Heightmaxxing WATERFALL

Dave1

Dave1

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Disclaimer:


This thread is for informational purposes only.
The use of any medications, including growth hormones, aromatase inhibitors (like anastrozole or letrozole), or other drugs, for the purpose of increasing height should only be done under the supervision of a qualified healthcare professional.
Self-prescribing or using medications without medical guidance can lead to serious health risks, including but not limited to hormonal imbalances, bone health issues, and long-term complications.
Using these drugs for non-medical purposes, such as attempting to increase height in healthy individuals, can result in unintended consequences and potential legal implications.
If you or someone you know is considering any form of height-enhancing treatment, it is essential to consult with a licensed healthcare provider (such as an endocrinologist) to discuss the risks, benefits, and suitability of such treatments.
The information shared in this thread is not intended as medical advice. Always seek professional guidance before making decisions related to medications or treatments.


All these drugs are to be used throughout the whole heightmaxxing timeframe except anavar. Doses of all these drugs can vary. There is no one-size-fits-all approach, and the doses given here are just examples.
These drugs will provide an increase in adult height ONLY if your growth plates are open.

Do NOT use anavar if you cannot use testosterone. Anavar will cause suppression of testosterone, the TRT dose is there to replace the testosterone you will not have once you start using anavar. Either use all of the drugs listed or only use HGH + AI.


Testosterone
(WARNING: WILL SUPRESS HPTA, PCT NECESSERY AFTER HEIGHTMAXXING PROTCOL ENDS)
(use only if you're ok with the risks, not compulsory)
Binds to androgen receptors (AR) on chondrocytes within long bones. Promotes chondrocyte proliferation, differentiation, and maturation. This causes hypertrophy in long bones causing growth. Testosterone works synergistically with HGH since it increases growth hormone receptor expression in chondrocytes, increasing localised igf1 production.
(1)
Usage:
Can differ between people but usually just above TRT dose such as 125 to 200mg. Pinning ED is important for stable testosterone blood levels.
Recommended regimen: 150mg testosterone cypionate pinned ED.


HGH (2)
HGH goes to your liver and makes it produce IGF-1. IGF1 directly promotes growth through chondrocyte proliferation and differentiation in the growth plates, leading to bone lengthening and height increase. However using IGF1 LR3 is suboptimal BECAUSE: chondrocytes have localised HGH receptors meaning when HGH binds to the IGF-1R receptors on chondrocytes it further stimulates local production of IGF1 directly promoting growth.
Recommended regimen: 5 (min) to 10 ius pinned ED before bed.


Anavar (3)
(WARNING: WILL SUPRESS HPTA, PCT NECESSERY AFTER HEIGHTMAXXING PROTCOL ENDS)
Increases protein synthesis, also in cartilage on long bones, contributing to elongation of bones.
Recommended regimen as used in study (3): 0.25 mg/kg/day e.g, if you're 65kg: 16.25mg ED
Anavar however is hepatoxic, to avoid this you can use anavar for 10 weeks then 10 weeks off. Counteracting hepatoxicity can be done with: (ran while on anavar)

  • Milk thistle
  • NAC
  • TUDCA

Aromatase inhibitors (AI):
Many AI's exist however studies on anastrozole prove it's effectiveness (4)
AI's are used since when testosterone is produced, it's converted into estrogen. Estrogen binds to receptors on growth plate chondrocytes such as ERα and ERβ. This promotes conversion of chondrocytes into osteoblasts. This change from cartilate to bone leads to the closure of growth plates. Furthermore, estrogen triggers apoptosis in growth plate cartilage cells. This contributes to the gradual disapearance of cartilage and the formation of bone, leading to growth plate fusion. For this reason AI's such as anastrozole are used since they reduce the conversion of testosterone to estrogen by reducing the amount of 5a reductase which is an enzyme that converts testosterone to estrogen.
Estrogen closes growth plates
(5)
Recommended regimen: 1mg ED, however blood tests can give a more precise dose as you want your estrogen to be between 10 to 15pg/mL.
Another AI that can be used is letrozole, studies prove IT'S effectiveness aswell.
(6)
Recommended regimen: 1mg ED


(1)
"Androgen itself also contributes to bone formation and the pubertal growth spurt, perhaps through a direct interaction with growth plate chondrocytes"

(2)
"HGH in studies is only used in prebutal kids!!"
"The age at start of treatment ranged between 4.7 years and 16 years, 11.9 ± 3.3 years for the 68 males and 12 ± 1.9 years for the 20 females."

