Guys, this is driving me absolutely crazy and no one can give a straight answer *HIGH IQ GTFIH*

[iblamemandible7]

Does DHT have any notable influence on bone mass/forward growth/facial structure, or is that testosterone's job?

My hairline started receding at 16, and Im now on finasteride at 17 to combat it. Im at norwood 2 right now, and have a strong family history of balding. My question is, will blocking type 2 5-alpha-reductase have a negative effect on the growth and masculinization of my facial bones specifically? Also my frame and growth plates.

DHT haters say: DHT is a paracrine and intracrine hormone that works in the male reproductive and integumentary system to act on the prostate, skin, and genitalia. Testosterone, not DHT, is the androgen that affects bones.

DHT supporters say: In addition to that, DHT also works as an androgen within the skeletal system to affect bone growth in a way that other hormones (testosterone, IGF1) cannot. Blocking 5 alpha reductase 2 will result in less bone growth and development than allowing the enzyme to interact with testosterone.

I want to know which of these stances is more physiologically correct. Please no tiktok knowledge, I want actual answers instead of slideshow knowledge. I know DHT has 5x more affinity for the androgen receptor than testosterone, but does it work on androgen receptors in a similar way to testosterone all throughout the body, or does it work on androgen receptors in a more specialized and isolated way differently than testosterone?

 

[ducksoover]

google

 

[iblamemandible7]

google

Bhai Ive googled this extensively and searched probably the entirety of pubmed and cant find one consistent answer. DHT deficiency leads to increased hip fractures and osteoporosis in old men, but is this just correlation or some other mechanism of action, or is it really because of DHT? Males with 5 alpha reductase deficiency are born as females, but develop male traits at puberty. Is this from testosterone? Will the same thing happen if you have DHT at first and then block it? Rats with type 1 5 alpha reductase deficiency have decreased bone mass, is this applicable to humans? Is it only type 1? Males treated with finasteride have the same spinal bone density as males who arent, is that only for the spine but also for the face and clavicles? For every study I find theres one that contradicts it. I need someone who knows endocrinology and the mechanism of action to help me out

 

[ducksoover]

if ur receding at 16 id nuke my DHT

 

[GreekGenes]

youre 17 already so who cares about DHT for bones beyond that point. its gonna kill your hair, thats for sure tho

 

[Сигма Бой]

 

[DR. NICKGA]

Most retarded thread ever

 

[shaggybaggy696]

Does DHT have any notable influence on bone mass/forward growth/facial structure, or is that testosterone's job?

My hairline started receding at 16, and Im now on finasteride at 17 to combat it. Im at norwood 2 right now, and have a strong family history of balding. My question is, will blocking type 2 5-alpha-reductase have a negative effect on the growth and masculinization of my facial bones specifically? Also my frame and growth plates.

DHT haters say: DHT is a paracrine and intracrine hormone that works in the male reproductive and integumentary system to act on the prostate, skin, and genitalia. Testosterone, not DHT, is the androgen that affects bones.

DHT supporters say: In addition to that, DHT also works as an androgen within the skeletal system to affect bone growth in a way that other hormones (testosterone, IGF1) cannot. Blocking 5 alpha reductase 2 will result in less bone growth and development than allowing the enzyme to interact with DHT.

I want to know which of these stances is more physiologically correct. Please no tiktok knowledge, I want actual answers instead of slideshow knowledge. I know DHT has 5x more affinity for the androgen receptor than testosterone, but does it work on androgen receptors in a similar way to testosterone all throughout the body, or does it work on androgen receptors in a more specialized and isolated way differently than testosterone?

yes. DHT is 10x more potent than testosterone and is far more important for male sexual characteristics like Penis length, facial bone mass, chin height, jaw length, brow ridge, orbitals, voice deepening, even height. testosterone has little to no effect on all of these things (although about 10% of testosterone gets converted to DHT), testosterone affects muscle building and physique. DHT for male sexual characteristics and dimorphism.

do NOT take a dht inhibitor until at least 22. if you take some form of exogenous DHT your face and body can change a lot until ~23/24

 

[iblamemandible7]

if ur receding at 16 id nuke my DHT

Thats what Im doing, I just need to know that Im not fucking myself out of facial gains

 

[iblamemandible7]

yes. DHT is 10x more potent than testosterone and is far more important for male sexual characteristics like Penis length, facial bone mass, voice deepening, even height. testosterone has little to no effect on all of these things (although about 10% of testosterone gets converted to DHT), testosterone affects muscle building and physique. DHT for male sexual characteristics and dimorphism.

