AMA + Update on upcoming threads

[couldbeenapimp]

You will grow but this ain’t really an ideal stack tbh


I haven’t really done much softmaxxing, but i mean using roids to improve physique, get lean and it changing my mindset, this was the best decision I’ve made

Other than that DIY teeth whitening is decent, growing lashes and eyebrows was really important and grooming them

Anything else is pretty much useless, will do DIY fat dissolvers, DIY PCL threads and DIY botox this summer and i think those are the best softmaxxes other than getting lean

are you taking fgfr3s?

 

[Zagro]

are you taking fgfr3s?

Very recently yes

 

[couldbeenapimp]

Very recently yes

Can you answer pm?

 

[Zagro]

Can you answer pm?

I'll answer it here, i do not share my stack anywhere

 

[couldbeenapimp]

I'll answer it here, i do not share my stack anywhere

how old are you?
And what compounds would you recommend to midiegate side effects on the brain while on an ai?

 

[infrainfra]

midiegate side effects on the brain while on an ai?

there is no brain damage

 

[Kojo]

This is a shit thread, theres no such thing as a genetic limit for height

I'm willing to challenge the claim that there's absolutely no genetic limit. I think you can optimise to the highest degree but everyone has a finite amount of skeletal stem cells. It all ends somewhere.

 

[Zagro]

I'm willing to challenge the claim that there's absolutely no genetic limit. I think you can optimise to the highest degree but everyone has a finite amount of skeletal stem cells. It all ends somewhere.

You can theoretically grow forever if your SSCs self-renewed faster than they differentiate

Don't challenge claims just because you feel like it, cause it's pointless, same shit argument that other guy made. When you begin binding emotions or seeing it as a "competition" of some sorts, you will not accept the truth and be biased till the end

 

[Kojo]

You can theoretically grow forever if your SSCs self-renewed faster than they differentiate

Theoreticals are just as good as saying something like if estradiol didn't exist you'd grow forever, Sure if that condition was fulfilled it'd be the case but that isn't going to happen. Same applies here.

Don't challenge claims just because you feel like it, cause it's pointless, same shit argument that other guy made. When you begin binding emotions or seeing it as a "competition" of some sorts, you will not accept the truth and be biased till the end

I don't even deny pharmaceutical intervention for height, it's still bullshit to say there's absolutely no limit when there is

 

[Kojo]

You can theoretically grow forever if your SSCs self-renewed faster than they differentiate

Don't challenge claims just because you feel like it, cause it's pointless, same shit argument that other guy made. When you begin binding emotions or seeing it as a "competition" of some sorts, you will not accept the truth and be biased till the end

Additionally, my question for thread would be examining the differences in e2 downregulation on the growth plates between different serum ranges.

Like nuking, heavy suppression, modest suppression and slight suppression of aromatase.

Also giving an updated take on what the best ai is for heightmaxxing

 

[Zagro]

Theoreticals are just as good as saying something like if estradiol didn't exist you'd grow forever, Sure if that condition was fulfilled it'd be the case but that isn't going to happen.

You can replicate zero E2 or aromatase deficient state easily with pharma, the rest ill respond to when i wake up but strawman arguments or whatever y’all call it like this is shit

There is a limit yes but it is far above the existing ideal upper range for heigth, you don’t have to worry even a tad bit

It’s like arguing just for the sake of arguing, like pointless if ykwim

 

[Kojo]

You can replicate zero E2 or aromatase deficient state easily with pharma, the rest ill respond to when i wake up but strawman arguments or whatever y’all call it like this is shit

Wasn't my point but it's irrelevant to go back and fourth, You can replicate zero e2 yes but the point i made is about making theoretical points without weighing out whether it's actually possible to replicate the theoretical is bad faith. I said if e2 never existed as a hormone that closes plates (in general). But e2 will obviously always be a thing.

 

[Zagro]

Wasn't my point but it's irrelevant to go back and fourth, You can replicate zero e2 yes but the point i made is about making theoretical points without weighing out whether it's actually possible to replicate the theoretical is bad faith. I said if e2 never existed as a hormone that closes plates (in general). But e2 will obviously always be a thing.

