[THEORY] The Genetic Lottery Pill | Chads are Made By Chance

ilovekolaches

ilovekolaches

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by @ilovekolaches

*This post will take three minutes to read, but if you're lazy the bare-bone minimum to understand are highlighted in green

Hypothesis: We all had a chance at being Chad's. That's the truth. You can all it a cope if you want, but it's true. Our genes always had the potential to be attractive and sought after. Let me explain.

When a mother and a father conceive a child that child is formed of 46 chromosomes, or 23 from each parent. These chromosomes are what makes up your genes, effecting your behaviour, blood type and genetics. But we all know this, this probably isn't anything new to you. But what you may not have known is that each gene or chromosome comes with multiple variants of the same traits. one chromosome can be expressed in multiple different forms, for example, your blood type can be A, AB, B or O and many times is something that neither of your parents have.

This is called Gene Expression.

*Gene expression is the process by which information from a gene is used in the synthesis of a functional gene product that enable to produce protein as the end product. These products are often proteins, but in non-protein-coding genes such as transfer RNA or small nuclear RNA genes, the product is a functional RNA.

But some genes do not end up expressed even if you have the chromosomes required for it. This is how deadly diseases such as cancer can be unknowingly transferred through a blood-line for years before actually becoming active in a person.


Gene expression control

But this isn't all, there also exists something called genetic variation,Genetic variation refers to diversity in gene frequencies. Genetic variation can refer to differences between individuals or to differences between populations. Mutation is the ultimate source of genetic variation, but mechanisms such as sexual reproduction and genetic drift contribute to it as well.

Paired chromosomes separate
Recombination














Genetic Variation, Gene Expression, Recombination as well as meiosis all contribute towards this randomization of trait combinations and possible proteins and features your genetics can express.

*Meiosis is a special type of cell division of germ cells in sexually-reproducing organisms used to produce the gametes, such as sperm or egg cells. It involves two rounds of division that ultimately result in four cells with only one copy of each paternal and maternal chromosome. Meiosis produces four haploid (n) gametes that are genetically unique from each other and the original parent (germ) cell.


Unknown










Even without this however there's already 8,324,608 possible combination for chromosomes.

FIG2
FIG1














And you weren't the only one who had 8 million possible variations of their genes, every single (non Autistic) person had a one in 8 million chance of looking how they do. In one of these 8 million you had the chance to be significantly more attractive. Maybe your genes for a negative canthal tilt are turned off and not expressed in one of these 8 million, maybe you don't have messed up teeth, maybe you have and extra inch or two on your height, maybe you have higher testosterone levels and more sexually day-orphic features in one of these realities.

If you have a sibling is your sibling more attractive than you? Do you mo
g them? Chad's, or good looking people are simply those who got the best out of their 8 million combinations and mutations.

*This means that there are a few combinations that would produce the most attractive facial
aesthetic, and one that would allow you to have easiest access to sex and resources.

These are the Odds of The Genetic Lottery, 1 in 8 Million.

The one in 8 million chance that you turned out how you did.



BONUS: AUTISTISM SHOWS STRONG
CORRELATION WITH THE DUPLICATION OF THE 22ND CHROMOSOMES AND THE DELETIONS OF SOME, BECAUSE OF THIS, AUTISTIC PEOPLE TYPICALLY HAVE A LOWER CHANCE IN THE GENETIC LOTTERY. https://www.spectrumnews.org/news/dna-doubling-on-chromosome-22-shows-strong-ties-to-autism/D

Sources:
 

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ilovekolaches
@ilovekolaches


*This post will take three minutes to read, but if you're lazy the bare-bone minimum to understand are highlighted in green

Hypothesis: We all had a chance at being Chad's. That's the truth. You can all it a cope if you want, but it's true. Our genes always had the potential to be attractive and sought after. Let me explain.

When a mother and a father conceive a child that child is formed of 46 chromosomes, or 23 from each parent. These chromosomes are what makes up your genes, effecting your behaviour, blood type and genetics. But we all know this, this probably isn't anything new to you. But what you may not have known is that each gene or chromosome comes with multiple variants of the same traits. one chromosome can be expressed in multiple different forms, for example, your blood type can be A, AB, B or O and many times is something that neither of your parents have.

This is called Gene Expression.

*Gene expression is the process by which information from a gene is used in the synthesis of a functional gene product that enable to produce protein as the end product. These products are often proteins, but in non-protein-coding genes such as transfer RNA or small nuclear RNA genes, the product is a functional RNA.

But some genes do not end up expressed even if you have the chromosomes required for it. This is how deadly diseases such as cancer can be unknowingly transferred through a blood-line for years before actually becoming active in a person.


Gene expression control

But this isn't all, there also exists something called genetic variation,Genetic variation refers to diversity in gene frequencies. Genetic variation can refer to differences between individuals or to differencesbetween populations. Mutation is the ultimate source of genetic variation, but mechanisms such as sexual reproduction and genetic drift contribute to it as well.

Paired chromosomes separate

Recombination














Genetic Variation, Gene Expression, Recombination as well as meiosis all contribute towards this randomization of trait combinations and possible proteins and features your genetics can express.

*Meiosis is a special type of cell division of germ cells in sexually-reproducing organisms used to produce the gametes, such as sperm or egg cells. It involves two rounds of division that ultimately result in four cells with only one copy of each paternal and maternal chromosome. Meiosis produces four haploid (n)gametes that are genetically unique from each other and the original parent (germ) cell.


Unknown










Even without this however there's already 8,324,608 possible combination for chromosomes.

FIG2

FIG1














And you weren't the only one who had 8 million possible variations of their genes, every single (non Autistic) person had a one in 8 million chance of looking how they do. In one of these 8 million you had the chance to be significantly more attractive. Maybe your genes for a negative canthal tilt are turned off and not expressed in one of these 8 million, maybe you don't have messed up teeth, maybe you have and extra inch or two on your height, maybe you have higher testosterone levels and more sexually day-orphic features in one of these realities.

If you have a sibling is your sibling more attractive than you? Do you mo
g them? Chad's, or good looking people are simply those who got the best out of their 8 million combinations and mutations.

*This means that there are a few combinations that would produce the most attractive facial
aesthetic, and one that would allow you to have easiest access to sex and resources.

These are the Odds of The Genetic Lottery, 1 in 8 Million.

The one in 8 million chance that you turned out how you did.


BONUS: AUTISTISM SHOWS STRONG
CORRELATIONWITH THE DUPLICATION OF THE 22ND CHROMOSOMES AND THE DELETIONS OF SOME, BECAUSE OF THIS, AUTISTIC PEOPLE TYPICALLYHAVE A LOWER CHANCE IN THE GENETIC LOTTERY. https://www.spectrumnews.org/news/dna-doubling-on-chromosome-22-shows-strong-ties-to-autism/D

Sources:


DNRD
 
Last edited:

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