My question is, how would you explain males born with 5 alpha reductase deficiency developing all of these characteristics listed aside from penis length? Height is unaffected in these men, their voices drop, and they have adult male faces. Also, do you mean its 10x more potent at doing what testosterone does, or is it 10x more potent at binding to ARs?

 

[FutureSlayer]

no role whatsoever. (As you correctly said it’s only involved in penis size)
nuke that dht asap

 

[shaggybaggy696]

My question is, how would you explain males born with 5 alpha reductase deficiency developing all of these characteristics listed aside from penis length? Height is unaffected in these men, their voices drop, and they have adult male faces. Also, do you mean its 10x more potent at doing what testosterone does, or is it 10x more potent at binding to ARs?

i assume you have seen this video .
dht is not the only androgen for facial masculinization, IGF-1 is crucially important as well. their voices should not drop to the full extent, testosterone has some importance to the deepening of the voice but nowhere near the levels of someone with high DHT, estrogen is also very important for bonemass

 

[shaggybaggy696]

Thats what Im doing, I just need to know that Im not fucking myself out of facial gains

you are 16, you are the perfect age for pubertymaxxing, you should be doing at least a low dose test base with DHT gel on your dick and maybe adams apple (or if you really want to go balls deep take some systemic DHT derivative like masteron to increase your DHT levels everywhere, but this could bald you so you should do a topical dht inhibitor on the scalp)
you should also be on HGH and an AI

 

[iblamemandible7]

you are 16, you are the perfect age for pubertymaxxing, you should be doing at least a low dose test base with DHT gel on your dick and maybe adams apple (or if you really want to go balls deep take some systemic DHT derivative like masteron to increase your DHT levels everywhere, but this could bald you so you should do a topical dht inhibitor on the scalp)
you should also be on HGH and an AI

Forgot to mention Im 17 now, but how would a topical treatment work if I was using DHT and DHT derivative hormones? Since those hormones dont go through the 5ar pathway, wouldnt it be impossible to block it from targeting the scalp?

 

[tweaqo]

Bhai Ive googled this extensively and searched probably the entirety of pubmed and cant find one consistent answer. DHT deficiency leads to increased hip fractures and osteoporosis in old men, but is this just correlation or some other mechanism of action, or is it really because of DHT? Males with 5 alpha reductase deficiency are born as females, but develop male traits at puberty. Is this from testosterone? Will the same thing happen if you have DHT at first and then block it? Rats with type 1 5 alpha reductase deficiency have decreased bone mass, is this applicable to humans? Is it only type 1? Males treated with finasteride have the same spinal bone density as males who arent, is that only for the spine but also for the face and clavicles? For every study I find theres one that contradicts it. I need someone who knows endocrinology and the mechanism of action to help me out

Just get hair transplant when ur older

 

[iblamemandible7]

i assume you have seen this video .
dht is not the only androgen for facial masculinization, IGF-1 is crucially important as well. their voices should not drop to the full extent, testosterone has some importance to the deepening of the voice but nowhere near the levels of someone with high DHT, estrogen is also very important for bonemass

Good video derek knows his shit but I didnt hear any mention of facial bones or skeletal bone mass at all from him so still curious.

 

[DR. NICKGA]

My question is, how would you explain males born with 5 alpha reductase deficiency developing all of these characteristics listed aside from penis length? Height is unaffected in these men, their voices drop, and they have adult male faces. Also, do you mean its 10x more potent at doing what testosterone does, or is it 10x more potent at binding to ARs?

Dht bind different at the androgen receptor then testosterone

My question is, how would you explain males born with 5 alpha reductase deficiency developing all of these characteristics listed aside from penis length? Height is unaffected in these men, their voices drop, and they have adult male faces

You got you answer, its useless after 18+

You face is done with developing (and frame) after 18+

 

[DR. NICKGA]

Good video derek knows his shit but I didnt hear any mention of facial bones or skeletal bone mass at all from him so still curious.

