Raw meat

iblamegenetics7681

iblamegenetics7681

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i just had my first ever raw meat it was tartar 200 grams just raw out of the package i wanna start eating other raw animal food like liver for example, is there anyone that can guide me, and tell me how they went about it, im 16 years old.
 
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Okay since you want to stay on topic, let’s stay on topic.

Raw meat or cooked meat, what is healthier for humans?

Your main arguments are that some nutrients, enzymes & amino acids are reduced, it’s our natural diet and cooking creates harmful compounds.

1. Raw meat commonly contains pathogenic bacteria. Yes we are made of bacteria, but there are good strains and bad strains of bacteria. Ecoli, salmonella, listeria etc. are not good for you and are harmful and bad for human health. Which is why safety of eating raw meat also translates to the health outcomes.
  • Food safety incidents in the red meat industry: A review of foodborne disease outbreaks linked to the consumption of red meat and its products, 1991 to 2021
  • Prevalence and Antimicrobial Susceptibility of Foodborne Pathogens from Raw Livestock Meat in China, 2021
  • Parasite risks from raw meat-based diets for companion animals
  • Health Risks Associated with Meat Consumption: A Review of Epidemiological Studies
  • Foodborne Parasites and Their Complex Life Cycles Challenging Food Safety in Different Food Chains
  • Predictors of Eating Raw or Undercooked Meat, Poultry, Seafood, and Eggs among Older Adults
  • Retail Meat Analyzed for Parasites

There have been thousands of deaths proven to be caused by salmonella, ecoli, listeria etc. The only reason our ancestors could eat raw meat instead of cooked meat was because they had a much better stomach than humans today. We have been eating cooked meat for almost a million years. We have evolved to eat cooked meat and not raw anymore. It’s why you wouldn't drink river water now instead of filtered. Natural doesn’t also doesn’t always mean good.

2. Nutrients are lost. Yes they are lost, but not by a significant amount. With your evidence, you are boiling the food. You can cook your foods in different ways and preserve the nutrients well. It is easy to eat a diet of less than 2000 calories and hit all your micronutrients. Vitamin C in organs isn’t in significant amounts anyways, so the loss isn’t a big deal. The idea of nutrient deficiency by cooking your food is stupid. Our ancestors were more nutrients deficient than a modern human today anyways.



3. Cooking does create harmful compounds when burnt to a fucking crisp. If you lightly cook your food at low temperatures, the amount of AGE’s, HCAs and oxidation is so insignificant when you do this. That your phone is around the same level on the carcinogenic scale then lightly cooked meat. 😹


I don’t see how you are this blind, to not notice the harmful effects of eating raw meat. People die from eating raw meat, even from good sources. Vegetables and fruits are good for you. No, you won’t become nutrient deficient.
:feelsuhh:
Plenty of cooked meat eaters are deficient in B vitamins, minerals, and fat-soluble vitamins. Cooking destroys heat-sensitive nutrients, thiamin (B1) losses up to 70–100% in boiling (Gerber 2009), B12 degraded by heat (Bito 2018), folate destroyed. You claim it’s “not significant,” but then admit you need to eat more food to make up for it. That’s the opposite of efficient. It is a waste of money, bad for the environment, waste of time etc etc.

Bacteria
You throw “salmonella” like it’s a trump card, but ignore that the majority of foodborne outbreaks come from spinach, salad, and bagged greens (CDC 2015). If bacteria = unhealthy, then vegetables are statistically more dangerous. You still haven’t proven that humans “lost” their ability to handle raw meat. Our gastric pH is 1–2, same as scavengers (Beasley 2015). That is hard physiological data. You still haven't proven anything with raw meat and bacteria, not even proven if bacteria is the reason for illness?. I know meat contains bacteria, all meat does so do yourself a favour and skip prooving that, I want to show me why it would be bad.


You claim we’ve been cooking for “almost a million years.” Source? The CNN link you dropped admits the earliest controlled fire evidence is ~780,000 years and patchy at best. This isn't even proof of cooking it is proof of burning fish remains btw. Even if it were a million, that’s less than 5% of our evolutionary timeline. Raw was the baseline, cooking is the deviation. If you admit “nature isn’t always better,” then you just destroyed your own appeal to evolution. Pick one.


No need to mention calories as they don't apply to organisms? And as I mentioned why would you waste more resources and so on to compensate + more toxins. And no it was not my evidence it was your own evidence that said that the sous vide was the least harmfull and other cooking methods being harmfull. Again most modern people are nutrient deficient in nutrients such as b12 because of cooking.We are talking about raw meat being healthier so insignificance is not a thing, even if raw meat is 0,001%healthier it is still healthier so shut up.

Toxins
Cooking creates poisons, AGEs, HCAs, PAHs, lipid peroxides. All linked in the literature to cancer, insulin resistance, cardiovascular disease, and aging. Your defense is literally “light cooking makes them insignificant.” Okay, so you admit they exist. What is healthier: no poison or little poison? Exactly. Phones have nothing to do with food, stop embarrassing yourself.


You still haven’t produced a single randomized controlled human trial or meta-analysis showing cooked > raw. You cite epidemiology about “foodborne illness” but ignore that those studies also include salads, processed meats, and bad storage. That doesn’t prove that raw meat itself is unhealthy, only that the industrial food system is.

Also why do you mention vegetables and fruit? we are talking about meat. and if you want to I can also destroy you in that department but again humans don't eat leaves so you start off with the burden of proof, but let us finish the meat thing first I will be sure to remember.
 
:feelsuhh:
Plenty of cooked meat eaters are deficient in B vitamins, minerals, and fat-soluble vitamins. Cooking destroys heat-sensitive nutrients, thiamin (B1) losses up to 70–100% in boiling (Gerber 2009), B12 degraded by heat (Bito 2018), folate destroyed. You claim it’s “not significant,” but then admit you need to eat more food to make up for it. That’s the opposite of efficient. It is a waste of money, bad for the environment, waste of time etc etc.

Bacteria
You throw “salmonella” like it’s a trump card, but ignore that the majority of foodborne outbreaks come from spinach, salad, and bagged greens (CDC 2015). If bacteria = unhealthy, then vegetables are statistically more dangerous. You still haven’t proven that humans “lost” their ability to handle raw meat. Our gastric pH is 1–2, same as scavengers (Beasley 2015). That is hard physiological data. You still haven't proven anything with raw meat and bacteria, not even proven if bacteria is the reason for illness?. I know meat contains bacteria, all meat does so do yourself a favour and skip prooving that, I want to show me why it would be bad.


You claim we’ve been cooking for “almost a million years.” Source? The CNN link you dropped admits the earliest controlled fire evidence is ~780,000 years and patchy at best. This isn't even proof of cooking it is proof of burning fish remains btw. Even if it were a million, that’s less than 5% of our evolutionary timeline. Raw was the baseline, cooking is the deviation. If you admit “nature isn’t always better,” then you just destroyed your own appeal to evolution. Pick one.


No need to mention calories as they don't apply to organisms? And as I mentioned why would you waste more resources and so on to compensate + more toxins. And no it was not my evidence it was your own evidence that said that the sous vide was the least harmfull and other cooking methods being harmfull. Again most modern people are nutrient deficient in nutrients such as b12 because of cooking.We are talking about raw meat being healthier so insignificance is not a thing, even if raw meat is 0,001%healthier it is still healthier so shut up.

Toxins
Cooking creates poisons, AGEs, HCAs, PAHs, lipid peroxides. All linked in the literature to cancer, insulin resistance, cardiovascular disease, and aging. Your defense is literally “light cooking makes them insignificant.” Okay, so you admit they exist. What is healthier: no poison or little poison? Exactly. Phones have nothing to do with food, stop embarrassing yourself.


You still haven’t produced a single randomized controlled human trial or meta-analysis showing cooked > raw. You cite epidemiology about “foodborne illness” but ignore that those studies also include salads, processed meats, and bad storage. That doesn’t prove that raw meat itself is unhealthy, only that the industrial food system is.

Also why do you mention vegetables and fruit? we are talking about meat. and if you want to I can also destroy you in that department but again humans don't eat leaves so you start off with the burden of proof, but let us finish the meat thing first I will be sure to remember.
First of all, stop with the boiling. No one boils their meat. Methods like sous vide will not make a meaningful difference in the nutritional profile. All the studies with significant loss is frying or boiling. Sure, many people don’t use sous vide for cooking but that is completely irrelevant. And yes you need to eat more food to get some of those nutrients back, but you don’t go out of your way to do it, you do it naturally to eat enough calories.

Salmonella is found on plants and does cause more salmonella outbreaks. But think about how many people eat plants every day. Almost the entire population. People who eat raw meat willingly, there are only a couple thousand. So of course there would be more salmonella breakouts with vegetables, but that doesn’t mean it’s more risky. The risk for raw meat is still way higher just less common due to sensible people not eating raw meat. And we do still have reactions to ecoli, salmonella, listeria etc. even though scavengers have the same pH level, their short intestines make it easier to pass through the harmful bacteria, while humans have longer intestines which make it very hard to get harmful bacteria out of our system. The scavengers also contain a very differently gut microbiome to us which allows them to break down the bacteria easier. These bacteria strains are harmful to us…

"Escherichia coli Infection" from StatPearls
"Salmonella" in Medical Microbiology
Mayo Clinic's overview of Salmonella infection
"Listeria monocytogenes: epidemiology, human disease, and mechanisms of brain invasion"
"Human Listeriosis" in the journal Pathogens:
The Link to Guillain-Barré Syndrome (GBS)


Also please explain why evidence is “patchy”. The burning of fish is it being cooked, pretty simple. And you can pretty surprised at how quickly species evolve. We will adapt if we continue doing something for so long. My point on natural isn’t always good, doesn’t go against evolution. It’s stating that something that artificial (which cooking isnt) is not always bad.

Most people aren’t eating quality meats, fruits, vegetables and dairy. So it’s completely stupid to use them as an example and then label them as cooked meat eaters. They aren’t deficient in B12 because of cooking, it’s because they don’t eat enough animal products. “Even if raw meat is 0.00001% healthier it is healthier”, that doesn’t matter at all if it has a tiny bit more nutrients because of the significant risk of bacteria.

Yes the compounds formed are harmful, but those studies heat them at high temperatures for a long time. A little heat for little time won’t form many. It’s very ironic to mention poison or no poison, because raw meat has risk of food POISONING. The compounds harms are so insignificant that they wouldn’t take more than a week of your life, which just isn't worth the risk of harmful bacteria. I only refereed to phones because you still use them and they are about as dangerous as slightly cooking food. Showing how useless the point is.

I think I’ve repeated the point almost three times and you just seem to ignore it. There are no solid RCT or meta analysis on raw vs cooked because they can’t put volunteers at the risk of eating raw meat because there is such an abundant amount of evidence showing that it is harmful. Studies on food-borne illness involve lots of different foods, but raw meat still has harmful bacteria, you said you know yourself. All harmful bacteria acts the same in all foods. Bad storage? You mean stored in fridges?

Once you realise you’re wrong, let’s move onto vegetables and fruits. Please read everything and respond to it all.
 
First of all, stop with the boiling. No one boils their meat. Methods like sous vide will not make a meaningful difference in the nutritional profile. All the studies with significant loss is frying or boiling. Sure, many people don’t use sous vide for cooking but that is completely irrelevant. And yes you need to eat more food to get some of those nutrients back, but you don’t go out of your way to do it, you do it naturally to eat enough calories.

