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Sending me a mouse cage sterilization study to argue about sprinting and HGH is typical 14 rep behavior jfl. That paper has nothing to do with exercise, growth hormone, or height, unless you think autoclaved corncob bedding boosts puberty gains. Read what you post before flexing citations you don’t understand


That’s not what the study says. The main point isn’t that "more rest = better recovery = more HGH." In fact, the sprints were separated by 4 hours, which is more than enough time for recovery and high exercise intensity. Yet, despite that rest, the GH response to the second sprint was still significantly lower, unless free fatty acid levels were chemically suppressed.

From the study:
"When two sprints are performed separated by 60 min or 240 min of recovery, the GH response to the second sprint is attenuated.”, and "The attenuation of the GH response to a second sprint... may be due to elevated concentrations of systemic FFA, which can inhibit GH release at the level of the pituitary."

The whole purpose of using nicotinic acid in the study was to block FFA release and test whether FFAs were the cause of GH suppression. The result:
"Suppressing lipolysis resulted in a significantly greater GH response to the second of two sprints, suggesting a potential role for serum FFA in negative feedback control of the GH response."


Multiple studies show high-intensity sprint exercise causes a significant acute surge in growth hormone levels. Example:
Godfrey et al. (2003) found that brief, all-out sprints produce large, transient spikes in GH that exceed levels seen in moderate exercise. This acute GH release is important because pulsatile GH secretion is the main physiological driver of growth during puberty.

GH stimulates the growth plates in long bones via IGF-1, promoting chondrocyte proliferation and bone elongation. The key paper (Nilsson & Ohlsson, 2006) clearly explains the biological mechanism by which GH and IGF-1 regulate longitudinal bone growth during adolescence.
The FFA feedback inhibition described in the study you mentioned (Stokes et al., 2008) relates specifically to GH secretion on subsequent sprints performed within hours. This means that if you do multiple sprints very close together, elevated free fatty acids can blunt the GH response in the second bout. But this does not mean sprinting doesn’t cause a GH surge overall. It just means there is a physiological feedback loop limiting continuous, repeated spikes in a short time frame.

Real-world training programs usually involve one or two sprint sessions per day with sufficient rest, which avoids the FFA feedback suppression. So, sprinting done properly will maximize GH release without FFA interference.
The acute GH increase from sprinting stimulates growth plates during puberty. FFA feedback only limits very frequent repeated sprints, not the overall GH boosting effect of sprinting. Therefore, sprinting remains an effective way to naturally enhance HGH levels and aid height growth.
 


Multiple studies show high-intensity sprint exercise causes a significant acute surge in growth hormone levels. Example:
Godfrey et al. (2003) found that brief, all-out sprints produce large, transient spikes in GH that exceed levels seen in moderate exercise. This acute GH release is important because pulsatile GH secretion is the main physiological driver of growth during puberty.

GH stimulates the growth plates in long bones via IGF-1, promoting chondrocyte proliferation and bone elongation. The key paper (Nilsson & Ohlsson, 2006) clearly explains the biological mechanism by which GH and IGF-1 regulate longitudinal bone growth during adolescence.
The FFA feedback inhibition described in the study you mentioned (Stokes et al., 2008) relates specifically to GH secretion on subsequent sprints performed within hours. This means that if you do multiple sprints very close together, elevated free fatty acids can blunt the GH response in the second bout. But this does not mean sprinting doesn’t cause a GH surge overall. It just means there is a physiological feedback loop limiting continuous, repeated spikes in a short time frame.

Real-world training programs usually involve one or two sprint sessions per day with sufficient rest, which avoids the FFA feedback suppression. So, sprinting done properly will maximize GH release without FFA interference.
The acute GH increase from sprinting stimulates growth plates during puberty. FFA feedback only limits very frequent repeated sprints, not the overall GH boosting effect of sprinting. Therefore, sprinting remains an effective way to naturally enhance HGH levels and aid height growth.
The GH released is so minimal you're better off napping instead of sprinting
 
The GH released is so minimal you're better off napping instead of sprinting
obviously alot of sleep helps with height growth in puberty but science has already shown that sprinting and increasing HGH helps with height growth, and hell call me crazy, even just a little bit more height growth is better than nothing, its like the jaw, every mm counts
 
I’m 13 wtf then you not read and I wanna be 6’6 so I idc and mk practically has no side effects I’m 5’8 my moms 5’9 and my dad is 6’1 can I make it to 6’6 naturally?
bro my mom is 5'3 my dad is 5'11 and none of my grandparents are tall and i was 5'5 at 15 now im 6'4 at 17, mk does have side effects youll just bloat and be a fat ass, and respectfully you dont have good bone mass the bloat would make you look retarded asf and moon faced. your on track to be tall already if your 13 and 5'8. stop being retarded
 
@Quncho @Orc @Bittersweet @Gengar @NumbThePain
 
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@Gengar ban
 
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Ur 13, go play football or something what are u even doung here. Just live ur life and let puberty do its work
 

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