hungry asf after first dose of reta

[ahhahahaha]

holy shit i never been so hungry in ages i took my first dose of reta yesterday .5mg is ts normal

 

[BlackFag]

bump

 

[BlackFag]

someone answer this nigga, taking reta in a few weeks and need an answer

 

[saff]

more than likely your body is adjusting to the new baseline blood glucose by releasing cortisol, should pass

 

[2s2f]

bump

yo terry

 

[Sub5kang]

more than likely your body is adjusting to the new baseline blood glucose by releasing cortisol, should pass

Or it could just be a low dose, I felt nothing for the first week on 1mg and I stupidly went to 2.5 and side effects hit in a couple days

 

[Sub5kang]

I started on 1 and didn't feel anything you need to increase dose incrementally weekly till you feel it

 

[aids]

Because the starting dose in clinical trials was 2mg and you obviously have no idea about a protocol to follow.
Disregard every retard above.

 

[grav]

yo terry

Yo chico

 

[saff]

Or it could just be a low dose, I felt nothing for the first week on 1mg and I stupidly went to 2.5 and side effects hit in a couple days

yeah anything under 2mg probably wont suppress appetite much, I think for the average non-obese person 3mg is the sweet spot for controlled fat loss mostly via insulin sensitivity and craving inhibition.

 

[saff]

Because the starting dose in clinical trials was 2mg and you obviously have no idea about a protocol to follow.
Disregard every retard above.

2mg was the starting dose because Eli Lilly used significantly overweight participants in trial groups, its an obesity medication. Obviously it will be dosed differently for those that are not obese.

 

[Trail4]

Start at 1mg minimum bro at 0.5 you wont get shit

 

[aids]

2mg was the starting dose because Eli Lilly used significantly overweight participants in trial groups, its an obesity medication. Obviously it will be dosed differently for those that are not obese.

Do you know why they used obese participants? Because the medication is not going to be prescribed to individuals that want to be hyper-lean. Thanks for the obvious statement.
Being of a higher bodyfat percentage does not necessarily mean you need a higher dose of Retatrutide.
Speak to me when you actually know what you're talking about.

 

[saff]

Do you know why they used obese participants? Because the medication is not going to be prescribed to individuals that want to be hyper-lean. Thanks for the obvious statement.
Being of a higher bodyfat percentage does not necessarily mean you need a higher dose of Retatrutide.
Speak to me when you actually know what you're talking about.

higher body fat percentage causes the drug to be utilized in the blood stream less efficiently due to insulin resistance

 

[aids]

higher body fat percentage causes the drug to be utilized in the blood stream less efficiently due to insulin resistance

I'm not even going to debate with a moron like you if that's your justification.

 

[saff]

I'm not even going to debate with a moron like you if that's your justification.

no counter-argument; retard detected

 

[aids]

higher body fat percentage causes the drug to be utilized in the blood stream less efficiently due to insulin resistance

I'm not even going to debate with a moron like you if that's your justification.

Actually, I will just briefly, so you can't claim that I don't know what I'm talking about and try to feign intellectualism over me.

Any GLP-1/GIP/Glucagon receptor agonism will help with insulin sensitivity (hence why GLP-1/GLP-1 & GIP agonists are used for weight loss i.e., Wegovy or T2D i.e., Ozempic, Mounjaro). That does not mean the drug is less efficacious, that simply implies that the increase in insulin sensitivity that a GLP-1 agonist might provide will not necessarily put somebody in what is considered a 'normal level' of insulin sensitivity.
The appetite suppression is the exact same.

no counter-argument; retard detected

There's your answer buddy. Predicted your bullshit because you're a low IQ gremlin.

 

[saff]

Actually, I will just briefly, so you can't claim that I don't know what I'm talking about and try to feign intellectualism over me.

Any GLP-1/GIP/Glucagon receptor agonism will help with insulin sensitivity (hence why GLP-1 agonists are used for weight loss i.e., Wegovy or T2D i.e., Ozempic, Mounjaro). That does not mean the drug is less efficacious, that simply implies that the increase in insulin sensitivity that a GLP-1 agonist might provide will not necessarily put somebody in what is considered a 'normal level' of insulin sensitivity.
The appetite suppression is the exact same.