If estrogen never existed something else would take its place it’s how our body has evolved to function, otherwise as i said earlier in this thread a stature too tall wouldn’t be healthy thus why shorter people also have a longer lifespan

What i said is possible, but not realistic. Zero E2 or replicating an aromatase deficiency on the other hand is realistic, possible and affordable

That analogy was shitty as what i said is literally possible, but estrogen or a hormone that closes the plates will always exist otherwise humans wouldn’t even live enough to reproduce

 

[arxangel6996]

so basically the stack for height should look something like this :
1. nuked e2
2. test base + NAAAS
3. high dose rHGH
4. fgfr3 inhibitor
ive also heard multiple individuals like fletchzilla talk about pth analogs for ihh loop

 

[Kojo]

If estrogen never existed something else would take its place it’s how our body has evolved to function, otherwise as i said earlier in this thread a stature too tall wouldn’t be healthy thus why shorter people also have a longer lifespan

What i said is possible, but not realistic. Zero E2 or replicating an aromatase deficiency on the other hand is realistic, possible and affordable

That analogy was shitty as what i said is literally possible, but estrogen or a hormone that closes the plates will always exist otherwise humans wouldn’t even live enough to reproduce

We can agree to disagree, I still don't think you're getting the core point of the analogy

 

[Zagro]

We can agree to disagree, I still don't think you're getting the core point of the analogy

No you’re just using nonsense, you aren’t even debating about pharmacology atp lol

 

[megapro981]

Your growth plate can synthesise estrogen locally through steroidogenesis from cholesterol, so you have local aromatisation that occurs in an autocrine and paracrine manner meaning both in the chondrocyte itself and in a cell-to-cell manner both without the help of anything exogenously from the bloodstream for the most part.

Aromatase inhibitors cant just reach inside the growth plates through the bloodstream as the growth plate is very avascular and isolated from anything outside significantly. So convection; the manner in which molecules travel through the bloodstream in a group to reach their target tissue, doesn’t work for the growth plates, but you need diffusion which is the molecules “diffusing” through the growth plate zones, just like ink fading through water, but still letrozole cant penetrate the cartilaginous tissue that well and only a small fraction of the molecules diffuse through and an even smaller portion make it through to the proliferating zone, so local E2 levels barely budge, but instead they increase the estrogen receptors sensitivity locally to compensate so you make the situation worse by amplifying the deleterious effect the local E2 has on your plates and barely changing the local levels of the ligand.

You also increase your testosterone endogenously through aromatase inhibitors increasing LH/FSH levels, and the more aromatase you inhibit=the more endogenous resources you have which directly contributed to more substrates locally for your growth plates to synthesise even more estrogen locally.

Post in thread 'do not blast test during puberty if u CARE ABOUT UR HEIGHT'
https://looksmax.org/threads/do-not...-u-care-about-ur-height.2086923/post-28851625

AIs do certainly reach the growth plate in meaningful concentrations, especially exemestane which we can use as an example, its molecular weight, structure and LogP is similar to various sex hormones and precursors that are confirmed to do this well. If u agree testosterone diffuses well through the environment u basically already agree that exemestane would too. Therefore aromatase enzyme in the growth plate is inhibited by a good amount, maybe not exactly the same amount reflected from e2 levels in systemic circulation but still very likely enough and it definitely works. Sure other AIs like letrozole and anastrozole might not do this just as well but then just stick to exemestane. Most of the signaling in the growth plates already are fueled by whats diffused through the vascular supply. The fact that the growth plate tissue is largely avascular doesnt mean that whats in circulation doesnt reach the plate to exert its signaling. Important endocrinological hormones are going to do this for certain which is confirmed, which exemestane can be grouped with in this case.

The idea that AIs would increase sensitivity to estrogen signaling due to the inhibition of aromatization obviously means that the AI would need to actually diffuse well into the tissue as well as this increase in sensitivity would be local and not systemic. And we can see that since AIs very clearly do reduce estrogen signaling signs and symptoms if its too much this over compensation by increasing sensitivity isnt gonna completely reverse the inhibition of estrogen signaling from AI usage. The idea that the increase in LH/FSH would increase and therefore more substrate for aromatization would exist is true but again this means uve accepted that sex hormones do reach the plate meaning u also accept exemestane does. And its also clear that signs of estrogen signaling decrease as aromatase inhibitors are administered, meaning it doesnt matter as the aromatase inhibition is countering this fully and more on top of that.