(3)
"Oxandrolone therapy resulted in a two-fold increase in mean growth velocity in the first six months of therapy and was an effective growth stimulant for the full four-year period."

(4)
"Anastrozole increases adult height potential of adolescent boys on GH therapy while maintaining normal pubertal progression after 2-3 yr."

(5)
"Estrogen determines the acceleration of bone elongation at puberty, epiphyseal closure"

(6)
"The final heights for Lz (letrozole) group were significantly (p=0.04) higher than the control group."

(pls rep :heart: )
 
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fire thread bro thank you ❤️‍🩹
 
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a thread worth reading 🥹
 
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i think theres older stuff on aromasin too
 
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i think theres older stuff on aromasin too
no actual studies confirming the efficacy of aromasin on height increase. it can still be used but idk i'd rather use the drugs used in studies
 
Genetics 😔
 
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Disclaimer:


This thread is for informational purposes only.
The use of any medications, including growth hormones, aromatase inhibitors (like anastrozole or letrozole), or other drugs, for the purpose of increasing height should only be done under the supervision of a qualified healthcare professional.
Self-prescribing or using medications without medical guidance can lead to serious health risks, including but not limited to hormonal imbalances, bone health issues, and long-term complications.
Using these drugs for non-medical purposes, such as attempting to increase height in healthy individuals, can result in unintended consequences and potential legal implications.
If you or someone you know is considering any form of height-enhancing treatment, it is essential to consult with a licensed healthcare provider (such as an endocrinologist) to discuss the risks, benefits, and suitability of such treatments.
The information shared in this thread is not intended as medical advice. Always seek professional guidance before making decisions related to medications or treatments.


All these drugs are to be used throughout the whole heightmaxxing timeframe except anavar. Doses of all these drugs can vary. There is no one-size-fits-all approach, and the doses given here are just examples.
These drugs will provide an increase in adult height ONLY if your growth plates are open.

Do NOT use anavar if you cannot use testosterone. Anavar will cause suppression of testosterone, the TRT dose is there to replace the testosterone you will not have once you start using anavar. Either use all of the drugs listed or only use HGH + AI.


Testosterone
(WARNING: WILL SUPRESS HPTA, PCT NECESSERY AFTER HEIGHTMAXXING PROTCOL ENDS)
(use only if you're ok with the risks, not compulsory)
Binds to androgen receptors (AR) on chondrocytes within long bones. Promotes chondrocyte proliferation, differentiation, and maturation. This causes hypertrophy in long bones causing growth. Testosterone works synergistically with HGH since it increases growth hormone receptor expression in chondrocytes, increasing localised igf1 production.
(1)
Usage:
Can differ between people but usually just above TRT dose such as 125 to 200mg. Pinning ED is important for stable testosterone blood levels.
Recommended regimen: 150mg testosterone cypionate pinned ED.


HGH (2)
HGH goes to your liver and makes it produce IGF-1. IGF1 directly promotes growth through chondrocyte proliferation and differentiation in the growth plates, leading to bone lengthening and height increase. However using IGF1 LR3 is suboptimal BECAUSE: chondrocytes have localised HGH receptors meaning when HGH binds to the IGF-1R receptors on chondrocytes it further stimulates local production of IGF1 directly promoting growth.
Recommended regimen: 5 (min) to 10 ius pinned ED before bed.


Anavar (3)
(WARNING: WILL SUPRESS HPTA, PCT NECESSERY AFTER HEIGHTMAXXING PROTCOL ENDS)
Increases protein synthesis, also in cartilage on long bones, contributing to elongation of bones.
Recommended regimen as used in study (3): 0.25 mg/kg/day e.g, if you're 65kg: 16.25mg ED
Anavar however is hepatoxic, to avoid this you can use anavar for 10 weeks then 10 weeks off. Counteracting hepatoxicity can be done with: (ran while on anavar)

