLS DO NOT FUCKING OVERDOSE ON THIS SHIT. IT IS EXTREMELY POTENT. PLEASE KEEP THAT IN MIND U STINKY NIGGER.
A certain study where Cabergoline was used to treat hyperprolactinemia in 455 patients showed that Cabergoline Normalized prolactin levels in 86% of patients.
pubmed.ncbi.nlm.nih.gov
FINAL THOUGHTS
All of what I just mentioned is cope and doesn't work and never will. All you can do is ldar and pray that you don't die whilegooning, no, I mean while on cycle.
pls ignore my crappy English. I tried my best, and I know it's horrendous, but pls bare with me. This is my first thread, and pls do not follow any of the advice I said on here since this is a very surface-level thread. I am going to be making a way more in-depth thread in detail explaining every cookie and crumb, and I promise I will make it as best as possible. I didn't include all of the compounds since I'm saving them for my next thread. Depending on how many reactions and the feedback from this thread, I might or might not be making a follow-up and actually put effort into it and make it professional. Thanks for reading ig. Now go jerk off u stinky Dalit.
DISCALIMER!!!
I want to apologize for my shitty English, my main language is Arabic. I have been self-teaching myself English for almost a year now, and I'm still in the process of learning, so pls bare with me.
Most of the people on here think that steroids are simple plug-and-play, but that couldn't be further from the truth. If you want to live past 40 while roiding, there needs to be some measures and precautions you should be taking to ensure you stay healthy while on cycle.We all know that steroid usage comes with a bunch of side effects, but most of you don't know how to properly mitigate these side effects in an effective way, ensuring your safety while on cycle.
In this thread, I am going to explain each major side effect that comes with certain steroids and how to mitigate them properly with the use of certain pharmaceutical compounds.
Buckle up, unzip your panties, cause this thread is gonna make u buuuuuuuuustttttt.
ESTROGENIC SIDE EFFECTS
I am not going to explain this, but we all know that testosterone converts into estrogen via the aromatase enzyme, which leads to gyno (man titties) and water retention.
For gynecomastia, the compound of choice would be raloxifene. What is raloxifene you may ask? it is a selective estrogen receptor modulator (SERM).
Basically, it blocks the estrogen receptors that are found in your breast tissue ( your big fat juicy titties ).
The reason why I prefer raloxifene over normal AIs is that raloxifene does not crush your estrogen systemically, unlike AIs.
Keep in mind that estrogen is very important for us, you need it for neuroprotection and libido, which is why systemically crushing estrogen is a bad move and why raloxifene is the preferred compound of choice for gynecomastia.
Also, raloxifene is more effective than tamoxifen, and studies show that raloxifene has fewer side effects compared to tamoxifen when treating pubertal/induced gynecomastia.
Beneficial effects of raloxifene and tamoxifen in the treatment of pubertal gynecomastia - PubMed
Inhibition of estrogen receptor action in the breast appears to be safe and effective in reducing persistent pubertal gynecomastia, with a better response to raloxifene than to tamoxifen. Further study is required to determine that this is truly a treatment effect.
pubmed.ncbi.nlm.nih.gov
onto dosaging, usually for prevention, the typical dose would be around 30mg/day, and if you have existing gyno, my recommendation would be around 60mg/day until your titties become non-existent, then taper off that bitch.
ESTROGEN CONTROLok, I know that it looks like I am contradicting myself because I literally just said that estrogen is crucial and should not be crushed, but keep in mind that excess estrogen is still brutal, and you'll be experiencing the same side effects as the ones I mentioned above.
Now the key here is to find the perfect dosage," the estrogenic balance".
One of the most important things in life is to find balance, and let me tell you something, do you ever wonder why you randomly start getting these gay thoughts in ur head, although you've been straight your whole life and never purposely had a gay thought before?
Well that my friend is your body trying to find balance, and it's totally normal don't be scared, it happens to all of us, but I think it happens to me the most, and now I'm starting to engage in weird gay shit.
