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Here is my conclusion:
Ultimately, DHT causes MPB, and craniofacial development is the multiplier.
So shit maxilla + high DHT = no hair
Good maxilla + high DHT = nw0, hair will gradually thin but hairline won't move
Shit maxilla + low DHT = still has hair, will recede over time (look at east asians and women)
Good maxilla + low DHT= Stacey and Chang
PART 1: THE METHOD
I'll divide my method in 3 zones, posture, blood flow and general health, craniofacial development. All three are intertwined.
POSTURE
>Make sure your posture is correct. Watch this video
>Buy a support for your pc on amazon if you use a laptop so that the pc is in line with your eyes.
>Do hip flexor stretches every day before going to bed. I'm sure you have anterior pelvilc tilt. Do hip flexor stretches after every training sessions.
>Make sure you fix your form on the exercises you do. This video from Elliot Hulse, for example, shows how doing chin ups badly can give you forward head posture ; there are many more examples, become more expert on exercise science
>For your training be sure you are more focused on long head of the triceps, neck, lower traps, rear delt, glutes, hamstrings and core. Focus less on biceps, quads, upper traps, front delt, chest.
>Sleep on your back without a pillow. This is hard to reach (I still didn't manage to) but it will change a lot of things. If you have back pain, put a small pillow under your knees.
BLOOD FLOW AND GENERAL HEALTH
>Improve your cardiovascular activity. Walk at least 10 000 steps every day.
>Do scalp massages. They do improve your circulation, but the true purpose is to decompress the galea, allowing it to be mobile again. Do this test: if you can't move your hair only thinking about it, you probably have a receiding hairline. Decompress the galea. Research very well how to do this. After your galea becomes mobile again, exercise it's movement everyday in your sparetime, to cause blood flow.
>Eat food containing aromatase inhibitors. Estrogen can cause hair loss in males, because estrogen is a stress hormone and stress can cause hair loss. In general have a good diet, consisting of non-processed food, meat and crude vegetables and fruits.
>Consider taking micronutrients integrators.
>Consider buying a water filter.
>Abstain from porn and masturbation.
>Abstain from activities that cause chronic stress you can avoid (internet usage, addictions, social medias, internet browsing, 4chan).
>Include more time with loved ones, if you can.
>Include a meditation routine into your life.
>Use a topic drug capable of inducing more blood flow to your scalp to avoid the situation becomes worse, like minoxidil.
CRANIOFACIAL DEVELOPMENT
This is the root cause. If you fix this, the others won't be necessary. But since it will take a while to fix this and you could not even be able to if you're too old or fucked up, you will still do the other two. The other two will actually accelerate the craniofacial process, making the improvement faster and better.
>Mewing. I'm listing this first because it's free. Learn to mew every 24/7 and how to swallow correctly.
>Surgery. If you have a deviated septum, get it fixed. It's free in most countries since it's not a cosmetic surgery, but a functional one. A deviated septum forces you to breath from your mouth since not enough air can enter your nose. This will help you with everything in your life, including posture, facial symmetry and forward maxillar development.
>Palate symmetry and expansion. Find a functional, safe appliance which can expand your palate both forward and laterally, while at the same time correcting the assymetry in your maxilla and jaw, which is the main cause of all these problems. There are many, and I'm still researching them. For now, the A.L.F. (Advanced Lightwire Functional appliance) looks the best. It will fix your face and your balding if it's not too late.
-----------------------------------------------------------------------------------------------------------------------------------------------------------------------------------
Blood vessels travel throught the scalp tissues fueling nutrients to the hair follicles. The galea aponeurotica is attached to the occipitofrontalis muscles. The temporalis muscles are also connected to the galea aponeurotica via the temporalis fascia. The galea aponeurotica can be stretched by the forces of muscular contraction.
Since muscles throughout the body are connected via myofascial meridians, their action cannot be seen in isolation. This explains why intensity of neck pain, forward head posture, chronic tension-type headache and migraine are strictly correlated.
