Height growth post-puberty possible? (Shortcels GTFIH)

[zeke.htn]

Height Growth as an Adult?

Once your growth plates are closed, most people think it's over unless you get LL Surgery. I may have found a way to elongate the bones, without endochondral ossification (growth plate growth).

Now we know banded sleeping works for spinal decompression and gives 1-2 inches over months, however this effect fades away over a few months when you decide to stop. I think this can possibly be turned to real permanent height. Most people don't know but bones have plasticity, they are able to bend and compress without breaking (ex. people with rickets). They can go through deformation.

Now there are two types of deformation,
- elastic deformation
- plastic deformation

Elastic is temporary (bone springs back into original shape). Plastic, however, is permanent, the bone stays in the state its deformed in. In theory, it is possible to permanently elongate bone through carefully applied, sustained axial tension. If a long bone is subjected to tension above its yield point but below the fracture threshold, it can plastically deform.

With induced microfractures in bone combined with banded sleeping, it could gradually lengthen by along that axis. Plastic deformation in this way is irreversible, and once the bone remodels under these new mechanical stresses, it could retain the longer shape as the material adapts to reinforce its structure along the new orientation. This principle is analogous to the surgical procedure of distraction osteogenesis, otherwise known as limb lengthening surgery, except that here it relies purely on mechanical stress instead of osteotomy to creating a physical gap.


Applying this concept to banded sleeping, the idea is that by stretching the bone during sleep WITH induced microfractures could induce sustained axial tension along the spine, femur, and tibia gradually stretching the vertebrae, intervertebral discs, and bone over months. In theory, if this tension were maintained consistently and precisely, the bones could undergo plastic deformation, while the discs adapt to the stress, potentially increasing end-to-end length. Over time, this process could create a permanent increase in stature, as the reinforced bones retain their new elongated geometry via osteogenesis.

I estimate this to cause 5-12mm per year of elongation (0.2-0.5in). It may not seem like much, but over a long period of time (ex. 10 years) it could give 37-92mm (1.5-3.6in) And that not accounting for the fast temporary spinal decompression gains of 1-2 inches in the first 3-6 months.

It may seem ridiculous and difficult but if you're a shortcel I wouldn't complain.

(just a bullshit theory, but I want to try to perform an experiment on myself to see if its really possible)

 

[HundredManSlayer]

it’s all theoretical though

10 years for 1.5-3.6 inches, maybe worth it maybe not

nonetheless it makes sense and i’ve seen the theory before

 

[zeke.htn]

it’s all theoretical though

10 years for 1.5-3.6 inches, maybe worth it maybe not

nonetheless it makes sense and i’ve seen the theory before

Ill make another post in 2-3 years and ill show the results on myself to see if its really possible

 

[Iblamegirthquake]

it’s all theoretical though

10 years for 1.5-3.6 inches, maybe worth it maybe not

nonetheless it makes sense and i’ve seen the theory before

Yeah, by the time your 35, you now 5'8 instead of 5'5.

 

[HundredManSlayer]

Ill make another post in 2-3 years and ill show the results on myself to see if its really possible

looking forward to it, you seem very high iq

very good threads so far

 

[DandyPickle91]

h

Ill make another post in 2-3 years and ill show the results on myself to see if its really possible

how old are you exactly?

 

[zeke.htn]

h

how old are you exactly?

I'm 16 almost 17 currently, I'd have to get a scan to be sure but I'm pretty sure my plates are closed or pretty much closed. I'll try to wait a bit to make sure puberty is not a factor. Currently at 5'9.

 

[DandyPickle91]

I'm 16 almost 17 currently, I'd have to get a scan to be sure but I'm pretty sure my plates are closed or pretty much closed. I'll try to wait a bit to make sure puberty is not a factor. Currently at 5'9.

also you did mention something about banded sleeping and I was wondering how much compression I should add since I was planning to start doing this.

 

[DandyPickle91]

Yeah, by the time your 35, you now 5'8 instead of 5'5.

nigga what are you saying

 

[zeke.htn]

also you did mention something about banded sleeping and I was wondering how much compression I should add since I was planning to start doing this.

40lb bands for both feet would be effective, but not too strong to yank you out of your bed. Which you wont have to worry about if its comined with a head mask pulling in the opossite direction.

 

[DandyPickle91]

40lb bands for both feet would be effective, but not too strong to yank you out of your bed. Which you wont have to worry about if its comined with a head mask pulling in the opossite direction.

tell me if I'm wrong but I thought when people would say to compress your legs to grow I thought you would put like a band around your quad, calfs, etc but wdym by bands on both feet.

