I am no professional but DHT is cancer, I think at 19 it should be enough. especially since you don't take dut but fin instead. if youre gonna roid u should really think about blocking dht since it might trigger norwood faster + acne
Yeah that's what I was thinking.
keep in mind tho u dont get the skin gains with fin whereas dut helps with that. but maybe start with fin on ur first cycle and see if u do get bad breakouts or not, ofc its up to u
I forgot does fin marginally lower dht that effects skin or does it just have 0 effect?
literally does nothing to bones
fin blocks the type II enzyme (70%) which is like the prostate, scalp (most importantly) and sum the type I is for the oil glands in the skin etc, that fin doesnt block.
I'll do my best to manage acne on cycle while I make sure fin gives no sides. If it's all good and I have some acne then I'll convert to dut after a month.
sounds like a good plan boss, always good to start with fin just to see how ur body reacts
pubmed.ncbi.nlm.nih.gov
Just use ru58841 and accutane.
Idk about accutane. I'm on tret right now but I can get really dry skin so idk if accutane is right for me.
U can drop tret and use low dose accutane on cycle
I'll def think about it. Idk if I can get prescribed bc I already have tret and no visible acne. Are most black/grey sources trustworthy or is it commonly faked?
Is easy to get
What will this do at 19 or 20 tho bro.Skeletal site-dependent expression of the androgen receptor in human osteoblastic cell populations - PubMed
There are obvious sexual differences in adult skeletal morphology which for the most part are related to differences in size. Higher androgen serum levels in males exert potent osteoanabolic effects and therefore may contribute to this sexual dimorphism of the skeleton. The presence of androgen...
Read the fucking study niiga
Still not the main factor in bones. Basically Negligible after 18. Ur not getting any visible jaw or bone change after 18 anyway
The study was in vitro (cell culture) and looked at receptor biology, not actual growth outcomes in living humans. Cope and seethe but DHT is still fucking useless, just blast T to be sure if you want that facial growth.Skeletal site-dependent expression of the androgen receptor in human osteoblastic cell populations - PubMed
There are obvious sexual differences in adult skeletal morphology which for the most part are related to differences in size. Higher androgen serum levels in males exert potent osteoanabolic effects and therefore may contribute to this sexual dimorphism of the skeleton. The presence of androgen...
Explain your points cuz this shit is pure retardation
You're saying that a study about injecting random cells DHT is a good argument against blocking DHT, thats insane. Most human facial bone growth is done by T and growth hormone, so if you really think that you can squeeze some facial masculinization, you should just blast GH and Testosterone.
Also fin blocks type 2 DHT which affects mostly hair/dick while type 1 is the one which mostly affects bone development
Not a single mechanistical argument.
Your bones need open plates to develop, even your facial ones, it doesnt matter if DHT develops isolated cells in vitro if they dont have room to grow on an actual human body. Most plates are 99% closed by 19. I thought that was so obvious i didn't need to explain.
No, nigga, bones get thicker through periosteal apposition
Which is mainly driven by testosterone and GH, just blast testosterone theory. JFL thinking that being bald is gonna make you gave a gigachad skull. Also you didnt even touch my argument about type I and type II DHT because you know I am right.
pubmed.ncbi.nlm.nih.gov
Which is mainly driven by testosterone and GH, just blast testosterone theory. JFL thinking that being bald is gonna make you gave a gigachad skull. Also you didnt even touch my argument about type I and type II DHT because you know I am right.
Finasteride therapy does not alter bone turnover in men with benign prostatic hyperplasia--a Clinical Research Center study - PubMed
Benign prostatic hyperplasia is often treated with finasteride, which inhibits the conversion of testosterone to dihydrotestosterone (DHT). Aside from the prostate, other androgen-dependent tissues seem to be unaffected by selective DHT deficiency, but the effect on bone density in humans has...