  • Milk thistle
  • NAC
  • TUDCA

Aromatase inhibitors (AI):
Many AI's exist however studies on anastrozole prove it's effectiveness (4)
AI's are used since when testosterone is produced, it's converted into estrogen. Estrogen binds to receptors on growth plate chondrocytes such as ERα and ERβ. This promotes conversion of chondrocytes into osteoblasts. This change from cartilate to bone leads to the closure of growth plates. Furthermore, estrogen triggers apoptosis in growth plate cartilage cells. This contributes to the gradual disapearance of cartilage and the formation of bone, leading to growth plate fusion. For this reason AI's such as anastrozole are used since they reduce the conversion of testosterone to estrogen by reducing the amount of 5a reductase which is an enzyme that converts testosterone to estrogen.
Estrogen closes growth plates
(5)
Recommended regimen: 1mg ED, however blood tests can give a more precise dose as you want your estrogen to be between 10 to 15pg/mL.
Another AI that can be used is letrozole, studies prove IT'S effectiveness aswell.
(6)
Recommended regimen: 1mg ED


(1)
"Androgen itself also contributes to bone formation and the pubertal growth spurt, perhaps through a direct interaction with growth plate chondrocytes"

(2)
"HGH in studies is only used in prebutal kids!!"
"The age at start of treatment ranged between 4.7 years and 16 years, 11.9 ± 3.3 years for the 68 males and 12 ± 1.9 years for the 20 females."

(3)
"Oxandrolone therapy resulted in a two-fold increase in mean growth velocity in the first six months of therapy and was an effective growth stimulant for the full four-year period."

(4)
"Anastrozole increases adult height potential of adolescent boys on GH therapy while maintaining normal pubertal progression after 2-3 yr."

(5)
"Estrogen determines the acceleration of bone elongation at puberty, epiphyseal closure"

(6)
"The final heights for Lz (letrozole) group were significantly (p=0.04) higher than the control group."

(pls rep :heart: )
nigga summarize it i aint reading allat
 
if ur serious about height then reading all of it would be helpful + its not that long
im a 5 9 manlet so ill read it soon
 
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High iq imo
 
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Needs more reps, nice thread bhai. Just don't forget the ezetimibe while on anavar for lipids and fish oils DHA and EPA:feelsautistic:
 
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Disclaimer:


This thread is for informational purposes only.
The use of any medications, including growth hormones, aromatase inhibitors (like anastrozole or letrozole), or other drugs, for the purpose of increasing height should only be done under the supervision of a qualified healthcare professional.
Self-prescribing or using medications without medical guidance can lead to serious health risks, including but not limited to hormonal imbalances, bone health issues, and long-term complications.
Using these drugs for non-medical purposes, such as attempting to increase height in healthy individuals, can result in unintended consequences and potential legal implications.
If you or someone you know is considering any form of height-enhancing treatment, it is essential to consult with a licensed healthcare provider (such as an endocrinologist) to discuss the risks, benefits, and suitability of such treatments.
The information shared in this thread is not intended as medical advice. Always seek professional guidance before making decisions related to medications or treatments.


All these drugs are to be used throughout the whole heightmaxxing timeframe except anavar. Doses of all these drugs can vary. There is no one-size-fits-all approach, and the doses given here are just examples.
These drugs will provide an increase in adult height ONLY if your growth plates are open.

Do NOT use anavar if you cannot use testosterone. Anavar will cause suppression of testosterone, the TRT dose is there to replace the testosterone you will not have once you start using anavar. Either use all of the drugs listed or only use HGH + AI.


Testosterone
(WARNING: WILL SUPRESS HPTA, PCT NECESSERY AFTER HEIGHTMAXXING PROTCOL ENDS)
(use only if you're ok with the risks, not compulsory)
Binds to androgen receptors (AR) on chondrocytes within long bones. Promotes chondrocyte proliferation, differentiation, and maturation. This causes hypertrophy in long bones causing growth. Testosterone works synergistically with HGH since it increases growth hormone receptor expression in chondrocytes, increasing localised igf1 production.
(1)
Usage:
Can differ between people but usually just above TRT dose such as 125 to 200mg. Pinning ED is important for stable testosterone blood levels.
Recommended regimen: 150mg testosterone cypionate pinned ED.


HGH (2)
HGH goes to your liver and makes it produce IGF-1. IGF1 directly promotes growth through chondrocyte proliferation and differentiation in the growth plates, leading to bone lengthening and height increase. However using IGF1 LR3 is suboptimal BECAUSE: chondrocytes have localised HGH receptors meaning when HGH binds to the IGF-1R receptors on chondrocytes it further stimulates local production of IGF1 directly promoting growth.
Recommended regimen: 5 (min) to 10 ius pinned ED before bed.


