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polarpop

polarpop

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how to lose fat safely and quickly as a teen in puberty without stunting growth a guide by polarpop

Yo wsg everyone, I was once in this exact position. I obviously wanted to grow taller and hit my absolute genetic limit, but I was carrying extra weight, feeling insecure about my looks, and honestly just didn't know wtf to do.
It feels like a massive trap. If you cut calories, all the mainstream advice tells you that you will permanently stunt your growth. But if you just keep eating like a slob hoping the height will stretch you out, you just stay chubby and miserable. I spent a massive amount of time digging into the actual clinical endocrinology and pediatric growth science because I wanted a real answer, not just fake bro science.
It turns out, staying chubby is actually way worse for your height, and you can absolutely shred down safely if you hack your biology the right way. Here is the deeply researched, A to Z guide on how to get lean, protect your gains, and actually delay your growth plates from fusing so you can maximize your height.

What Actually Stunts Growth: what actual stunting is and how long it actually takes first off we need to clear up wtf actual stunting is cause bros think missing one meal or running a cut for a few weeks permanently thinks them to actually permanently stunt your genetic height. you have to be in a state of severe chronic starvation for months or even year sim talking actual third world famine levels like a starving kid in gaza or severe clinical anorexia where you are literally skin and bones. clinically this is called severe protein energy malnutrition or pem what happens is your body completely runs out of adipose fat tissue to burn so it enters a permanent biological survival panic it starts cannibalizing your muscle tissue and your internal organs just to keep your heart beating and your brain working when you are in this state of literal starvation for months on end your chronic cortisol levels stay so astronomically high that they permanently damage the growth plates the osteoblasts which are the cells that build bone matrix literally shut down and the cartilage cells permanently die off.
  1. The Fat & Estrogen Trap (Why staying chubby kills your height)
    Most bros think testosterone is what fuses your growth plates, but they are dead wrong. Estrogen, specifically estradiol, is the primary hormone responsible for epiphyseal fusion, which is the permanent calcification and closing of your growth plates. Here is the massive problem with carrying extra weight. Body fat isn't just dead tissue, it is a highly active endocrine gland. Fat tissue contains an enzyme called aromatase, which literally acts like a parasite, taking your circulating testosterone and converting it directly into estrogen.
Clinical pediatric endocrinology studies, specifically those by Grumbach regarding estrogen and bone maturation, show that excess fat accelerates skeletal aging. The more fat you carry, the more aromatase you have, wich means your system is flooded with way more estrogen. For you guys who are severely overweight or morbidly obese, this is absolutely critical because you are dealing with hyperestrogenism. This means your plates are getting signaled to close way faster than a lean kid. The fix is cutting down to around 15ish % body fat so you systematically eradicate that aromatase tissue. You drop your estrogen levels to rock bottom, which keeps the close signal away from your plates and buys your bones more time to grow.
  1. Pausing (How a deficit delays growth safely)
    When you go into a severe caloric deficit, you aren't permanently stunting your growth. You are literally just hitting the pause button on your growth. Linear bone growth is driven by igf1 When you are in a deep deficit, your body senses an energy crisis and creates a state of hepatic growth hormone resistance. Your liver temporarily stops making igf1 and bone lengthening halts because your body redirects energy to keep your organs running instead.
This is a clinically documented biological phenomenon called dietary restriction-induced growth attenuation. You are trading growth right now for massive growth later. Studies by Boersma and Wit on the mechanics of catch-up growth prove that when the deficit ends and you introduce a clean caloric surplus, the liver's resistance breaks and igf1 levels absolutely skyrocket. Because you used the cutting phase to completely nuke your estrogen, your growth plates are still open and completely primed to absorb those nutrients and rapidly elongate.
  1. The Math On The Seymour Alpert Limit
    You can't just starve yourself like a dumbass, or your body will panic and eat its own muscle and organ tissue to survive. You have to calculate the exact maximum amount of fat your body can burn in a single day. In 2005, a researcher named Seymour Alpert published a highly cited study in the Journal of Theoretical Biology, proving mathematically that the human body can only extract a maximum of 31.4 calories per pound of fat mass per day.
To use this science, you just run your own numbers. Let's say you weigh 220 lbs and are around 24 percent body fat. That means you are carrying about 52 lbs of pure fat tissue. Multiply 52 by 31.4, and you get roughly 1,600 calories. That means your absolute maximum biological daily calorie deficit is 1,600. If you stay just under that limit, like running a 1,400 calorie deficit, your body uses your genuine fat as pure fuel, melting it away without losing a single ounce of muscle or triggering a permanent starvation response. Now, if you are morbidly obese carrying say 100 lbs of fat, your Alpert limit is massive, like a 3,140 calorie deficit. This means heavier bros can safely run an extremely aggressive cut and drop weight insanely fast without risking muscle loss or bone health, all because they have a massive fat tank to power the deficit.
  1. Training (Stop doing intense cardio and just walk bro)
    This is exactly where most teens completely mess up their biology. They go on a massive diet and start doing hours of intense boxing,, sprints, jogging, or insane cardio circuits. Do not do this while in a deep deficit. Severe caloric restriction combined with highly intense, glycolytic cardio massively spikes cortisol, which is your primary stress hormone.
Studies on glucocorticoids, which includes cortisol, show that chronic high levels are absolutely devastating to height. High cortisol directly inhibits chondrocyte proliferation. Chondrocytes are the cartilage cells inside your growth plates that actually multiply to make you taller. Cortisol can literally induce chondrocyte apoptosis, which means it causes those crucial growth cells to literally die off. The fix is low intensity steady state cardio. Just walk, bro. Walking keeps your heart rate low in the exact zone for lipid oxidation, meaning it burns the fat directly without signaling a systemic survival panic. It keeps your cortisol at rock bottom, completely protecting the cellular health of your growth plates while the fat melts off.
  1. Vital Micros You Gotta Hit
    While your upward growth is temporarily paused during the cut, you have to protect your bone density at all costs or your bones get incredibly weak. Even if your daily calories are super low to hit that Alpert limit, you must hit your recommended daily intake for all the crucial micronutrients to feed your osteoblasts, which are the cells that actually build bone matrix.
You need heavy protein to protect your muscle mass from breaking down. You need high doses of Calcium and Magnesium to maintain the actual structural matrix of your bones. You absolutely need Vitamin D3 paired with K2, because D3 absorbs the calcium and K2 acts like a traffic cop, directing that calcium straight into the bones instead of letting it calcify in your arteries. Finally, you need Zinc, which is scientifically crucial for natural testosterone production and optimal cellular growth.
  1. The Exit Way (The Catch Up Phase)
    You can't cut forever. A solid biological window is about 50 days of strict discipline to drop down to that 15 percent body fat range, or longer if you started morbidly obese and need more time to clear the estrogen. But what you do on the exact day you finish the cut dictates if this entire guide works or fails.
If you finish the cut and just start dirty bulking and eating junk food, your highly insulin sensitive body will instantly store it all as new fat. Your aromatase will return, your estrogen will spike, and you will permanently fuse your plates, ruining all your hard work. You need to reverse diet, bro. Rapidly bring your calories back up to maintenance, but do it exclusively with clean, nutrient-dense whole foods. Once your metabolism stabilizes over a week or two. Your body will act like an absolute sponge. With your estrogen gone and your insulin sensitivity fully reset, that clean surplus will cause a massive spike in igf1, triggering the exact catch up growth you safely delayed during the cut. You get lean, you feel less insecure and better about yourself, you clear the estrogen, you protect the muscle, and then you finally feed the growth.