 

[zeke.htn]

tell me if I'm wrong but I thought when people would say to compress your legs to grow I thought you would put like a band around your quad, calfs, etc but wdym by bands on both feet.

Banded sleeping is more like this; it's about stretching your body not squeezing. Your probably thinking of somethin else.

 

[amineamine00]

Banded sleeping is more like this; it's about stretching your body not squeezing. Your probably thinking of somethin else.

This is all good , the point is how much force do u need to put on the bone for it to actually respond , and let's suppose for the sake of this theory that u figure out the right amount of force needed, can ur body sustain it ?

 

[zeke.htn]

This is all good , the point is how much force do u need to put on the bone for it to actually respond , and let's suppose for the sake of this theory that u figure out the right amount of force needed, can ur body sustain it ?

The actual force required would be most likely more than your body can sustain, but In theory with induced microfractures, the threshold for plastic deformation of bone would be lower, allowing for them to elongate without damaging your body.

 

[CyprusGD]

Me when I don’t know how bones work.

 

[amineamine00]

Me when I don’t know how bones work.

Me when idk what plastic deformation is.

 

[CyprusGD]

Me when idk what plastic deformation is.

Plastic deformation is an injury, it has nothing to do with bone elongation you low iq mutt

 

[zeke.htn]

Plastic deformation is an injury, it has nothing to do with bone elongation you low iq mutt

deformation can be anything, a bend, a twist, and it can theoretically elongate your bones as well under the right conditions

 

[CyprusGD]

deformation can be anything, a bend, a twist, and it can theoretically elongate your bones as well under the right conditions

Plastic deformation for bones is specifically an injury. It cannot lengthen bones. Bones do not stretch or elongate especially from bands. By this logic standing all day would make our bones very short but that doesn’t happen.

 

[CyprusGD]

deformation can be anything, a bend, a twist, and it can theoretically elongate your bones as well under the right conditions

And clearly you haven’t gained any height from this

 

[zeke.htn]

Plastic deformation for bones is specifically an injury. It cannot lengthen bones. Bones do not stretch or elongate especially from bands. By this logic standing all day would make our bones very short but that doesn’t happen.

And clearly you haven’t gained any height from this

Plastic deformation is able to manipulate the bone in anyway, the plastic deformation zone will never go away no matter how much the bone is manipulated or in whichever direction it is being manipulated in, so is the proper force is applied in the right direction then the bone can stretch (theoretically).

So far I'm about 1.8 cm taller morning height in 2.5 months, my study is still going on I'm not going to make a post for another couple months and until I verify that it is permanent and not temporary. It may just be spinal decompression and fluid retention but just wait till I finish my experiment boyo.

 

[CyprusGD]

Plastic deformation is able to manipulate the bone in anyway, the plastic deformation zone will never go away no matter how much the bone is manipulated or in whichever direction it is being manipulated in, so is the proper force is applied in the right direction then the bone can stretch (theoretically).

So far I'm about 1.8 cm taller morning height in 2.5 months, my study is still going on I'm not going to make a post for another couple months and until I verify that it is permanent and not temporary. It may just be spinal decompression and fluid retention but just wait till I finish my experiment boyo.

You are one retarded mfer. The 1.8 cm is pure decompression, posture fix, measurement error and/or puberty.

banded sleeping claims that wearing elastic or inelastic bands around limbs or torso during sleep provides a sustained low magnitude tensile load allegedly stimulating longitudinal bone growth via:

- Mechanotransduction causing osteoblast proliferation
- "creep" of bone matrix
- chondrocyte stretch in remaining growth plates

problem is that bone is not viscoelastic in the way required for creep elongation. true creep (time dependent plastic strain under constant load) occurs in polymers, metals above half their melting point (homologous temperature >0.5) and soft tissues like tendon. at 37°C bone’s homologous temperature is like 0.289x its melting point (hydroxyapatite decomposes at over 800°C). no dislocation climb, no grain boundary sliding, no time dependent elongation in mineralized tissue at body temperature.


2. Tensile vs. compressive loading

wolff’s law (actual formulation is yhat bone adapts to habitual mechanical environment) operates primarily under compression and shear and not sustained pure tension. why tho?

- osteocytes detect fluid flow in canaliculi driven by strain gradients. constant tension without cyclic variation means adaptation stalls within hours due to mechanosensory desensitization (Piezo1/TRPV4 channels inactivate)

- in fact sustained tension without rest periods triggers disuse osteopenia paradoxically, that means bone resorption via RANK L upregulation.