Salmonella is found on plants and does cause more salmonella outbreaks. But think about how many people eat plants every day. Almost the entire population. People who eat raw meat willingly, there are only a couple thousand. So of course there would be more salmonella breakouts with vegetables, but that doesn’t mean it’s more risky. The risk for raw meat is still way higher just less common due to sensible people not eating raw meat. And we do still have reactions to ecoli, salmonella, listeria etc. even though scavengers have the same pH level, their short intestines make it easier to pass through the harmful bacteria, while humans have longer intestines which make it very hard to get harmful bacteria out of our system. The scavengers also contain a very differently gut microbiome to us which allows them to break down the bacteria easier. These bacteria strains are harmful to us…

"Escherichia coli Infection" from StatPearls
"Salmonella" in Medical Microbiology
Mayo Clinic's overview of Salmonella infection
"Listeria monocytogenes: epidemiology, human disease, and mechanisms of brain invasion"
"Human Listeriosis" in the journal Pathogens:
The Link to Guillain-Barré Syndrome (GBS)


Also please explain why evidence is “patchy”. The burning of fish is it being cooked, pretty simple. And you can pretty surprised at how quickly species evolve. We will adapt if we continue doing something for so long. My point on natural isn’t always good, doesn’t go against evolution. It’s stating that something that artificial (which cooking isnt) is not always bad.

Most people aren’t eating quality meats, fruits, vegetables and dairy. So it’s completely stupid to use them as an example and then label them as cooked meat eaters. They aren’t deficient in B12 because of cooking, it’s because they don’t eat enough animal products. “Even if raw meat is 0.00001% healthier it is healthier”, that doesn’t matter at all if it has a tiny bit more nutrients because of the significant risk of bacteria.

Yes the compounds formed are harmful, but those studies heat them at high temperatures for a long time. A little heat for little time won’t form many. It’s very ironic to mention poison or no poison, because raw meat has risk of food POISONING. The compounds harms are so insignificant that they wouldn’t take more than a week of your life, which just isn't worth the risk of harmful bacteria. I only refereed to phones because you still use them and they are about as dangerous as slightly cooking food. Showing how useless the point is.

I think I’ve repeated the point almost three times and you just seem to ignore it. There are no solid RCT or meta analysis on raw vs cooked because they can’t put volunteers at the risk of eating raw meat because there is such an abundant amount of evidence showing that it is harmful. Studies on food-borne illness involve lots of different foods, but raw meat still has harmful bacteria, you said you know yourself. All harmful bacteria acts the same in all foods. Bad storage? You mean stored in fridges?

Once you realise you’re wrong, let’s move onto vegetables and fruits. Please read everything and respond to it all.
Alright I will be addressing everything but since you don't seem to be reading and understanding everything please read everything and also maybe read your own response before sending it lol
"First of all, stop with the boiling. No one boils their meat. Methods like sous vide will not make a meaningful difference in the nutritional profile. All the studies with significant loss is frying or boiling. Sure, many people don’t use sous vide for cooking but that is completely irrelevant"
You brought up the boiling and sous vide and you would know if you had read your "evidence" before sending it to me, not my fault. And as your "evidence" actually proves it is not "irrelevant" what method you choose as it greatly influences the degree of "unfavourable changes". All of this has been concluded on the basis of your own study my guy.

"And yes you need to eat more food to get some of those nutrients back, but you don’t go out of your way to do it, you do it naturally to eat enough calories."
And what is your point here? Adress my argument: The more cooked meat you eat to compensate for the nutrient loss the more harmfull compounds you consume, therefore more unhealthy than raw. The increase also leeds to increased negative impact on the environment and would cost more time and money. Either you agree or you make it clear to me that I am wrong and why. Furthermore stop talking about calories as they don't apply to organisms, how many times do I have to tell you, same thing with all my arguments you keep circling around like an idiot.

I think we can move on from the nutrient argument now, is that correct? We have established a loss in nutrients, increase in harmfull particles, cost more, worse for environment more time consuming and things I mentioned earlier from cooking (not directly health related but I think we can agree that environment and stress from money and time etc does influence health), so just shortly confirm in the next answer if you agree. Otherwise make a valid argument ofc.

next.

"
Salmonella is found on plants and does cause more salmonella outbreaks. But think about how many people eat plants every day. Almost the entire population. People who eat raw meat willingly, there are only a couple thousand. So of course there would be more salmonella breakouts with vegetables, but that doesn’t mean it’s more risky. The risk for raw meat is still way higher just less common due to sensible people not eating raw meat. And we do still have reactions to ecoli, salmonella, listeria etc. even though scavengers have the same pH level, their short intestines make it easier to pass through the harmful bacteria, while humans have longer intestines which make it very hard to get harmful bacteria out of our system. The scavengers also contain a very differently gut microbiome to us which allows them to break down the bacteria easier. These bacteria strains are harmful to us…

"Escherichia coli Infection" from StatPearls
"Salmonella" in Medical Microbiology
Mayo Clinic's overview of Salmonella infection
"Listeria monocytogenes: epidemiology, human disease, and mechanisms of brain invasion"
"Human Listeriosis" in the journal Pathogens:
The Link to Guillain-Barré Syndrome (GBS)

"

Why the fuck do you use the "less cases does not mean less risky" rebuttal and then link a study for vegetables. You are confusing case counts with actual relative risk. The CDC and EFSA outbreak data show that vegetables like sprouts, lettuce, melons and tomatoes are consistently the leading sources of Salmonella and E coli outbreaks in the modern world. That is not just because more people eat plants, it is because they are a highly vulnerable vector. If risk was purely proportional to how many eat them, the raw dairy and raw meat outbreaks would still dominate given the small niche that eats them. They do not.

Your citations are generic textbook descriptions of pathogens. They prove nothing about relative risk of raw vs cooked animal foods in healthy humans. Mayo Clinic and StatPearls are patient info summaries, not comparative studies. Drevets 2008 on Listeria is a mechanistic review of brain invasion pathways, not an argument that raw meat is uniquely unsafe. You are throwing scary pathogen names around without a single human trial comparing outcomes.

The scavenger argument is also weak. Humans do not have herbivore guts. Our intestines are short relative to body size and our gastric acid is at pH 1 to 2 in fasting state. That is a direct adaptation to handling animal tissue.

So to recap: you claim that just because there are less cases of raw meat being the reason of these pathogens than of vegetables it is still more risky but you did not prove this in any way so that point remains false. Then you talk about human anatomy and claim we have longer intenstines (like herbivores) without any proof, and other animals gut microbiome is irrelevant so let us just look at humans (yes I earlier compared humans to scavengers earlier because we are alike which I wanted to make you realise).

next.

"Also please explain why evidence is “patchy”. The burning of fish is it being cooked, pretty simple. And you can pretty surprised at how quickly species evolve. We will adapt if we continue doing something for so long. My point on natural isn’t always good, doesn’t go against evolution. It’s stating that something that artificial (which cooking isnt) is not always bad."

First off, one account of burned fish remains does not relate to cooked meat being healthier than raw meat. I will happily teach you why the evidence is utter shit for proving anything but I think it is completely irrelevant to the cooked meat being healthier than raw meat debate.
Secondly you need to learn to clearly state your point so that I can tell what you are talking about because you are mostly just rambling like a schizophrenic TikTok conspiracy theorist. It seems like you want to talk about nature and evolution? Just make this point clear in the next argument, maybe google how to argument too lol:ROFLMAO:


JFL cause ur an idiot:
"It’s stating that something that artificial (which cooking isnt:soy::soy::soy:) is not always bad.":feelsuhh:
This is the definition of artificial:
"made or produced by human beings rather than occurring naturally, especially as a copy of something natural."


"Most people aren’t eating quality meats, fruits, vegetables and dairy. So it’s completely stupid to use them as an example and then label them as cooked meat eaters(first claim). They aren’t deficient in B12 because of cooking, it’s because they don’t eat enough animal products(second claim). “Even if raw meat is 0.00001% healthier it is healthier”, that doesn’t matter at all if it has a tiny bit more nutrients because of the significant risk of bacteria."

Coherence level 0
First claim needs proof and when did I use them as an example.
Second claim also need proof, and how does this relate to the point again, I hope I don't need to remind you but you are trying to prove that cooked meat is healthier than raw.
And you cannot use the bacteria being risky argument here because you didn't prove it, also risk of food poisoning is also part of something being healthier, not admitting that bacteria is risky but that seemed like your point (any risk lowers health so no need to mention riskines making the healthier choice less advantageous)


"Yes the compounds formed are harmful, but those studies heat them at high temperatures for a long time. A little heat for little time won’t form many. It’s very ironic to mention poison or no poison, because raw meat has risk of food POISONING. The compounds harms are so insignificant that they wouldn’t take more than a week of your life, which just isn't worth the risk of harmful bacteria. I only refereed to phones because you still use them and they are about as dangerous as slightly cooking food. Showing how useless the point is."

Again, you need to show how raw meat is poisonous or risky to use the argument. You don't need to mention the poisonous effects of cooking any more as we have already established that the only thing you have left to prove is the risk of illness. It is easy to establish that the poisonous compounds from cooking makes it unhealthy therefore waste of time, doesn't matter if it is just as much as your phone or whatever, 0 is better than 0,1."Yes the compounds formed are harmful" then don't use the fucking argument.

"I think I’ve repeated the point almost three times and you just seem to ignore it. There are no solid RCT or meta analysis on raw vs cooked because they can’t put volunteers at the risk of eating raw meat because there is such an abundant amount of evidence showing that it is harmful. Studies on food-borne illness involve lots of different foods, but raw meat still has harmful bacteria, you said you know yourself. All harmful bacteria acts the same in all foods. Bad storage? You mean stored in fridges?"

1. you simply have the burden of proof so that is your problem.
2. show how rawmeat bacteria is harmfull. I don't think bacteria is bad.

Show me 1 single piece of evigende if "there is such an abundant amount of evidence showing that it is harmful", seems like a waste of time if there is sooooo much evidence right?:feelshah:


“So far you admitted nutrient losses exist, harmful compounds form with heat, and your only argument left is ‘risk’. But you have not posted a single piece of human evidence proving raw meat is uniquely risky compared to plants, dairy, or anything else. If you had the evidence you would have posted it already. Either bring real data or concede.”

what an utter disgrace to your genetic pool, must feel good
Please read everything and respond:lul:
 
Alright I will be addressing everything but since you don't seem to be reading and understanding everything please read everything and also maybe read your own response before sending it lol
"First of all, stop with the boiling. No one boils their meat. Methods like sous vide will not make a meaningful difference in the nutritional profile. All the studies with significant loss is frying or boiling. Sure, many people don’t use sous vide for cooking but that is completely irrelevant"
You brought up the boiling and sous vide and you would know if you had read your "evidence" before sending it to me, not my fault. And as your "evidence" actually proves it is not "irrelevant" what method you choose as it greatly influences the degree of "unfavourable changes". All of this has been concluded on the basis of your own study my guy.

"And yes you need to eat more food to get some of those nutrients back, but you don’t go out of your way to do it, you do it naturally to eat enough calories."
And what is your point here? Adress my argument: The more cooked meat you eat to compensate for the nutrient loss the more harmfull compounds you consume, therefore more unhealthy than raw. The increase also leeds to increased negative impact on the environment and would cost more time and money. Either you agree or you make it clear to me that I am wrong and why. Furthermore stop talking about calories as they don't apply to organisms, how many times do I have to tell you, same thing with all my arguments you keep circling around like an idiot.

I think we can move on from the nutrient argument now, is that correct? We have established a loss in nutrients, increase in harmfull particles, cost more, worse for environment more time consuming and things I mentioned earlier from cooking (not directly health related but I think we can agree that environment and stress from money and time etc does influence health), so just shortly confirm in the next answer if you agree. Otherwise make a valid argument ofc.

next.