There's your answer buddy. Predicted your bullshit because you're a low IQ gremlin.

and the obese experimental group in trials are insulin-resistant, hence why they require a more significant dose to obtain said normal level of insulin sensitivity. you haven't disproven a single statement from me.

 

[aids]

and the obese experimental group in trials are insulin-resistant, hence why they require a more significant dose to obtain said normal level of insulin sensitivity. you haven't disproven a single statement from me.

A higher dose does not put them into a normal level of insulin sensitivity.
You act like GLP-1/double/triple receptor agonists are perfectly compensatory and put the body back into homeostasis.

No, they do not, they solely enable individuals to lose weight while helping to bring the body closer to an ideal level of insulin sensitivity until what can be considered a healthy weight is reached where it is implicit that said individual will be of that ideal insulin sensitivity assuming they are not T1D.
That does not mean the drug is utilised less efficiently. It just means that the increase in insulin sensitivity does not necessarily bring the individual to ideal levels, as aforementioned.
The appetite suppression is comparable for an individual that is overweight or underweight because Retatrutide is not lipophilic.
Do fat people have significantly larger bloodstreams to the extent where it is relevant, if that even mattered? No, stupid cunt.

In before you argue semantics with me because you have nothing else to say other than "yeah but it at least compensates somewhat".
Now I won't debate with you any further.

 

[saff]

A higher dose does not put them into a normal level of insulin sensitivity.
You act like GLP-1/double/triple receptor agonists are perfectly compensatory and put the body back into homeostasis.

No, they do not, they solely enable individuals to lose weight while helping to bring the body closer to an ideal level of insulin sensitivity until what can be considered a healthy weight is reached where it is implicit that said individual will be of that ideal insulin sensitivity assuming they are not T1D.
That does not mean the drug is utilised less efficiently. It just means that the increase in insulin sensitivity does not necessarily bring the individual to ideal levels, as aforementioned.
The appetite suppression is comparable for an individual that is overweight or underweight because Retatrutide is not lipophilic.
Do fat people have significantly larger bloodstreams to the extent where it is relevant, if that even mattered? No, stupid cunt.

In before you argue semantics with me because you have nothing else to say other than "yeah but it at least compensates somewhat".
Now I won't debate with you any further.

If a patient who has developed a resistance to insulin due to extreme fat accumulation Inquires for glp-1 type medication their bmi is taken into consideration when developing the dose protocol. Just like a t2d would require more insulin to level out to an acceptable measurement of blood glucose, the same general rule applies with glp or glucagon receptor agonists. A higher baseline hba1c would call for a more aggressive treatment for any glucose metabolization related compound. Yes, the appetite suppression and gastric emptying effects will be similar to those in use without the medical necessity, however the dose used in protocols for leaning down an already somewhat slim physique is not the ideal dose for those who aquire the peptide as a prescription, therefore making the dosages of said compounds context-dependant.

 

[aids]

If a patient who has developed a resistance to insulin due to extreme fat accumulation Inquires for glp-1 type medication their bmi is taken into consideration when developing the dose protocol. Just like a t2d would require more insulin to level out to an acceptable measurement of blood glucose, the same general rule applies with glp or glucagon receptor agonists. A higher baseline hba1c would call for a more aggressive treatment for any glucose metabolization related compound. Yes, the appetite suppression and gastric emptying effects will be similar to those in use without the medical necessity, however the dose used in protocols for leaning down an already somewhat slim physique is not the ideal dose for those who aquire the peptide as a prescription, therefore making the dosages of said compounds context-dependant.

Brutal ChatGPT reliance pill for your argument.
Nobody uses that verbiage.

 

[saff]

Brutal ChatGPT reliance pill for your argument.
Nobody uses that verbiage.

I write sports med articles and study endocrinology, cope

 

[dachi101]

do at least 5mg reta weekly