3. Band tensile forces

a typical elastic band provides like 75N at 100% elongation. but:

- for longitudinal strain (ε = σ/E) even 100N on a femur (CSA 4 cm² of cortical bone) gives axial stress σ = 0.25 MPa. bone’s elastic modulus E = 17 GPa means ε = 1.47 × 10^-5 (0.0015%). that’s like 100x below the mechanosensitive threshold (0.1% strain).

- growth plate cartilage (even if open) requires dynamic compressive strains of 5 - 15% to stimulate chondrocyte proliferation. static tension does nothing except maybe trigger apoptosis in chondrocytes


4. "Sleep duration = muh 8 hours of continuous loading"

tissues require unloading periods for fluid transport and waste clearance. eight hours of constant tension means:

- venous and lymphatic obstruction which means compartment syndrome risk if bands are tight (over 40 mmHg)
- nerve compression (peroneal, tibial, femoral) which causes paresthesias, foot drop or Saturday night palsy analogues
- muscle contracture causes sarcomere length adaptation in series and not bone growth. muscles would shorten under sustained low tension

even Ilizarov distraction (the only proven limb lengthening method) requires intermittent distraction (0.25mm every 6 hours) with daily compression phases and rigid external fixation and not elastic bands during sleep. this is actually funny lol, just imagine the difference.

through apatite lattice. at 37°C:

- diffusion coefficient of Ca2+ in hydroxyapatite will be 10^-20 m²/s (for comparison in water it’s like 10^-9 m²/s)
- characteristic time for 1% creep strain via diffusion is >10^9 years (I'm actually crying :banderas::banderas::banderas

so even if you applied 10x the yield stress continuously, bone would fracture violently before any measurable creep elongation occurred. the only known biological tensile creep is in dentin (due to collagen fibril sliding) but that saturates at like 0.1% strain and is damage lol not growth

in controlled in vitro experiments a fresh bone can be plastically deformed. but, in in vivo this isn't controllable. applying tension over its yield point means you're micro damaging the bone. body's response would be a pain induced muscular spasm to stop the stress, then there'd be an inflammatory healing response to repair the fuck up not elongate the bone.
plastic deformation is a constitutive material and not a biologically permissible adaptation in living bone. the moment cortical or trabecular bone approaches it's yield strain in vivo the body classifies it as structural damage instead of remodeling stimulus. osteocytes are mechanosensors and respond to supraphysiologic strain by starting targeted remodeling aimed at RESTORING PRE INJURY GEOMETRY, no preserving the deformation.
empirically, adult cortical bone yield strain is 0.75-0.89% (if i recall correctly) and fracture occurs at like 1.7% or something. there is no physiological window where sustained strain above yield strain can be applied again n again without triggering nociception, reflexive muscle contraction, inflammatory signaling and osteoclastic resorption followed by osteoblastic infill. this converges back to the original length, but, if i recall correctly, with higher cross sectional moment of inertia (thicker/stiffer bone)
and an important point is that plastic deformation observed in ex vivo bone samples is only possible because neural pain signaling is absent, muscular guarding is absent, cellular remodeling pathways are inactive.

once bone is living, plastic deformation can't accumulate longitudinally because remodeling enforces geometric homeostasis. this is why no longitudinal elongation of intact adult bones has ever been documented outside distraction osteogenesis (LL) or some pathological deformity
exercise induced "weakening" of bone produces diffuse microcracks (45-100 micrometer, iirc.) that activate targeted remodeling units. these cracks are spatially random, non collinear and discontinuous. the biological response is osteoclastic resorption followed by osteoblastic deposition that restores original length and alignment while increasing mineral density and cortical thickness. this is a protective adaptation.

intact long bones don't experience any useful axial tensile load during sleep. using whatever retarded distraction frame you have in mind would result in termination by pain mediated muscle contraction before yield strain is reached

why distraction osteogenesis succeeds and your idea fails because an osteotomy creates a low strain interfragmentary gap, mechanical fixation prevents collapse, controlled distraction of 1mm/day maintains strain within osteogenic window (2-10% iirc), bone forms by intramembranous ossification within the gap not by deformation of intact cortex. sorry son, but stretching an intact and fatigued bone doesn't place it into an osteogenic stretch regime. there's only an injury-repair loop and its outcome is reinforcement and not axial elongation.

bone remodels after damage and often becomes stronger but the strengthening occurs via increase in mineral density, cortical thickening and altered trabecular orientation and not via axial elongation.
osteocyte mediated remodeling is constrained by geometric homeostasis, remodeling units operate locally and restore baseline length and alignment and any increase in "size" documented in studies refers to CSA, moment of inertia or volumetric BMD and not length
this is why wolff’s law and frost’s mechanostat predict radial hypertrophy under load and not longitudinal growth. longitudinal growth is biologically restricted to endochondral ossification at growth plates which are absent in adults

 

[CyprusGD]

Plastic deformation is able to manipulate the bone in anyway, the plastic deformation zone will never go away no matter how much the bone is manipulated or in whichever direction it is being manipulated in, so is the proper force is applied in the right direction then the bone can stretch (theoretically).