Thats not a problem if you take finasteride

It dont block type enzyme 1, that is present at the bones

As i said, facial bones is done developing after 18+ even after 15

 

[shaggybaggy696]

You face is done with developing (and frame) after 18+

this is true (depending on the ethnicity) that your face is roughly fully developed by 18, but your facial androgen receptors are still sensitive and receptive to change until 24/25 if you take exogenous hormones.

in my case my facial changes from 18-20 were significant especially the lower third. but im caucasian and whites have extended puberties finishing later

 

[iblamemandible7]

Thats not a problem if you take finasteride

It dont block type enzyme 1, that is present at the bones

As i said, facial bones is done developing after 18+ even after 15

Ok good to hear but I just want to stay unbiased, do you have a source for type 1 5ar being present in bone? I know a study about it being in mice bones but not humans

 

[Jonas2k7]

Does DHT have any notable influence on bone mass/forward growth/facial structure, or is that testosterone's job?

My hairline started receding at 16, and Im now on finasteride at 17 to combat it. Im at norwood 2 right now, and have a strong family history of balding. My question is, will blocking type 2 5-alpha-reductase have a negative effect on the growth and masculinization of my facial bones specifically? Also my frame and growth plates.

DHT haters say: DHT is a paracrine and intracrine hormone that works in the male reproductive and integumentary system to act on the prostate, skin, and genitalia. Testosterone, not DHT, is the androgen that affects bones.

DHT supporters say: In addition to that, DHT also works as an androgen within the skeletal system to affect bone growth in a way that other hormones (testosterone, IGF1) cannot. Blocking 5 alpha reductase 2 will result in less bone growth and development than allowing the enzyme to interact with testosterone.

I want to know which of these stances is more physiologically correct. Please no tiktok knowledge, I want actual answers instead of slideshow knowledge. I know DHT has 5x more affinity for the androgen receptor than testosterone, but does it work on androgen receptors in a similar way to testosterone all throughout the body, or does it work on androgen receptors in a more specialized and isolated way differently than testosterone?

The studies are contradicting themselves, there is no real answer. What is factual though, is that DHT has other effects on gene expression than testosterone as they have different affinity to the androgen receptor. I wouldn't risk blocking DHT during puberty, if you are able to I'd recommend using growth factors and minoxidil for hair growth.

But if you are really NW2 right now at 17, I'd recommend hopping on Dutasteride over Finasteride as it should have less side effects as Dutasteride is blocking all types of 5AR not just one like Finasteride. Also PFS is only with Finasteride and not with Dutasteride.

 

[shaggybaggy696]

Ok good to hear but I just want to stay unbiased, do you have a source for type 1 5ar being present in bone? I know a study about it being in mice bones but not humans

dont do fin until later bro. its not only about bones, you are possibly stunting penile growth and other dimorphic features.
look into topical DHT blockers if you are desperate (i have not read much about these)

 

[iblamemandible7]

dont do fin until later bro. its not only about bones, you are possibly stunting penile growth and other dimorphic features.
look into topical DHT blockers if you are desperate (i have not read much about these)

My dick is already grown and my hair is much more important to me than getting a beard or more body hair. Topical 5ar inhibitors still go systemic, and the only effective topical antiandrogen, ru55841, can destroy your heart so its not worth it. Tbh bhai being a norwood 2 at 17 I think my only option is to take it as long as it doesnt stunt my bones, being bald would absolutely kill my looks

 

[iblamemandible7]

The studies are contradicting themselves, there is no real answer. What is factual though, is that DHT has other effects on gene expression than testosterone as they have different affinity to the androgen receptor. I wouldn't risk blocking DHT during puberty, if you are able to I'd recommend using growth factors and minoxidil for hair growth.

But if you are really NW2 right now at 17, I'd recommend hopping on Dutasteride over Finasteride as it should have less side effects as Dutasteride is blocking all types of 5AR not just one like Finasteride. Also PFS is only with Finasteride and not with Dutasteride.

Someone else said type 1 5AR is active within the bone in a way type 2 isnt, do you agree?

Also I would use minoxidil but my dad hit norwood 3 by his early 20s and my grandpa went bald around then so I have to get to the root cause before its too late yk

 

[shaggybaggy696]

My dick is already grown and my hair is much more important to me than getting a beard or more body hair. Topical 5ar inhibitors still go systemic, and the only effective topical antiandrogen, ru55841, can destroy your heart so its not worth it. Tbh bhai being a norwood 2 at 17 I think my only option is to take it as long as it doesnt stunt my bones, being bald would absolutely kill my looks

have you looked into topical fin? I seriously dont recommend taking 5ar inhibitors at 16

 

[iblamemandible7]

have you looked into topical fin? I seriously dont recommend taking 5ar inhibitors at 16

Topical fin goes systemic its proven to lower serum DHT the exact same at the 1 month mark as oral. 17 btw but yeah Im definitely young but I can deal with stopping my dick growth right now not my skeleton tho

 

[Jonas2k7]

Someone else said type 1 5AR is active within the bone in a way type 2 isnt, do you agree?

