Cycle Help

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MediterraneanPill

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I am 17 and planning on running my first cycle, but don't really know what would be optimal to run. I am assuming I have some residual height growth left so my main anabolism should likely come from a NAAAS, but I would appreciate some help on what to run. My goals are better physique (duh), voice, athletic performance, and dimorphism (If it's possible by my age).
 
Non anabolic anabolic androgenic steroids? What is that lmao
 
"im 17 and dont know shit and dont do my own research" simple answer tren and test
 
If it's non anabolic that would mean that it wouldn't help with hypertrophy whatsoever buddy boy
 
Just take test with low dose AI should be fine brocel
 
I am 17 and planning on running my first cycle, but don't really know what would be optimal to run. I am assuming I have some residual height growth left so my main anabolism should likely come from a NAAAS, but I would appreciate some help on what to run. My goals are better physique (duh), voice, athletic performance, and dimorphism (If it's possible by my age).
why are you planning to run a cycle if you don’t know shit retard.
 
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why are you planning to run a cycle if you don’t know shit retard.
I know plenty and there are a few good choices. The point of the post was to get advice from people who know more than me dumbass
 
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If it's non anabolic that would mean that it wouldn't help with hypertrophy whatsoever buddy boy
NAAAS - non aromitizing anabolic androgenic steroid
 
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"im 17 and dont know shit and dont do my own research" simple answer tren and test
yeah I was considering that but the brain issues seem to be extremely hard to mitigate
 
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I know plenty and there are a few good choices. The point of the post was to get advice from people who know more than me dumbass
if you knew plenty you wouldn’t have to ask others let’s be real. you could potentially take halo, tren, var, there’s much more.
 
ai's dont prevent local aromatization though
Tell me how? AI s literally bind to aromatase such that any other compound would get blocked from it. It introduces competition for the aromatase molecule
 
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yeah I was considering that but the brain issues seem to be extremely hard to mitigate
Either way, there is no complete side effect profile free cycle. Your genetics left you behind, you think you can just take certain compound to balance out genetics side effect free? Sorry buddy, doesn't work that way
 
Either way, there is no complete side effect profile free cycle. Your genetics left you behind, you think you can just take certain compound to balance out genetics side effect free? Sorry buddy, doesn't work that way
yeah I am not too worried about the side effects for the most part, with my inhibition it's just hard to get past the brain stuff
 
yeah it includes anything that doesnt aromatize like dht derivatives and some 19 nors
Fair ig, this term just isn't used like at all. Idk in what kind of study you read that JFL
 
yeah I am not too worried about the side effects for the most part, with my inhibition it's just hard to get past the brain stuff
I mean I have been low e2. In general if you weigh like 80kgs, you take 500mg test with 10mg aromasin, you'll be fine
 
Tell me how? AI s literally bind to aromatase such that any other compound would get blocked from it. It introduces competition for the aromatase molecule
AIs bind to the aromatase enzyme directly and block it everywhere in the body. If local estrogen still happens on high-dose testosterone, it's because not every enzyme molecule got blocked, not because the AI missed that tissue.
 
But also you can titration dosage down as well, get a pill cutter online and try to source tablets in a dosages that would be easier to adjust, maybe like 5mg tablets
 
if you knew plenty you wouldn’t have to ask others let’s be real. you could potentially take halo, tren, var, there’s much more.
or maybe I know a lot, yet there are people with first hand experience using them that know more than me???
 
AIs bind to the aromatase enzyme directly and block it everywhere in the body. If local estrogen still happens on high-dose testosterone, it's because not every enzyme molecule got blocked, not because the AI missed that tissue.
Yeah but there is no reason to believe/assume this would happen more in tissues where it would be extra undesirable like growth plates. Assuming testosterone is equally divided In all tissues/body parts
 
Either way you want some estrogen expression right? Isn't it favorable that aromatase isn't blocked everywhere 100% in your body?
 
You just want your e2 probably slightly below baseline?
 
Non anabolic anabolic androgenic steroids? What is that lmao
Non aromatising anabolic androgenic steroids idiot

Use context clues, this is unironically a de-facto iq test:lul:
 
I am 17 and planning on running my first cycle, but don't really know what would be optimal to run. I am assuming I have some residual height growth left so my main anabolism should likely come from a NAAAS, but I would appreciate some help on what to run. My goals are better physique (duh), voice, athletic performance, and dimorphism (If it's possible by my age).
Very low trt dose test halo anavar and gh ai, don't run any of this without an xray
 
Non aromatising anabolic androgenic steroids idiot

Use context clues, this is unironically a de-facto iq test:lul:
finally somebody uses their brain lmao
 
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You don't have any idea on nothing we can work with? Bruh
 
yeah I was considering that but the brain issues seem to be extremely hard to mitigate
retard u don't know shit JFL its not extremely hard to mitigate u just haven't done enough research :ROFLMAO:
 
memantine, pregnenolone, astaxanthin, nac, melatonin?
 
if you knew plenty you wouldn’t have to ask others let’s be real. you could potentially take halo, tren, var, there’s much more.
This is such a low iq take
 
Either way you want some estrogen expression right? Isn't it favorable that aromatase isn't blocked everywhere 100% in your body?
Why would you want e2 expression? I'm not saying to nuke e2 but you don't really need too much of it, AI's barely pierce the blood brain barrier ; E2 is locally synthesised in brain tissue anyways so it doesn't rely on serum levels.
 
Look into selective estrogen receptor degraders like fulvestrant
wouldn't thaty just have a similar effect to an ai )dif mechanism but u get the point) since it's not growth plate specific?
 
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wouldn't thaty just have a similar effect to an ai )dif mechanism but u get the point) since it's not growth plate specific?
It's not about growth plate specificity, when you see people running around with the phrase "AI's don't affect plates" its false but technically true. As always we have people running around on the forum and social media saying things but not explaining.

AI's do get into the bones and cartilage, that's literally why people get temporary osteoporosis and joint pains from really low/nuked e2. The issue is, when estrogen is decreased in the plates, the estrogen receptors just decide to incredibly upregulate their sensitivity to small trace amounts of estrogen left as a feedback loop.

This is why SERDs are useful for actually degrading the receptor so that the sensitivity doesn't increase. There's also another negative feedback loop with AI's that's actually a net negative for bone growth, the hpga sees there isn't enough estrogen so it upregulates lh and fsh to increase androgens for more aromatisation into estrogen which results in even more aromatisation into estrogen within the GP.

This is why it is optimal to shut off the HPGA with exogenous test at really low trt doses to avoid the e2 feedback loop.

So yes TLDR AI's are also growth plate specific but it practically doesn't decrease plate e2 mainly due to the negative feedback loop upregulating receptor sensitivity and as a biproduct increasing e2 from hpga.
 
Toral cope, anyone that has actually crushed e2 knows there are side effects to it. It might be locally produced as well, of course. But that doesn't mean the e2 in the blood is negligible when it comes to neuropathic side effects.
 

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