great
why?jealous of you bro
miring bra
probably my fav compound
any metformin or a sglt 2 inhib?
Up the test, drop the clen & var. Add in a statin and have caber on hand if you don't already and you're g2g.
cuz im broke asf and cant afford shit
that is my first time hearing about those, why would I add them?
to lower blood sugar from the gh
don't wanna close my plates through local atomization, why drop the clan and var? I have caber on hand, why statins I already have ezemtibe that lowers bad cholesterol low enough lol
im only running 5iu ?
youll probabl;y be fine without them
92-95% of men have fused growth plates by 16, you can fact check this. Touching AAS before your plates are fused on it's own is pretty retarded imo but that's your decision.
Clen is possibly the lowest ROI fat burner to ever exist, jumping up and down would be more effective.
Statin + ezetimibe lowers ApoB synergistically compared to monotherapy, your lipids are the most essential health marker to keep intact on cycle since it's virtually the only noteworthy long-term risk. Why would you want to control cholesterol "enough" rather than crush it and allow you to extend the duration of your cycle?
I got an X-ray a week ago and mine are still open lol
clen burns like 300 extra cals daily, which is all I need, have you used anavar before ?
which statins do you recommend, statins have way bigger side effect profile then something like ezemtibe, ill just up the dose of it?
What other PEDs would you recommend that don’t raise estrogen and can be ran long term that provide real muscle building, I really can’t think of any
That's literally a 30 minute walk lol. I've never taken an oral nor do I plan on it. Lipids are law.
Pitavastatin & Rosuvostatin are fine. Upping the dose of ezetimibe will do nothing since the drugs effects plateau at 5mg's, with -> 10mg's showing no clinical significance. All it does is inhibit the absorption of dietary cholestral which has a far more strict cut off range than statins, for example. 5mg's of both ezetimibe & rosuvostatin is my go-to and is an incredible option.
The only PEDs you should ever be taking in terms of AAS are honestly test and optionally tren/eq. E2 is highly lipid-protective, matching or surpassing that of most ancillary stacks, so you should keep your E2 sky high just enough to not cause gyno.
So do you think that local atomization at the gp actually happens? I feel like every person says a different answer, with my research it does happen even if estrogen is low
are my neroprotection ancillears good enough for tren? what about the mg is that good to?