The Actual Science (References)​

For the people who want to read the clinical data themselves, here are the actual studies backing up everything in this guide:
  1. Estrogen and Growth Plate Fusion: Grumbach, M. M. (2000). Estrogen, bone, growth and sex: a sea change in conventional wisdom. Journal of Pediatric Endocrinology and Metabolism.
    Link: https://pubmed.ncbi.nlm.nih.gov/11228079/
  2. Catch-Up Growth and IGF-1 Delay:
    Boersma, B., & Wit, J. M. (1997). Catch-up growth. Endocrine Reviews.
    Link: https://pubmed.ncbi.nlm.nih.gov/9331549/
  3. The Maximum Fat Burning Limit (Alpert Limit):
    Alpert, S. S. (2005). A limit on the energy transfer rate from the human fat store in hypophagia. Journal of Theoretical Biology.
    Link: https://pubmed.ncbi.nlm.nih.gov/15615615/
  4. Cortisol (Glucocorticoids) Killing Growth Plate Cells (Apoptosis):
    Chrysis, D., Ritzen, E. M., & Savendahl, L. (2003). Growth retardation induced by dexamethasone is associated with increased apoptosis of the growth plate chondrocytes. Journal of Endocrinology.
    Link: https://pubmed.ncbi.nlm.nih.gov/12558587/
investing
 
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Just eat less
 
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how to lose fat safely and quickly as a teen in puberty without stunting growth a guide by polarpop

Yo wsg everyone, I was once in this exact position. I obviously wanted to grow taller and hit my absolute genetic limit, but I was carrying extra weight, feeling insecure about my looks, and honestly just didn't know wtf to do.
It feels like a massive trap. If you cut calories, all the mainstream advice tells you that you will permanently stunt your growth. But if you just keep eating like a slob hoping the height will stretch you out, you just stay chubby and miserable. I spent a massive amount of time digging into the actual clinical endocrinology and pediatric growth science because I wanted a real answer, not just fake bro science.
It turns out, staying chubby is actually way worse for your height, and you can absolutely shred down safely if you hack your biology the right way. Here is the deeply researched, A to Z guide on how to get lean, protect your gains, and actually delay your growth plates from fusing so you can maximize your height.

