Noah Boat's leanmaxxing transformation

[Seth Walsh]

Pretty crazy. Do you think GLPs were involved?

 

[Deleted member 55437]

Looks the same

 

[oily703]

noah bloat

 

[Deleted member 77820]

Pretty crazy. Do you think GLPs were involved?

Nigga im gonna kill you

Do you think anyone who do transformation videos are on ozempic jfl

 

[oily703]

Nigga im gonna kill you

Do you think anyone who do transformation videos are on ozempic jfl

LA influencer
its more likely they were than not jfl

 

[chadisbeingmade]

Pretty crazy. Do you think GLPs were involved?

Nigga im gonna kill you

Do you think anyone who do transformation videos are on ozempic jfl

Yall talking like Sema/GLP1s is a big deal. It’s just a fucking apatite suppressant. It’s not like bros blasting Test. Ozempic is barely cheating.

 

[Deleted member 77820]

LA influencer
its more likely they were than not jfl

Your retarded

The transformation isn’t that impressive

 

[Deleted member 77820]

Yall talking like Sema/GLP1s is a big deal. It’s just a fucking apatite suppressant. It’s not like bros blasting Test. Ozempic is barely cheating.

Normies don’t use glp-1 bro

 

[Seth Walsh]

Nigga im gonna kill you

Do you think anyone who do transformation videos are on ozempic jfl

Yeah, especially this one.


If not, then props to him for the insane insane willpower.

David Dobrik did it too under the same trainer, who promotes his workout/diet plan, supplements and calorie tracking app.

Not being on GLPs would make it 500x harder and the daily cravings would be near unbearable unless each person actually has insane self restraint.

Nonetheless. Serious props to Noah. Looksmaxxed better than 99.999% of the forum in 4 months. This is how gymmaxxing should be done.


Clinical.

 

[Deleted member 77820]

I mean imagine using glps to go from 20 to 12% in fucking nearly 18 weeks thats if he didn’t mie about the time frame

 

[Seth Walsh]

Normies don’t use glp-1 bro

Everyone in hollywood or on social media would. Ozempic is really really popular worldwide. It's not as if it's some underground bodybuilding secret only found in fringe internet forums.

 

[Seth Walsh]

I mean imagine using glps to go from 20 to 12% in fucking nearly 18 weeks thats if he didn’t mie about the time frame

Yeah seems realistic. Are you saying the timeframe seems too short or too long?

 

[Deleted member 77820]

Yeah, especially this one.


If not, then props to him for the insane insane willpower.

David Dobrik did it too under the same trainer, who promotes his workout/diet plan, supplements and calorie tracking app.

Not being on GLPs would make it 500x harder and the daily cravings would be near unbearable unless each person actually has insane self restraint.

Nonetheless. Serious props to Noah. Looksmaxxed better than 99.999% of the forum in 4 months. This is how gymmaxxing should be done.


Clinical.

We are 50000 in this forum bro I assure you their is more than 50 people who leanmaxxed succesfully
Including myself

 

[chadisbeingmade]

Everyone in hollywood or on social media would. Ozempic is really really popular worldwide. It's not as if it's some underground bodybuilding secret only found in fringe internet forums.

Normies don’t use glp-1 bro

There is literally 0 reason not to use it. Willpower is retarded. This is one of the few things I agree with Mike on.

 

[oily703]

Your retarded

The transformation isn’t that impressive

so what if its not that impressive
it would be easier if he used these compounds so why wouldnt he use these compounds?
that paired with the fact that he is a fucking LAcel i couldnt think of a reason he woulnt use it

 

[Deleted member 77820]

Everyone in hollywood or on social media would. Ozempic is really really popular worldwide. It's not as if it's some underground bodybuilding secret only found in fringe internet forums.

On hollywodd yeah basicaly all of the celebrities
Agree
But not those shitty ahh youtubers they either lie about the time frame or they just use lighting to their advantages

 

[Seth Walsh]

On hollywodd yeah basicaly all of the celebrities
Agree
But not those shitty ahh youtubers they either lie about the time frame or they just use lighting to their advantages

Loveliveserve are turbo blackpilled. Pretty sure they've read through the forums at one point. A lot of their content is just BP and looks/dating related stuff.