ANYWAY, back to the main topic, estrogen shouldn't be nuked rather must be controlled smartly. Now I'm not going to spoonfeed you and tell you exactly what to do since there are a bunch of threads talking about this, but basically get your baseline E2 levels checked and start with the typical dosage, then from there it's just trial and error until you find the perfect range for YOU. everyone has a different range of E2 that their body responds perfectly to, so keep that in mind as well.
For estrogen control, the compound of choice would be Aromasin (exemestane). The reason why I chose this over Arimidex because it's a suicide inhibitor that permanently disables the aromatase enzyme, unlike Arimidex, which can cause estrogen rebound after you stop it. Also, it won't affect your lipids as much as Arimidex does.
For dosing, the typical dose is usually 12.5mg EOD, or even better, just use it when needed if you start noticing high E2 sides, gyno water retention, etc, and pls keep in mind, do not crush ur E2, it is very crucial and needed for your joints and brain health, and many other shit in your body.
Also, for all you heightmaxing faggots, I would recommend using Arimidex over aromasin since it's a non-steroidal AI that doesn't have any androgenic side effects and is easier to dose compared to aromasin.
btw, all of you gay heightmaxers aren't gaining an inch of height, keep coping you utter subhuman manlet nigger retarded faggot.
MINI DISCLAIMER!!!Keep in mind that most of you incels wouldn't even need an AI if you have an appropriate protocol. I started noticing many people on here were switching from testosterone enanthate/cypionate to testosterone propionate and going from pinning 2x a week to daily. The reason why this protocol is much more appropriate than the others is that test p is a very short ester compared to the rest, and combine that with daily pinning, you're going to have a stable release of testosterone which mimics ur body's natural shit, but obv you'll still have higher T levels therefore there would be no massive spikes in E2 which means no high E2 side effects so no man tittties for you lil nigga.
But let's be honest, most of you incels can't even manage to do basic self-care care so I highly doubt that you'd have the willpower to pin on a daily basis, so just kys lil nigga.
(Saint ER may he rest in peace
) Also, it's always beneficial to keep an AI on hand in case of an emergency, but I can assure you if you're pinning daily, you prob won't even need to use it, but just keep that in mind.
Hair Loss & Acne This doesn't need an explanation, we all know that DHT binds to your scalp follicles, which causes androgenic alopecia (hair loss), although androgenic alopecia is a mix of genetics and certain hormones, you can still prevent it and control it with the use of certain pharmaceutical compounds, and as a looksmaxer, that should be the first thing you should be doing since we all know that if you have no hair you have no life. over for balding manlet cells thinking they can "heightmax" and "hairmax", just get LL and hair transplant and call it a day lil nigga, but no, these little shitlets thinking that they're high iq pharmaceutical masterminds as if they can iq mog the one and only @Micrognathic, little do we know that all of us are sub 80 iq manlet gpt abusers jfl.
For hair loss, the compound of choice would be no other than dutasteride. The reason why I chose this over finasteride is that dut inhibits both type 1 and type 2 alpha reductase enzymes, whereas finasteride only inhibits type 2. Also, dut reduces serum DHT by 90% vs fin 70%.
onto dosing, the typical dose that is prescribed for dutasteride by jew doctors is 0.5 mg ED, and let me tell you that this dosage is utter dogshit and won't do shit, jk it's a good dose, but the IDEAL dosage would be 2.5 mg ED. Why is this the ideal dosage you may ask?
2.5mg provides an extra 30% scalp DHT suppression while changing serum levels by around 4% compared to 0.5mg, so there is literally 0 reason not to be using 2.5mg of dutasteride.