When neck muscles are in continuous tension, their action propagates to the head, stretching and tightening the galea against the underlying layers of the scalp. The underlying structure is rich of blood vessels that are compressed, blocking blood flow towards the hair follicles. The restriction in blood supply to tissues is called ischemia: this leads to insufficiency of oxygen (hypoxia), reduced availability of nutrients and inadequate removal of metabolites. This obviously leads to the death of tissues, thus including the hair follicles (hair loss) and surrounding structures. This is also reflected in the presence of dandruff (excessive shedding of dead cells from the scalp).
When tissues are damaged, an inflammatory response is activated. The function of inflammation is to clear out necrotic cells and damaged tissues. The classical signs of inflammation are heat, pain and redness. These elements describe symptoms of scalp sensitivity and trichodynia.
Since the muscle tension that tight the galea is always present, the inflammation is long-term and chronic, causing fibrosis and calcification. This further decreases the blood flow into the scalp, promoting ulterior cells death, leading to a closed-loop chain of events depicted in the diagram I posted, reason why hair loss progresses with individuals becoming older.
Obviously a bad diet, lack of physical exercise and rise in estrogen levels brought an higher level of inflamation. But the main cause here is the tightening of those muscles. Before I explain why those muscles became tight in the first place, I'll first debunk the genetics/dht cope.
Current theories see the dihydrotestosterone (DHT) hormone as the main responsible for hair loss. As the name suggests, there are two supposed causes for androgenetic alopecia: the action of hormones (androgens) and action derived from genetic.
The theory started to arise after observations made on Eunuchs (i.e. men who have been castrated). Already in 400 BCE, Hippocrates noticed that Eunuchs do not become bald, with formal conclusions made by James B. Hamilton between 40s and 60s: eunuchoid and prepubertally castrated men fails to achieve sexual maturation and show complete retention of head hair. Baldness can then be induced by following administration of male hormone substances to sexually immature men who otherwise would not have become bald. In 1974, a study has generally observed a higher formation of 5α-reduced metabolites and 17-ketosteroid metabolites at all sites of the scalp of bald men as compared to hair obtained from the corresponding sites of women and nonbalding men. So, from here it has been assumed the importance of DHT in baldness.
However, many arguments are against the role of DHT as primary cause in hair loss:
>DHT stimulates the production of pigmented terminal hair in many areas after puberty, including pubic and axillary hair in both sexes and beard growth in men. But both beard growth and balding can occur on the same person demonstrating a paradox
>Serum androgen concentrations in men with a disposition to balding is lower than in men with no reduction of scalp hair.
>The prevalence of Male Pattern Baldness increases with age. However, serum testosterone production declines with aging, together with lower serum DHT.
>Female androgenetic alopecia can develop in the absence of detectable levels of circulating androgens or other signs of postpubertal androgenization, showing that this pattern of hair loss is not necessarily androgen dependent. Not only, female patients with androgen deficiency reports hair regrowth on testosterone therapy.
We can see that Male Pattern Baldness only affects hair on top of the head, while hair on sides and back does not fall. So, how can DHT cause hair in one area of the scalp to fall out but have no effect on hair follicles just a few millimeters further down?
Here is where the genetic part comes into action. According to the current theory, hair follicles on top of the head show increased expression of the androgen receptor gene, i.e. the top of the head is more sensitive to androgens action than hair on the sides and back of the head. However, higher levels of androgen receptors are also found in follicles of beard, scrotum and pubis, reinforcing the paradox. Genetics also suggests that the probability of male pattern hair loss is dependent on family history and age. However, in case of identical male twins, one can bald significantly faster than his counterpart. These together with many other unknowns bring the necessity to define a new model for the explanation of baldness.