 

[Deleted member 77820]

so what if its not that impressive
it would be easier if he used these compounds so why wouldnt he use these compounds?
that paired with the fact that he is a fucking LAcel i couldnt think of a reason he woulnt use it

Waste of money in my opinion
If he could afford it good for him

I tried generic semag and it completely nuked my appetite but i didn’t lose visceral fat or anything crazy it just make me stick to my diet

If i used my money on shot that actually make you lose fat (but has sides) it would have helped me way more

 

[sub5outsider]

hes the definition of white nigger

 

[Seth Walsh]

@Dyorotic potential Reta use?

I need to know more about the benefits. Only one of the 3 I never tried.

 

[Deleted member 77820]

Yeah seems realistic. Are you saying the timeframe seems too short or too long?

Its realistic its nothing crazy he doesn’t have to be in a insane caloric deficit he doesn’t need to be on Glp-1s would it be easier if he used glp-1s
Sure thing

 

[Deleted member 77820]

@Dyorotic potential Reta use?

I need to know more about the benefits. Only one of the 3 I never tried.

Its overrated trust me its just a propaganda stick to semaglutide by far the best ROI Glp-1

 

[Seth Walsh]

Its overrated trust me its just a propaganda stick to semaglutide by far the best ROI Glp-1

Why, did you use it extensively and found Semaglutide more effective?

 

[Seth Walsh]

Its realistic its nothing crazy he doesn’t have to be in a insane caloric deficit he doesn’t need to be on Glp-1s would it be easier if he used glp-1s
Sure thing

Well the drug would help him stay in a caloric deficit, which is the hard thing. Eating boring foods, sticking to a tough routine.

 

[DarkAscender]

Looks achievable in that time frame. Losing bodyfat % bove 15% bf is pretty easy. Afterwards its a bit slower, but 3 months seems realistic.

 

[fogdart]

it's not that hard to just put the burger and fries away and eat real food.

 

[Deleted member 55437]

Your retarded

The transformation isn’t that impressive

He looks better physically but facially hard to see much change

 

[Deleted member 77820]

Why, did you use it extensively and found Semaglutide more effective?

No
But would do the experiment if i get the money i swear to god
Do you know any method that can make me make 25$/day online while im 16 yo in morroco

 

[Dyorotic]

Its overrated trust me its just a propaganda stick to semaglutide by far the best ROI Glp-1

If your sole goal is weight loss at all cost, regardless of sides, then yeah Semaglutide is the strongest mg for mg at stimulating GLP1 and also causes the most weight loss (again mg for mg, Semaglutide is active in the microgram range), but GLP1 is only one factor. At the end of the day the GLP1 receptor is doing most of the work here because of it's effect on gastric emptying and satiety, so of course you're going to lose weight when you literally hurl at the thought of food (common amongst Semaglutide users). However it is absolutely the worst in regards to side effects because it doesn't have the anti-emetic component of Reta/Tirz, speaking of, Tirz has a weaker affinity for GIPR than endogenous GIP does and it isn't even a full agonist (partial agonist), so really Tirz is just a more expensive and weaker version of Semaglutide. I wouldn't use Tirz willingly if I had access to Semaglutide and Retatrutide.

Anyway with GIPR agonism you get an array of benefits that obviously mono-agonists (Sema) don't produce. GIPR is highly synergistic with GLP1R. The neuroprotective effect of GLP1R agonism is potentiated by GIP receptors in the brain, it also increases insulin sensitivity within the brain because it lowers hepatic glucose output to cerebral tissue specifically. Obviously dual agonism enhances satiety signalling in the brain as well. GIP lowers glucose uptake into muscle but it also increases insulin sensitivity to skeletal muscle. Insulin synthesis, insulin secretion and beta cell survival are also significantly enhanced with GIP1/GIP agonsim and from what I've read/seen GIP seems to be highly protective of the pancreas compared to Semaglutide (still majorly protective). Most of it's action occurs within the pancreas, brain and adipose tissue. This is where it gets interesting for me, the GIPR seems to be highly active within adipose tissue, majorly increasing insulin sensitivity, increasing blood flow to adipocytes, decreasing pro-inflammatory adipokines whilst simultaneously increasing anti-inflammatory ones (adiponectin). GIP and PPAR gamma also induce the expression of one another and GIP agonism also increases adipogenesis which is the most metabolically sound state of weight gain (the inverse being adipocyte hypertrophy which upregulates pro-inflammatory markers (Visfatin, Apelin and Resistin) and decreases insulin sensitivity)

