Is raw carnivore diet cope?

[hxmzx00]

is raw carnivore cope or is it really the optimal diet, What do y’all think?

 

[hej1377]

Yes cope

 

[SvtvnTr14d]

is raw carnivore cope or is it really the optimal diet, What do y’all think?

only good side is gut health won't do anything else really

 

[htntown]

is raw carnivore cope or is it really the optimal diet, What do y’all think?

try it

 

[KellSS]

is raw carnivore cope or is it really the optimal diet, What do y’all think?

You also need to live in middle of forest and live like a prehistoric nigga for very high testosterone not only raw meat

 

[BoneLord67]

is raw carnivore cope or is it really the optimal diet, What do y’all think?

kinda, depends how you think of the benefits and downsides.
Pros: high nutrient density, glycemic stability (no carbs = no insulin spikes), no antinutrients from plants (it can reduce GI distress in IBS and stuff which is cool)
Cons: pathogen risk (salmonella, E. coli, parasites, and many other dangerous diseases), micronutrient deficiency (like vitamin C, Magnesium, Potassium, Folate, and iodine are usually common), lipid profile and vascular risk (raw saturated fat intake can elevate LDL-C and ApoB in genetically susceptible individuals, and no fiber = reduced bile acid clearance = potential cholesterol dysregulation), gut microbiome collapse (intestinal barrier collapse, inflammation spike, skin and immune health worsen), reduced t3 hormone (elevates cortisol and impairs sleep quality and recovery)

 

[hxmzx00]

try it

I’m on a carnivore diet rn and I’m slowly transitioning to raw meat

 

[KellSS]

kinda, depends how you think of the benefits and downsides.
Pros: high nutrient density, glycemic stability (no carbs = no insulin spikes), no antinutrients from plants (it can reduce GI distress in IBS and stuff which is cool)
Cons: pathogen risk (salmonella, E. coli, parasites, and many other dangerous diseases), micronutrient deficiency (like vitamin C, Magnesium, Potassium, Folate, and iodine are usually common), lipid profile and vascular risk (raw saturated fat intake can elevate LDL-C and ApoB in genetically susceptible individuals, and no fiber = reduced bile acid clearance = potential cholesterol dysregulation), gut microbiome collapse (intestinal barrier collapse, inflammation spike, skin and immune health worsen), reduced t3 hormone (elevates cortisol and impairs sleep quality and recovery)

 

[abuse]

is raw carnivore cope or is it really the optimal diet, What do y’all think?

100%, being in a state of ketosis is super suboptimal

 

[htntown]

I’m on a carnivore diet rn and I’m slowly transitioning to raw meat

whats your goal

 

[hxmzx00]

whats your goal

wdym?

 

[lit]

kinda, depends how you think of the benefits and downsides.
Pros: high nutrient density, glycemic stability (no carbs = no insulin spikes), no antinutrients from plants (it can reduce GI distress in IBS and stuff which is cool)
Cons: pathogen risk (salmonella, E. coli, parasites, and many other dangerous diseases), micronutrient deficiency (like vitamin C, Magnesium, Potassium, Folate, and iodine are usually common), lipid profile and vascular risk (raw saturated fat intake can elevate LDL-C and ApoB in genetically susceptible individuals, and no fiber = reduced bile acid clearance = potential cholesterol dysregulation), gut microbiome collapse (intestinal barrier collapse, inflammation spike, skin and immune health worsen), reduced t3 hormone (elevates cortisol and impairs sleep quality and recovery)

Clearly you lack understanding of metabolic biochemistry, I’ll dive into the primary misleading claims.



i) Human Data

LDL-C isn’t inherently problematic on a raw carnivore/keto diet, and much of the association between LDL, ApoB, and cardiovascular disease is epidemiological and not mechanistic.

A 1-year study in lean, metabolically healthy individuals on a ketogenic diet found that neither baseline levels nor changes in LDL-C and ApoB were associated with plaque progression: https://pubmed.ncbi.nlm.nih.gov/40192608/​

"In lean metabolically healthy people on KD, neither total exposure nor changes in baseline levels of ApoB and LDL-C were associated with changes in plaque.”

In the current best study, which is a follow-up (~4.7 years) used coronary CT scans to directly measure atherosclerotic plaque progression in metabolically healthy individuals: https://pubmed.ncbi.nlm.nih.gov/39372369/​

“Coronary plaque in metabolically healthy individuals with carbohydrate restriction-induced LDL-C ≥190 mg/dL on KETO for a mean of 4.7 years is not greater than a matched cohort with 149 mg/dL lower average LDL-C. There is no association between LDL-C and plaque burden in either cohort.”

Even with the keto group having "chronically high LDL" there was no association between LCL-C and plaque progression in either cohort.



ii) Thyroid Function on Low-Carb


Lower T3 on very low-carbohydrate diets does not automatically indicate pathology. In fat-adapted states, reduced T3 commonly reflects increased metabolic efficiency. Less T3 is required when the body is efficiently oxidizing fatty acids rather than glucose.

https://pmc.ncbi.nlm.nih.gov/articles/PMC12468144/



iii) Vitamin C and Glucose Competition for GLUT4

Glucose and vitamin C compete for the same GLUT4 transporter. Glucose inhibits vitamin C uptake at the cellular level, which explains why individuals with hyperglycemia can exhibit functional vitamin C deficiency despite exogenous intake. This mechanism explains the historical absence of scurvy in low-carb animal based populations such as the Inuit. Furtherly, chromium further upregulates GLUT4 expression, improving vitamin C uptake, and is found in animal foods like oysters, shrimp, beef, and eggs. Not to mention that vitamin C is in animal products and is preserved raw since vitamin C is a water soluble vitamin, meaning that none will be lost to heat.

https://pubmed.ncbi.nlm.nih.gov/19391462/



iv) Dietary Fiber

Ho KS, Tan CYM, Mohd Daud MA, Seow-Choen F. Stopping or reducing dietary fiber intake reduces constipation and its associated symptoms. World J Gastroenterol 2012; 18(33): 4593-4596 [PMID: 22969234 DOI: 10.3748/wjg.v18.i33.4593]

1- "Idiopathic constipation and its associated symptoms can be effectively reduced by stopping or even lowering the intake of dietary fiber.”
2- "For symptoms of bloating, all of those on a high fiber diet continued to be symptomatic, while only 31.3% in the reduced fiber group and none of the no fiber group had symptoms.”
3-“These symptoms are caused by the fermentation of dietary fiber by colonic bacteria, which produces hydrogen, carbon dioxide and methane.”

 

[htntown]

wdym?

what are you trying to improve

 

[hxmzx00]

better gut health so I get less acne and I’m trying to gain weight