Not surprisingly, it has been found that subcutaneous blood flow in the scalp of patients with early male pattern baldness is much lower than the values found in the normal individuals . This would explain the aforementioned higher formation of 5α-reduced metabolites and 17-ketosteroid metabolites at all sites of the scalp of bald men at the basis of the androgenetic theory.
The restriction in blood supply to tissues is called ischemia: this leads to insufficiency of oxygen (hypoxia). Not surprisingly again, men suffering from androgenic alopecia have significantly lower oxygen partial pressure (meaning microvascular insufficiency and hypoxia) in the areas of their scalp affected by balding (frontal and vertex regions) versus unaffected areas (temporal and occipital regions). Moreover, balding men have significantly lower oxygen partial pressure in the areas of balding scalp than the same areas of non-bald people.
Minoxidil solution, which is one of the commercially available medications for hair loss, stimulates the microcirculation of the bald scalp, effectively promoting hair growth . Patients should be warned about increased shedding in the first months of treatment, that is seen as a positive response to the treatment. How can this increased shedding be explained? Could it be the so called reperfusion injury, i.e. the tissue damage caused when blood supply returns to the tissue after a period of ischemia?
In case of reduced blood supply, tissues are damaged and die, thus including the hair follicles and surrounding structures. A proof of this is the associated excessive dandruff found in hair loss: the last layer of the skin, called stratum corneum, consists of dead cells (corneocytes) that are regularly replaced through desquamation. However, certain conditions trigger a more rapid turnover, leading to a larger shedding recognized as dandruff.
When tissues are damaged, an inflammatory response is activated. The function of inflammation is to clear out necrotic cells and damaged tissues. The classical signs of inflammation are heat, pain and redness, situation recognized as scalp sensitivity (burning, stinging, dry scalp, vague discomfort in the scalp, and sometimes, trichodynia) that is associated with hair loss. And guess what? Many studies have shown DHT plays an important role in inflammations, regulating its activity
When the inflammation is long-term and chronic, it causes fibrosis and calcification. Indeed, miniaturization of hair follicles has been associated with deposits of densely packed collagen bundles in the follicular dermal sheath, called perifollicular fibrosis. This further decreases the blood flow into the scalp, promoting ulterior cells death.
Conclusion: hair loss is not a problem of the hair itself but was a problem of the surrounding tissues. Take two seeds of a genetically identical corn, place one seed in a temperate climate and one in an arid climate: the one in the arid climate only grows to half the height of the one in the temperate climate due to lack of water and nutrients in its environment. This is exactly what happens with our hair and there are studies confirming it: miniaturized hair follicles of pattern alopecia can quickly regenerate once removed from the human scalp and can grow as well as or better than terminal follicles from the same individual. And not only miniaturized hair can regrowth, but also terminal hairs can shrink in difficult recipient sites, impacting their growth and survival rates.
The cause of bad head posture, other than bad habits, is bad craniofacial development, caused by various things, such as biberons, mouthbreathing, early hard feeding, allergies (caused by the modern world).
This is a topic too long to explain now, and I reccomend the blog I took the info from (www.tmdocclusion.com).
There's not a single bald person with good craniofacial development. As you can see here, both Statham and the Guy in the OP pic here have a very tight neck (look at the folds on the neck. This is how you determine if someone has a tight neck or not) and most importantly a totally assymetric and badly developed face.
Already in 1939 Weston A. Price wrote about his travel around the world where he discovered that jaw problems were not present in tribes of isolated areas far from civilization. In a simplistic view, maxilla is primarily remodeled by forces coming from the tongue and teeth (through masseter muscles) and the causes of a poor craniofacial development have to be found in modern lifestyle, in particular in:
>Increasing of bottle-feeding over breast-feeding that influences the swallowing pattern and all oral muscles.
>Use of pacifiers/dummies that mainly affect tongue posture.
>Trend towards industrial soft high-calorie food that hits chewing effort and muscles action.
>Reduced nasal airways, increasing of allergies and nasal congestion that leads to mouth breathing affecting both tongue posture and muscles action.