De novo estrogen synthesis locally in the plates also always relies on aromatase enzyme, even if its made from other precursors or downstream of cholesterol steroidogenesis it needs to be converted with aromatase to become estrogen. As we already established that aromatase enzyme is going to be inhibited systemically and locally in the growth plates we can know that all de novo synthesis of estrogen is going to be reduced. If we for some reason instead ignore all the facts stated earlier, cholesterol doesnt diffuse well through the growth plate tissue, its role in the growth plate is regulated by de novo synthesis of cholesterol in the tissue. For it to then convert all the way to estrogen it needs to go through multiple steps and the enzymes and proteins needed for these steps like the rate limiting factors of cholesterol steroidogenesis, StAR and CYP11A1, are very likely not highly expressed there in comparison to tissues which do express them more like the testes. Even if they were expressed at high amounts, cholesterols main job in the growth plates is to work with and control hedgehog signaling which is what they would mainly be doing.

If e2 levels in blood work is reduced with use of an AI like exemestane and a good protocol we can also be more or less certain that its going to be reduced in the growth plates, how perfectly this compares to the reduction in systemic e2 levels and how this scales with larger dosages of exogenous hormones or precursors is less certain but we can at least establish that aromatase inhibitors most likely work for decreasing estrogen signaling in the growth plates and does inhibit local e2 synthesis. And cholesterol steroidogenesis is mostly irrelevant here.

 

[Kojo]

No you’re just using nonsense, you aren’t even debating about pharmacology atp lol

Regardless of what you want to call it, any discussion uses logic in which the logic isn't adding up for the argument you made.

It doesn't have to *reducibly* be about pharmacology or physiology lol

 

[Zagro]

Regardless of what you want to call it, any discussion uses logic in which the logic isn't adding up for the argument you made.

It doesn't have to *reducibly* be about pharmacology or physiology lol

That’s why you will never learn about pharmacology or refute a claim your dumbass doesn’t even understand it, and you seem to be proud of that fact too what a fucking disappointment you are

“any discussion uses logic” yeah bro religion is very logical innit

Genuinely made me laugh

 

[Zagro]

AIs do certainly reach the growth plate in meaningful concentrations, especially exemestane which we can use as an example, its molecular weight, structure and LogP is similar to various sex hormones and precursors that are confirmed to do this well. If u agree testosterone diffuses well through the environment u basically already agree that exemestane would too. Therefore aromatase enzyme in the growth plate is inhibited by a good amount, maybe not exactly the same amount reflected from e2 levels in systemic circulation but still very likely enough and it definitely works. Sure other AIs like letrozole and anastrozole might not do this just as well but then just stick to exemestane. Most of the signaling in the growth plates already are fueled by whats diffused through the vascular supply. The fact that the growth plate tissue is largely avascular doesnt mean that whats in circulation doesnt reach the plate to exert its signaling. Important endocrinological hormones are going to do this for certain which is confirmed, which exemestane can be grouped with in this case.

The idea that AIs would increase sensitivity to estrogen signaling due to the inhibition of aromatization obviously means that the AI would need to actually diffuse well into the tissue as well as this increase in sensitivity would be local and not systemic. And we can see that since AIs very clearly do reduce estrogen signaling signs and symptoms if its too much this over compensation by increasing sensitivity isnt gonna completely reverse the inhibition of estrogen signaling from AI usage. The idea that the increase in LH/FSH would increase and therefore more substrate for aromatization would exist is true but again this means uve accepted that sex hormones do reach the plate meaning u also accept exemestane does. And its also clear that signs of estrogen signaling decrease as aromatase inhibitors are administered, meaning it doesnt matter as the aromatase inhibition is countering this fully and more on top of that.

De novo estrogen synthesis locally in the plates also always relies on aromatase enzyme, even if its made from other precursors or downstream of cholesterol steroidogenesis it needs to be converted with aromatase to become estrogen. As we already established that aromatase enzyme is going to be inhibited systemically and locally in the growth plates we can know that all de novo synthesis of estrogen is going to be reduced. If we for some reason instead ignore all the facts stated earlier, cholesterol doesnt diffuse well through the growth plate tissue, its role in the growth plate is regulated by de novo synthesis of cholesterol in the tissue. For it to then convert all the way to estrogen it needs to go through multiple steps and the enzymes and proteins needed for these steps like the rate limiting factors of cholesterol steroidogenesis, StAR and CYP11A1, are very likely not highly expressed there in comparison to tissues which do express them more like the testes. Even if they were expressed at high amounts, cholesterols main job in the growth plates is to work with and control hedgehog signaling which is what they would mainly be doing.

