why do white boys have feminine high pitched gay voices and ethnics have low pitched masculine high t bvlls voices

[160cmcurry]

?

 

[Maxillular]

It's the other way around JFL, only Blacks have deeper voices than Whites.

Spics, gooks, sand niggers and jeets all sound nasally and if they try to project their voice loudly it immediately goes up octaves. Maybe their lungs or vocal cords are small since theyre framecel manlets idk.

 

[ryuken]

View attachment 4519271

It always frustrates me that this nigga has a dick bigger than mine

 

[trump]

Youre jewish

 

[wewuzaryans134]

"feminine gay whiteboy"

 

[160cmcurry]

It's the other way around JFL, only Blacks have deeper voices than Whites.

Spics, gooks, sand niggers and jeets all sound nasally and if they try to project their voice loudly it immediately goes up octaves. Maybe their lungs or vocal cords are small since theyre framecel manlets idk.

nah

lack of natural t development during puberty can fuck up their vocal thickness

a lot of it is caused by stress, high cholesterol, which blunts their t development making them not produce enough t for their vocal thickness to get affected

white boys arent genetically predisposed to have a voice with resonance even if they have high levels of t lol

 

[160cmcurry]

It always frustrates me that this nigga has a dick bigger than mine

why does it matter if i’ll chop it off

he can’t defend himself

 

[160cmcurry]

View attachment 4519279
"feminine gay whiteboy"

white boy

get down on your knees
go make me my sandwich tyler

 

[Former Shortcel]

Nobody thinks currys have “low pitched masculine high t bvll voices”

 

[Kelly Oubre Jr]

It always frustrates me that this nigga has a dick bigger than mine

why you been looking at femboy dick brochacho

 

[Lefty Rankin]

View attachment 4519267View attachment 4519268View attachment 4519269
?
View attachment 4519270View attachment 4519271

Not all whites.

 

[Luquier]

Lol just faggots.org atp

And I though this forum couldn’t be more dead

 

[160cmcurry]

Nobody thinks currys have “low pitched masculine high t bvll voices”
View attachment 4519449

tales

all curries are high t

 

[Former Shortcel]

tales
View attachment 4519487
all curries are high t

Then why are you 160cm?

 

[160cmcurry]

Then why are you 160cm?

height doesn’t correlate with t pinkie

now go make me my turkey sandwhich peter

 

[Former Shortcel]

height doesn’t correlate with t pinkie
View attachment 4519492
now go make me my turkey sandwhich peter

More testosterone = Boosted HGH and IGF-1

Also do i have to make you a sandwich because you cant reach the kitchen counter?

 

[160cmcurry]

More testosterone = Boosted HGH and IGF-1

boosted hgh and igf levels doesn’t correlate whether you’ll be tall or not

it’ll just make you reach your genetic celling faster

Also do i have to make you a sandwich because you cant reach the kitchen counter?

no

you’re my slave

 

[ryuken]

why you been looking at femboy dick brochacho

I've seen him once by mistake and the size with the face just bamboozled me

 

[aethus]

You are porn addicted @160cmcurry

 

[5OuttaTen]

I think it’s an accent thing, people speak with different inflections etc. my city is a good example there are 2 main accents, people from the north side of the city speak very high and nasally whereas people on the south side speak more from their chest and sound deeper

 

[160cmcurry]

I've seen him once by mistake and the size with the face just bamboozled me

are you sure about that

 

[sick kunt31]

More testosterone = Boosted HGH and IGF-1

Also do i have to make you a sandwich because you cant reach the kitchen counter?

its literally the opposite, testerone closes the growth plates while hgh elongates them, ut its high levels also signal growth plates to mature and fuse, ending height growth; HGH, in contrast, directly drives bone elongation, and while it needs normal testosterone/estrogen for proper growth plate function, Growth hormone (GH) elongates bones during childhood and adolescence by stimulating cell activity at the epiphyseal growth plates, the areas of cartilage located near the ends of long bones. This process, called endochondral ossification, relies on a combination of direct GH action and its primary mediator, insulin-like growth factor-I (IGF-I),

• Stimulation of Chondrocytes: GH directly stimulates the division and differentiation of precursor cartilage cells (chondrocytes) in the upper, or germinal and proliferative, zones of the growth plate.
• IGF-I Production: GH also triggers the production and release of IGF-I, primarily from the liver (endocrine effect) and locally within the bone and growth plate tissue itself (autocrine/paracrine effect).
• Chondrocyte Proliferation and Hypertrophy: IGF-I then acts on the chondrocytes in the proliferative zone, promoting their rapid multiplication and subsequent enlargement (hypertrophy). This pushes the ends of the bone (epiphyses) further away from the center shaft (diaphysis).
• Ossification: The enlarged cartilage cells eventually die, and the matrix they leave behind becomes calcified and is invaded by blood vessels, osteoblasts (bone-forming cells), and osteoclasts (bone-resorbing cells). The osteoblasts then deposit new bone tissue, effectively replacing the cartilage with bone and lengthening the bone shaft.