"
Salmonella is found on plants and does cause more salmonella outbreaks. But think about how many people eat plants every day. Almost the entire population. People who eat raw meat willingly, there are only a couple thousand. So of course there would be more salmonella breakouts with vegetables, but that doesn’t mean it’s more risky. The risk for raw meat is still way higher just less common due to sensible people not eating raw meat. And we do still have reactions to ecoli, salmonella, listeria etc. even though scavengers have the same pH level, their short intestines make it easier to pass through the harmful bacteria, while humans have longer intestines which make it very hard to get harmful bacteria out of our system. The scavengers also contain a very differently gut microbiome to us which allows them to break down the bacteria easier. These bacteria strains are harmful to us…

"Escherichia coli Infection" from StatPearls
"Salmonella" in Medical Microbiology
Mayo Clinic's overview of Salmonella infection
"Listeria monocytogenes: epidemiology, human disease, and mechanisms of brain invasion"
"Human Listeriosis" in the journal Pathogens:
The Link to Guillain-Barré Syndrome (GBS)

"

Why the fuck do you use the "less cases does not mean less risky" rebuttal and then link a study for vegetables. You are confusing case counts with actual relative risk. The CDC and EFSA outbreak data show that vegetables like sprouts, lettuce, melons and tomatoes are consistently the leading sources of Salmonella and E coli outbreaks in the modern world. That is not just because more people eat plants, it is because they are a highly vulnerable vector. If risk was purely proportional to how many eat them, the raw dairy and raw meat outbreaks would still dominate given the small niche that eats them. They do not.

Your citations are generic textbook descriptions of pathogens. They prove nothing about relative risk of raw vs cooked animal foods in healthy humans. Mayo Clinic and StatPearls are patient info summaries, not comparative studies. Drevets 2008 on Listeria is a mechanistic review of brain invasion pathways, not an argument that raw meat is uniquely unsafe. You are throwing scary pathogen names around without a single human trial comparing outcomes.

The scavenger argument is also weak. Humans do not have herbivore guts. Our intestines are short relative to body size and our gastric acid is at pH 1 to 2 in fasting state. That is a direct adaptation to handling animal tissue.

So to recap: you claim that just because there are less cases of raw meat being the reason of these pathogens than of vegetables it is still more risky but you did not prove this in any way so that point remains false. Then you talk about human anatomy and claim we have longer intenstines (like herbivores) without any proof, and other animals gut microbiome is irrelevant so let us just look at humans (yes I earlier compared humans to scavengers earlier because we are alike which I wanted to make you realise).

next.

"Also please explain why evidence is “patchy”. The burning of fish is it being cooked, pretty simple. And you can pretty surprised at how quickly species evolve. We will adapt if we continue doing something for so long. My point on natural isn’t always good, doesn’t go against evolution. It’s stating that something that artificial (which cooking isnt) is not always bad."

First off, one account of burned fish remains does not relate to cooked meat being healthier than raw meat. I will happily teach you why the evidence is utter shit for proving anything but I think it is completely irrelevant to the cooked meat being healthier than raw meat debate.
Secondly you need to learn to clearly state your point so that I can tell what you are talking about because you are mostly just rambling like a schizophrenic TikTok conspiracy theorist. It seems like you want to talk about nature and evolution? Just make this point clear in the next argument, maybe google how to argument too lol:ROFLMAO:


JFL cause ur an idiot:
"It’s stating that something that artificial (which cooking isnt:soy::soy::soy:) is not always bad.":feelsuhh:
This is the definition of artificial:
"made or produced by human beings rather than occurring naturally, especially as a copy of something natural."


"Most people aren’t eating quality meats, fruits, vegetables and dairy. So it’s completely stupid to use them as an example and then label them as cooked meat eaters(first claim). They aren’t deficient in B12 because of cooking, it’s because they don’t eat enough animal products(second claim). “Even if raw meat is 0.00001% healthier it is healthier”, that doesn’t matter at all if it has a tiny bit more nutrients because of the significant risk of bacteria."

Coherence level 0
First claim needs proof and when did I use them as an example.
Second claim also need proof, and how does this relate to the point again, I hope I don't need to remind you but you are trying to prove that cooked meat is healthier than raw.
And you cannot use the bacteria being risky argument here because you didn't prove it, also risk of food poisoning is also part of something being healthier, not admitting that bacteria is risky but that seemed like your point (any risk lowers health so no need to mention riskines making the healthier choice less advantageous)


"Yes the compounds formed are harmful, but those studies heat them at high temperatures for a long time. A little heat for little time won’t form many. It’s very ironic to mention poison or no poison, because raw meat has risk of food POISONING. The compounds harms are so insignificant that they wouldn’t take more than a week of your life, which just isn't worth the risk of harmful bacteria. I only refereed to phones because you still use them and they are about as dangerous as slightly cooking food. Showing how useless the point is."

Again, you need to show how raw meat is poisonous or risky to use the argument. You don't need to mention the poisonous effects of cooking any more as we have already established that the only thing you have left to prove is the risk of illness. It is easy to establish that the poisonous compounds from cooking makes it unhealthy therefore waste of time, doesn't matter if it is just as much as your phone or whatever, 0 is better than 0,1."Yes the compounds formed are harmful" then don't use the fucking argument.

"I think I’ve repeated the point almost three times and you just seem to ignore it. There are no solid RCT or meta analysis on raw vs cooked because they can’t put volunteers at the risk of eating raw meat because there is such an abundant amount of evidence showing that it is harmful. Studies on food-borne illness involve lots of different foods, but raw meat still has harmful bacteria, you said you know yourself. All harmful bacteria acts the same in all foods. Bad storage? You mean stored in fridges?"

1. you simply have the burden of proof so that is your problem.
2. show how rawmeat bacteria is harmfull. I don't think bacteria is bad.

Show me 1 single piece of evigende if "there is such an abundant amount of evidence showing that it is harmful", seems like a waste of time if there is sooooo much evidence right?:feelshah:


“So far you admitted nutrient losses exist, harmful compounds form with heat, and your only argument left is ‘risk’. But you have not posted a single piece of human evidence proving raw meat is uniquely risky compared to plants, dairy, or anything else. If you had the evidence you would have posted it already. Either bring real data or concede.”

what an utter disgrace to your genetic pool, must feel good
Please read everything and respond:lul:
You just keep repeating the same point. Yes there is nutrients loss. Im admitting it. But there isn’t a significant amount. There is not significant amount of nutrients loss that a healthy individual who consumes a well balanced diet would become deficient. There just isn’t a problem with that. Yes it’s technically “unideal”. But unideal doesn’t mean bad. It’s unideal that meat does not have enough nutrients for an optimal diet by itself, but that doesn’t make it bad. The nutrient argument is completely irrelevant. Stop bringing it up. Our ancestors were more deficient than we were. Prove that wrong.

The more cooked meat you eat the more harmful compounds you consume. Again I literally just dismissed that the compounds created have such little effect on human health, even if it’s a little, it doesn’t contribute to a significant amount. And holy shit you are actually more brain dead than i thought. Calories don’t apply to organisms, please provide some solid research to back up that totally false claim. And we are talking about health, not the environment, give that up. A tiny, minuscule effect on the environment and maybe an extra 5$ a week wont affect your stress and health. Don’t even.

Salmonella & Ecoli are caused by the large amount of people who eat plants. Not by the risk. If you calculate the amount of people who eat plants and the amount that get sick. You would see that it is much lower to people who eat raw meat & dairy and get sick. It’s not a hard concept. There are no good studies comparing the effects of ecoli, salmonella, listeria etc from raw meat. But the effect of these strands are the same consumed from any type of organism. The studies I provided earlier prove undeniably that these harmful bacteria strands are bad for humans. Here are the same and some more:

  • "Human Health Hazards from Antimicrobial-Resistant Escherichia coli of Animal Origin" - Clinical Infectious Diseases - Oxford Academic
  • "E. coli: Infection, Causes, Symptoms & Treatment" - Cleveland Clinic
  • "Salmonellosis: An Overview of Epidemiology, Pathogenesis, and Innovative Approaches to Mitigate the Antimicrobial Resistant Infections" - PMC
  • "Salmonella (non-typhoidal)" - World Health Organization (WHO)
  • "Salmonella" - EFSA - European Union
  • "Listeria monocytogenes" - StatPearls - NCBI Bookshelf
  • "Listeria (Listeriosis)" - FDA
  • "Listeriosis" - World Health Organization (WHO)
  • "An Exploration of Listeria monocytogenes, Its Influence on the UK Food Industry and Future Public Health Strategies" - MDPI
  • "A systematic review of the methodological considerations in Campylobacter burden of disease studies" - Research journals - PLOS
  • "Campylobacteriosis: A rising threat in foodborne illnesses" - PMC
  • "Prevalence and risk factors associated with the occurrence of Campylobacter sp. in children aged 6–24 months in peri-urban Nairobi, Kenya" - Frontiers"Clinical Overview of Campylobacter" - CDC
Vultures, true scavengers, have a much stronger acid defense and a unique gut microbiome. A study in Nature Communications comparing the gut microbiomes of vultures and other birds showed that vultures have a highly specialized gut flora dominated by Clostridia and Fusobacteria. These bacteria, which are lethal to humans, help the vultures neutralize harmful pathogens in the carrion they consume. This is a crucial adaptation that humans completely lack.

Scavengers also eat their meat soon after their prey are killed or die. Meaning bacteria has less time to form if it’s not there already. Unlike humans who eat meat hours to days to even weeks after the slaughter of the animal.

The human digestive system is highly effective at breaking down the proteins and fats in fresh, clean meat. Our short intestines are efficient for nutrient absorption from such a diet. However, raw carrion is a fundamentally different food source, teeming with pathogens and toxins. A study in the American Journal of clinical nutrition shows that while our guts are carnivore-like, but this doesn't mean they are immune to bacteria like listeria and salmonella that proliferate in decomposing meat.

We can move on from the topic of when we started cooking our meat. But I only brought that up to show that we have evolved to eat cooked meat and not raw. I’m not trying to talk about nature and evolution. I was again just saying that we have evolved to eat cooked meat. But let’s stay on topic then and use actual science.

You said people in the world are deficient in B12 because of cooking their meat. Absolutely zero evidence towards this by the way. People are deficient because of their poor diet and not enough meat, regardless raw or cooked. I’m only saying this to show that cooked meat doesn’t mean you would be deficient.

If you believe these bacteria are not risky, can you provide a single peer-reviewed study or a medical consensus report that supports that claim? The evidence I've presented clearly shows the opposite.

I’ve provided numerous studies and more again this time, that show without a doubt that raw meat is risky due to presence of harmful bacteria. Yes a little toxin is worse than no toxin. But raw meat contains many poisonous when not cooked. I’ve showed the data, you just keep ignoring it. Try and disprove the evidence, go on bro.

No, because their is proof that raw meat is harmful to human in consumption. I have proved it, unless you have a good solid reason against all the evidence…

I’ve showed the evidence multiple times, look carefully through each one and tell me why it’s wrong.

Raw meat carries the same risk as raw dairy. I’ve already told you why raw meat is more risky than vegetables. It’s plain and simple. Less people eat raw meat, so of course there will be less cases. Thousands vs billions of who consume raw meat vs vegetables. The risk is high. You have already admitted that you acknowledge the bacteria is there, you are just saying it’s not harmful. So why are you now trying to say it’s hard to get the bacteria from it?

Stop going around and around.
 
You just keep repeating the same point. Yes there is nutrients loss. Im admitting it. But there isn’t a significant amount. There is not significant amount of nutrients loss that a healthy individual who consumes a well balanced diet would become deficient. There just isn’t a problem with that. Yes it’s technically “unideal”. But unideal doesn’t mean bad. It’s unideal that meat does not have enough nutrients for an optimal diet by itself, but that doesn’t make it bad. The nutrient argument is completely irrelevant. Stop bringing it up. Our ancestors were more deficient than we were. Prove that wrong.

The more cooked meat you eat the more harmful compounds you consume. Again I literally just dismissed that the compounds created have such little effect on human health, even if it’s a little, it doesn’t contribute to a significant amount. And holy shit you are actually more brain dead than i thought. Calories don’t apply to organisms, please provide some solid research to back up that totally false claim. And we are talking about health, not the environment, give that up. A tiny, minuscule effect on the environment and maybe an extra 5$ a week wont affect your stress and health. Don’t even.