Multiple types of 5AR are in the bone tissue.

Also I would use minoxidil but my dad hit norwood 3 by his early 20s and my grandpa went bald around then so I have to get to the root cause before its too late yk

The minoxidil is just to save your hair some time until you can get to the 5AR inhibitors.

 

[iblamemandible7]

Multiple types of 5AR are in the bone tissue.

The minoxidil is just to save your hair some time until you can get to the 5AR inhibitors.

"5α-reductase type 2 is predominantly expressed in male reproductive tissues whereas 5α-reductase type 1 is highly expressed in liver and moderately expressed in several other tissues including bone"

Reduced Bone Mass and Muscle Strength in Male 5α-Reductase Type 1 Inactivated Mice - PMC

Androgens are important regulators of bone mass but the relative importance of testosterone (T) versus dihydrotestosterone (DHT) for the activation of the androgen receptor (AR) in bone is unknown. 5α-reductase is responsible for the irreversible ...

pmc.ncbi.nlm.nih.gov

In mice it seems like type 2 is almost negligent in the bone while type 1 and 3 are much more present, thoughts?

Also I heard minoxidil just hides hair loss and all the hair will fall out with years if DHT isnt treated, I know my hair will probably end up falling out even with treatment I just want to get the most longevity possible

 

[forestanon]

inject test 500 nigga

 

[iblamemandible7]

inject test 500 nigga

Im still 5'9 negroid

 

[forestanon]

Im still 5'9 negroid

inject MSC's into your vertebrae stop complaining JFL

 

[Newday*V3]

yes. DHT is 10x more potent than testosterone and is far more important for male sexual characteristics like facial bone mass, chin height, jaw length, brow ridge, orbitals, even height. testosterone has little to no effect on all of these things

 

[iblamemandible7]

Yeah that sounds like bullshit to me I thought testosterone is the main skeletal androgen but who fucking knows atp

 

[Newday*V3]

Yeah that sounds like bullshit to me I thought testosterone is the main skeletal androgen but who fucking knows atp

He is blabbering, DHT is a massive bone ager, shit will close growth plates in the jaw, maxilla sutures, and growth plates in the body if under the right conditions. DHT is still really good for bone density though but same as estrogen, in a way they are very similar

 

[shaggybaggy696]

He is blabbering, DHT is a massive bone ager, shit will close growth plates in the jaw, maxilla sutures, and growth plates in the body if under the right conditions. DHT is still really good for bone density though but same as estrogen, in a way they are very similar

DHT does NOT close growth plates. Estrogen does, DHT increases your height growth velocity by upregulating chrondocytes in the growth plates.
.
Testosterone gets converted into both DHT and Estrogen, people with low aromatase conversion efficiency (more test gets converted to DHT rather than estrogen) grow taller. DHT is not responsible for the fusion of any growth plate lmao, estrogen is.

Sex-specific effects of 17β-estradiol and dihydrotestosterone (DHT) on growth plate chondrocytes are dependent on both ERα and ERβ and require palmitoylation to translocate the receptors to the plasma membrane

Rat costochondral cartilage growth plate chondrocytes exhibit cell sex-specific responses to 17β-estradiol (E2), testosterone, and dihydrotestosterone…

www.sciencedirect.com

your lack of knowledge is humiliating

 

[shaggybaggy696]

Yeah that sounds like bullshit to me I thought testosterone is the main skeletal androgen but who fucking knows atp

dht = dihydrotestosterone

Reduced Bone Mass and Muscle Strength in Male 5α-Reductase Type 1 Inactivated Mice

Androgens are important regulators of bone mass but the relative importance of testosterone (T) versus dihydrotestosterone (DHT) for the activation of the androgen receptor (AR) in bone is unknown. 5α-reductase is responsible for the irreversible conversion of T to the more potent AR activator...