Basically adipocyte hypertrophy is highly linked to central/visceral obesity, which a lot of men have because androgens modulate the expression of beta receptors (increased sensitivity to catecholamines) and are highly involved in lipolysis/hypertrophy of VAT adipocytes rather than SAT tissue. Basically if you do fuck up your diet somehow on a GIP agonist you can be assured that the weight gain will mostly be in the form of subcutaneous tissue osed to obesity, ERa singalling seems to induce PPARy expression, whereas AR signalling seems to do the inverse (hence why men gain intra-abdominal, visceral and liver/organ fat preferentially). It's always healthier to hold some subcutaneous fat, this is why women are less predisposed to diabetes). E2 also upregulates anti-lipolytic alpha-2-adrenergic receptors selectively within subcutaneous depots which results in less lipolysis in subcutaneous tissue. There's a reason women preferentially gain weight on their lower body, sometimes you even come across women who have seemingly lean navals/midsections but huge thighs and ass, this is all due to ERa & PPARy expression.

Enough of the soy science though, something a lot of people complain about in regards to Semaglutide is the acid reflux, of which GIPR agonists seem to abate quite significantly due to the HCL reduction, I never once had acid reflux on Reta but when I tried Sema it was pretty persistent. Vomitting and nausea, being tangentially related to acid reflux, also doesn't occur much on Retatrutide because of the Glucagon component. Unlike GIP, Glucagon is seemingly active everywhere in the body. The thing about Glucagon is it doesn't just decrease satiety, it seems to be highly lipolytic compared to GIP and GLP1. In brown adipose tissue it increases thermogenesis, in white it increases lipolyis AND thermogenesis, it also increases basal metabolic rate significantly via an increase in heart rate (can be uncomfortable but it's nowhere near as bad as the nause/reflux from sema). It's highly hepatoprotective (modulation of lipid synthesis, LDL down, HDL up). Besides its effects on the liver, glucagon is also known to influence kidney function, studies showed that it increases glomerular filtration rate (GFR) and natriuresis (GCGR gene deletion causes major dysregulation of the kidneys and is highly nephrotoxic). In other words, glucagon acts like a lowgrade diuretic (JFL). It's just too good.

Also there's nothing wrong with combining GLP1s. A low dose of sema + regular Reta dose would enhance the gastric emptying effect whilst retaining all the benefits of GIP/GCGR. From my experience though Cagrilintide is by far the most potent appetite crusher. 125mcg + my normal Reta dose basically made it impossible to eat for three days. So there's that

I always ramble but I hope you understand the point. Sema is fantastic but Reta is overall a better compound regardless of whether Semaglutide is more effacacious clincially (for total body mass loss specifically).

@Seth Walsh

 

[Dyorotic]

The main takeaway should be that if you're already lean and don't want to relapse, take Reta and maintain your weight. You'll still have an appetite but it won't be voracious, but all of your good biomarkers go up, so there's no reason not to use it (especially over Tirz).

Tirz won't be used when Retatrutide is officially avaliable.

 

[Seth Walsh]

If your sole goal is weight loss at all cost, regardless of sides, then yeah Semaglutide is the strongest mg for mg at stimulating GLP1 and also causes the most weight loss (again mg for mg, Semaglutide is active in the microgram range), but GLP1 is only one factor. At the end of the day the GLP1 receptor is doing most of the work here because of it's effect on gastric emptying and satiety, so of course you're going to lose weight when you literally hurl at the thought of food (common amongst Semaglutide users). However it is absolutely the worst in regards to side effects because it doesn't have the anti-emetic component of Reta/Tirz, speaking of, Tirz has a weaker affinity for GIPR than endogenous GIP does and it isn't even a full agonist (partial agonist), so really Tirz is just a more expensive and weaker version of Semaglutide. I wouldn't use Tirz willingly if I had access to Semaglutide and Retatrutide.