If e2 levels in blood work is reduced with use of an AI like exemestane and a good protocol we can also be more or less certain that its going to be reduced in the growth plates, how perfectly this compares to the reduction in systemic e2 levels and how this scales with larger dosages of exogenous hormones or precursors is less certain but we can at least establish that aromatase inhibitors most likely work for decreasing estrogen signaling in the growth plates and does inhibit local e2 synthesis. And cholesterol steroidogenesis is mostly irrelevant here.

“gigantism lit proves it works for height?”

I don’t think I’ll even bother replying to you, you think gigantism proves the usage of rhGH for increasing final height

 

[Kojo]

That’s why you will never learn about pharmacology or refute a claim your dumbass doesn’t even understand it, and you seem to be proud of that fact too what a fucking disappointment you are

You're genuinely sub-human in iq levels if you think I was proudly saying pharmacology doesn't matter, Zagro you have really terrible comprehension skills it's actually baffling.

I don't have to restrictively debate about the pharmacology of something when the usage of the compound for a specific result is practically a pseudoscience. I can judge it based on the theories you're making, it's all theoretical using connections to say it works.

If I say the logic to get you to that 'it works' doesn't add up, it doesn't have to specifically be about pharma so you failed there.

“any discussion uses logic” yeah bro religion is very logical innit

Genuinely made me laugh

Also religion is very logical, you're just a normie that doesn't know anything about it Philosophers and theologians argue using logically deductive arguments to justify the position.

I doubt you could even give me one syllogism type yet you're here trying to clown the use of logic and religion

Stay in your place and keep talking about rhGH that's all you're good for

 

[Zagro]

You're genuinely sub-human in iq levels if you think I was proudly saying pharmacology doesn't matter, Zagro you have really terrible comprehension skills it's actually baffling.

I don't have to restrictively debate about the pharmacology of something when the usage of the compound for a specific result is practically a pseudoscience. I can judge it based on the theories you're making, it's all theoretical using connections to say it works.

If I say the logic to get you to that 'it works' doesn't add up, it doesn't have to specifically be about pharma so you failed there.

Also religion is very logical, you're just a normie that doesn't know anything about it Philosophers and theologians argue using logically deductive arguments to justify the position.

I doubt you could even give me one syllogism type yet you're here trying to clown the use of logic and religion

Stay in your place and keep talking about rhGH that's all you're good for

“The Calling: Rocks and trees will call out: "O Muslim, O servant of Allah, there is a Jew behind me; come and kill him!”

Yeah dude religion is logical dude yeah, the trees talking to us and snitching on jews sounds pretty viable yeah

You don’t know anything about pharmacology i can confidently say i can rape you in any topic regarding longitudinal growth like anything, if you ever think you know like even 10% of what i know you should kill yourself, and this is the first and last time I’ve ever said such a thing.

You never debate about the topic itself you dumb pseudo-intellectual fucktard, you’re so fucking embarrassing

Do you know what pseudoscience means? “result is practically a pseudoscience” we have evidence for all of this, every result every outcome possible can be predicted and is proven

“It doesn't have to *reducibly* be about pharmacology or physiology lol”

I really had hope in you dude even though you don’t even know what chondrocyte proliferation meant, you spoke nicely

“I can judge it based on the theories you're making, it's all theoretical using connections to say it works.” zero theoretical connections it’s all proven you haven’t read a singular paper

 

[Zagro]

You're genuinely sub-human in iq levels if you think I was proudly saying pharmacology doesn't matter, Zagro you have really terrible comprehension skills it's actually baffling.

I don't have to restrictively debate about the pharmacology of something when the usage of the compound for a specific result is practically a pseudoscience. I can judge it based on the theories you're making, it's all theoretical using connections to say it works.

If I say the logic to get you to that 'it works' doesn't add up, it doesn't have to specifically be about pharma so you failed there.

Also religion is very logical, you're just a normie that doesn't know anything about it Philosophers and theologians argue using logically deductive arguments to justify the position.

I doubt you could even give me one syllogism type yet you're here trying to clown the use of logic and religion

Stay in your place and keep talking about rhGH that's all you're good for

@laplacist is this how niggas debate? Bluds prompt ai watch a few podcasts, interviews and videos read 2-3 books and become fucking philosophers in their own mind

 

[Kojo]

“The Calling: Rocks and trees will call out: "O Muslim, O servant of Allah, there is a Jew behind me; come and kill him!”

Yeah dude religion is logical dude yeah, the trees talking to us and snitching on jews sounds pretty viable yeah

I'm not a muslim but this verse seems metaphorical and not literal, it's pretty obvious

Also yeah religion isn't nomological if you know what that means, but it is logical. Also can be logically justified.