( I STOLE THIS PIECE OF INFO FROM SOME FAMOUS NIGGA ON HERE, BUT I HIGHLY DOUBT THAT HE'LL SEE THIS THREAD, SO I SHOULD BE IN THE CLEAR. IF YOU ARE SEEING THIS, THANKS FOR THE FREE INFO LIL NIGGA, I'M NOT GIVING YOU CREDIT. )
( NEVERMIND, I JUST FOUND OUT THIS NIGGA IS A MOD. I AM SO SORRY, PLS DON'T ATTACK ME ALL CREDITS GO TO @chadisbeingmade
IN ALL SERIOUSNESS, GENUINELY THANK YOU FOR THIS PIECE OF INFO
)Also, another good compound is RU58841, which is a topical androgen receptor antagonist that basically targets DHT on the scalp rather than systemically blocking DHT, as is the case with dutasteride. ig the reason why someone would want to use this over dut is to maybe preserve libido, but I have seen some anecdotes saying that RU58841 can go systemic, so take that with a grain of salt.
Also, most of the erectile dysfunction caused by dut is prob fake reports made by Jewish researchers trying to fearmonger you. Trust me, there's nothing to worry about, and even if you get ED, my dick is always free at your disposal you stinky cuck.
For dosing, the typical dose would be around 50-100mg of RU58841 to the scalp daily.
I ALSO STOLE THIS FROM @chadisbeingmade once again, thank you lil nigga.
ACNE Both testosterone and DHT bind to the androgen receptors that are found in your sebaceous glands, which forces them to enlarge and buuuuuuusstttt the excess oil. Once again, I am not going to explain this any further cuz im too lazy, and I'm saving most of the info for the actual thread I'm going to make.
The only compound you should be using for acne, whether it's from steroids, hormonal imbalances, or too much jerking off, is isotretinoin. No other compound will work better for acne than isotretinoin, so pls don't bother wasting ur time with the rest.
now the dose is really tricky since there is no one dose applicable to all, it varies from person to person. I would recommend that you use this website to calculate your cumulative and daily dose of Accutane.
ig im going to give you guys a baseline dosage for all the lazy fuckers out there.
for severe acne, my recommendation would be 40-80mg, and for prevention/mild acne, 10-20mg should work well. aswell u can microdose isotretinoin for antiaging and collagen-boosting effects, which many celebrities do. The typical dose for that is 5-10 mg.
CARDIOVASCULAR SUPPORT
(LEFT VENTRICULAR HYPERTROPHY, HIGH BP, FUCKED UP LIPIDS, HIGH LDL/ LOW HDL)
This is what gets most people killed from steroid usage. Do you ever wonder why your favourite gymbro died at the age of 40? Most prob it's because of this.
BP & LEFT VENTRICULAR HYPERTROPHY PROTECTION
Certain steroids stimulate the mineralocorticoid receptors that are found in your kidneys, which forces them to hold onto sodium and water, therefore increasing your blood volume and blood pressure, basically giving you hypertension.
The compound of choice to prevent this side effect would be Telmisartan. What is Telmisartan, you may ask? it is an Angiotensin 2 Receptor Blocker (ARB).
The reason why I chose Telmisartan is that it selectively reduces your visceral fat, while improving your insulin sensitivity, and it specifically prevents left ventricular hypertrophy caused by androgens.
the typical dosage of Telmisartan would be around 40 mg-80 mg, usually taken at night.
A certain study, where Telmisartan was used to treat hypertension, showed that 80mg of Telmisartan outperformed a well-known drug used to treat high bp called Ramipril, and had a significantly better 24-hour control than Losartan/Valsartan, which is basically Telmisartan's brother from another mother.
A Review of Telmisartan in the Treatment of Hypertension: Blood Pressure Control in the Early Morning Hours - PMC
Measurement of blood pressure in the clinic may provide a false impression of blood pressure control. Ambulatory blood pressure monitoring (ABPM) allows the automatic recording of the circadian variation in blood pressure and evaluation of the ...
pmc.ncbi.nlm.nih.gov
Lipids (Cholesterol)Certain steroids, especially orals such as Anavar, Winstrol, and Dianabol, massively stimulate HTGL (Hepatic Triglyceride Lipase) in your liver. This particular enzyme rapes the living shit out of ur HDL, which is ur good cholesterol. It breaks down these particles so fast to a point where ur own body can't even replace them, causing ur HDL levels to drop significantly into the single-digit range, which leads to ur bad cholesterol (LDL) building up in your arteries.