>Trend towards an earlier or too early weaning, influencing swallowing pattern.
When one or more of these factors are present, the maxilla drops down and back, reducing the eye support, flattening the cheekbones, narrowing the nasal airway, lengthening the mid facial third, and lowering the palate, which narrows and create malocclusion.
SOURCES :
>Godse, Kiran, and Vijay Zawar. “Sensitive scalp.” International journal of trichology 4.2 (2012): 102.
>Misery, Laurent, et al. “Sensitive scalp: does this condition exist? An epidemiological study.” Contact Dermatitis 58.4 (2008): 234-238.
>Willimann, Barbara, and Ralph M. Trüeb. “Hair pain (trichodynia): frequency and relationship to hair loss and patient gender.” Dermatology 205.4 (2002): 374-377.
>ALEXANBER, SUZANNE. “Loss of hair and dandruff.” British Journal of Dermatology 79.10 (1967): 549-552.
>Rossi, Alfredo, et al. “Minoxidil use in dermatology, side effects and recent patents.” Recent patents on inflammation & allergy drug discovery 6.2 (2012): 130-136.
>Avci, Pinar, et al. “Low‐level laser (light) therapy (LLLT) for treatment of hair loss.” Lasers in surgery and medicine 46.2 (2014): 144-151.
>Choy, H. “Detumescence Therapy of Human Scalp for Natural Hair Regrowth.” J Clin Exp Dermatol Res 3.138 (2012): 2.
>Mew, M. “Craniofacial dystrophy. A possible syndrome?.” British dental journal 216.10 (2014): 555-558.
>Gonzalez, Humberto E., and Arturo Manns. “Forward head posture: its structural and functional influence on the stomatognathic system, a conceptual study.” CRANIO® 14.1 (1996): 71-80.
>Solow, Beni, and Antje Tallgren. “Head posture and craniofacial morphology.” American Journal of Physical Anthropology 44.3 (1976): 417-435.
>Strokon, Dennis. “Correction of Dental and Cranial Sidebend with ALF.” IJOM 21 (2010): 3.
>Darnell, Michael W. “A proposed chronology of events for forward head posture.” Journal of craniomandibular practice1.4 (1983): 49-54.
>Ernest III, Edwin A., and W. Ernest. “Splenius Capitis.”
>Simons, David G., Janet G. Travell, and Lois S. Simons. Travell & Simons’ myofascial pain and dysfunction: upper half of body. Vol. 1. Lippincott Williams & Wilkins, 1999.
>Myers, Thomas W. “The ‘anatomy trains’.” Journal of Bodywork and Movement Therapies 1.2 (1997): 91-101.
>Haughie, Laura J., Ira M. Fiebert, and Kathryn E. Roach. “Relationship of forward head posture and cervical backward bending to neck pain.” Journal of Manual & Manipulative Therapy 3.3 (1995): 91-97.
>Fernández‐de‐las‐Peñas, César, et al. “Trigger Points in the Suboccipital Muscles and Forward Head Posture in Tension‐Type Headache.” Headache: The Journal of Head and Face Pain 46.3 (2006): 454-460.
>Fernández-de-las-Penas, César, et al. “Referred pain areas of active myofascial trigger points in head, neck, and shoulder muscles, in chronic tension type headache.” Journal of bodywork and movement therapies 14.4 (2010): 391-396.
>Fernández‐de‐las‐Peñas, César, Maria L. Cuadrado, and Juan A. Pareja. “Myofascial trigger points, neck mobility, and forward head posture in episodic tension‐type headache.” Headache: The Journal of Head and Face Pain 47.5 (2007): 662-672.
>Freund, Brian J., and Marvin Schwartz. “Treatment of male pattern baldness with botulinum toxin: a pilot study.” Plastic and reconstructive surgery 126.5 (2010): 246e-248e.