Anavar (3)
(WARNING: WILL SUPRESS HPTA, PCT NECESSERY AFTER HEIGHTMAXXING PROTCOL ENDS)
Increases protein synthesis, also in cartilage on long bones, contributing to elongation of bones.
Recommended regimen as used in study (3): 0.25 mg/kg/day e.g, if you're 65kg: 16.25mg ED
Anavar however is hepatoxic, to avoid this you can use anavar for 10 weeks then 10 weeks off. Counteracting hepatoxicity can be done with: (ran while on anavar)

  • Milk thistle
  • NAC
  • TUDCA

Aromatase inhibitors (AI):
Many AI's exist however studies on anastrozole prove it's effectiveness (4)
AI's are used since when testosterone is produced, it's converted into estrogen. Estrogen binds to receptors on growth plate chondrocytes such as ERα and ERβ. This promotes conversion of chondrocytes into osteoblasts. This change from cartilate to bone leads to the closure of growth plates. Furthermore, estrogen triggers apoptosis in growth plate cartilage cells. This contributes to the gradual disapearance of cartilage and the formation of bone, leading to growth plate fusion. For this reason AI's such as anastrozole are used since they reduce the conversion of testosterone to estrogen by reducing the amount of 5a reductase which is an enzyme that converts testosterone to estrogen.
Estrogen closes growth plates
(5)
Recommended regimen: 1mg ED, however blood tests can give a more precise dose as you want your estrogen to be between 10 to 15pg/mL.
Another AI that can be used is letrozole, studies prove IT'S effectiveness aswell.
(6)
Recommended regimen: 1mg ED


(1)
"Androgen itself also contributes to bone formation and the pubertal growth spurt, perhaps through a direct interaction with growth plate chondrocytes"

(2)
"HGH in studies is only used in prebutal kids!!"
"The age at start of treatment ranged between 4.7 years and 16 years, 11.9 ± 3.3 years for the 68 males and 12 ± 1.9 years for the 20 females."

(3)
"Oxandrolone therapy resulted in a two-fold increase in mean growth velocity in the first six months of therapy and was an effective growth stimulant for the full four-year period."

(4)
"Anastrozole increases adult height potential of adolescent boys on GH therapy while maintaining normal pubertal progression after 2-3 yr."

(5)
"Estrogen determines the acceleration of bone elongation at puberty, epiphyseal closure"

(6)
"The final heights for Lz (letrozole) group were significantly (p=0.04) higher than the control group."

(pls rep :heart: )
Good thread but why dose Test C ED if it has like the longest half life, why not do test P EOD. test c ED I’m pretty sure will make ur dick stop working rofl
 
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Disclaimer:


This thread is for informational purposes only.
The use of any medications, including growth hormones, aromatase inhibitors (like anastrozole or letrozole), or other drugs, for the purpose of increasing height should only be done under the supervision of a qualified healthcare professional.
Self-prescribing or using medications without medical guidance can lead to serious health risks, including but not limited to hormonal imbalances, bone health issues, and long-term complications.
Using these drugs for non-medical purposes, such as attempting to increase height in healthy individuals, can result in unintended consequences and potential legal implications.
If you or someone you know is considering any form of height-enhancing treatment, it is essential to consult with a licensed healthcare provider (such as an endocrinologist) to discuss the risks, benefits, and suitability of such treatments.
The information shared in this thread is not intended as medical advice. Always seek professional guidance before making decisions related to medications or treatments.


All these drugs are to be used throughout the whole heightmaxxing timeframe except anavar. Doses of all these drugs can vary. There is no one-size-fits-all approach, and the doses given here are just examples.
These drugs will provide an increase in adult height ONLY if your growth plates are open.

Do NOT use anavar if you cannot use testosterone. Anavar will cause suppression of testosterone, the TRT dose is there to replace the testosterone you will not have once you start using anavar. Either use all of the drugs listed or only use HGH + AI.


Testosterone
(WARNING: WILL SUPRESS HPTA, PCT NECESSERY AFTER HEIGHTMAXXING PROTCOL ENDS)
(use only if you're ok with the risks, not compulsory)
Binds to androgen receptors (AR) on chondrocytes within long bones. Promotes chondrocyte proliferation, differentiation, and maturation. This causes hypertrophy in long bones causing growth. Testosterone works synergistically with HGH since it increases growth hormone receptor expression in chondrocytes, increasing localised igf1 production.
(1)
Usage:
Can differ between people but usually just above TRT dose such as 125 to 200mg. Pinning ED is important for stable testosterone blood levels.
Recommended regimen: 150mg testosterone cypionate pinned ED.