You don’t know anything about pharmacology i can confidently say i can rape you in any topic regarding longitudinal growth like anything, if you ever think you know like even 10% of what i know you should kill yourself, and this is the first and last time I’ve ever said such a thing.

You never debate about the topic itself you dumb pseudo-intellectual fucktard, you’re so fucking embarrassing

Do you know what pseudoscience means? “result is practically a pseudoscience” we have evidence for all of this, every result every outcome possible can be predicted and is proven

“It doesn't have to *reducibly* be about pharmacology or physiology lol”

I really had hope in you dude even though you don’t even know what chondrocyte proliferation meant, you spoke nicely

“I can judge it based on the theories you're making, it's all theoretical using connections to say it works.” zero theoretical connections it’s all proven you haven’t read a singular paper

Just a pseudo rant, boasting about knowledge is kind of corny, even if you did have so much knowledge and I knew only a small fraction of what you know in this field there's always a bigger fish everywhere so it's not something that hurts my pride or ego.

Knowledge is knowledge, I rather being intelligent as knowledge isn't necessarily intelligence. Intelligence is being able to logically interpret knowledge/information/Literature and potentially make other good conclusions from it.

Also I know what pseudoscience means, this is also pretty contradictory as you said prior that we can't really 100% prove certain things about rhGH's relationship to FAH when we discussed a few weeks ago.

Also I do know what chondrocyte proliferation is? You might be misremembering me with someone else I don't recall misrepresenting what that is. It's pretty basic anyways, chondrocytes multiply and stack in rows..

Edit: These back and fourths are like a giga time waste over asinine topics like it's really childish.. "i HaVe MoRe KnOwLeDgE!!" Like okay zagro, You have the most knowledge move on and make the informative thread we're asking for no one cares anymore

Edit #2: You need to start controlling your emotions better

 

[Kojo]

@laplacist is this how niggas debate? Bluds prompt ai watch a few podcasts, interviews and videos read 2-3 books and become fucking philosophers in their own mind

Huh? I've been studying philo for 3 years lmao and I don't watch podcasts or use ai, I only use ai for basic silly info. I've also read like 100x that but go off ig

Also sure? everything is happening in the mind anyways I'm not sure what the get back was for this one it's really immature.

These are my ai prompts btw and they were like months ago

 

[Zagro]

I'm not a muslim but this verse seems metaphorical and not literal, it's pretty obvious

Also yeah religion isn't nomological if you know what that means, but it is logical. Also can be logically justified.

Just a pseudo rant, boasting about knowledge is kind of corny, even if you did have so much knowledge and I knew only a small fraction of what you know in this field there's always a bigger fish everywhere so it's not something that hurts my pride or ego.

Knowledge is knowledge, I rather being intelligent as knowledge isn't necessarily intelligence. Intelligence is being able to logically interpret knowledge/information/Literature and potentially make other good conclusions from it.

Also I know what pseudoscience means, this is also pretty contradictory as you said prior that we can't really 100% prove certain things about rhGH's relationship to FAH when we discussed a few weeks ago.

Also I do know what chondrocyte proliferation is? You might be misremembering me with someone else I don't recall misrepresenting what that is. It's pretty basic anyways, chondrocytes multiply and stack in rows..

Edit: These back and fourths are like a giga time waste over asinine topics like it's really childish.. "i HaVe MoRe KnOwLeDgE!!" Like okay zagro, You have the most knowledge move on and make the informative thread we're asking for no one cares anymore

Edit #2: You need to start controlling your emotions better

I genuinely never reply back like that dude i hopped off almost almost a gram of tren cold-turkey and now just messing around with 0 T and 0 E2, pretty much a fucking plant at this point, and also the reason I’ve made multiple “I’m leaving the forum” threads lately. It’s like having a boy period, you become a whining bitch

You can genuinely see when I’m on and off stack based off of how i reply to people on here

I literally hate people who claim to know it all and have never even implied that I’m even the most knowledgeable or even knowledgeable at all, ignore allat previous replies i will reply back when calm

 

[Kojo]

I genuinely never reply back like that dude i hopped off almost almost a gram of tren cold-turkey and now just messing around with 0 T and 0 E2, pretty much a fucking plant at this point, and also the reason I’ve made multiple “I’m leaving the forum” threads lately. It’s like having a boy period, you become a whining bitch

You can genuinely see when I’m on and off stack based off of how i reply to people on here