>Klemp, Per, Kurt Peters, and Birgitte Hansted. “Subcutaneous blood flow in early male pattern baldness.” Journal of investigative dermatology 92.5 (1989): 725-726.
>Goldman, Boris E., David M. Fisher, and Steven L. Ringler. “Transcutaneous PO2 of the scalp in male pattern baldness: a new piece to the puzzle.” Plastic and reconstructive surgery97.6 (1996): 1109-16.
>Wester, Ronald C., et al. “Minoxidil stimulates cutaneous blood flow in human balding scalps: pharmacodynamics measured by laser Doppler velocimetry and photopulse plethysmography.” Journal of investigative dermatology 82.5 (1984): 515-517.
>Toshitani, Shoji, et al. “A New Apparatus for Hair Regrowth in Male‐pattern Baldness.” The Journal of dermatology 17.4 (1990): 240-246.
>Whiting, David A. “Diagnostic and predictive value of horizontal sections of scalp biopsy specimens in male pattern androgenetic alopecia.” Journal of the American Academy of Dermatology 28.5 (1993): 755-763.
>Whiting, David A. “Chronic telogen effluvium: increased scalp hair shedding in middle-aged women.” Journal of the American Academy of Dermatology 35.6 (1996): 899-906.
>Hoelzel, Frederick. “Baldness and Calcification of the Ivory Dome.” Journal of the American Medical Association 119.12 (1942): 968-968.
TLDR:
So shit maxilla + high DHT = no hair
Good maxilla + high DHT = nw0, hair will gradually thin but hairline won't move
Shit maxilla + low DHT = still has hair, will recede over time (look at east asians and women)
Good maxilla + low DHT= Stacey and Chang
Ultimately, DHT causes MPB, and craniofacial development is the multiplier.
So shit maxilla + high DHT = no hair
Good maxilla + high DHT = nw0, hair will gradually thin but hairline won't move
Shit maxilla + low DHT = still has hair, will recede over time (look at east asians and women)
Good maxilla + low DHT= Stacey and Chang
A Different Take on Curing MBP
PART 1: THE METHOD
I'll divide my method in 3 zones, posture, blood flow and general health, craniofacial development. All three are intertwined.
POSTURE
>Make sure your posture is correct. Watch this video
>Buy a support for your pc on amazon if you use a laptop so that the pc is in line with your eyes.
>Do hip flexor stretches every day before going to bed. I'm sure you have anterior pelvilc tilt. Do hip flexor stretches after every training sessions.
>Make sure you fix your form on the exercises you do. This video from Elliot Hulse, for example, shows how doing chin ups badly can give you forward head posture ; there are many more examples, become more expert on exercise science
>For your training be sure you are more focused on long head of the triceps, neck, lower traps, rear delt, glutes, hamstrings and core. Focus less on biceps, quads, upper traps, front delt, chest.
>Sleep on your back without a pillow. This is hard to reach (I still didn't manage to) but it will change a lot of things. If you have back pain, put a small pillow under your knees.
BLOOD FLOW AND GENERAL HEALTH
>Improve your cardiovascular activity. Walk at least 10 000 steps every day.
>Do scalp massages. They do improve your circulation, but the true purpose is to decompress the galea, allowing it to be mobile again. Do this test: if you can't move your hair only thinking about it, you probably have a receiding hairline. Decompress the galea. Research very well how to do this. After your galea becomes mobile again, exercise it's movement everyday in your sparetime, to cause blood flow.
>Eat food containing aromatase inhibitors. Estrogen can cause hair loss in males, because estrogen is a stress hormone and stress can cause hair loss. In general have a good diet, consisting of non-processed food, meat and crude vegetables and fruits.
>Consider taking micronutrients integrators.
>Consider buying a water filter.
>Abstain from porn and masturbation.
>Abstain from activities that cause chronic stress you can avoid (internet usage, addictions, social medias, internet browsing, 4chan).
>Include more time with loved ones, if you can.