HGH (2)
HGH goes to your liver and makes it produce IGF-1. IGF1 directly promotes growth through chondrocyte proliferation and differentiation in the growth plates, leading to bone lengthening and height increase. However using IGF1 LR3 is suboptimal BECAUSE: chondrocytes have localised HGH receptors meaning when HGH binds to the IGF-1R receptors on chondrocytes it further stimulates local production of IGF1 directly promoting growth.
Recommended regimen: 5 (min) to 10 ius pinned ED before bed.


Anavar (3)
(WARNING: WILL SUPRESS HPTA, PCT NECESSERY AFTER HEIGHTMAXXING PROTCOL ENDS)
Increases protein synthesis, also in cartilage on long bones, contributing to elongation of bones.
Recommended regimen as used in study (3): 0.25 mg/kg/day e.g, if you're 65kg: 16.25mg ED
Anavar however is hepatoxic, to avoid this you can use anavar for 10 weeks then 10 weeks off. Counteracting hepatoxicity can be done with: (ran while on anavar)

  • Milk thistle
  • NAC
  • TUDCA

Aromatase inhibitors (AI):
Many AI's exist however studies on anastrozole prove it's effectiveness (4)
AI's are used since when testosterone is produced, it's converted into estrogen. Estrogen binds to receptors on growth plate chondrocytes such as ERα and ERβ. This promotes conversion of chondrocytes into osteoblasts. This change from cartilate to bone leads to the closure of growth plates. Furthermore, estrogen triggers apoptosis in growth plate cartilage cells. This contributes to the gradual disapearance of cartilage and the formation of bone, leading to growth plate fusion. For this reason AI's such as anastrozole are used since they reduce the conversion of testosterone to estrogen by reducing the amount of 5a reductase which is an enzyme that converts testosterone to estrogen.
Estrogen closes growth plates
(5)
Recommended regimen: 1mg ED, however blood tests can give a more precise dose as you want your estrogen to be between 10 to 15pg/mL.
Another AI that can be used is letrozole, studies prove IT'S effectiveness aswell.
(6)
Recommended regimen: 1mg ED


(1)
"Androgen itself also contributes to bone formation and the pubertal growth spurt, perhaps through a direct interaction with growth plate chondrocytes"

(2)
"HGH in studies is only used in prebutal kids!!"
"The age at start of treatment ranged between 4.7 years and 16 years, 11.9 ± 3.3 years for the 68 males and 12 ± 1.9 years for the 20 females."

(3)
"Oxandrolone therapy resulted in a two-fold increase in mean growth velocity in the first six months of therapy and was an effective growth stimulant for the full four-year period."

(4)
"Anastrozole increases adult height potential of adolescent boys on GH therapy while maintaining normal pubertal progression after 2-3 yr."

(5)
"Estrogen determines the acceleration of bone elongation at puberty, epiphyseal closure"

(6)
"The final heights for Lz (letrozole) group were significantly (p=0.04) higher than the control group."

(pls rep :heart: )
What AI is better ? Letrozole or exemestane because @MyDreamIsToBe183CM uses letrozole instead of exemestane which I thought was better. I am confused rn
 
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WHERE CAN I BUY HGH
 
WHERE CAN I BUY HGH
 
What AI is better ? Letrozole or exemestane because @MyDreamIsToBe183CM uses letrozole instead of exemestane which I thought was better. I am confused rn
probably letrozole since theres studies on it
 
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Reactions: theübermenschboy
Good thread but why dose Test C ED if it has like the longest half life, why not do test P EOD. test c ED I’m pretty sure will make ur dick stop working rofl
none make ur dick stop working, both make ur balls stop working which is why PCT after your heightmaxxing protocol is necessery.
test c ED since the slightly longer half life makes it have the most stable blood testosterone levels esp since it's pinned ED. furthermore test p EOD is very wrong propionate has roughly a day half life
 
none make ur dick stop working, both make ur balls stop working which is why PCT after your heightmaxxing protocol is necessery.
test c ED since the slightly longer half life makes it have the most stable blood testosterone levels esp since it's pinned ED. furthermore test p EOD is very wrong propionate has roughly a day half life
yea thats what i ment by dick not working your balls will flatline and u cant nut im on test e but still why test c ED if the half life is so long im retardmaxxing rn but it acc dosent make any sense rofl
 
