Cycle Advice? Roidcels GTFIH

Are you fucking retarded?

Do you want my dick up your ass?

Stop asking GPT for answers it's too obvious that you are.
jfl. Whats ur opinion on my stack? I've seen u around hella so ik u know your stuff; your advice would be greatly appreciated.
 
why pct bro:lul::lul::lul:
loving the cycle so far even at moderate dosages so maybe i'll just cruise. This cycle is basically intended as a trial run of sorts until I go into college.
 
Never really considered proviron, thanks for the suggestion. I assume this would be used in conjunction with HCG? I appreciate your advice and will be sure to look into it. Also, for your proposed "natty+ stack", it seems valid but, because my goal is to gain dimo, I would also include AAS, in my case test, for the androgenic effects.

If you’re running Test and HCG, even better. Proviron will certainly cause dimorphism. Estered DHT prop would be more effective tho. Just gotta be proactive about hairloss if you’re susceptible to it. I have one of/if not the best source for estered DHT just in case. Make sure to pair this with Tesamorelin or GH to get the most dimorphism possible since gene transcription involves androgens + igf-1.
 
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If you’re running Test and HCG, even better. Proviron will certainly cause dimorphism. Estered DHT prop would be more effective tho. Just gotta be proactive about hairloss if you’re susceptible to it. I have one of/if not the best source for estered DHT just in case. Make sure to pair this with Tesamorelin or GH to get the most dimorphism possible since gene transcription involves androgens + igf-1.
Makes sense to me. My current philosophy is that I am increasing DHT levels by inhibiting aromatase as well as supplementing testosterone, thus providing ample substrate for the 5ar enzyme, but I am more than open to supplementing exogenous DHT as well; I already have Anavar on the way, though I recognize it's not as bioavailable as DHT itself.. would you be opposed to providing your source in PMs? Finally, do you suppose RU58841 would be valid for preventing DHT-mediated hairloss? If not, what would you suggest?
 
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Makes sense to me. My current philosophy is that I am increasing DHT levels by inhibiting aromatase as well as supplementing testosterone, thus providing ample substrate for the 5ar enzyme, but I am more than open to supplementing exogenous DHT as well; I already have Anavar on the way, though I recognize it's not as bioavailable as DHT itself.. would you be opposed to providing your source in PMs? Finally, do you suppose RU58841 would be valid for preventing DHT-mediated hairloss? If not, what would you suggest?

Gotchu bro just hit me. I’ve been using 8% RU, microneedling with Derminator 2 inconsistently tho, 2% keto shampoo, and I just started oral 2.5mg/day minoxidil. RU seems to be weak so I still need more DHT and AR blocking so I finally gave in and ordered topical fin mixed with spirinolactone and tret. Since I’m using DHT, proviron, androE I’m not too worried about the fin.

For dimorphism specifically, Proviron and DHT esters will be more effective than Anavar which was designed to be more anabolic/peripheral tissue targeted than androgenic/CNS targeted. There will certainly be more gene transcription with bioidentical DHT since it is more flexible and fits the receptor much more effectively than rigid synthetic DHT derivatives. Mast seems to behave the most like DHT, something I haven’t tried yet and don’t intend to with DHT-P available.
 
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Reactions: acromegalic
Gotchu bro just hit me. I’ve been using 8% RU, microneedling with Derminator 2 inconsistently tho, 2% keto shampoo, and I just started oral 2.5mg/day minoxidil. RU seems to be weak so I still need more DHT and AR blocking so I finally gave in and ordered topical fin mixed with spirinolactone and tret. Since I’m using DHT, proviron, androE I’m not too worried about the fin.

For dimorphism specifically, Proviron and DHT esters will be more effective than Anavar which was designed to be more anabolic/peripheral tissue targeted than androgenic/CNS targeted. There will certainly be more gene transcription with bioidentical DHT since it is more flexible and fits the receptor much more effectively than rigid synthetic DHT derivatives. Mast seems to behave the most like DHT, something I haven’t tried yet and don’t intend to with DHT-P available.
Tysm bro I really appreciate it. Mirin the hair loss protocol also, I'm sure mine will end up looking quite similar. Not sure about the fin though, since you're already bypassing 5ar anyway by using exogenous DHT; you know your stuff tho so i'll take your word. Something like topilutamide may also be worth looking into.

I agree with you on the anavar, I intend to use it more as just a mild cutting compound as opposed to an androgenic agent. Thought it's worth mentioning on the topic of DHT derivs tho. Mast could be intriguing but DHT would surely be better for its androgenic effects for the reason you stated, as well as the fact that DHT would metabolize much more cleanly since it's surely more bioavailable than synthetics like mast. I'm interested to hear the reason why you opt for DHT prop, as opposed to a longer esther. If its anything like testosterone, frequent injections with a longer esther would allow for more stable serum levels. I'm not too well researched on injectable DHT though tbf.
 

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