This cycle of cartilage growth, maturation, calcification, and replacement by bone continues until the end of puberty, when the growth plates close (fuse) and are replaced by solid bone, at which point further longitudinal elongation is no longer possible.
Growth hormone (GH) contributes to a more masculine face by promoting the growth and development of specific facial bones and soft tissues, particularly during developmental years, leading to larger, broader, and more rugged features.

Effects on Bone Structure
GH, often mediated by Insulin-like Growth Factor 1 (IGF-1), is a critical regulator of bone growth. In the face, its influence contributes to:

• Increased size of the mandible: This results in a larger, more prominent jawline and chin.
• Wider facial structure: It increases total facial height and width.
• Prominent features: The nose can become wider, and overall facial bone structure appears more substantial and chiseled.

These changes occur because while long bones (arms, legs) seal off after puberty, the flat bones of the skull can continue to grow or be influenced by hormones throughout life, albeit less dramatically in adulthood.

Effects on Soft Tissue and Skin
Beyond bone structure, GH influences soft tissues in ways that contribute to a "masculine" appearance:

• Skin thickening: GH and testosterone can thicken the skin and widen pores, contributing to a coarser texture.
• Tissue hypertrophy: High levels can cause soft tissue hypertrophy (enlargement) and edema (swelling), which can give the face a fuller, sometimes "bloated" or rougher look.
• Ruddy complexion: Increased blood vessel creation can lead to a ruddy or red look to the skin.

 

[sick kunt31]

View attachment 4519279
"feminine gay whiteboy"

why is this nigga seething so much

 

[Former Shortcel]

its literally the opposite, testerone closes the growth plates while hgh elongates them, ut its high levels also signal growth plates to mature and fuse, ending height growth; HGH, in contrast, directly drives bone elongation, and while it needs normal testosterone/estrogen for proper growth plate function, Growth hormone (GH) elongates bones during childhood and adolescence by stimulating cell activity at the epiphyseal growth plates, the areas of cartilage located near the ends of long bones. This process, called endochondral ossification, relies on a combination of direct GH action and its primary mediator, insulin-like growth factor-I (IGF-I),

• Stimulation of Chondrocytes: GH directly stimulates the division and differentiation of precursor cartilage cells (chondrocytes) in the upper, or germinal and proliferative, zones of the growth plate.
• IGF-I Production: GH also triggers the production and release of IGF-I, primarily from the liver (endocrine effect) and locally within the bone and growth plate tissue itself (autocrine/paracrine effect).
• Chondrocyte Proliferation and Hypertrophy: IGF-I then acts on the chondrocytes in the proliferative zone, promoting their rapid multiplication and subsequent enlargement (hypertrophy). This pushes the ends of the bone (epiphyses) further away from the center shaft (diaphysis).
• Ossification: The enlarged cartilage cells eventually die, and the matrix they leave behind becomes calcified and is invaded by blood vessels, osteoblasts (bone-forming cells), and osteoclasts (bone-resorbing cells). The osteoblasts then deposit new bone tissue, effectively replacing the cartilage with bone and lengthening the bone shaft.

This cycle of cartilage growth, maturation, calcification, and replacement by bone continues until the end of puberty, when the growth plates close (fuse) and are replaced by solid bone, at which point further longitudinal elongation is no longer possible.
Growth hormone (GH) contributes to a more masculine face by promoting the growth and development of specific facial bones and soft tissues, particularly during developmental years, leading to larger, broader, and more rugged features.

Effects on Bone Structure
GH, often mediated by Insulin-like Growth Factor 1 (IGF-1), is a critical regulator of bone growth. In the face, its influence contributes to:

• Increased size of the mandible: This results in a larger, more prominent jawline and chin.
• Wider facial structure: It increases total facial height and width.
• Prominent features: The nose can become wider, and overall facial bone structure appears more substantial and chiseled.

These changes occur because while long bones (arms, legs) seal off after puberty, the flat bones of the skull can continue to grow or be influenced by hormones throughout life, albeit less dramatically in adulthood.

Effects on Soft Tissue and Skin
Beyond bone structure, GH influences soft tissues in ways that contribute to a "masculine" appearance:

• Skin thickening: GH and testosterone can thicken the skin and widen pores, contributing to a coarser texture.
• Tissue hypertrophy: High levels can cause soft tissue hypertrophy (enlargement) and edema (swelling), which can give the face a fuller, sometimes "bloated" or rougher look.
• Ruddy complexion: Increased blood vessel creation can lead to a ruddy or red look to the skin.

Correct, i just wanted to justify the original joke i made

 

[sick kunt31]

Correct, i just wanted to justify the original joke i made

idk even I just copied grok.ai

 

[walee]

View attachment 4519270View attachment 4519271

What soy milk causes:

 

[walee]

Cope nigger