is it possible that chronically elevated levels of igf-1 (which cjc causes) could cause receptors to become resistant to it? wondering if it works in the same way as insulin resistance
and if this is true, would it mean that regular pulsatile secretion could be better for maximum effect of igf-1? not sure what methods enhance that though
and if this is true, would it mean that regular pulsatile secretion could be better for maximum effect of igf-1? not sure what methods enhance that though