So far I'm about 1.8 cm taller morning height in 2.5 months, my study is still going on I'm not going to make a post for another couple months and until I verify that it is permanent and not temporary. It may just be spinal decompression and fluid retention but just wait till I finish my experiment boyo.

You should’ve measured your legs specifically before and after that way you’d know if it was actual bone lengthening or just some other shit

 

[zeke.htn]

You should’ve measured your legs specifically before and after that way you’d know if it was actual bone lengthening or just some other shit

Well, if it can't lengthen bone I still think it can add up to 1.5 inches to your height with spinal decompression and fluid retention, even if its temporary. I'll try to prove that with my experiment, mirin the effort you took to debunk me tho, I guess I still have more research to do.

 

[CyprusGD]

Well, if it can't lengthen bone I still think it can add up to 1.5 inches to your height with spinal decompression and fluid retention, even if its temporary. I'll try to prove that with my experiment, mirin the effort you took to debunk me tho, I guess I still have more research to do.

Thanks bro. Also to test if it’s real height or permanent you should stop banded sleeping for like a couple days to a week and measure ur height afterwards

 

[CyprusGD]

Plastic deformation is able to manipulate the bone in anyway, the plastic deformation zone will never go away no matter how much the bone is manipulated or in whichever direction it is being manipulated in, so is the proper force is applied in the right direction then the bone can stretch (theoretically).

So far I'm about 1.8 cm taller morning height in 2.5 months, my study is still going on I'm not going to make a post for another couple months and until I verify that it is permanent and not temporary. It may just be spinal decompression and fluid retention but just wait till I finish my experiment boyo.

Also u said after 2.5 months, ur 1.8 cm taller in the morning. What about ur evening height?

 

[LeAwesomism]

You are one retarded mfer. The 1.8 cm is pure decompression, posture fix, measurement error and/or puberty.

banded sleeping claims that wearing elastic or inelastic bands around limbs or torso during sleep provides a sustained low magnitude tensile load allegedly stimulating longitudinal bone growth via:

- Mechanotransduction causing osteoblast proliferation
- "creep" of bone matrix
- chondrocyte stretch in remaining growth plates

problem is that bone is not viscoelastic in the way required for creep elongation. true creep (time dependent plastic strain under constant load) occurs in polymers, metals above half their melting point (homologous temperature >0.5) and soft tissues like tendon. at 37°C bone’s homologous temperature is like 0.289x its melting point (hydroxyapatite decomposes at over 800°C). no dislocation climb, no grain boundary sliding, no time dependent elongation in mineralized tissue at body temperature.


2. Tensile vs. compressive loading

wolff’s law (actual formulation is yhat bone adapts to habitual mechanical environment) operates primarily under compression and shear and not sustained pure tension. why tho?

- osteocytes detect fluid flow in canaliculi driven by strain gradients. constant tension without cyclic variation means adaptation stalls within hours due to mechanosensory desensitization (Piezo1/TRPV4 channels inactivate)

- in fact sustained tension without rest periods triggers disuse osteopenia paradoxically, that means bone resorption via RANK L upregulation.


3. Band tensile forces

a typical elastic band provides like 75N at 100% elongation. but:

- for longitudinal strain (ε = σ/E) even 100N on a femur (CSA 4 cm² of cortical bone) gives axial stress σ = 0.25 MPa. bone’s elastic modulus E = 17 GPa means ε = 1.47 × 10^-5 (0.0015%). that’s like 100x below the mechanosensitive threshold (0.1% strain).