Salmonella & Ecoli are caused by the large amount of people who eat plants. Not by the risk. If you calculate the amount of people who eat plants and the amount that get sick. You would see that it is much lower to people who eat raw meat & dairy and get sick. It’s not a hard concept. There are no good studies comparing the effects of ecoli, salmonella, listeria etc from raw meat. But the effect of these strands are the same consumed from any type of organism. The studies I provided earlier prove undeniably that these harmful bacteria strands are bad for humans. Here are the same and some more:

  • "Human Health Hazards from Antimicrobial-Resistant Escherichia coli of Animal Origin" - Clinical Infectious Diseases - Oxford Academic
  • "E. coli: Infection, Causes, Symptoms & Treatment" - Cleveland Clinic
  • "Salmonellosis: An Overview of Epidemiology, Pathogenesis, and Innovative Approaches to Mitigate the Antimicrobial Resistant Infections" - PMC
  • "Salmonella (non-typhoidal)" - World Health Organization (WHO)
  • "Salmonella" - EFSA - European Union
  • "Listeria monocytogenes" - StatPearls - NCBI Bookshelf
  • "Listeria (Listeriosis)" - FDA
  • "Listeriosis" - World Health Organization (WHO)
  • "An Exploration of Listeria monocytogenes, Its Influence on the UK Food Industry and Future Public Health Strategies" - MDPI
  • "A systematic review of the methodological considerations in Campylobacter burden of disease studies" - Research journals - PLOS
  • "Campylobacteriosis: A rising threat in foodborne illnesses" - PMC
  • "Prevalence and risk factors associated with the occurrence of Campylobacter sp. in children aged 6–24 months in peri-urban Nairobi, Kenya" - Frontiers"Clinical Overview of Campylobacter" - CDC
Vultures, true scavengers, have a much stronger acid defense and a unique gut microbiome. A study in Nature Communications comparing the gut microbiomes of vultures and other birds showed that vultures have a highly specialized gut flora dominated by Clostridia and Fusobacteria. These bacteria, which are lethal to humans, help the vultures neutralize harmful pathogens in the carrion they consume. This is a crucial adaptation that humans completely lack.

Scavengers also eat their meat soon after their prey are killed or die. Meaning bacteria has less time to form if it’s not there already. Unlike humans who eat meat hours to days to even weeks after the slaughter of the animal.

The human digestive system is highly effective at breaking down the proteins and fats in fresh, clean meat. Our short intestines are efficient for nutrient absorption from such a diet. However, raw carrion is a fundamentally different food source, teeming with pathogens and toxins. A study in the American Journal of clinical nutrition shows that while our guts are carnivore-like, but this doesn't mean they are immune to bacteria like listeria and salmonella that proliferate in decomposing meat.

We can move on from the topic of when we started cooking our meat. But I only brought that up to show that we have evolved to eat cooked meat and not raw. I’m not trying to talk about nature and evolution. I was again just saying that we have evolved to eat cooked meat. But let’s stay on topic then and use actual science.

You said people in the world are deficient in B12 because of cooking their meat. Absolutely zero evidence towards this by the way. People are deficient because of their poor diet and not enough meat, regardless raw or cooked. I’m only saying this to show that cooked meat doesn’t mean you would be deficient.

If you believe these bacteria are not risky, can you provide a single peer-reviewed study or a medical consensus report that supports that claim? The evidence I've presented clearly shows the opposite.

I’ve provided numerous studies and more again this time, that show without a doubt that raw meat is risky due to presence of harmful bacteria. Yes a little toxin is worse than no toxin. But raw meat contains many poisonous when not cooked. I’ve showed the data, you just keep ignoring it. Try and disprove the evidence, go on bro.

No, because their is proof that raw meat is harmful to human in consumption. I have proved it, unless you have a good solid reason against all the evidence…

I’ve showed the evidence multiple times, look carefully through each one and tell me why it’s wrong.

Raw meat carries the same risk as raw dairy. I’ve already told you why raw meat is more risky than vegetables. It’s plain and simple. Less people eat raw meat, so of course there will be less cases. Thousands vs billions of who consume raw meat vs vegetables. The risk is high. You have already admitted that you acknowledge the bacteria is there, you are just saying it’s not harmful. So why are you now trying to say it’s hard to get the bacteria from it?

Stop going around and around.

You brought up the nutrient argument in the first place, actually i think it was your first line of defence. We established that nutritionally raw meat is healthier and I gave you the opportunity to agree, now you have and I will not bring it up again and so should you not. Why would you mention the ancestors being deficient in this debate? We are debating if cooked meat is healthier than raw meat, so I don’t see the relevance, you can elaborate if you think it will prove your point, otherwise I don’t give a fuck what you think about my ancestors.


Harmful compounds argument clearly shows that raw meat is healthier, end of discussion ecxept if you can prove that >0 of all toxic and poisonous products of cooking is healthier than 0. Otherwise, do not bring it up and do not say it is irrelevant, just agree or give the proof of the earlier mentioned.


Unrelated to the point but you requested me to explain what calories are: Calories measure heat from burning food in a closed chamber. The body isn’t a closed firebox but an open biochemical system, so the theory does not apply. To elaborate, when you eat the food does not enter a chamber where the food is burned and water is heated and from there energy extracted, this is because organisms are open biochemical systems not closed systems like combustion chambers. If you think it in any way relates to your point you can mention that, otherwise don’t waste my time as this is very simple science and I don’t care about your opinion. bomb calorimeter calories ≠ human metabolism

Health is highly dependent on the environment. More meat means negative environment impact. More negative environment impact means less health



Maybe you don’t have a problem paying an extra 5$ a week but some people live on under 2,15$ a day (including everything), you clearly don’t know how much some people struggle even in richer countries. Health is highly dependent on stress. More money more stress. More stress less health.



“Salmonella & Ecoli are caused by the large amount of people who eat plants”. Bacteria are not caused by the size of people who eat plants, bacteria are microorganisms, and I don’t think you could really say they are caused by anyone? The effects of consumption of strands cannot be generalized for all organisms unless proof is provided from you as it is your claim, and it is irrelevant to mention.



I don’t see how vultures relate to your point in any way. Why do you state that humans lack the adaptation of having Clostridia and Fusobacteria in their gut flora being crucial? Is your argument that raw meat is unhealthy because humans are not adapted to the gut flora of vultrues? If that is not your argument then tell me what it is except if it is unrelated to the main point.



What is ”raw carrion” and why do you mention it? What does ”teeming wich pathogens and toxins” mean and why is it relevant to our debate?



Link to the study ” A study in the American Journal of clinical nutrition shows that while our guts are carnivore-like, but this doesn't mean they are immune to bacteria like listeria and salmonella that proliferate in decomposing meat.”





You claim that b12 deficiency stems from poor diet (which is???) and not enough meat. First of you did not show evidence of this. Second of a poor diet is most likely a less heathy diet (I amt trying to prove something being healthier than what you are claiming, see the relation?)?, Third you mention not enough meat, would getting 50% more b12 from the same amount of meat not result in less deficiency, making not enough meat a bad argument and making the fact that nutrient deficient meat could be the reason? Also modern medicine and cofactors play a role in the absorption of b12. In summary, yes people are deficient from cooking, simple equation:

A person who eats 100 g of raw meat containing 2 µg of B12 → could reach the recommended daily intake (~2.4 µg).
If the same meat is cooked and loses, say, 40% → only 1.2 µg remains, which is below the recommendation.
Over time this can lead to a gradual deficiency, even though the person technically “eats animal food.”



Your point is that people in the world are not deficient because of cooking, i would say that people who eat 100g of cooked beef daily on a regular basis exist and those people would get deficient.



“If you believe these bacteria are not risky, can you provide a single peer-reviewed study or a medical consensus report that supports that claim?”You cannot seriously think bacteria are risky? 90% of the body is bacteria, is that risky? also have to disprove asymptomatic carrier state with ecoli salmonella etc.



If you beleve that i did not fuck your mom, can you provide a single peer-reviewed study or a medical consensus report that supports that claim? Otherwise the opposite must be true right

”But raw meat contains many poisonous when not cooked”. Grammatically challenged sentencene but i think you mean raw meat contains posion, if so name one.


Why do you mention raw dairy? Are you saying that babies should not be fed raw breast milk but instead pasteurised breast milk, which is raw dary, OH NO RAW IT MUST BE SO BAD THE BABY COULD DIE FROM INVISIBLE BACTERIA AND POISON LET US REMOVE THE NUTRIENTS SO IT BECOMES A BLACPILLED SUB5 INCEL WHEN IT GROWS UP (it wont literraly grow because of the lack of nutrients, you cannot build a house without the materials and the same goes for a pretty scull). Bacteria is necessary for life so of course it is not hard to get it and i did not claim that.

Your sources only describe hazards and outbreaks. Presence of a bacterium in patients or food is association, not proof of cause. Healthy humans often carry Salmonella and E. coli without symptoms. Unless Koch’s postulates are fulfilled in humans, you cannot jump from “detected” to “caused.” None of your citations provide comparative human risk per serving of raw versus cooked meat. At best, they show correlation and hazard potential, not direct evidence that raw meat bacteria are inherently pathogenic.



You keep spamming names of bacteria as if the word “Salmonella” or “E. coli” is an automatic checkmate, but you clearly don’t even understand the basics of microbiology. Every single organism you listed is either a natural commensal in the human body or an environmental symbiont that most people carry without ever getting sick.

E. coli? It lives in all humans from birth, makes vitamin K, competes against actual pathogens and keeps your gut stable. Remove it and you literally die. Only a few mutated lab-selected strains (like O157:H7) are problematic, and even those only cause illness in the presence of other stressors.

Salmonella? Tens of millions of people are asymptomatic carriers. It sits in the gut and only becomes an issue if your immune system is already compromised. Calling Salmonella “bad” by definition is like calling firemen guilty of arson just because they show up at fires.

Listeria? It’s everywhere in soil, plants, water, animals, and yes, in us. Healthy people handle it fine because it’s just part of the microbial environment. It only becomes a problem in extreme edge cases (pregnancy, immunodeficiency). To label it as inherently “dangerous” is dishonest fear-mongering.

Campylobacter? Same story. Found in tons of healthy individuals without symptoms. Again, only becomes a problem under certain conditions like massive overgrowth or poor gut balance. Without constant low-level exposure to microbes like these, our immune system literally atrophies – this is the hygiene hypothesis, accepted even in mainstream science.

So what exactly are you proving with your list? That humans live in symbiosis with bacteria and that our health depends on them? Because that’s exactly what the evidence says. What you have not shown is a single human study proving that these bacteria, when present in raw meat, automatically cause disease in healthy individuals. You’re just listing names out of a textbook and pretending that proves causation. It doesn’t.

Unless you can demonstrate in humans that raw-meat bacteria are uniquely and causally pathogenic (not just correlated, not just detected), your entire “safety” argument collapses. Right now you’re literally arguing against human physiology itself.
 
i just had my first ever raw meat it was tartar 200 grams just raw out of the package i wanna start eating other raw animal food like liver for example, is there anyone that can guide me, and tell me how they went about it, im 16 years old.
when eatin raw liver for the first time you will maybe feel a little bit dizzy thats because ur body hasnt recognized your natural diet yet and watch it as a threat, and you dont need that much raw liver because its so nutritous and u will get full fast
 
Vegetables and fruits are good for you. No, you won’t become nutrient deficient.
 
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Reactions: WaterDog
You brought up the nutrient argument in the first place, actually i think it was your first line of defence. We established that nutritionally raw meat is healthier and I gave you the opportunity to agree, now you have and I will not bring it up again and so should you not. Why would you mention the ancestors being deficient in this debate? We are debating if cooked meat is healthier than raw meat, so I don’t see the relevance, you can elaborate if you think it will prove your point, otherwise I don’t give a fuck what you think about my ancestors.


Harmful compounds argument clearly shows that raw meat is healthier, end of discussion ecxept if you can prove that >0 of all toxic and poisonous products of cooking is healthier than 0. Otherwise, do not bring it up and do not say it is irrelevant, just agree or give the proof of the earlier mentioned.