What Actually Stunts Growth: what actual stunting is and how long it actually takes first off we need to clear up wtf actual stunting is cause bros think missing one meal or running a cut for a few weeks permanently thinks them to actually permanently stunt your genetic height. you have to be in a state of severe chronic starvation for months or even year sim talking actual third world famine levels like a starving kid in gaza or severe clinical anorexia where you are literally skin and bones. clinically this is called severe protein energy malnutrition or pem what happens is your body completely runs out of adipose fat tissue to burn so it enters a permanent biological survival panic it starts cannibalizing your muscle tissue and your internal organs just to keep your heart beating and your brain working when you are in this state of literal starvation for months on end your chronic cortisol levels stay so astronomically high that they permanently damage the growth plates the osteoblasts which are the cells that build bone matrix literally shut down and the cartilage cells permanently die off.
  1. The Fat & Estrogen Trap (Why staying chubby kills your height)
    Most bros think testosterone is what fuses your growth plates, but they are dead wrong. Estrogen, specifically estradiol, is the primary hormone responsible for epiphyseal fusion, which is the permanent calcification and closing of your growth plates. Here is the massive problem with carrying extra weight. Body fat isn't just dead tissue, it is a highly active endocrine gland. Fat tissue contains an enzyme called aromatase, which literally acts like a parasite, taking your circulating testosterone and converting it directly into estrogen.
Clinical pediatric endocrinology studies, specifically those by Grumbach regarding estrogen and bone maturation, show that excess fat accelerates skeletal aging. The more fat you carry, the more aromatase you have, wich means your system is flooded with way more estrogen. For you guys who are severely overweight or morbidly obese, this is absolutely critical because you are dealing with hyperestrogenism. This means your plates are getting signaled to close way faster than a lean kid. The fix is cutting down to around 15ish % body fat so you systematically eradicate that aromatase tissue. You drop your estrogen levels to rock bottom, which keeps the close signal away from your plates and buys your bones more time to grow.
  1. Pausing (How a deficit delays growth safely)
    When you go into a severe caloric deficit, you aren't permanently stunting your growth. You are literally just hitting the pause button on your growth. Linear bone growth is driven by igf1 When you are in a deep deficit, your body senses an energy crisis and creates a state of hepatic growth hormone resistance. Your liver temporarily stops making igf1 and bone lengthening halts because your body redirects energy to keep your organs running instead.
This is a clinically documented biological phenomenon called dietary restriction-induced growth attenuation. You are trading growth right now for massive growth later. Studies by Boersma and Wit on the mechanics of catch-up growth prove that when the deficit ends and you introduce a clean caloric surplus, the liver's resistance breaks and igf1 levels absolutely skyrocket. Because you used the cutting phase to completely nuke your estrogen, your growth plates are still open and completely primed to absorb those nutrients and rapidly elongate.
  1. The Math On The Seymour Alpert Limit
    You can't just starve yourself like a dumbass, or your body will panic and eat its own muscle and organ tissue to survive. You have to calculate the exact maximum amount of fat your body can burn in a single day. In 2005, a researcher named Seymour Alpert published a highly cited study in the Journal of Theoretical Biology, proving mathematically that the human body can only extract a maximum of 31.4 calories per pound of fat mass per day.
To use this science, you just run your own numbers. Let's say you weigh 220 lbs and are around 24 percent body fat. That means you are carrying about 52 lbs of pure fat tissue. Multiply 52 by 31.4, and you get roughly 1,600 calories. That means your absolute maximum biological daily calorie deficit is 1,600. If you stay just under that limit, like running a 1,400 calorie deficit, your body uses your genuine fat as pure fuel, melting it away without losing a single ounce of muscle or triggering a permanent starvation response. Now, if you are morbidly obese carrying say 100 lbs of fat, your Alpert limit is massive, like a 3,140 calorie deficit. This means heavier bros can safely run an extremely aggressive cut and drop weight insanely fast without risking muscle loss or bone health, all because they have a massive fat tank to power the deficit.
  1. Training (Stop doing intense cardio and just walk bro)
    This is exactly where most teens completely mess up their biology. They go on a massive diet and start doing hours of intense boxing,, sprints, jogging, or insane cardio circuits. Do not do this while in a deep deficit. Severe caloric restriction combined with highly intense, glycolytic cardio massively spikes cortisol, which is your primary stress hormone.
Studies on glucocorticoids, which includes cortisol, show that chronic high levels are absolutely devastating to height. High cortisol directly inhibits chondrocyte proliferation. Chondrocytes are the cartilage cells inside your growth plates that actually multiply to make you taller. Cortisol can literally induce chondrocyte apoptosis, which means it causes those crucial growth cells to literally die off. The fix is low intensity steady state cardio. Just walk, bro. Walking keeps your heart rate low in the exact zone for lipid oxidation, meaning it burns the fat directly without signaling a systemic survival panic. It keeps your cortisol at rock bottom, completely protecting the cellular health of your growth plates while the fat melts off.
  1. Vital Micros You Gotta Hit
    While your upward growth is temporarily paused during the cut, you have to protect your bone density at all costs or your bones get incredibly weak. Even if your daily calories are super low to hit that Alpert limit, you must hit your recommended daily intake for all the crucial micronutrients to feed your osteoblasts, which are the cells that actually build bone matrix.
You need heavy protein to protect your muscle mass from breaking down. You need high doses of Calcium and Magnesium to maintain the actual structural matrix of your bones. You absolutely need Vitamin D3 paired with K2, because D3 absorbs the calcium and K2 acts like a traffic cop, directing that calcium straight into the bones instead of letting it calcify in your arteries. Finally, you need Zinc, which is scientifically crucial for natural testosterone production and optimal cellular growth.
  1. The Exit Way (The Catch Up Phase)
    You can't cut forever. A solid biological window is about 50 days of strict discipline to drop down to that 15 percent body fat range, or longer if you started morbidly obese and need more time to clear the estrogen. But what you do on the exact day you finish the cut dictates if this entire guide works or fails.
If you finish the cut and just start dirty bulking and eating junk food, your highly insulin sensitive body will instantly store it all as new fat. Your aromatase will return, your estrogen will spike, and you will permanently fuse your plates, ruining all your hard work. You need to reverse diet, bro. Rapidly bring your calories back up to maintenance, but do it exclusively with clean, nutrient-dense whole foods. Once your metabolism stabilizes over a week or two. Your body will act like an absolute sponge. With your estrogen gone and your insulin sensitivity fully reset, that clean surplus will cause a massive spike in igf1, triggering the exact catch up growth you safely delayed during the cut. You get lean, you feel less insecure and better about yourself, you clear the estrogen, you protect the muscle, and then you finally feed the growth.