- growth plate cartilage (even if open) requires dynamic compressive strains of 5 - 15% to stimulate chondrocyte proliferation. static tension does nothing except maybe trigger apoptosis in chondrocytes


4. "Sleep duration = muh 8 hours of continuous loading"

tissues require unloading periods for fluid transport and waste clearance. eight hours of constant tension means:

- venous and lymphatic obstruction which means compartment syndrome risk if bands are tight (over 40 mmHg)
- nerve compression (peroneal, tibial, femoral) which causes paresthesias, foot drop or Saturday night palsy analogues
- muscle contracture causes sarcomere length adaptation in series and not bone growth. muscles would shorten under sustained low tension

even Ilizarov distraction (the only proven limb lengthening method) requires intermittent distraction (0.25mm every 6 hours) with daily compression phases and rigid external fixation and not elastic bands during sleep. this is actually funny lol, just imagine the difference.

through apatite lattice. at 37°C:

- diffusion coefficient of Ca2+ in hydroxyapatite will be 10^-20 m²/s (for comparison in water it’s like 10^-9 m²/s)
- characteristic time for 1% creep strain via diffusion is >10^9 years (I'm actually crying :banderas::banderas::banderas

so even if you applied 10x the yield stress continuously, bone would fracture violently before any measurable creep elongation occurred. the only known biological tensile creep is in dentin (due to collagen fibril sliding) but that saturates at like 0.1% strain and is damage lol not growth

in controlled in vitro experiments a fresh bone can be plastically deformed. but, in in vivo this isn't controllable. applying tension over its yield point means you're micro damaging the bone. body's response would be a pain induced muscular spasm to stop the stress, then there'd be an inflammatory healing response to repair the fuck up not elongate the bone.
plastic deformation is a constitutive material and not a biologically permissible adaptation in living bone. the moment cortical or trabecular bone approaches it's yield strain in vivo the body classifies it as structural damage instead of remodeling stimulus. osteocytes are mechanosensors and respond to supraphysiologic strain by starting targeted remodeling aimed at RESTORING PRE INJURY GEOMETRY, no preserving the deformation.
empirically, adult cortical bone yield strain is 0.75-0.89% (if i recall correctly) and fracture occurs at like 1.7% or something. there is no physiological window where sustained strain above yield strain can be applied again n again without triggering nociception, reflexive muscle contraction, inflammatory signaling and osteoclastic resorption followed by osteoblastic infill. this converges back to the original length, but, if i recall correctly, with higher cross sectional moment of inertia (thicker/stiffer bone)
and an important point is that plastic deformation observed in ex vivo bone samples is only possible because neural pain signaling is absent, muscular guarding is absent, cellular remodeling pathways are inactive.

once bone is living, plastic deformation can't accumulate longitudinally because remodeling enforces geometric homeostasis. this is why no longitudinal elongation of intact adult bones has ever been documented outside distraction osteogenesis (LL) or some pathological deformity
exercise induced "weakening" of bone produces diffuse microcracks (45-100 micrometer, iirc.) that activate targeted remodeling units. these cracks are spatially random, non collinear and discontinuous. the biological response is osteoclastic resorption followed by osteoblastic deposition that restores original length and alignment while increasing mineral density and cortical thickness. this is a protective adaptation.

intact long bones don't experience any useful axial tensile load during sleep. using whatever retarded distraction frame you have in mind would result in termination by pain mediated muscle contraction before yield strain is reached

why distraction osteogenesis succeeds and your idea fails because an osteotomy creates a low strain interfragmentary gap, mechanical fixation prevents collapse, controlled distraction of 1mm/day maintains strain within osteogenic window (2-10% iirc), bone forms by intramembranous ossification within the gap not by deformation of intact cortex. sorry son, but stretching an intact and fatigued bone doesn't place it into an osteogenic stretch regime. there's only an injury-repair loop and its outcome is reinforcement and not axial elongation.

bone remodels after damage and often becomes stronger but the strengthening occurs via increase in mineral density, cortical thickening and altered trabecular orientation and not via axial elongation.
osteocyte mediated remodeling is constrained by geometric homeostasis, remodeling units operate locally and restore baseline length and alignment and any increase in "size" documented in studies refers to CSA, moment of inertia or volumetric BMD and not length
this is why wolff’s law and frost’s mechanostat predict radial hypertrophy under load and not longitudinal growth. longitudinal growth is biologically restricted to endochondral ossification at growth plates which are absent in adults

"what abt muh bone mashing "

 

[zeke.htn]

Also u said after 2.5 months, ur 1.8 cm taller in the morning. What about ur evening height?

In the evenings I'm much taller probably because of retaining the morning height, my evening height before was 175 (morning ~176) now my evening height is ~177.5. (and mornings around 177.8 almost 178)

 

[CyprusGD]

In the evenings I'm much taller probably because of retaining the morning height, my evening height before was 175 (morning ~176) now my evening height is ~177.5. (and mornings around 177.8 almost 178)

Too bad you didn’t measure ur legs