Fire thread check ur dms
 
yes you can, without lh and fsh testosterone and sperm drops yes but doesnt mean u dont still produce semen but i get what u mean. u can use test e ED yes but test c will be slightly more stable in blood levels
 

Disclaimer:


This thread is for informational purposes only.
The use of any medications, including growth hormones, aromatase inhibitors (like anastrozole or letrozole), or other drugs, for the purpose of increasing height should only be done under the supervision of a qualified healthcare professional.
Self-prescribing or using medications without medical guidance can lead to serious health risks, including but not limited to hormonal imbalances, bone health issues, and long-term complications.
Using these drugs for non-medical purposes, such as attempting to increase height in healthy individuals, can result in unintended consequences and potential legal implications.
If you or someone you know is considering any form of height-enhancing treatment, it is essential to consult with a licensed healthcare provider (such as an endocrinologist) to discuss the risks, benefits, and suitability of such treatments.
The information shared in this thread is not intended as medical advice. Always seek professional guidance before making decisions related to medications or treatments.


All these drugs are to be used throughout the whole heightmaxxing timeframe except anavar. Doses of all these drugs can vary. There is no one-size-fits-all approach, and the doses given here are just examples.
These drugs will provide an increase in adult height ONLY if your growth plates are open.

Do NOT use anavar if you cannot use testosterone. Anavar will cause suppression of testosterone, the TRT dose is there to replace the testosterone you will not have once you start using anavar. Either use all of the drugs listed or only use HGH + AI.


Testosterone
(WARNING: WILL SUPRESS HPTA, PCT NECESSERY AFTER HEIGHTMAXXING PROTCOL ENDS)
(use only if you're ok with the risks, not compulsory)
Binds to androgen receptors (AR) on chondrocytes within long bones. Promotes chondrocyte proliferation, differentiation, and maturation. This causes hypertrophy in long bones causing growth. Testosterone works synergistically with HGH since it increases growth hormone receptor expression in chondrocytes, increasing localised igf1 production.
(1)
Usage:
Can differ between people but usually just above TRT dose such as 125 to 200mg. Pinning ED is important for stable testosterone blood levels.
Recommended regimen: 150mg testosterone cypionate pinned ED.


HGH (2)
HGH goes to your liver and makes it produce IGF-1. IGF1 directly promotes growth through chondrocyte proliferation and differentiation in the growth plates, leading to bone lengthening and height increase. However using IGF1 LR3 is suboptimal BECAUSE: chondrocytes have localised HGH receptors meaning when HGH binds to the IGF-1R receptors on chondrocytes it further stimulates local production of IGF1 directly promoting growth.
Recommended regimen: 5 (min) to 10 ius pinned ED before bed.


Anavar (3)
(WARNING: WILL SUPRESS HPTA, PCT NECESSERY AFTER HEIGHTMAXXING PROTCOL ENDS)
Increases protein synthesis, also in cartilage on long bones, contributing to elongation of bones.
Recommended regimen as used in study (3): 0.25 mg/kg/day e.g, if you're 65kg: 16.25mg ED
Anavar however is hepatoxic, to avoid this you can use anavar for 10 weeks then 10 weeks off. Counteracting hepatoxicity can be done with: (ran while on anavar)

  • Milk thistle
  • NAC
  • TUDCA

Aromatase inhibitors (AI):
Many AI's exist however studies on anastrozole prove it's effectiveness (4)
AI's are used since when testosterone is produced, it's converted into estrogen. Estrogen binds to receptors on growth plate chondrocytes such as ERα and ERβ. This promotes conversion of chondrocytes into osteoblasts. This change from cartilate to bone leads to the closure of growth plates. Furthermore, estrogen triggers apoptosis in growth plate cartilage cells. This contributes to the gradual disapearance of cartilage and the formation of bone, leading to growth plate fusion. For this reason AI's such as anastrozole are used since they reduce the conversion of testosterone to estrogen by reducing the amount of 5a reductase which is an enzyme that converts testosterone to estrogen.
Estrogen closes growth plates
(5)
Recommended regimen: 1mg ED, however blood tests can give a more precise dose as you want your estrogen to be between 10 to 15pg/mL.
Another AI that can be used is letrozole, studies prove IT'S effectiveness aswell.
(6)
Recommended regimen: 1mg ED


(1)
"Androgen itself also contributes to bone formation and the pubertal growth spurt, perhaps through a direct interaction with growth plate chondrocytes"

(2)
"HGH in studies is only used in prebutal kids!!"
"The age at start of treatment ranged between 4.7 years and 16 years, 11.9 ± 3.3 years for the 68 males and 12 ± 1.9 years for the 20 females."

