pumpeduppecs
Diamond
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No need to take tranny drugs we can just use stem cells.
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Its just hair bro36 wtf
Roids36 wtf
Do you have more information on this subject? I heard that malocclusion also plays a role in balding.Unless genetically prone to balding (having hypersensitivity to dht in your scalp), hairline recession is usually a telltale sign of maxillary recession. Notice how one of the first places you lose hair is your temples? Its due to the fact that as the cranium undergoes a CW rotation (downward growth) which causes the temporal peaks to go back (Recede).
Finasteride's mechanism of action works via killing DHT receptors and decreasing androgen sensitivity altogether. The issue is, finasteride is administered orally (whether this is done or purpose or not I don't know) and this administration of a DHT blocker orally means it decreases androgen sensitivity all over the body, not just the scalp.
I would assume locally administering finasteride could negate the effects of PFS and or other ED-adjacent symptoms.
Having High T is actually good for keeping your hair, and neither is DHT the problem. Nine times out of 10 the problem is a CW rotated cranium or being genetically prone to balding.
No need to take tranny drugs we can just use stem cells.
if stem cells can save a roidcell then they’ve got to save a natty baldcell right?“I tried every hair medication and none of it worked”
Dude was clearly on roids what did he expect?
Okay, so why does taking 5AR inhibitors stop hair loss and often cause hair regrowth if the reason isn’t DHT acting on the AR receptors within the scalp? Fin and DUT work for 90 percent of people and not to mention that MTF trannymorphs grow back hair when they’re stripped of androgens so your CW rotated cranium theory goes out the window and it’s clear that DHT and AR sensitivity is the main driving force behind hair loss.Unless genetically prone to balding (having hypersensitivity to dht in your scalp), hairline recession is usually a telltale sign of maxillary recession. Notice how one of the first places you lose hair is your temples? Its due to the fact that as the cranium undergoes a CW rotation (downward growth) which causes the temporal peaks to go back (Recede).
Finasteride's mechanism of action works via killing DHT receptors and decreasing androgen sensitivity altogether. The issue is, finasteride is administered orally (whether this is done or purpose or not I don't know) and this administration of a DHT blocker orally means it decreases androgen sensitivity all over the body, not just the scalp.
I would assume locally administering finasteride could negate the effects of PFS and or other ED-adjacent symptoms.
Having High T is actually good for keeping your hair, and neither is DHT the problem. Nine times out of 10 the problem is a CW rotated cranium or being genetically prone to balding.
Wrong and wrong:Finasteride's mechanism of action works via killing DHT receptors and decreasing androgen sensitivity altogether. The
Although there may be some merit regarding skull shape and blood flow which is important for healthy hair.Okay, so why does taking 5AR inhibitors stop hair loss and often cause hair regrowth if the reason isn’t DHT acting on the AR receptors within the scalp? Fin and DUT work for 90 percent of people and not to mention that MTF trannymorphs grow back hair when they’re stripped of androgens so your CW rotated cranium theory goes out the window and it’s clear that DHT and AR sensitivity is the main driving force behind hair loss.
Your temples recede first as they’re filled with the most AR receptors for DHT to bind to. Simple as that.
Wrong and wrong:
Fin doesn’t kill DHT receptors which aren’t even a real thing, there’s only Androgen receptors. It also doesn’t decrease Androgen receptor sensitivity. It works by stopping the enzyme which converts test into DHT. That’s why it’s called a 5 Alpha reductase inhibitor.
2 months so far, we cannot say for sure atm. He could just lose all by 6th month.if stem cells can save a roidcell then they’ve got to save a natty baldcell right?
Yep, people will always find weird theories. instead of actual thing that work.Okay, so why does taking 5AR inhibitors stop hair loss and often cause hair regrowth if the reason isn’t DHT acting on the AR receptors within the scalp? Fin and DUT work for 90 percent of people and not to mention that MTF trannymorphs grow back hair when they’re stripped of androgens so your CW rotated cranium theory goes out the window and it’s clear that DHT and AR sensitivity is the main driving force behind hair loss.
Your temples recede first as they’re filled with the most AR receptors for DHT to bind to. Simple as that.