I literally hate people who claim to know it all and have never even implied that I’m even the most knowledgeable or even knowledgeable at all, ignore allat previous replies i will reply back when calm

All good

 

[infrainfra]

I already do and have been for a long time


1. 0.07mg/kg and above

2. Pre-bed

3. Insulin sensitivity is really important so we do have to use something, i don’t think berberine is that bad, maybe dihydroberberine and retatrutide. Empagliflozin seems interesting but have to look more into it

4. Beta-blockers are a MUST have, then the usuals and a statin should not be overlooked, and memantine

5. Yeah i have been looking into it and might buy that injectable beginning with “d” if ykwim

hey can u respond discord? actually kinda important

 

[megapro981]

“gigantism lit proves it works for height?”

I don’t think I’ll even bother replying to you, you think gigantism proves the usage of rhGH for increasing final height

Are u on the side of that ahmed guy? Ur clearly not so u just have something against my argument with gigantism, yet u share no argument against it? I saw on ahmeds post that apparently u were the one teaching him how "acromegalic niggas cant compare to an healthy adolescent" quoted from u. I assume with acromegaly u mean gigantism since surely ahmed would already know why someone acromegalic (closed plate) doesnt compare to a healthy adolescent (open plate) when it comes to this debate. Meaning u must be the one who supplied him with this shitty arguments against me? The one about the feedback mechanisms? U need to prove u have better arguments against my claim, because if thats ur only counter argument ur already proved wrong.

I guess ur having ur boy period cause of ur hormonal issues like u said but dont act like ur above me, and if ur lazy just argue against my points that are the weakest in ur eyes, the ones u can very easily counter argue against and prove me wrong in. If u provide valid points and u feel I somehow dont accept them then just stop replying. The main points I argued for which u disagree to are that 1. exemestane does diffuse well into the avascular growth plate enviroment, and that 2. gigantism would work similarly to how high doses of relatively frequently injected rhgh subq would work for growth plate effects without the growth response being inhibited in either of the cases by feedback mechanisms more than the other case (ofc assuming that the rhgh dosage is enough to mimic gh levels in an individual with gigantism which isnt even that high of a dosage) and gigantism being mainly the cause of pituitary tumors which do quite frequently only interact noticeably with the HPG axis of the pituitary. Both of these points are fairly easy to understand and find the truth on, theyre not even deeply mechanistic theories, its just some pretty simple biochemistry.

 

[vroom]

@Zagro can you reply to my last msg. I want to hop on frgr3s but not sure if its worth it at my age. If I should, which one do you recommend how would I dose it?

 

[Kainep]

Just out of curiosity since my plates r already closed
would hgh have worked for me -a hgh sufficient short person-?

Why HGH is cope and will NOT work for you!!!! (HGH USERS GTFIH!!!)

A lot of People continue to believe that healthy children who lack growth hormone deficiency will experience height increases through HGH injections. It won’t. Why? Let’s get into this 1. How Growth Hormone Actually Works 2. What the Studies ACTUALLY Show 3. Why HGH does NOT work...

looksmax.org

Regarding this thread that shows that with hgh you cant surpass your genetical height but just reach the genetical height.

what kinda retarded question is that how do u expect them to grow then the plates already fused

 

[Jesus_ist_König]

what kinda retarded question is that how do u expect them to grow then the plates already fused

Its not for knowledge and just out of "curiosity" in the sense of out of curiosity since i cant grow anymore
retard

 

[Kainep]

Its not for knowledge and just out of "curiosity" in the sense of out of curiosity since i cant grow anymore
retard

i asked the same question about two years ago i think i was 19 back then i can assure you nothing changed

 

[Jesus_ist_König]

i asked the same question about two years ago i think i was 19 back then

i can assure you nothing changed

No shit bro

 

[duduboy]

Ask me anything here as then i wouldn’t have to repeat myself to others in threads and dms as much, you can ask whatever even personal experiences.

Also any questions about my upcoming threads; rhGH thread update, aromatase inhibitors thread, FGFR inhibitors thread and may include and finish my HDACi threads, please ask them and let me know your suggestions

Just want to know when youre going to post

 

[Xazzy]

Ask me anything here as then i wouldn’t have to repeat myself to others in threads and dms as much, you can ask whatever even personal experiences.

Also any questions about my upcoming threads; rhGH thread update, aromatase inhibitors thread, FGFR inhibitors thread and may include and finish my HDACi threads, please ask them and let me know your suggestions

are aromatase inhibitors on its own during puberty good