The compound of choice would be Ezetimibe. I will not be going into detail, but basically, it prevents the absorption of cholesterol in the intestines.
The typical dosage for Ezetimibe would be around 10mg daily.
In a certain study, Ezetimibe lowered LDL by 18% when used by itself, and when combined with a low-dose statin, it decreased LDL by around 40%.
I would recommend you to read this thread made by the user @SlayerJonas, he goes into much more detail and explains it way better than me.
Thread 'How to manage your cholesterol effectively - The last secret about longevity'
How to manage your cholesterol effectively - The last secret about longevity
How to manage your cholesterol effectively - The last secret about longevity What is cholesterol? Disclaimer: For the sake of this thread, I want you to forget everything you’ve ever heard about cholesterol. Cholesterol is a fatty substance essential for many processes in the body and is...
looksmax.org
The second compound of choice would be Citrus Bergamot, which basically acts like a weak statin.
For dosing, the typical dose of Citrus Bergamot would be around 1000mg daily, but pls keep in mind it must be a high-quality extract and not some random shitty one from Indiamart. Although Indiamart is a good and cheap place to buy ur shit, you can just buy Citrus Bergamot OTC.
In a certain study, Citrus bergamot dosed at 500-1000mg/day reduced LDL by around 24-40% and Triglycerides by around 30%
Also, this is safer than Rosuvastatin since it doesn't stress your liver, whereas Rosuvastatin rapes the living shit out of it, although Rosuvastatin is more potent in lowering LDL cholesterol, so keep that in mind.
Unveiling the Power of Bergamot: Beyond Lipid-Lowering Effects - PMC
Dyslipidemia is a leading risk factor for cardiovascular diseases. Nutraceuticals for the management of dyslipidemia are gaining growing attention. Derived from food sources, they represent a promising adjunctive or alternative therapeutic option in ...
pmc.ncbi.nlm.nih.gov
ORGAN SUPPORT (Liver & Kidneys)
Hepatotoxicity
We all know that orals like Dbol, Anadrol, and Winstrol fuck ur liver up, and the reason why this happens is that there is a chemical modification made to these certain steroids called 17-alpha alkylated. This chemical modification allows these compounds to pass through ur liver without getting degraded, therefore causing cholestasis, which is a condition where the bile flow stops, causing the toxic bile acids to build up in your liver cells, literally poisoning your liver cells from the inside out.
The compound of choice to prevent Hepatotoxicity while on cycle would be Tudca, which is a bile acid that prevents cholestasis.
Why I chose Tudca over NAC is that Tudca specifically treats cholestasis, whereas NAC is used for general oxidative stress. These two compounds are usually combined, but I wouldn't recommend overusing antioxidants since they can fuck u up even more. I advise you to read this thread made by the user @7evenvox22.
Thread 'Do antioxidants secretly damage your health?'
Do antioxidants secretly damage your health?
Thread Song: Do antioxidants secretly damage your health? Unfortunately, they actually could be based on the way many utilize them, but let me elucidate this point first so there's no misunderstanding, and subsequently tell you how to fix and avoid it. Different types of...
looksmax.org
A certain study showed a 40% decrease in ALP levels ( the bile buildup marker ) and an 80% decrease in ALT levels in patients with liver cirrhosis.
KIDNEY DEFENSE
Now we all know that steroids will massively increase lean muscle mass and bp, but did you know that due to this, it will force your kidneys to filter the blood at an unsustainable rate. This stress will physically damage the delicate filters called glomeruli that are found in your kidneys.