journals.plos.org

"Androgens are important regulators of bone mass but the relative importance of testosterone (T) versus dihydrotestosterone (DHT) for the activation of the androgen receptor (AR) in bone is unknown. 5α-reductase is responsible for the irreversible conversion of T to the more potent AR activator DHT. There are two well established isoenzymes of 5α-reductase (type 1 and type 2), encoded by separate genes (Srd5a1 and Srd5a2). 5α-reductase type 2 is predominantly expressed in male reproductive tissues whereas 5α-reductase type 1 is highly expressed in liver and moderately expressed in several other tissues including bone"
" In conclusion, 5α-reductase type 1 inactivated male mice have reduced bone mass "
this is nothing new. why do i have to be explaining this JFL

 

[Newday*V3]

DHT does NOT close growth plates. Estrogen does, DHT increases your height growth velocity by upregulating chrondocytes in the growth plates.
.
Testosterone gets converted into both DHT and Estrogen, people with low aromatase conversion efficiency (more test gets converted to DHT rather than estrogen) grow taller. DHT is not responsible for the fusion of any growth plate lmao, estrogen is.

Sex-specific effects of 17β-estradiol and dihydrotestosterone (DHT) on growth plate chondrocytes are dependent on both ERα and ERβ and require palmitoylation to translocate the receptors to the plasma membrane

Rat costochondral cartilage growth plate chondrocytes exhibit cell sex-specific responses to 17β-estradiol (E2), testosterone, and dihydrotestosterone…

www.sciencedirect.com

your lack of knowledge is humiliating

Wow, a study done on rats (an animal that has there growth plates open for the majority on there life) amazing , no fucking shit they would just keep growing, because they don’t experience bone aging like humans do.

Androgens are important regulators of bone mass

MASSSSS U FOOOOOLLL MASSSS, NOT LONGITUDINAL GROWTH THROUGH THE JAW, GROWTH PLATES OR SUTURES, MASS/APPOSITIONNNN.

 

[Newday*V3]

DHT does NOT close growth plates. Estrogen does, DHT increases your height growth velocity

And also, keyword VELOCITY VELOCITY VELOCITY

 

[JizzyMonohydrate]

Bhai Ive googled this extensively and searched probably the entirety of pubmed and cant find one consistent answer. DHT deficiency leads to increased hip fractures and osteoporosis in old men, but is this just correlation or some other mechanism of action, or is it really because of DHT? Males with 5 alpha reductase deficiency are born as females, but develop male traits at puberty. Is this from testosterone? Will the same thing happen if you have DHT at first and then block it? Rats with type 1 5 alpha reductase deficiency have decreased bone mass, is this applicable to humans? Is it only type 1? Males treated with finasteride have the same spinal bone density as males who arent, is that only for the spine but also for the face and clavicles? For every study I find theres one that contradicts it. I need someone who knows endocrinology and the mechanism of action to help me out

@m0ss26 help this tard

 

[shaggybaggy696]

Wow, a study done on rats (an animal that has there growth plates open for the majority on there life) amazing , no fucking shit they would just keep growing, because they don’t experience bone aging like humans do.

MASSSSS U FOOOOOLLL MASSSS, NOT LONGITUDINAL GROWTH THROUGH THE JAW, GROWTH PLATES OR SUTURES, MASS/APPOSITIONNNN.

because thats the responsibility of IGF-1 (insulin like growth factor 1), the precurser to HGH. holy shit you surely cant be this stupid right?
in regards to height and growth in surtures GROWTH FACTORS is what grows this. DHT increases the velocity and thus final outcome of the growth paired with IGF-1

bros argument is "im wrong! b-b-but rats! "
"Mice and rats have long served as the preferred species for biomedical research animal models due to their anatomical, physiological, and genetic similarity to humans"
its over for your knowledge bro

 

[Newday*V3]

because thats the responsibility of IGF-1 (insulin like growth factor 1), the precurser to HGH. holy shit you surely cant be this stupid right?