The Actual Science (References)​

For the people who want to read the clinical data themselves, here are the actual studies backing up everything in this guide:
  1. Estrogen and Growth Plate Fusion: Grumbach, M. M. (2000). Estrogen, bone, growth and sex: a sea change in conventional wisdom. Journal of Pediatric Endocrinology and Metabolism.
    Link: https://pubmed.ncbi.nlm.nih.gov/11228079/
  2. Catch-Up Growth and IGF-1 Delay:
    Boersma, B., & Wit, J. M. (1997). Catch-up growth. Endocrine Reviews.
    Link: https://pubmed.ncbi.nlm.nih.gov/9331549/
  3. The Maximum Fat Burning Limit (Alpert Limit):
    Alpert, S. S. (2005). A limit on the energy transfer rate from the human fat store in hypophagia. Journal of Theoretical Biology.
    Link: https://pubmed.ncbi.nlm.nih.gov/15615615/
  4. Cortisol (Glucocorticoids) Killing Growth Plate Cells (Apoptosis):
    Chrysis, D., Ritzen, E. M., & Savendahl, L. (2003). Growth retardation induced by dexamethasone is associated with increased apoptosis of the growth plate chondrocytes. Journal of Endocrinology.
    Link: https://pubmed.ncbi.nlm.nih.gov/12558587/
dnr
 
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fcking ai slop
 
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investing

1779038830797


it can't be

he would never

tell me it's a joke :feelswhy:
 
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Hes asking you @Idontknow-
 
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jesus christ bro got so smoked he had to delete the entire shit
 
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jesus christ bro got so smoked he had to delete the entire shit
Nah no point of providing decent threads if I get called out for ai for simply just utilizing good formatting and medical terms lmao
 
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Nah no point of providing decent threads if I get called out for ai for simply just utilizing good formatting and medical terms lmao
Crying
 
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