Unrelated to the point but you requested me to explain what calories are: Calories measure heat from burning food in a closed chamber. The body isn’t a closed firebox but an open biochemical system, so the theory does not apply. To elaborate, when you eat the food does not enter a chamber where the food is burned and water is heated and from there energy extracted, this is because organisms are open biochemical systems not closed systems like combustion chambers. If you think it in any way relates to your point you can mention that, otherwise don’t waste my time as this is very simple science and I don’t care about your opinion. bomb calorimeter calories ≠ human metabolism

Health is highly dependent on the environment. More meat means negative environment impact. More negative environment impact means less health



Maybe you don’t have a problem paying an extra 5$ a week but some people live on under 2,15$ a day (including everything), you clearly don’t know how much some people struggle even in richer countries. Health is highly dependent on stress. More money more stress. More stress less health.



“Salmonella & Ecoli are caused by the large amount of people who eat plants”. Bacteria are not caused by the size of people who eat plants, bacteria are microorganisms, and I don’t think you could really say they are caused by anyone? The effects of consumption of strands cannot be generalized for all organisms unless proof is provided from you as it is your claim, and it is irrelevant to mention.



I don’t see how vultures relate to your point in any way. Why do you state that humans lack the adaptation of having Clostridia and Fusobacteria in their gut flora being crucial? Is your argument that raw meat is unhealthy because humans are not adapted to the gut flora of vultrues? If that is not your argument then tell me what it is except if it is unrelated to the main point.



What is ”raw carrion” and why do you mention it? What does ”teeming wich pathogens and toxins” mean and why is it relevant to our debate?



Link to the study ” A study in the American Journal of clinical nutrition shows that while our guts are carnivore-like, but this doesn't mean they are immune to bacteria like listeria and salmonella that proliferate in decomposing meat.”





You claim that b12 deficiency stems from poor diet (which is???) and not enough meat. First of you did not show evidence of this. Second of a poor diet is most likely a less heathy diet (I amt trying to prove something being healthier than what you are claiming, see the relation?)?, Third you mention not enough meat, would getting 50% more b12 from the same amount of meat not result in less deficiency, making not enough meat a bad argument and making the fact that nutrient deficient meat could be the reason? Also modern medicine and cofactors play a role in the absorption of b12. In summary, yes people are deficient from cooking, simple equation:

A person who eats 100 g of raw meat containing 2 µg of B12 → could reach the recommended daily intake (~2.4 µg).
If the same meat is cooked and loses, say, 40% → only 1.2 µg remains, which is below the recommendation.
Over time this can lead to a gradual deficiency, even though the person technically “eats animal food.”



Your point is that people in the world are not deficient because of cooking, i would say that people who eat 100g of cooked beef daily on a regular basis exist and those people would get deficient.



“If you believe these bacteria are not risky, can you provide a single peer-reviewed study or a medical consensus report that supports that claim?”You cannot seriously think bacteria are risky? 90% of the body is bacteria, is that risky? also have to disprove asymptomatic carrier state with ecoli salmonella etc.



If you beleve that i did not fuck your mom, can you provide a single peer-reviewed study or a medical consensus report that supports that claim? Otherwise the opposite must be true right

”But raw meat contains many poisonous when not cooked”. Grammatically challenged sentencene but i think you mean raw meat contains posion, if so name one.


Why do you mention raw dairy? Are you saying that babies should not be fed raw breast milk but instead pasteurised breast milk, which is raw dary, OH NO RAW IT MUST BE SO BAD THE BABY COULD DIE FROM INVISIBLE BACTERIA AND POISON LET US REMOVE THE NUTRIENTS SO IT BECOMES A BLACPILLED SUB5 INCEL WHEN IT GROWS UP (it wont literraly grow because of the lack of nutrients, you cannot build a house without the materials and the same goes for a pretty scull). Bacteria is necessary for life so of course it is not hard to get it and i did not claim that.

Your sources only describe hazards and outbreaks. Presence of a bacterium in patients or food is association, not proof of cause. Healthy humans often carry Salmonella and E. coli without symptoms. Unless Koch’s postulates are fulfilled in humans, you cannot jump from “detected” to “caused.” None of your citations provide comparative human risk per serving of raw versus cooked meat. At best, they show correlation and hazard potential, not direct evidence that raw meat bacteria are inherently pathogenic.



You keep spamming names of bacteria as if the word “Salmonella” or “E. coli” is an automatic checkmate, but you clearly don’t even understand the basics of microbiology. Every single organism you listed is either a natural commensal in the human body or an environmental symbiont that most people carry without ever getting sick.

E. coli? It lives in all humans from birth, makes vitamin K, competes against actual pathogens and keeps your gut stable. Remove it and you literally die. Only a few mutated lab-selected strains (like O157:H7) are problematic, and even those only cause illness in the presence of other stressors.

Salmonella? Tens of millions of people are asymptomatic carriers. It sits in the gut and only becomes an issue if your immune system is already compromised. Calling Salmonella “bad” by definition is like calling firemen guilty of arson just because they show up at fires.

Listeria? It’s everywhere in soil, plants, water, animals, and yes, in us. Healthy people handle it fine because it’s just part of the microbial environment. It only becomes a problem in extreme edge cases (pregnancy, immunodeficiency). To label it as inherently “dangerous” is dishonest fear-mongering.

Campylobacter? Same story. Found in tons of healthy individuals without symptoms. Again, only becomes a problem under certain conditions like massive overgrowth or poor gut balance. Without constant low-level exposure to microbes like these, our immune system literally atrophies – this is the hygiene hypothesis, accepted even in mainstream science.

So what exactly are you proving with your list? That humans live in symbiosis with bacteria and that our health depends on them? Because that’s exactly what the evidence says. What you have not shown is a single human study proving that these bacteria, when present in raw meat, automatically cause disease in healthy individuals. You’re just listing names out of a textbook and pretending that proves causation. It doesn’t.

Unless you can demonstrate in humans that raw-meat bacteria are uniquely and causally pathogenic (not just correlated, not just detected), your entire “safety” argument collapses. Right now you’re literally arguing against human physiology itself.
Okay fine, let’s stop going on about the nutritional content and the compounds created. I think we both agree that nutrients are lost and harmful compounds are created but they make no significant impact. Again, i only brought up ancestors because that’s one reason you stated for us to eat raw meat, because our ancestors naturally did. But let’s move on then.

Yes calories are slightly irrelevant to what we are talking about, but unless you can find one study or research paper that shows calories don’t actually matter when tracked and measured. Don’t say they apply to organisms. Unless you can find one. Don’t bother talking about it. Here are some studies to prove they do work in humans through food and exercise :

  • "Energy expenditure by doubly labeled water: validation in humans and proposed calculation" American Journal of Physiology (1986).
  • "Discrepancy between self-reported and actual caloric intake and exercise in obese subjects." New England Journal of Medicine (1992).
  • "Energy expenditure by doubly labeled water: validation in lean and obese subjects" American Journal of Physiology. Endocrinology and Metabolism (1991).
  • "Predicting metabolic adaptation, body weight change, and energy intake in humans" PLoS Computational Biology(2010).
  • "Doubly Labeled Water Is a Validated and Verified Reference Standard in Nutrition Research" The Journal of Nutrition (2014).
  • "Indirect Calorimetry in Clinical Practice" Nutrients (2019).
  • "The energy balance model of obesity: beyond calories in, calories out" Nature Reviews Endocrinology (2022).
  • "Analysis of energy metabolism in humans: A review of methodologies" Nutrition and Metabolism (2016).
  • "Validation of the doubly labeled water method using off-axis integrated cavity output spectroscopy and isotope ratio mass spectrometry" American Journal of Physiology. Endocrinology and Metabolism (2018).
  • "Energy expenditure measurements are reproducible in different whole‐room indirect calorimeters in humans"Clinical and Translational Science (2022).
  • "Energy Value of Foods" USDA Agriculture Handbook No. 74 (1955).
  • "Physical activity and physical activity induced energy expenditure in humans: measurement, determinants, and effects" Frontiers in Physiology (2013).

Stop talking about the effect on the environment and money. It doesn’t make a big impact. And doesn’t relate to if raw meat or cooked meat is healthier. Leave it out.

I was talking about vultures because you mentioned that if we have the same pH stomach acid as scavengers then we should be able to handle raw meat. And I just simply responded by saying how we lack what scavengers have so we therefore can’t safely eat raw meat. There is no denying that evidence. These are only relevant because you brought up scavengers and how it “proves” that we can safely eat raw meat.

  • Meat Consumption and Gut Microbiota: a Scoping Review of Literature and Systematic Review of Randomized Controlled Trials in Adults (Published in AJCN, 22 May 2023)
  • A prospective study of meat intake and gut microbiota in US women (Published in AJCN, May 2021)
B12 deficiency is from a poor diet of candy, junk food, chips etc. There is so much evidence to suggest that we consume way too much of this stuff:

  • A Systematic Review of Worldwide Consumption of Ultra-Processed Foods: Findings and Criticisms (Nutrients, 2021).
  • Ultra-processed food staples dominate mainstream U.S. supermarkets. Americans more than Europeans forced to choose between health and cost (medRxiv, 2024).
  • Trends in Consumption of Ultra-Processed Foods Among Adults in Southern China: Analysis of Serial Cross-Sectional Health Survey Data 2002–2022 (Nutrients, 2024).

Yeah people could eat less meat and get your B12, but B12 is such an easy nutrient to get through meat that if your diet is based around meat or only even 10% you would get your B12. If you have some common sense you realise that if people are actively trying to be healthy they will eat enough meat to get plenty of B12. Modern people you can’t use as an example, because they eat terrible quality meat and it’s because they don’t eat enough. You can’t hit all your RDI’s from 100g of beef in one day anyways. Raw or cooked.


I said : “If you believe these bacteria are not risky, can you provide a single peer-reviewed study or a medical consensus report that supports that claim?”

“You cannot seriously think bacteria are risky? 90% of the body is bacteria, is that risky? also have to disprove asymptomatic carrier state with ecoli salmonella etc”

Notice you didn’t reply with a study 😹. If you can’t reply with a study next time, don’t bother, you would have officially lost the argument. Yes our body is made of bacteria. But if yo listened to what i said the first time when I replied to this. Our body is made of good and bad bacteria. Some is good for us and some is bad.

  • The Human Gut Microbiome in Health and Disease (The New England Journal of Medicine, 2013)
  • Gut microbiota: the neglected organ and its role in immune and inflammatory conditions (British Medical Bulletin, 2015)
  • An altered gut microbiota in individuals with type 2 diabetes is associated with more efficient energy harvest(Genome Medicine, 2012)
  • Role of commensal bacteria in the regulation of intestinal inflammation (Trends in Immunology, 2007)
  • Role of Lactobacillus and Bifidobacterium in human health (International Dairy Journal, 2004)
  • The human intestinal Bacteroides genus: a functional analysis (Current Opinion in Microbiology, 2006)
  • Clostridium difficile Infection: Mechanisms, Epidemiology, and Interventions (Clinical Infectious Diseases, 2008)
  • Microbial ecology: Helicobacter pylori and the human gut microbiota (Nature Reviews Gastroenterology & Hepatology, 2015)
  • Probiotics and prebiotics: effects on diarrhea (The Journal of Nutrition, 2007)
  • Dietary modulation of the human gut microbiota: an update for the clinician (The Journal of Clinical Investigation, 2018)
  • Pathogenesis of Escherichia coli infections (Clinical Science (London), 1998)
  • Butyrate and its implications for human health (Current Opinion in Clinical Nutrition and Metabolic Care, 22)* (2019)



Raw meat contains ecoli, salmonella, listeria, campylobacter etc. These are essentially poisonous, because they are what cause food poisonings…

I only mentioned raw milk regarding statistics. And if you had basic knowledge in this topics you would know that raw milk is not sterile and is meant for calves. Breast milk is basically sterile because it contains microbes, and has bacteria made for the baby.