Anyway with GIPR agonism you get an array of benefits that obviously mono-agonists (Sema) don't produce. GIPR is highly synergistic with GLP1R. The neuroprotective effect of GLP1R agonism is potentiated by GIP receptors in the brain, it also increases insulin sensitivity within the brain because it lowers hepatic glucose output to cerebral tissue specifically. Obviously dual agonism enhances satiety signalling in the brain as well. GIP lowers glucose uptake into muscle but it also increases insulin sensitivity to skeletal muscle. Insulin synthesis, insulin secretion and beta cell survival are also significantly enhanced with GIP1/GIP agonsim and from what I've read/seen GIP seems to be highly protective of the pancreas compared to Semaglutide (still majorly protective). Most of it's action occurs within the pancreas, brain and adipose tissue. This is where it gets interesting for me, the GIPR seems to be highly active within adipose tissue, majorly increasing insulin sensitivity, increasing blood flow to adipocytes, decreasing pro-inflammatory adipokines whilst simultaneously increasing anti-inflammatory ones (adiponectin). GIP and PPAR gamma also induce the expression of one another and GIP agonism also increases adipogenesis which is the most metabolically sound state of weight gain (the inverse being adipocyte hypertrophy which upregulates pro-inflammatory markers (Visfatin, Apelin and Resistin) and decreases insulin sensitivity)

Basically adipocyte hypertrophy is highly linked to central/visceral obesity, which a lot of men have because androgens modulate the expression of beta receptors (increased sensitivity to catecholamines) and are highly involved in lipolysis/hypertrophy of VAT adipocytes rather than SAT tissue. Basically if you do fuck up your diet somehow on a GIP agonist you can be assured that the weight gain will mostly be in the form of subcutaneous tissue osed to obesity, ERa singalling seems to induce PPARy expression, whereas AR signalling seems to do the inverse (hence why men gain intra-abdominal, visceral and liver/organ fat preferentially). It's always healthier to hold some subcutaneous fat, this is why women are less predisposed to diabetes). E2 also upregulates anti-lipolytic alpha-2-adrenergic receptors selectively within subcutaneous depots which results in less lipolysis in subcutaneous tissue. There's a reason women preferentially gain weight on their lower body, sometimes you even come across women who have seemingly lean navals/midsections but huge thighs and ass, this is all due to ERa & PPARy expression.

Enough of the soy science though, something a lot of people complain about in regards to Semaglutide is the acid reflux, of which GIPR agonists seem to abate quite significantly due to the HCL reduction, I never once had acid reflux on Reta but when I tried Sema it was pretty persistent. Vomitting and nausea, being tangentially related to acid reflux, also doesn't occur much on Retatrutide because of the Glucagon component. Unlike GIP, Glucagon is seemingly active everywhere in the body. The thing about Glucagon is it doesn't just decrease satiety, it seems to be highly lipolytic compared to GIP and GLP1. In brown adipose tissue it increases thermogenesis, in white it increases lipolyis AND thermogenesis, it also increases basal metabolic rate significantly via an increase in heart rate (can be uncomfortable but it's nowhere near as bad as the nause/reflux from sema). It's highly hepatoprotective (modulation of lipid synthesis, LDL down, HDL up). Besides its effects on the liver, glucagon is also known to influence kidney function, studies showed that it increases glomerular filtration rate (GFR) and natriuresis (GCGR gene deletion causes major dysregulation of the kidneys and is highly nephrotoxic). In other words, glucagon acts like a lowgrade diuretic (JFL). It's just too good.

Also there's nothing wrong with combining GLP1s. A low dose of sema + regular Reta dose would enhance the gastric emptying effect whilst retaining all the benefits of GIP/GCGR. From my experience though Cagrilintide is by far the most potent appetite crusher. 125mcg + my normal Reta dose basically made it impossible to eat for three days. So there's that

I always ramble but I hope you understand the point. Sema is fantastic but Reta is overall a better compound regardless of whether Semaglutide is more effacacious clincially (for total body mass loss specifically).

@Seth Walsh

You’re a star. Thank you, will read it later. It’s great to have your experience as feedback in addition to your knowledge

 

[EliDKing]

If your sole goal is weight loss at all cost, regardless of sides, then yeah Semaglutide is the strongest mg for mg at stimulating GLP1 and also causes the most weight loss (again mg for mg, Semaglutide is active in the microgram range), but GLP1 is only one factor. At the end of the day the GLP1 receptor is doing most of the work here because of it's effect on gastric emptying and satiety, so of course you're going to lose weight when you literally hurl at the thought of food (common amongst Semaglutide users). However it is absolutely the worst in regards to side effects because it doesn't have the anti-emetic component of Reta/Tirz, speaking of, Tirz has a weaker affinity for GIPR than endogenous GIP does and it isn't even a full agonist (partial agonist), so really Tirz is just a more expensive and weaker version of Semaglutide. I wouldn't use Tirz willingly if I had access to Semaglutide and Retatrutide.