>Include a meditation routine into your life.
>Use a topic drug capable of inducing more blood flow to your scalp to avoid the situation becomes worse, like minoxidil.
CRANIOFACIAL DEVELOPMENT
This is the root cause. If you fix this, the others won't be necessary. But since it will take a while to fix this and you could not even be able to if you're too old or fucked up, you will still do the other two. The other two will actually accelerate the craniofacial process, making the improvement faster and better.
>Mewing. I'm listing this first because it's free. Learn to mew every 24/7 and how to swallow correctly.
>Surgery. If you have a deviated septum, get it fixed. It's free in most countries since it's not a cosmetic surgery, but a functional one. A deviated septum forces you to breath from your mouth since not enough air can enter your nose. This will help you with everything in your life, including posture, facial symmetry and forward maxillar development.
>Palate symmetry and expansion. Find a functional, safe appliance which can expand your palate both forward and laterally, while at the same time correcting the assymetry in your maxilla and jaw, which is the main cause of all these problems. There are many, and I'm still researching them. For now, the A.L.F. (Advanced Lightwire Functional appliance) looks the best. It will fix your face and your balding if it's not too late.
-----------------------------------------------------------------------------------------------------------------------------------------------------------------------------------
Blood vessels travel throught the scalp tissues fueling nutrients to the hair follicles. The galea aponeurotica is attached to the occipitofrontalis muscles. The temporalis muscles are also connected to the galea aponeurotica via the temporalis fascia. The galea aponeurotica can be stretched by the forces of muscular contraction.
Since muscles throughout the body are connected via myofascial meridians, their action cannot be seen in isolation. This explains why intensity of neck pain, forward head posture, chronic tension-type headache and migraine are strictly correlated.
When neck muscles are in continuous tension, their action propagates to the head, stretching and tightening the galea against the underlying layers of the scalp. The underlying structure is rich of blood vessels that are compressed, blocking blood flow towards the hair follicles. The restriction in blood supply to tissues is called ischemia: this leads to insufficiency of oxygen (hypoxia), reduced availability of nutrients and inadequate removal of metabolites. This obviously leads to the death of tissues, thus including the hair follicles (hair loss) and surrounding structures. This is also reflected in the presence of dandruff (excessive shedding of dead cells from the scalp).
When tissues are damaged, an inflammatory response is activated. The function of inflammation is to clear out necrotic cells and damaged tissues. The classical signs of inflammation are heat, pain and redness. These elements describe symptoms of scalp sensitivity and trichodynia.
Since the muscle tension that tight the galea is always present, the inflammation is long-term and chronic, causing fibrosis and calcification. This further decreases the blood flow into the scalp, promoting ulterior cells death, leading to a closed-loop chain of events depicted in the diagram I posted, reason why hair loss progresses with individuals becoming older.
Obviously a bad diet, lack of physical exercise and rise in estrogen levels brought an higher level of inflamation. But the main cause here is the tightening of those muscles. Before I explain why those muscles became tight in the first place, I'll first debunk the genetics/dht cope.
Current theories see the dihydrotestosterone (DHT) hormone as the main responsible for hair loss. As the name suggests, there are two supposed causes for androgenetic alopecia: the action of hormones (androgens) and action derived from genetic.
The theory started to arise after observations made on Eunuchs (i.e. men who have been castrated). Already in 400 BCE, Hippocrates noticed that Eunuchs do not become bald, with formal conclusions made by James B. Hamilton between 40s and 60s: eunuchoid and prepubertally castrated men fails to achieve sexual maturation and show complete retention of head hair. Baldness can then be induced by following administration of male hormone substances to sexually immature men who otherwise would not have become bald. In 1974, a study has generally observed a higher formation of 5α-reduced metabolites and 17-ketosteroid metabolites at all sites of the scalp of bald men as compared to hair obtained from the corresponding sites of women and nonbalding men. So, from here it has been assumed the importance of DHT in baldness.