  • The microbiota of human milk and their implications for infant health (Cell Host & Microbe, 2017)
  • Human Milk: A Source of Microbes and Developmental Signals for the Neonate (Pediatric Research, 2017)
  • Diversity of the human milk microbiome and its association with maternal and infant factors (The American Journal of Clinical Nutrition, 2016)
  • Microbial composition of human breast milk: Tapping into a reservoir of potential probiotics (The New England Journal of Medicine, 2013)
  • Evidence of a bacterial transfer from the mother's mouth to the infant's gut via breast milk (Pediatric Research, 2009)

Raw milk is also harmful but that’s something else we can talk about later…

  • Risk of Human Illness Associated with the Consumption of Raw Milk and Raw Milk Cheeses (Clinical Infectious Diseases, 2011)
  • Raw milk associated outbreaks of infectious disease in the United States (Emerging Infectious Diseases, 2010)
  • Microbiological quality and safety of raw milk in retail markets in the United States (Journal of Food Protection, 22)* (2015)
  • Bacterial pathogens in bulk tank milk from US dairy farms: a systematic review and meta-analysis (Preventive Veterinary Medicine, 2017)
  • A review of the microbiological hazards of raw milk (International Journal of Food Microbiology, 2004)

Yes bacteria is needed to grow, but that’s good bacteria not harmful and pathogenic bacteria. This is basic knowledge by now. There is no proof that pathogenic bacteria helps us grow, it can actually stunt our growth.

This just proves that you’re lying and you’re ignorant. The studies show that these bacteria are harmful, regardless of where they come from. You said you already admitted this, you do not deny the presence of these pathogens.

Ecoli found in all foods are pathogenic. No denying it, look through the multiple studies I provided. We have non pathogenic ecoli found in our body. But they are different strands found in food. The ones found in food are natural, not lab mutated.

its the dose of salmonella, people do carry salmonella already but in tiny amounts that aren’t found in food. When a person eats food that has the presence of salmonella it would be in high amounts, causing the symptoms of salmonella.

Listeria is found in the ground. Well done! That doesn’t prove anything. Listeria isnt very harmful to healthy people. But still is harmful, like you said yourself, little poison or no poison. And it’s extremely harmful to young, old or pregnant people. It is dangerous, just like your argument with the PHA’s, AGE’s etc. Even though this is way more significant.

It's absolutely true that our immune systems need to be "trained" by constant, low-level exposure to a diverse microbial world—this is the meaning of the hypothesis. However, its wrong to say "training" microbes with pathogens like Campylobacter. Our immune system is primarily trained by beneficial and environmental microbes that are non-disease-causing, not by foodborne pathogens. Campylobacter is not a bacteria our body wants to use. While some people may carry it asymptomatically, it is still classified as a pathogen because its biological purpose is to cause disease in a susceptible host. Relying on exposure to a bacterium that is the leading cause of bacterial foodborne illness globally, and a known trigger for a severe paralyzing condition like Guillian Barre syndrome, is like arguing that you should regularly practice crossing a busy highway to keep their reflexes sharp. Good argument! You get the training you need from sidewalk navigation, not by inviting catastrophic risk.

We can't show a single study where scientists deliberately infect a healthy person to prove that contaminated raw meat automatically causes illness, as that would be unethical and not allowed. So public health experts rely on Quantitative Risk Assessment—

Risk Assessment for Escherichia coli O157:H7 in Ground Beef and the methods described in Quantitative Risk Assessment: ”
An Emerging Tool for Emerging Foodborne Pathogens, proves the cause-and-effect relationship, showing that the risk is not just an assumption, but a verifiable number that essentially disappears the moment that meat is cooked to a safe temperature”

Again raw meat pathogens is the same as any other plant, seafood pathogen. You can’t just give someone a pathogen for a study.

Before you respond:

Find a study that shows that these pathogenic bacteria have no negative effects on humans.

Prove that calories don’t work when tracked and measured in humans.

You keep on making up false points by using “logic” which is outdated and misrepresented. You are just essentially lying every time you talk, and to yourself. I know you will never admit to being wrong because you are too embarrassed and ignorant. Unless you can find studies proving you are right, you shouldn’t respond. You just take one study out of over 10 I provide, say something idiotic that has no meaning and easily proven wrong. This is what we have been doing back and forth. You just make so many small little lies and misinformation and drive away from the real point. JFL
 
Dnr shr rthread
 
Relevant:
I can very easily prove that bacteria are not risky but to do something as pointless you have to realise how much of a fucking idiot you have to be to actually request something like that, you should really reconsider something fundamental in your life at this point. You even admitted it yourself already btw. Let me begin:

Your point: “bacteria are not risky"

1. First of I will show you where you already admitted that bacteria are not risky yourself: “Our body is made of good and bad bacteria”, “Yes bacteria is needed to grow”, “It's absolutely true that our immune systems need to be "trained" by constant, low-level exposure to a diverse microbial world”

How can good bacteria be risky? And not related to yourself sabotage but, our body is “made” of bad bacteria?
You even linked a lot of studies yourself proving it so this whole answer is useless, how stupid can you be?

2. Burden of proof

You have the burden of proof because humans are heavily dependent on bacteria:

(Sender et al. 2016, Cell).

(Koh et al. 2016, Nat Rev Gastroenterol Hepatol)

(Bentley & Meganathan 1982, Microbiol Rev)

(Strachan 1989, BMJ)

(Cryan & Dinan 2012, Nat Rev Neurosci)

(Stein et al. 2016, NEJM)


You keep demanding “one peer-reviewed study” proving bacteria aren’t risky. That’s the dumbest framing I’ve ever read. You’re literally asking me to prove that oxygen isn’t poisonous in normal doses. The burden is on you to show that ubiquitous commensals like E. coli or Salmonella automatically cause disease in healthy humans. Otherwise you are just fear-mongering. “Good vs bad bacteria” is not science, it’s baby talk. The same species can be harmless in one host and helpful in another, and only cause trouble under stress conditions. That’s why asymptomatic carriage of Salmonella, Campylobacter, Listeria, and E. coli is well-documented in tens of millions of people. If they were inherently “bad,” those carriers would all be dead. They’re not.


1. All your “good vs bad” talk collapses the second you read your own citations. Every single one shows that microbes are context-dependent. Lactobacillus and Bifidobacterium are “good” because they balance the system – but in the wrong context even they can translocate and cause problems. Same with E. coli, Salmonella, Listeria, Campylobacter. They are not inherently poisonous, they are normal environmental and commensal species. Whether they cause symptoms depends on host health, dose, strain, and environment. That’s the entire point of microbiome science.

2. Your own NEJM review (2013) literally calls the microbiome a functional organ. If bacteria are an organ, then by your logic we should label our own liver or kidneys as “poisonous” just because organ failure sometimes kills people. Dumb framing.

3. Asymptomatic carriage is the nail in your coffin. Tens of millions of humans carry “your” scary names right now with zero symptoms. That fact alone destroys the “essentially poisonous” claim. If something is inherently poisonous, it kills on contact. These bacteria clearly do not. They only flip pathogenic under extreme conditions (immunodeficiency, imbalance, lab-selected strains like O157:H7).

4. You haven’t shown a single RCT or prospective human trial where healthy people eating hygienic raw meat get sick from these organisms. Presence ≠ poison. Koch’s postulates 101. Listing bacteria names from PubMed does not prove causation in humans.


You have to be academically illiterate if you actually think the burden of proof is on me to show bacteria are risky.


“Raw meat contains ecoli, salmonella, listeria, campylobacter etc. These are essentially poisonous, because they are what cause food poisonings…”
- Prove it


” Listeria is found in the ground. Well done! That doesn’t prove anything. Listeria isnt very harmful to healthy people. But still is harmful, like you said yourself, little poison or no poison. And it’s extremely harmful to young, old or pregnant people. It is dangerous, just like your argument with the PHA’s, AGE’s etc. Even though this is way more significant.”

- Okay well done yourself, it is irrelevant to raw meat then haha



You are confusing labels with reality. Calling some microbes “good” and others “bad” is not science, it’s kindergarten talk. The exact same species can be harmless, beneficial or problematic depending on host state, dose and context. That’s why asymptomatic carriage of Salmonella, Campylobacter, Listeria and E. coli is documented in tens of millions of healthy people. If they were “essentially poisonous” like you keep saying, all those carriers would be sick or dead. They’re not.

E. coli – Wrong again. Not all E. coli “found in food” are pathogenic. O157:H7 is one mutated lab-selected strain, most are harmless commensals or even protective. The human body depends on E. coli for vitamin K synthesis and for blocking pathogen overgrowth. To call the entire genus “poison” is just ignorance.

Salmonella – “Only in tiny amounts”? No. Studies show long-term asymptomatic carriers excreting significant loads without symptoms. The variable is not presence vs absence, but host immune status and gut balance. Presence ≠ disease.

Listeria – You literally admit it: “not very harmful to healthy people.” Thanks for proving my point. That means your own example is irrelevant for raw meat safety in healthy individuals. Edge cases (pregnancy, elderly, immunocompromised) do not define baseline human physiology.

Campylobacter – Saying its “biological purpose” is to cause disease is laughable. Bacteria don’t have “purposes.” They have ecological niches. Campylobacter is carried asymptomatically in humans, poultry, cattle and pets worldwide. If its sole “purpose” was disease, every carrier would be hospitalized. They’re not. The hygiene hypothesis (Strachan 1989, BMJ; Stein et al. 2016, NEJM) is mainstream: low-level exposure to all environmental microbes, including so-called “pathogens,” is required for proper immune training.

Quantitative Risk Assessment – That’s not causation. That’s modeling assumptions, not controlled proof. You even admit: “We can’t show a single study where scientists deliberately infect a healthy person.” Exactly. Which means your claim that raw-meat microbes are inherently poisonous is unproven. Modeling probabilities is not the same as demonstrating inevitability.


Less relevant
If you want knowledge about something as simple as calories go research it yourself first, literally just how they are measured and if you think that applies to organisms or you can save that for another debate like the vegetables or whatever it was.

>0 negative impact on health = less healthy, therefore:

” Stop talking about the effect on the environment and money. It doesn’t make a big impact. And doesn’t relate to if raw meat or cooked meat is healthier. Leave it out.”


Dont tell me to stop talking about it, either accept that it has >0 negative impact on health or dont repeat my point.


“These are only relevant because you brought up scavengers and how it “proves” that we can safely eat raw meat.”

- Do not misquote me, I never said that it “” proves”” anything. I was trying to make you understand that humans are carnivorous animals, and every carnivorous animal eats raw meat, humans no different. Ditch the vulture thing I don’t know why you keep nailing at it as if it is some big flaw in my point? And saying that we lack something vultures have negatively influences our ability to “handle” raw meat is false, if so show how every single carnivorous animal possesses those things but humans lack them and that exactly that is the reason, not just an exception.

In short I did not use scavengers as prove, just an exampe, however you do claim that humans lacking something from vultures IS “evidence” that humans cannot handle raw meat. Yes it is irrelevant so you don’t have to mention it anymore except if you don’t want to except your claim being false and then you can show some evidence, and please read this again so you understand the error in your point.


Thought we agreed on the superior nutrient point, so the only rational reason for you to keep mentioning it is if you think nutrients don’t matter for health. Remember the point you are trying to defend and ask yourself if your argument is relevant.

Stop requesting irrelevant explanations of fucking logical things


So let’s recap.

1. You concede nutrients are lost in cooking.

2. You concede harmful compounds form in cooking.

3. You admit your own pathogen examples (Listeria, Campylobacter, Salmonella, E. coli) can all exist in healthy carriers without symptoms.

That destroys the idea that they are inherently “poisonous.” Until you show me direct causal human evidence, your argument is nothing but fear-mongering with scary names.


” You just make so many small little lies and misinformation and drive away from the real point. JFL” shut the fuck up btw i am the one who keeps redirecting you towards the point, you even try to make me talk about calories in the previous paragraph, just shut the fuck up and talk science, you talk about so much irrelevant ilogical stuf? I am the one who uses logic between the two of us? You clearly don't understand simple concepts such as burden of proof, again if I claim to have fucked your mom who has the burden of proof and how can it be fair for me to demand you of proof or I have won that point?