Anyway with GIPR agonism you get an array of benefits that obviously mono-agonists (Sema) don't produce. GIPR is highly synergistic with GLP1R. The neuroprotective effect of GLP1R agonism is potentiated by GIP receptors in the brain, it also increases insulin sensitivity within the brain because it lowers hepatic glucose output to cerebral tissue specifically. Obviously dual agonism enhances satiety signalling in the brain as well. GIP lowers glucose uptake into muscle but it also increases insulin sensitivity to skeletal muscle. Insulin synthesis, insulin secretion and beta cell survival are also significantly enhanced with GIP1/GIP agonsim and from what I've read/seen GIP seems to be highly protective of the pancreas compared to Semaglutide (still majorly protective). Most of it's action occurs within the pancreas, brain and adipose tissue. This is where it gets interesting for me, the GIPR seems to be highly active within adipose tissue, majorly increasing insulin sensitivity, increasing blood flow to adipocytes, decreasing pro-inflammatory adipokines whilst simultaneously increasing anti-inflammatory ones (adiponectin). GIP and PPAR gamma also induce the expression of one another and GIP agonism also increases adipogenesis which is the most metabolically sound state of weight gain (the inverse being adipocyte hypertrophy which upregulates pro-inflammatory markers (Visfatin, Apelin and Resistin) and decreases insulin sensitivity)

Basically adipocyte hypertrophy is highly linked to central/visceral obesity, which a lot of men have because androgens modulate the expression of beta receptors (increased sensitivity to catecholamines) and are highly involved in lipolysis/hypertrophy of VAT adipocytes rather than SAT tissue. Basically if you do fuck up your diet somehow on a GIP agonist you can be assured that the weight gain will mostly be in the form of subcutaneous tissue osed to obesity, ERa singalling seems to induce PPARy expression, whereas AR signalling seems to do the inverse (hence why men gain intra-abdominal, visceral and liver/organ fat preferentially). It's always healthier to hold some subcutaneous fat, this is why women are less predisposed to diabetes). E2 also upregulates anti-lipolytic alpha-2-adrenergic receptors selectively within subcutaneous depots which results in less lipolysis in subcutaneous tissue. There's a reason women preferentially gain weight on their lower body, sometimes you even come across women who have seemingly lean navals/midsections but huge thighs and ass, this is all due to ERa & PPARy expression.

Enough of the soy science though, something a lot of people complain about in regards to Semaglutide is the acid reflux, of which GIPR agonists seem to abate quite significantly due to the HCL reduction, I never once had acid reflux on Reta but when I tried Sema it was pretty persistent. Vomitting and nausea, being tangentially related to acid reflux, also doesn't occur much on Retatrutide because of the Glucagon component. Unlike GIP, Glucagon is seemingly active everywhere in the body. The thing about Glucagon is it doesn't just decrease satiety, it seems to be highly lipolytic compared to GIP and GLP1. In brown adipose tissue it increases thermogenesis, in white it increases lipolyis AND thermogenesis, it also increases basal metabolic rate significantly via an increase in heart rate (can be uncomfortable but it's nowhere near as bad as the nause/reflux from sema). It's highly hepatoprotective (modulation of lipid synthesis, LDL down, HDL up). Besides its effects on the liver, glucagon is also known to influence kidney function, studies showed that it increases glomerular filtration rate (GFR) and natriuresis (GCGR gene deletion causes major dysregulation of the kidneys and is highly nephrotoxic). In other words, glucagon acts like a lowgrade diuretic (JFL). It's just too good.

Also there's nothing wrong with combining GLP1s. A low dose of sema + regular Reta dose would enhance the gastric emptying effect whilst retaining all the benefits of GIP/GCGR. From my experience though Cagrilintide is by far the most potent appetite crusher. 125mcg + my normal Reta dose basically made it impossible to eat for three days. So there's that

I always ramble but I hope you understand the point. Sema is fantastic but Reta is overall a better compound regardless of whether Semaglutide is more effacacious clincially (for total body mass loss specifically).

@Seth Walsh

high iq, mirin

 

[Sorrowandsuffering]

Pretty crazy. Do you think GLPs were involved?

Sprite

 

[copercel123]

Pretty crazy. Do you think GLPs were involved?

Who cares about gymcels

 

[superpsycho]

mirin