(3)
"Oxandrolone therapy resulted in a two-fold increase in mean growth velocity in the first six months of therapy and was an effective growth stimulant for the full four-year period."

(4)
"Anastrozole increases adult height potential of adolescent boys on GH therapy while maintaining normal pubertal progression after 2-3 yr."

(5)
"Estrogen determines the acceleration of bone elongation at puberty, epiphyseal closure"

(6)
"The final heights for Lz (letrozole) group were significantly (p=0.04) higher than the control group."

(pls rep :heart: )
Holy thread
 

Disclaimer:


This thread is for informational purposes only.
The use of any medications, including growth hormones, aromatase inhibitors (like anastrozole or letrozole), or other drugs, for the purpose of increasing height should only be done under the supervision of a qualified healthcare professional.
Self-prescribing or using medications without medical guidance can lead to serious health risks, including but not limited to hormonal imbalances, bone health issues, and long-term complications.
Using these drugs for non-medical purposes, such as attempting to increase height in healthy individuals, can result in unintended consequences and potential legal implications.
If you or someone you know is considering any form of height-enhancing treatment, it is essential to consult with a licensed healthcare provider (such as an endocrinologist) to discuss the risks, benefits, and suitability of such treatments.
The information shared in this thread is not intended as medical advice. Always seek professional guidance before making decisions related to medications or treatments.


All these drugs are to be used throughout the whole heightmaxxing timeframe except anavar. Doses of all these drugs can vary. There is no one-size-fits-all approach, and the doses given here are just examples.
These drugs will provide an increase in adult height ONLY if your growth plates are open.

Do NOT use anavar if you cannot use testosterone. Anavar will cause suppression of testosterone, the TRT dose is there to replace the testosterone you will not have once you start using anavar. Either use all of the drugs listed or only use HGH + AI.


Testosterone
(WARNING: WILL SUPRESS HPTA, PCT NECESSERY AFTER HEIGHTMAXXING PROTCOL ENDS)
(use only if you're ok with the risks, not compulsory)
Binds to androgen receptors (AR) on chondrocytes within long bones. Promotes chondrocyte proliferation, differentiation, and maturation. This causes hypertrophy in long bones causing growth. Testosterone works synergistically with HGH since it increases growth hormone receptor expression in chondrocytes, increasing localised igf1 production.
(1)
Usage:
Can differ between people but usually just above TRT dose such as 125 to 200mg. Pinning ED is important for stable testosterone blood levels.
Recommended regimen: 150mg testosterone cypionate pinned ED.


HGH (2)
HGH goes to your liver and makes it produce IGF-1. IGF1 directly promotes growth through chondrocyte proliferation and differentiation in the growth plates, leading to bone lengthening and height increase. However using IGF1 LR3 is suboptimal BECAUSE: chondrocytes have localised HGH receptors meaning when HGH binds to the IGF-1R receptors on chondrocytes it further stimulates local production of IGF1 directly promoting growth.
Recommended regimen: 5 (min) to 10 ius pinned ED before bed.


Anavar (3)
(WARNING: WILL SUPRESS HPTA, PCT NECESSERY AFTER HEIGHTMAXXING PROTCOL ENDS)
Increases protein synthesis, also in cartilage on long bones, contributing to elongation of bones.
Recommended regimen as used in study (3): 0.25 mg/kg/day e.g, if you're 65kg: 16.25mg ED
Anavar however is hepatoxic, to avoid this you can use anavar for 10 weeks then 10 weeks off. Counteracting hepatoxicity can be done with: (ran while on anavar)