However, many arguments are against the role of DHT as primary cause in hair loss:
>DHT stimulates the production of pigmented terminal hair in many areas after puberty, including pubic and axillary hair in both sexes and beard growth in men. But both beard growth and balding can occur on the same person demonstrating a paradox
>Serum androgen concentrations in men with a disposition to balding is lower than in men with no reduction of scalp hair.
>The prevalence of Male Pattern Baldness increases with age. However, serum testosterone production declines with aging, together with lower serum DHT.
>Female androgenetic alopecia can develop in the absence of detectable levels of circulating androgens or other signs of postpubertal androgenization, showing that this pattern of hair loss is not necessarily androgen dependent. Not only, female patients with androgen deficiency reports hair regrowth on testosterone therapy.
We can see that Male Pattern Baldness only affects hair on top of the head, while hair on sides and back does not fall. So, how can DHT cause hair in one area of the scalp to fall out but have no effect on hair follicles just a few millimeters further down?
Here is where the genetic part comes into action. According to the current theory, hair follicles on top of the head show increased expression of the androgen receptor gene, i.e. the top of the head is more sensitive to androgens action than hair on the sides and back of the head. However, higher levels of androgen receptors are also found in follicles of beard, scrotum and pubis, reinforcing the paradox. Genetics also suggests that the probability of male pattern hair loss is dependent on family history and age. However, in case of identical male twins, one can bald significantly faster than his counterpart. These together with many other unknowns bring the necessity to define a new model for the explanation of baldness.
Not surprisingly, it has been found that subcutaneous blood flow in the scalp of patients with early male pattern baldness is much lower than the values found in the normal individuals . This would explain the aforementioned higher formation of 5α-reduced metabolites and 17-ketosteroid metabolites at all sites of the scalp of bald men at the basis of the androgenetic theory.
The restriction in blood supply to tissues is called ischemia: this leads to insufficiency of oxygen (hypoxia). Not surprisingly again, men suffering from androgenic alopecia have significantly lower oxygen partial pressure (meaning microvascular insufficiency and hypoxia) in the areas of their scalp affected by balding (frontal and vertex regions) versus unaffected areas (temporal and occipital regions). Moreover, balding men have significantly lower oxygen partial pressure in the areas of balding scalp than the same areas of non-bald people.
Minoxidil solution, which is one of the commercially available medications for hair loss, stimulates the microcirculation of the bald scalp, effectively promoting hair growth . Patients should be warned about increased shedding in the first months of treatment, that is seen as a positive response to the treatment. How can this increased shedding be explained? Could it be the so called reperfusion injury, i.e. the tissue damage caused when blood supply returns to the tissue after a period of ischemia?
In case of reduced blood supply, tissues are damaged and die, thus including the hair follicles and surrounding structures. A proof of this is the associated excessive dandruff found in hair loss: the last layer of the skin, called stratum corneum, consists of dead cells (corneocytes) that are regularly replaced through desquamation. However, certain conditions trigger a more rapid turnover, leading to a larger shedding recognized as dandruff.
When tissues are damaged, an inflammatory response is activated. The function of inflammation is to clear out necrotic cells and damaged tissues. The classical signs of inflammation are heat, pain and redness, situation recognized as scalp sensitivity (burning, stinging, dry scalp, vague discomfort in the scalp, and sometimes, trichodynia) that is associated with hair loss. And guess what? Many studies have shown DHT plays an important role in inflammations, regulating its activity
When the inflammation is long-term and chronic, it causes fibrosis and calcification. Indeed, miniaturization of hair follicles has been associated with deposits of densely packed collagen bundles in the follicular dermal sheath, called perifollicular fibrosis. This further decreases the blood flow into the scalp, promoting ulterior cells death.