Also, recent studies confirm that steroids are directly toxic to your kidney cells, which leads to a specific type of scarring called focal segmental glomerulosclerosis (FSGS), which is a permanent type of scarring and can lead to kidney failure, brutal.
The compound of choice to combat this would be Astragalus Membranaceus. It works by increasing your renal blood flow and reduces the protein in your urine.
Typical dosage would be around 4g-15g.
A certain study showed that Astragalus Membranaceus significantly stabilized eGFR ( your kidney filtration rate ) as well as delaying the decline compared to the other controlled groups.
Add-on astragalus in type 2 diabetes and chronic kidney disease: A multi-center, assessor-blind, randomized controlled trial - PubMed
In patients with type 2 diabetes, stage 2 to 3 CKD and macroalbuminuria, add-on astragalus for 48 weeks further stabilized kidney function on top of standard care.
pubmed.ncbi.nlm.nih.gov
HPTA & FERTILITY (preserving your stinky Dalit Jewish balllzz)
we all know that exogenous testosterone causes Testicular atrophy and makes you shoot blanks lmao.
Testicular Maintenance ( maintaining your stinky Dalit Jewish balllzz)
The compound of choice for maintaining your stinky Dalit Jewish balllzz would be HCG (Human Chorionic Gonadotropin).
Basically, it works by mimicking LH, therefore keeping your Leydig cells active, which preserves your stinky Dalit Jewish balllzz.
The recommended dose would be somewhere around 250IU to 500IU EOD.
A certain study showed that Men on TRT + 250IU HCG EOD maintained intratesticular testosterone levels only 7% below baseline, whereas TRT-only patients dropped by 94% and Men on 500IU EOD actually increased it by 26%.
Indications for the use of human chorionic gonadotropic hormone for the management of infertility in hypogonadal men - PMC
Hypogonadism among men desiring fertility preservation presents a unique challenge to physicians. Over the past decade the number of younger men with hypogonadism has increased dramatically. These men are often treated with testosterone replacement ...
pmc.ncbi.nlm.nih.gov
PROLACTIN CONTROL
(19-Nors / Deca / Tren)
If you have browsed a BB forum before, or even on here, you have probably stumbled on the term "Deca Dick." This term basically refers to sexual dysfunction caused by elevated prolactin levels, which not only fucks ur dick up but also makes ur titties lactate lmao.
Dopamine Agonist
The compound of choice to combat this is going to be cabergoline.
How does it work? it works by agonizing your dopamine receptors, which directly inhibits prolactin secretion from the pituitary.
For dosing, my recommendation would be 0.25mg to 0.5mg once per week.
PLS DO NOT FUCKING OVERDOSE ON THIS SHIT. IT IS EXTREMELY POTENT. PLEASE KEEP THAT IN MIND U STINKY NIGGER.
A certain study where Cabergoline was used to treat hyperprolactinemia in 455 patients showed that Cabergoline Normalized prolactin levels in 86% of patients.
Cabergoline in the treatment of hyperprolactinemia: a study in 455 patients - PubMed
Cabergoline is a new long-acting dopamine agonist that is very effective and well tolerated in patients with pathological hyperprolactinemia. The aim of this study was to examine, in a very large number of hyperprolactinemic patients, the ability to normalize PRL levels with cabergoline, to...
pubmed.ncbi.nlm.nih.gov
FINAL THOUGHTS
All of what I just mentioned is cope and doesn't work and never will. All you can do is ldar and pray that you don't die while
pls ignore my crappy English. I tried my best, and I know it's horrendous, but pls bare with me. This is my first thread, and pls do not follow any of the advice I said on here since this is a very surface-level thread. I am going to be making a way more in-depth thread in detail explaining every cookie and crumb, and I promise I will make it as best as possible. I didn't include all of the compounds since I'm saving them for my next thread. Depending on how many reactions and the feedback from this thread, I might or might not be making a follow-up and actually put effort into it and make it professional. Thanks for reading ig. Now go jerk off u stinky Dalit.