Important, You need from now on understand:

• Burden of proof – You are the one claiming “cooked meat is healthier than raw.” Therefore you must provide positive evidence for this claim. My job is only to evaluate and falsify. Listing hazards or repeating “it’s risky” is not evidence.

• Causation vs. correlation – Presence of bacteria in patients or food does not prove causation. Unless Koch’s postulates (or equivalent modern criteria) are met in humans, your citations remain associative. Correlation, risk models, and outbreak reports ≠ causal evidence.

• Relevance – Only bring material that directly addresses the central claim (cooked > raw in terms of health). Tangents about ancestors, calories, vultures, environment etc. are red herrings. Keep focus on the actual claim.

You claim cooked is better than raw, prove it with evidence. And understand what evidence is please, the bacteria being present in raw meat and in a sick individual is not proof as an example.
 
  • +1
Reactions: iblamegenetics7681
Relevant:
I can very easily prove that bacteria are not risky but to do something as pointless you have to realise how much of a fucking idiot you have to be to actually request something like that, you should really reconsider something fundamental in your life at this point. You even admitted it yourself already btw. Let me begin:

Your point: “bacteria are not risky"

1. First of I will show you where you already admitted that bacteria are not risky yourself: “Our body is made of good and bad bacteria”, “Yes bacteria is needed to grow”, “It's absolutely true that our immune systems need to be "trained" by constant, low-level exposure to a diverse microbial world”

How can good bacteria be risky? And not related to yourself sabotage but, our body is “made” of bad bacteria?
You even linked a lot of studies yourself proving it so this whole answer is useless, how stupid can you be?

2. Burden of proof

You have the burden of proof because humans are heavily dependent on bacteria:

(Sender et al. 2016, Cell).

(Koh et al. 2016, Nat Rev Gastroenterol Hepatol)

(Bentley & Meganathan 1982, Microbiol Rev)

(Strachan 1989, BMJ)

(Cryan & Dinan 2012, Nat Rev Neurosci)

(Stein et al. 2016, NEJM)


You keep demanding “one peer-reviewed study” proving bacteria aren’t risky. That’s the dumbest framing I’ve ever read. You’re literally asking me to prove that oxygen isn’t poisonous in normal doses. The burden is on you to show that ubiquitous commensals like E. coli or Salmonella automatically cause disease in healthy humans. Otherwise you are just fear-mongering. “Good vs bad bacteria” is not science, it’s baby talk. The same species can be harmless in one host and helpful in another, and only cause trouble under stress conditions. That’s why asymptomatic carriage of Salmonella, Campylobacter, Listeria, and E. coli is well-documented in tens of millions of people. If they were inherently “bad,” those carriers would all be dead. They’re not.


1. All your “good vs bad” talk collapses the second you read your own citations. Every single one shows that microbes are context-dependent. Lactobacillus and Bifidobacterium are “good” because they balance the system – but in the wrong context even they can translocate and cause problems. Same with E. coli, Salmonella, Listeria, Campylobacter. They are not inherently poisonous, they are normal environmental and commensal species. Whether they cause symptoms depends on host health, dose, strain, and environment. That’s the entire point of microbiome science.

2. Your own NEJM review (2013) literally calls the microbiome a functional organ. If bacteria are an organ, then by your logic we should label our own liver or kidneys as “poisonous” just because organ failure sometimes kills people. Dumb framing.

3. Asymptomatic carriage is the nail in your coffin. Tens of millions of humans carry “your” scary names right now with zero symptoms. That fact alone destroys the “essentially poisonous” claim. If something is inherently poisonous, it kills on contact. These bacteria clearly do not. They only flip pathogenic under extreme conditions (immunodeficiency, imbalance, lab-selected strains like O157:H7).

4. You haven’t shown a single RCT or prospective human trial where healthy people eating hygienic raw meat get sick from these organisms. Presence ≠ poison. Koch’s postulates 101. Listing bacteria names from PubMed does not prove causation in humans.


You have to be academically illiterate if you actually think the burden of proof is on me to show bacteria are risky.


“Raw meat contains ecoli, salmonella, listeria, campylobacter etc. These are essentially poisonous, because they are what cause food poisonings…”
- Prove it


” Listeria is found in the ground. Well done! That doesn’t prove anything. Listeria isnt very harmful to healthy people. But still is harmful, like you said yourself, little poison or no poison. And it’s extremely harmful to young, old or pregnant people. It is dangerous, just like your argument with the PHA’s, AGE’s etc. Even though this is way more significant.”

- Okay well done yourself, it is irrelevant to raw meat then haha



You are confusing labels with reality. Calling some microbes “good” and others “bad” is not science, it’s kindergarten talk. The exact same species can be harmless, beneficial or problematic depending on host state, dose and context. That’s why asymptomatic carriage of Salmonella, Campylobacter, Listeria and E. coli is documented in tens of millions of healthy people. If they were “essentially poisonous” like you keep saying, all those carriers would be sick or dead. They’re not.

E. coli – Wrong again. Not all E. coli “found in food” are pathogenic. O157:H7 is one mutated lab-selected strain, most are harmless commensals or even protective. The human body depends on E. coli for vitamin K synthesis and for blocking pathogen overgrowth. To call the entire genus “poison” is just ignorance.

Salmonella – “Only in tiny amounts”? No. Studies show long-term asymptomatic carriers excreting significant loads without symptoms. The variable is not presence vs absence, but host immune status and gut balance. Presence ≠ disease.

Listeria – You literally admit it: “not very harmful to healthy people.” Thanks for proving my point. That means your own example is irrelevant for raw meat safety in healthy individuals. Edge cases (pregnancy, elderly, immunocompromised) do not define baseline human physiology.

Campylobacter – Saying its “biological purpose” is to cause disease is laughable. Bacteria don’t have “purposes.” They have ecological niches. Campylobacter is carried asymptomatically in humans, poultry, cattle and pets worldwide. If its sole “purpose” was disease, every carrier would be hospitalized. They’re not. The hygiene hypothesis (Strachan 1989, BMJ; Stein et al. 2016, NEJM) is mainstream: low-level exposure to all environmental microbes, including so-called “pathogens,” is required for proper immune training.

Quantitative Risk Assessment – That’s not causation. That’s modeling assumptions, not controlled proof. You even admit: “We can’t show a single study where scientists deliberately infect a healthy person.” Exactly. Which means your claim that raw-meat microbes are inherently poisonous is unproven. Modeling probabilities is not the same as demonstrating inevitability.


Less relevant
If you want knowledge about something as simple as calories go research it yourself first, literally just how they are measured and if you think that applies to organisms or you can save that for another debate like the vegetables or whatever it was.

>0 negative impact on health = less healthy, therefore:

” Stop talking about the effect on the environment and money. It doesn’t make a big impact. And doesn’t relate to if raw meat or cooked meat is healthier. Leave it out.”


Dont tell me to stop talking about it, either accept that it has >0 negative impact on health or dont repeat my point.


“These are only relevant because you brought up scavengers and how it “proves” that we can safely eat raw meat.”

- Do not misquote me, I never said that it “” proves”” anything. I was trying to make you understand that humans are carnivorous animals, and every carnivorous animal eats raw meat, humans no different. Ditch the vulture thing I don’t know why you keep nailing at it as if it is some big flaw in my point? And saying that we lack something vultures have negatively influences our ability to “handle” raw meat is false, if so show how every single carnivorous animal possesses those things but humans lack them and that exactly that is the reason, not just an exception.

In short I did not use scavengers as prove, just an exampe, however you do claim that humans lacking something from vultures IS “evidence” that humans cannot handle raw meat. Yes it is irrelevant so you don’t have to mention it anymore except if you don’t want to except your claim being false and then you can show some evidence, and please read this again so you understand the error in your point.


Thought we agreed on the superior nutrient point, so the only rational reason for you to keep mentioning it is if you think nutrients don’t matter for health. Remember the point you are trying to defend and ask yourself if your argument is relevant.

Stop requesting irrelevant explanations of fucking logical things


So let’s recap.

1. You concede nutrients are lost in cooking.

2. You concede harmful compounds form in cooking.

3. You admit your own pathogen examples (Listeria, Campylobacter, Salmonella, E. coli) can all exist in healthy carriers without symptoms.

That destroys the idea that they are inherently “poisonous.” Until you show me direct causal human evidence, your argument is nothing but fear-mongering with scary names.


” You just make so many small little lies and misinformation and drive away from the real point. JFL” shut the fuck up btw i am the one who keeps redirecting you towards the point, you even try to make me talk about calories in the previous paragraph, just shut the fuck up and talk science, you talk about so much irrelevant ilogical stuf? I am the one who uses logic between the two of us? You clearly don't understand simple concepts such as burden of proof, again if I claim to have fucked your mom who has the burden of proof and how can it be fair for me to demand you of proof or I have won that point?


Important, You need from now on understand:

• Burden of proof – You are the one claiming “cooked meat is healthier than raw.” Therefore you must provide positive evidence for this claim. My job is only to evaluate and falsify. Listing hazards or repeating “it’s risky” is not evidence.

• Causation vs. correlation – Presence of bacteria in patients or food does not prove causation. Unless Koch’s postulates (or equivalent modern criteria) are met in humans, your citations remain associative. Correlation, risk models, and outbreak reports ≠ causal evidence.

• Relevance – Only bring material that directly addresses the central claim (cooked > raw in terms of health). Tangents about ancestors, calories, vultures, environment etc. are red herrings. Keep focus on the actual claim.

You claim cooked is better than raw, prove it with evidence. And understand what evidence is please, the bacteria being present in raw meat and in a sick individual is not proof as an example.
You just iq mogged him bhai
 
  • JFL
Reactions: WaterDog
Relevant:
I can very easily prove that bacteria are not risky but to do something as pointless you have to realise how much of a fucking idiot you have to be to actually request something like that, you should really reconsider something fundamental in your life at this point. You even admitted it yourself already btw. Let me begin:

Your point: “bacteria are not risky"

1. First of I will show you where you already admitted that bacteria are not risky yourself: “Our body is made of good and bad bacteria”, “Yes bacteria is needed to grow”, “It's absolutely true that our immune systems need to be "trained" by constant, low-level exposure to a diverse microbial world”

How can good bacteria be risky? And not related to yourself sabotage but, our body is “made” of bad bacteria?
You even linked a lot of studies yourself proving it so this whole answer is useless, how stupid can you be?

2. Burden of proof

You have the burden of proof because humans are heavily dependent on bacteria:

(Sender et al. 2016, Cell).

(Koh et al. 2016, Nat Rev Gastroenterol Hepatol)

(Bentley & Meganathan 1982, Microbiol Rev)

(Strachan 1989, BMJ)

(Cryan & Dinan 2012, Nat Rev Neurosci)

(Stein et al. 2016, NEJM)


You keep demanding “one peer-reviewed study” proving bacteria aren’t risky. That’s the dumbest framing I’ve ever read. You’re literally asking me to prove that oxygen isn’t poisonous in normal doses. The burden is on you to show that ubiquitous commensals like E. coli or Salmonella automatically cause disease in healthy humans. Otherwise you are just fear-mongering. “Good vs bad bacteria” is not science, it’s baby talk. The same species can be harmless in one host and helpful in another, and only cause trouble under stress conditions. That’s why asymptomatic carriage of Salmonella, Campylobacter, Listeria, and E. coli is well-documented in tens of millions of people. If they were inherently “bad,” those carriers would all be dead. They’re not.


1. All your “good vs bad” talk collapses the second you read your own citations. Every single one shows that microbes are context-dependent. Lactobacillus and Bifidobacterium are “good” because they balance the system – but in the wrong context even they can translocate and cause problems. Same with E. coli, Salmonella, Listeria, Campylobacter. They are not inherently poisonous, they are normal environmental and commensal species. Whether they cause symptoms depends on host health, dose, strain, and environment. That’s the entire point of microbiome science.

2. Your own NEJM review (2013) literally calls the microbiome a functional organ. If bacteria are an organ, then by your logic we should label our own liver or kidneys as “poisonous” just because organ failure sometimes kills people. Dumb framing.