Wrong and wrong:
Fin doesn’t kill DHT receptors which aren’t even a real thing, there’s only Androgen receptors. It also doesn’t decrease Androgen receptor sensitivity. It works by stopping the enzyme which converts test into DHT. That’s why it’s called a 5 Alpha reductase inhibitor.
hair system? you mean a wig?Or just get a hair system and have mogger hair which you would never be able to have naturally.
I am getting one soon.
Not exactly a wig, you go to hair system saloon or whatever, they do some measurements, discuss with you what hairstyle you want, and then order a hair system.hair system? you mean a wig?
don’t hair look fake? I’m blasting tranny drugs idcNot exactly a wig, you go to hair system saloon or whatever, they do some measurements, discuss with you what hairstyle you want, and then order a hair system.
They basically glue artificial hair on your scalp and it stays there, you can wash it, swim with it, do everything regularly, plus you will have main character hair:
View attachment 2541645
I decided to go for it instead of taking tranny drugs and butchering myself with surgeries. The only downside is that you need to maintain it every 6-8 months, but are we here to looksmax at any cost or what?
It actually looks more natural than transplant, but go become a tranny and mutate your future kids.don’t hair look fake? I’m blasting tranny drugs idc
my kids? lol tho nigga thinks i’m having kids after being such a subhuman wasteIt actually looks more natural than transplant, but go become a tranny and mutate your future kids.
also by the time i’ll want to have kids you will have the chance to choose the embrions that you prefer and you will be able to choose your kids stats (if you have money that is.)my kids? lol tho nigga thinks i’m having kids after being such a subhuman waste
wtf is that first paragraph? youre a fucking retard holy shitUnless genetically prone to balding (having hypersensitivity to dht in your scalp), hairline recession is usually a telltale sign of maxillary recession. Notice how one of the first places you lose hair is your temples? Its due to the fact that as the cranium undergoes a CW rotation (downward growth) which causes the temporal peaks to go back (Recede).
Finasteride's mechanism of action works via killing DHT receptors and decreasing androgen sensitivity altogether. The issue is, finasteride is administered orally (whether this is done or purpose or not I don't know) and this administration of a DHT blocker orally means it decreases androgen sensitivity all over the body, not just the scalp.
I would assume locally administering finasteride could negate the effects of PFS and or other ED-adjacent symptoms.
Having High T is actually good for keeping your hair, and neither is DHT the problem. Nine times out of 10 the problem is a CW rotated cranium or being genetically prone to balding.
You will forever live in torment if you don't have kids.my kids? lol tho nigga thinks i’m having kids after being such a subhuman waste
maybe, who knowsYou will forever live in torment if you don't have kids.
Unless genetically prone to balding (having hypersensitivity to dht in your scalp), hairline recession is usually a telltale sign of maxillary recession. Notice how one of the first places you lose hair is your temples? Its due to the fact that as the cranium undergoes a CW rotation (downward growth) which causes the temporal peaks to go back (Recede).
Finasteride's mechanism of action works via killing DHT receptors and decreasing androgen sensitivity altogether. The issue is, finasteride is administered orally (whether this is done or purpose or not I don't know) and this administration of a DHT blocker orally means it decreases androgen sensitivity all over the body, not just the scalp.
I would assume locally administering finasteride could negate the effects of PFS and or other ED-adjacent symptoms.
Having High T is actually good for keeping your hair, and neither is DHT the problem. Nine times out of 10 the problem is a CW rotated cranium or being genetically prone to balding.
Someone here needs to make a megathread about hair systems it’s hard to find information about them and I agree with you on the drugs I don’t wanna mess with my bodies natural chemistryNot exactly a wig, you go to hair system saloon or whatever, they do some measurements, discuss with you what hairstyle you want, and then order a hair system.
They basically glue artificial hair on your scalp and it stays there, you can wash it, swim with it, do everything regularly, plus you will have main character hair:
View attachment 2541645
I decided to go for it instead of taking tranny drugs and butchering myself with surgeries. The only downside is that you need to maintain it every 6-8 months, but are we here to looksmax at any cost or what?