  • Milk thistle
  • NAC
  • TUDCA

Aromatase inhibitors (AI):
Many AI's exist however studies on anastrozole prove it's effectiveness (4)
AI's are used since when testosterone is produced, it's converted into estrogen. Estrogen binds to receptors on growth plate chondrocytes such as ERα and ERβ. This promotes conversion of chondrocytes into osteoblasts. This change from cartilate to bone leads to the closure of growth plates. Furthermore, estrogen triggers apoptosis in growth plate cartilage cells. This contributes to the gradual disapearance of cartilage and the formation of bone, leading to growth plate fusion. For this reason AI's such as anastrozole are used since they reduce the conversion of testosterone to estrogen by reducing the amount of 5a reductase which is an enzyme that converts testosterone to estrogen.
Estrogen closes growth plates
(5)
Recommended regimen: 1mg ED, however blood tests can give a more precise dose as you want your estrogen to be between 10 to 15pg/mL.
Another AI that can be used is letrozole, studies prove IT'S effectiveness aswell.
(6)
Recommended regimen: 1mg ED


(1)
"Androgen itself also contributes to bone formation and the pubertal growth spurt, perhaps through a direct interaction with growth plate chondrocytes"

(2)
"HGH in studies is only used in prebutal kids!!"
"The age at start of treatment ranged between 4.7 years and 16 years, 11.9 ± 3.3 years for the 68 males and 12 ± 1.9 years for the 20 females."

(3)
"Oxandrolone therapy resulted in a two-fold increase in mean growth velocity in the first six months of therapy and was an effective growth stimulant for the full four-year period."

(4)
"Anastrozole increases adult height potential of adolescent boys on GH therapy while maintaining normal pubertal progression after 2-3 yr."

(5)
"Estrogen determines the acceleration of bone elongation at puberty, epiphyseal closure"

(6)
"The final heights for Lz (letrozole) group were significantly (p=0.04) higher than the control group."

(pls rep :heart: )
hm but why would you take test daily? what about 500 mg weekly?
 
500 is too high to be sustained long term. daily injections ensures stable testosterone blood levels
No, Most testosterone esters have a half life of 1 week so thats already stable levels. I do agree there will be insane aromatization from high test but you can cope with aromasin, and 500mg should get you around 2k test, which is what Clav ran for a long time
 
thank you chatgpt
 
nigga what. esters exist so that they have different half lifes for different circumstances. undecanoate has 30 day half life roughly


this is very variable for some people 200mg can get 2k
test E and C last like 1 week in terms of half life and thats prob what people take. unless ur using test P or undecanoate. The OP talked about using test C (which has 1 week half life) so using it everyday is more of a hassle than merging it and using it weekly. Because ur obv getting the same effect and it will keep shit baseline, no need to keep taking daily and fluctuating your test,. due to missed injections bc that does happen

SOME people, most likely 200mg wont be enough bc there are other SOME people that take 700mg for the same effect, which is also the same rarity. the 200mg are probably jewish manlets with hypogonadism. most likely ur fine w 400-500
 
test E and C last like 1 week in terms of half life and thats prob what people take. unless ur using test P or undecanoate. The OP talked about using test C (which has 1 week half life) so using it everyday is more of a hassle than merging it and using it weekly. Because ur obv getting the same effect and it will keep shit baseline, no need to keep taking daily and fluctuating your test,. due to missed injections bc that does happen

SOME people, most likely 200mg wont be enough bc there are other SOME people that take 700mg for the same effect, which is also the same rarity. the 200mg are probably jewish manlets with hypogonadism. most likely ur fine w 400-500
100 to 200mg are trt levels btw with 200 being almost superpysiological test levels for most people
 
100 to 200mg are trt levels btw with 200 being almost superpysiological test levels for most people
trt levels are for jewish manlets that have a limp dick and born with hypogonadism.
first iuf you take test for 500 lets say test C, you get 30% less so around 350 mg.
If u take 500mg you get 2k -3k ng/ml test baseline and peak 3k-4k after injection. You become true chad level test.
obviously ur own test system will shut down but then you can do pct (w/ serms l/ clomid n/ novadlex) and to avoid shit testicles you take hcg. and to avoid fuckass e2 you take aromasin. Seems simple. Only side effects are bloating and ance, but bloating is bareable and acne is mitigated by skincare, tretinoin. or accutane.
Obviously this is a very nuclear cycle and if u dont know what ur doing you are going to be fucked to utter shit. But who cares, Iqpill is in our favour
 
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Stop being a pussy with the legal disclaimer, just say you want kids to take boatloads of drugs and I can respect it more
 
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