Cjc1295 with DAC or HGH for height maxing (14yo 5’5)

[Temporalcel]

I’m 14 and my plates are still open. My dad is 5’10 and my mom is 5’4 my dad was nutrient deprived as a child as he grew up in the third world and so was my mom. I want to heightmog as I am a 4.5/10 facially. I want to do hgh but there is the downside of cartilage growth with would make my ears and nose bigger. I know cjc1295 is less effective and ill atmost grow 1-2 past height potential but it is safer. Are there any alternatives?

 

[BR32]

For the love of God stop using AI

@Zagro this dude can't even respond without AI

View attachment 4888197

This isn't AI nigga I wrote this myslef

Are you unfamiliar with commas

 

[BR32]

@Deroga stop calling out to daddy every 5 minutes and think for yourself

 

[Deroga]

This isn't AI nigga I wrote this myslef

Are you unfamiliar with commas

"No congenital deficiency, no missing receptor, and no HPG suppression. Just high T, blocked conversion to E2, bones stop developing properly."

Are you telling me this Bullshit isn't AI? Yeah bro the AI detector is definitely wrong and we SHOULD TRUST YOU

 

[Zagro]

Fair on the first three, but you skipped the Cutler study entirely.

Boys with precocious puberty and testosterone already in the adult male range. They blocked aromatization with testolactone. Testosterone stayed high the whole time, only E2 was reduced. Growth velocity normalized and bone maturation slowed.

No congenital deficiency, no missing receptor, and no HPG suppression. Just high T, blocked conversion to E2, bones stop developing properly.

Please explain this to me because your critiques don't explain it

“These remarkable cases provide compelling evidence that androgen alone, even at levels above the normal adult male range, does not fuse the epiphyses. Rather, it is estrogen that fuses the epiphyses and terminates linear growth.” This means that androgens themselves don't contribute to fusion, but it's estrogen that fuses and terminates the growth velocity.

"Administration of the antiandrogen, spironolactone, was less effective in decreasing growth rate, and had no apparent effect on the markedly elevated rate of bone maturation." This proves even further that androgens themselves aren't the problem and inhibiting them doesn't fix anything, the devil here is estrogen.

“Administration of the aromatase inhibitor, testolactone, caused a marked reduction of both growth velocity and bone maturation rate to nearly normal prepubertal levels” Aromatase inhibitor corrects the precocious pubertal state to normal prepubertal state, reduces the bone maturation rate which is accelerated through adult leves of testosterone being produced and reduces the growth velocity to normal prepubertal/pre growth spurt state, as essentially the amount of androgens contributed to a "growth spurt" like effect in them but the outcome would be that their final height decreasing: transient significant height growth but epiphyses ossify earlier than normal.

What do you want me to explain here? This is good and needed for precocious puberty, "bones stop developing properly." do you know that accelerated bone maturation rate caused by adult sex hormone levels in a fucking 9yo isn't "proper"? Bone maturation acceleration was caused by increased aromatisation so an aromatase inhibitors fixed that and prevented any more decrease in final adult height, it also normalised the growth velocity. You're embarrassing yourself by speaking on topics you have no knowledge about.

"The dose-response curve for the growth-promoting action of estrogen is biphasic, with maximal stimulation of growth at estradiol levels of approximately 3-4 pg/ml (11-15 pmol/1)." Okay so what does this tell us? Let me explain son, the effect of estrogen on growth-rate is biphasic meaning low levels= bad but also high levels=bad, meaning a moderate level is optimal. "but why is it like this, didn't you tell people to nuke E2?" Yes I did and still do, the only growth-promoting effect of estrogen is not directly by estrogen itself but rather it's effects on IGF-1, so if exogenous supplementation/administration of IGF-1/GH is used estrogen has zero usage for your height and only downsides.

Also funny how maximal stimulation of growth only required 3-4 pg/ml, this is already nuked.

Also @Zagro

Were not even talking about all of the other reasons you want normal serum concentrations of E2 during puberty.

Even if an AI did boost growth is it worth all of the horrific side effects that will cause?

Cognitive development, libido/sexual development, Cardiovascular funciton, Joint health, mental health. basic stuff

I'm purely open to having a civil conversation, I value your input

JFL at "horrific side-effects"

1. Zero evidence of it being bad for cognitive development, literally zero.
2. "Sexual development" son you aren't 8 years old it's only partially needed for formation of some penile tissue after that there is no use, do aromatase deficient males have wonky dicks?
3. Provide one single paper on it being bad for cardiovascular function, at most it's the slightest shit ever and can be fully prevented.
4. Yes trade centimeters of height for temporary joint pain, mirin iq.
5. Provide evidence on it being bad for mental health, I've rather seen improvement in life quality.

You don't know anything you shouldn't continue and I do not want a civil conversation as I'm literally refuting ChatGPT when talking to you and I've done this 100x times, it gets tiring