No shit, that is what I am implying? When did I say this isn’t the case

in regards to height and growth in surtures GROWTH FACTORS is what grows this. DHT increases the velocity and thus final outcome of the growth paired with IGF-1

VELOCITY (Do you know why?) because it accelerates BONE AGE through mineralization, T3 also does this, it accelerates growth while also accelerating ossification.

bros argument is "im wrong! b-b-but rats! "
"Mice and rats have long served as the preferred species for biomedical research animal models due to their anatomical, physiological, and genetic similarity to humans"
its over for your knowledge bro

 

[Seong Gi-Hun]

you need to remember that T turns into DHT

 

[shaggybaggy696]

VELOCITY (Do you know why?) because it accelerates BONE AGE through mineralization, T3 also does this, it accelerates growth while also accelerating ossification.

and do you know what else does? IGF-1. you are clearly trying to drift off topic in a sad attempt to win this argument you clearly know nothing about

is the topic of this thread about Height growth? or facial bone mass? thats right
mice DO have growth plates, they just dont fuse, making them great for research.
once again "Mice and rats have long served as the preferred species for biomedical research animal models due to their anatomical, physiological, and genetic similarity to humans" keyword: similarity

 

[Newday*V3]

and do you know what else does? IGF-1. you are clearly trying to drift off topic in a sad attempt to win this argument you clearly know nothing about

Mogged by chatgpt

is the topic of this thread about Height growth? or facial bone mass? thats right

yes. DHT is 10x more potent than testosterone and is far more important for male sexual characteristics like Penis length, facial bone mass, chin height, jaw length, brow ridge, orbitals, voice deepening, even height.

Just highlighted the 2 longitudinally growing parts of the body you previously typed in in order to refresh your short-term memory

mice DO have growth plates,

No shit

they just dont fuse,

No shit, that is the entire point, they don’t fuse under any accelerant so it is incomparable to a human

making them great for research.

No, since mice growth plates don’t fuse, then virtually any growth accelerant that promotes bone mineralization would never be able to stop the mice from growing, they could use estrogen ffs and they’d still be growing

This is not the case in humans since human’s growth plates actually age through sex hormones unlike mice.

 

[shaggybaggy696]

No, since mice growth plates don’t fuse, then virtually any growth accelerant that promotes bone mineralization would never be able to stop the mice from growing, they could use estrogen ffs and they’d still be growing

decades of scientific research concluding mice are ideal for research due to their anatomical, physiological, and genetic similarity to humans proven wrong by an incel online

you have outdone yourself sir

 

[Newday*V3]

decades of scientific research concluding mice are ideal for research due to their anatomical, physiological, and genetic similarity to humans proven wrong by an incel online

you have outdone yourself sir

It’s like you cannot read or something

Here is from chatgpt for your sub100 iq brain

“Well, muh mice is used for scientific sheit, that makes it accurate for everything cuh me say so! me just ignore everything, mice and human same!”

 

[shaggybaggy696]

It’s like you cannot read or something
View attachment 3511942View attachment 3511943
View attachment 3511945

Here is from chatgpt for your sub100 iq brain
View attachment 3511949

“Well, muh mice is used for scientific sheit, that makes it accurate for everything cuh me say so! me just ignore everything, mice and human same!”

no shit mice dont have growth plate fusion. what is bro even on about

mention once in OPs title where he talks about height growth. its not like the literal title of this thread is "Does DHT have any notable influence on bone mass/forward growth/facial structure, or is that testosterone's job?"

 

[latent]

finasteride inhibits type 2 5-alpha-reductase reduces DHT production mainly in tissues like the scalp and prostate, facial bones are likely fused at 17 so no it doesn't affect it, Bone expresses type 1 5-alpha-reductase Finasteride’s weak effect on type 1 means DHT production in bone is hugely preserved, your growth plates have likely fused but clavicle bones end growth at 25 same with spine so it might effect your shoulder width, still bone relies on type 1 5-alpha-reductase not finasterides target but still affects it a little bit. your take on dht supporters is so overestimated it's funny, bone relies more on testosterone for masculinity than DHT it's still up to you in using it or not.

 

[Newday*V3]

no shit mice dont have growth plate fusion. what is bro even on about

Im convinced ur just trolling now

mention once in OPs title where he talks about height growth. its not like the literal title of this thread is "Does DHT have any notable influence on bone mass/forward growth/facial structure, or is that testosterone's job?"

Forward growth of the jaw is primarily longitudinal
“Facial structure” can include maxillary sutures which is longitudinal
and also, you were the one who included height for some reason, which is longitudinal

yes. DHT is 10x more potent than testosterone and is far more important for male sexual characteristics like Penis length, facial bone mass, chin height, jaw length, brow ridge, orbitals, voice deepening, even height.

 

[vtribal]

ur balding and worried about 2 mm of bone in your face