Conclusion: hair loss is not a problem of the hair itself but was a problem of the surrounding tissues. Take two seeds of a genetically identical corn, place one seed in a temperate climate and one in an arid climate: the one in the arid climate only grows to half the height of the one in the temperate climate due to lack of water and nutrients in its environment. This is exactly what happens with our hair and there are studies confirming it: miniaturized hair follicles of pattern alopecia can quickly regenerate once removed from the human scalp and can grow as well as or better than terminal follicles from the same individual. And not only miniaturized hair can regrowth, but also terminal hairs can shrink in difficult recipient sites, impacting their growth and survival rates.
The cause of bad head posture, other than bad habits, is bad craniofacial development, caused by various things, such as biberons, mouthbreathing, early hard feeding, allergies (caused by the modern world).
This is a topic too long to explain now, and I reccomend the blog I took the info from (www.tmdocclusion.com).
There's not a single bald person with good craniofacial development. As you can see here, both Statham and the Guy in the OP pic here have a very tight neck (look at the folds on the neck. This is how you determine if someone has a tight neck or not) and most importantly a totally assymetric and badly developed face.
Already in 1939 Weston A. Price wrote about his travel around the world where he discovered that jaw problems were not present in tribes of isolated areas far from civilization. In a simplistic view, maxilla is primarily remodeled by forces coming from the tongue and teeth (through masseter muscles) and the causes of a poor craniofacial development have to be found in modern lifestyle, in particular in:
>Increasing of bottle-feeding over breast-feeding that influences the swallowing pattern and all oral muscles.
>Use of pacifiers/dummies that mainly affect tongue posture.
>Trend towards industrial soft high-calorie food that hits chewing effort and muscles action.
>Reduced nasal airways, increasing of allergies and nasal congestion that leads to mouth breathing affecting both tongue posture and muscles action.
>Trend towards an earlier or too early weaning, influencing swallowing pattern.
When one or more of these factors are present, the maxilla drops down and back, reducing the eye support, flattening the cheekbones, narrowing the nasal airway, lengthening the mid facial third, and lowering the palate, which narrows and create malocclusion.
SOURCES :
>Godse, Kiran, and Vijay Zawar. “Sensitive scalp.” International journal of trichology 4.2 (2012): 102.
>Misery, Laurent, et al. “Sensitive scalp: does this condition exist? An epidemiological study.” Contact Dermatitis 58.4 (2008): 234-238.
>Willimann, Barbara, and Ralph M. Trüeb. “Hair pain (trichodynia): frequency and relationship to hair loss and patient gender.” Dermatology 205.4 (2002): 374-377.
>ALEXANBER, SUZANNE. “Loss of hair and dandruff.” British Journal of Dermatology 79.10 (1967): 549-552.
>Rossi, Alfredo, et al. “Minoxidil use in dermatology, side effects and recent patents.” Recent patents on inflammation & allergy drug discovery 6.2 (2012): 130-136.
>Avci, Pinar, et al. “Low‐level laser (light) therapy (LLLT) for treatment of hair loss.” Lasers in surgery and medicine 46.2 (2014): 144-151.
>Choy, H. “Detumescence Therapy of Human Scalp for Natural Hair Regrowth.” J Clin Exp Dermatol Res 3.138 (2012): 2.
>Mew, M. “Craniofacial dystrophy. A possible syndrome?.” British dental journal 216.10 (2014): 555-558.
>Gonzalez, Humberto E., and Arturo Manns. “Forward head posture: its structural and functional influence on the stomatognathic system, a conceptual study.” CRANIO® 14.1 (1996): 71-80.
>Solow, Beni, and Antje Tallgren. “Head posture and craniofacial morphology.” American Journal of Physical Anthropology 44.3 (1976): 417-435.
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TLDR:
So shit maxilla + high DHT = no hair
Good maxilla + high DHT = nw0, hair will gradually thin but hairline won't move
Shit maxilla + low DHT = still has hair, will recede over time (look at east asians and women)
Good maxilla + low DHT= Stacey and Chang
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