3. Asymptomatic carriage is the nail in your coffin. Tens of millions of humans carry “your” scary names right now with zero symptoms. That fact alone destroys the “essentially poisonous” claim. If something is inherently poisonous, it kills on contact. These bacteria clearly do not. They only flip pathogenic under extreme conditions (immunodeficiency, imbalance, lab-selected strains like O157:H7).

4. You haven’t shown a single RCT or prospective human trial where healthy people eating hygienic raw meat get sick from these organisms. Presence ≠ poison. Koch’s postulates 101. Listing bacteria names from PubMed does not prove causation in humans.


You have to be academically illiterate if you actually think the burden of proof is on me to show bacteria are risky.


“Raw meat contains ecoli, salmonella, listeria, campylobacter etc. These are essentially poisonous, because they are what cause food poisonings…”
- Prove it


” Listeria is found in the ground. Well done! That doesn’t prove anything. Listeria isnt very harmful to healthy people. But still is harmful, like you said yourself, little poison or no poison. And it’s extremely harmful to young, old or pregnant people. It is dangerous, just like your argument with the PHA’s, AGE’s etc. Even though this is way more significant.”

- Okay well done yourself, it is irrelevant to raw meat then haha



You are confusing labels with reality. Calling some microbes “good” and others “bad” is not science, it’s kindergarten talk. The exact same species can be harmless, beneficial or problematic depending on host state, dose and context. That’s why asymptomatic carriage of Salmonella, Campylobacter, Listeria and E. coli is documented in tens of millions of healthy people. If they were “essentially poisonous” like you keep saying, all those carriers would be sick or dead. They’re not.

E. coli – Wrong again. Not all E. coli “found in food” are pathogenic. O157:H7 is one mutated lab-selected strain, most are harmless commensals or even protective. The human body depends on E. coli for vitamin K synthesis and for blocking pathogen overgrowth. To call the entire genus “poison” is just ignorance.

Salmonella – “Only in tiny amounts”? No. Studies show long-term asymptomatic carriers excreting significant loads without symptoms. The variable is not presence vs absence, but host immune status and gut balance. Presence ≠ disease.

Listeria – You literally admit it: “not very harmful to healthy people.” Thanks for proving my point. That means your own example is irrelevant for raw meat safety in healthy individuals. Edge cases (pregnancy, elderly, immunocompromised) do not define baseline human physiology.

Campylobacter – Saying its “biological purpose” is to cause disease is laughable. Bacteria don’t have “purposes.” They have ecological niches. Campylobacter is carried asymptomatically in humans, poultry, cattle and pets worldwide. If its sole “purpose” was disease, every carrier would be hospitalized. They’re not. The hygiene hypothesis (Strachan 1989, BMJ; Stein et al. 2016, NEJM) is mainstream: low-level exposure to all environmental microbes, including so-called “pathogens,” is required for proper immune training.

Quantitative Risk Assessment – That’s not causation. That’s modeling assumptions, not controlled proof. You even admit: “We can’t show a single study where scientists deliberately infect a healthy person.” Exactly. Which means your claim that raw-meat microbes are inherently poisonous is unproven. Modeling probabilities is not the same as demonstrating inevitability.


Less relevant
If you want knowledge about something as simple as calories go research it yourself first, literally just how they are measured and if you think that applies to organisms or you can save that for another debate like the vegetables or whatever it was.

>0 negative impact on health = less healthy, therefore:

” Stop talking about the effect on the environment and money. It doesn’t make a big impact. And doesn’t relate to if raw meat or cooked meat is healthier. Leave it out.”


Dont tell me to stop talking about it, either accept that it has >0 negative impact on health or dont repeat my point.


“These are only relevant because you brought up scavengers and how it “proves” that we can safely eat raw meat.”

- Do not misquote me, I never said that it “” proves”” anything. I was trying to make you understand that humans are carnivorous animals, and every carnivorous animal eats raw meat, humans no different. Ditch the vulture thing I don’t know why you keep nailing at it as if it is some big flaw in my point? And saying that we lack something vultures have negatively influences our ability to “handle” raw meat is false, if so show how every single carnivorous animal possesses those things but humans lack them and that exactly that is the reason, not just an exception.

In short I did not use scavengers as prove, just an exampe, however you do claim that humans lacking something from vultures IS “evidence” that humans cannot handle raw meat. Yes it is irrelevant so you don’t have to mention it anymore except if you don’t want to except your claim being false and then you can show some evidence, and please read this again so you understand the error in your point.


Thought we agreed on the superior nutrient point, so the only rational reason for you to keep mentioning it is if you think nutrients don’t matter for health. Remember the point you are trying to defend and ask yourself if your argument is relevant.

Stop requesting irrelevant explanations of fucking logical things


So let’s recap.

1. You concede nutrients are lost in cooking.

2. You concede harmful compounds form in cooking.

3. You admit your own pathogen examples (Listeria, Campylobacter, Salmonella, E. coli) can all exist in healthy carriers without symptoms.

That destroys the idea that they are inherently “poisonous.” Until you show me direct causal human evidence, your argument is nothing but fear-mongering with scary names.


” You just make so many small little lies and misinformation and drive away from the real point. JFL” shut the fuck up btw i am the one who keeps redirecting you towards the point, you even try to make me talk about calories in the previous paragraph, just shut the fuck up and talk science, you talk about so much irrelevant ilogical stuf? I am the one who uses logic between the two of us? You clearly don't understand simple concepts such as burden of proof, again if I claim to have fucked your mom who has the burden of proof and how can it be fair for me to demand you of proof or I have won that point?


Important, You need from now on understand:

• Burden of proof – You are the one claiming “cooked meat is healthier than raw.” Therefore you must provide positive evidence for this claim. My job is only to evaluate and falsify. Listing hazards or repeating “it’s risky” is not evidence.

• Causation vs. correlation – Presence of bacteria in patients or food does not prove causation. Unless Koch’s postulates (or equivalent modern criteria) are met in humans, your citations remain associative. Correlation, risk models, and outbreak reports ≠ causal evidence.

• Relevance – Only bring material that directly addresses the central claim (cooked > raw in terms of health). Tangents about ancestors, calories, vultures, environment etc. are red herrings. Keep focus on the actual claim.

You claim cooked is better than raw, prove it with evidence. And understand what evidence is please, the bacteria being present in raw meat and in a sick individual is not proof as an example.
The burden of proof, you idiot, is not on the person stating that high-consequence pathogens are dangerous; it is on the one claiming that consuming food laced with them is healthier than not. The global health apparatus already confirms the risk: the WHO estimates that foodborne diseases cause 600 million illnesses and 420,000 deaths annually worldwide, resulting in 33 million Disability-Adjusted Life Years (DALYs) lost. You want to argue that this massive, persistent biological catastrophe is less significant than preserving some vitamin C? That's not a healthy choice; that's suicidal recklessness backed by profound scientific illiteracy. The fact that animal-source foods account for roughly one-third of the total global foodborne disease burden just slams the door on your raw meat fantasy. In the US, meat and poultry are linked to 29% of all foodborne deaths, primarily due to the specific organisms you are minimizing.
Let's dissect the molecular warfare these pathogens employ, proving they are not just context-dependent commensals, but genetically armed killers that necessitate a thermal "kill step."
Take Shiga toxin-producing Escherichia coli (STEC), particularly O157:H7, which is frequently linked to raw ground beef dishes like steak tartare or cannibal sandwiches.[1, 2] You think this is just benign bacteria? STEC's lethality comes from its virulence factors, notably the Shiga toxin (Stx) and the Locus of Enterocyte Effacement (LEE).[3] The LEE pathogenicity island allows the bacteria to colonize the bowel by creating attaching and effacing (A/E) lesions on the enterocytes.[4] The real terror, though, is the Stx. Once the bacterial cells are lysed (often triggered by certain environmental factors), the Stx is released into the gut lumen.[4] This toxin is composed of an enzymatically active A subunit and a pentameric B subunit.[4] The B subunit specifically binds to the glycosphingolipid receptor globotriaosylceramide (\text{Gb}_3), which is highly expressed on the surface of human vascular endothelial cells, particularly in the kidneys.[5, 4] The toxin is internalized via endocytosis and then undergoes retrograde transport to the endoplasmic reticulum, completely avoiding the normal lysosomal degradation pathway.[5, 6] Once the active A1 fragment translocates into the cytoplasm, it functions as an RNA N-glycosidase, cleaving one specific adenine residue from the 28S rRNA of the 60S ribosomal subunit.[5, 4, 6] This molecular sabotage irreversibly inhibits host protein synthesis, triggering the ribotoxic and endoplasmic reticulum stress responses, which directly leads to endothelial cell apoptosis and subsequent activation of complement pathways.[5, 6] This systemic damage to the kidney's microvasculature is the exact mechanism that causes Hemolytic Uremic Syndrome (HUS), leading to acute renal failure, which occurs in 15–20% of infected children and has a 3% mortality rate in young children and 20% in older adults. This is a direct, quantifiable poison, operating at an estimated infectious dose as low as 2 to less than 9 Colony-Forming Units (CFU), according to outbreak data. Your "healthy gut" cannot handle a pathogenic dose that low, and cooking is the required step to eliminate it.[7]
Now, let's talk about Listeria monocytogenes, the pathogen you condescendingly admitted is irrelevant to "baseline human physiology." Listeriosis is the third leading cause of death from foodborne illness, with a terrifying case-fatality rate of about 20%. Why is it so lethal? Because it is a facultative intracellular pathogen, meaning it has evolved an intricate molecular arsenal to invade and spread inside host cells, bypassing humoral immunity.[8, 9] The invasion is orchestrated by surface proteins, primarily Internalin A (InlA) and Internalin B (InlB), which force the host cell to engulf the bacteria.[8, 10] Once internalized, it relies on the secreted pore-forming toxin, Listeriolysin O (LLO), which disrupts the phagosome membrane, allowing the pathogen to escape directly into the host cell cytoplasm. Inside the cytoplasm, the bacteria hijacks the host's cytoskeleton using the ActA protein to generate an actin comet tail. This ActA-mediated motility is a biological jet propulsion system that allows the Listeria to push itself into adjacent cells, forming a double-membraned secondary vacuole which is then lysed by LLO and phospholipases, completing the cell-to-cell spread without ever exposing itself to the immune system.[9] This molecular stealth is why Listeria is neuroinvasive, causing meningitis, which carries a staggering consequence: a French national prospective study showed that 44% of patients who survive neurolisteriosis suffer long-term neurological sequelae. Furthermore, nearly 25% of pregnancy-associated cases result in fetal loss or the death of the newborn. To call this a non-significant, "edge case" risk when it causes irreversible neurological damage and massive infant mortality is ethically indefensible and scientifically negligent.
And finally, Campylobacter jejuni, another ubiquitous contaminant of raw poultry and meat that you think is just building up the immune system. Campylobacter infection is the leading cause of Guillain-Barré Syndrome (GBS), the most common cause of acute flaccid paralysis worldwide. This isn't a direct bacterial attack; it’s an autoimmune catastrophe caused by molecular mimicry. The lipopolysaccharides (LPS) on the surface of neuropathy-associated C. jejuni strains share specific ganglioside-like epitopes with the myelin sheath and axons of human peripheral nerves. When the body mounts an immune response to the bacteria ingested from raw meat, the resulting antibodies cross-react with and attack the host's own nerve tissues, leading to demyelination and paralysis. This is a profound, chronic, and severe sequela, with approximately 1 in every 1,000 people infected with Campylobacter developing GBS. This is a direct, causal link between a raw meat pathogen and life-altering paralysis, and it is a major contributor to the DALY burden from foodborne disease.
The entire argument that a loss of a minimal amount of vitamins during a Kill Step—which regulatory bodies like the USDA and FDA mandate to achieve a verified reduction of pathogens to acceptable levels—is offset by the risk of HUS, paralysis, or fetal death is idiotic. Cooking is the only validated control that stops these organisms from unleashing their molecular weapons on the host. Stop talking about irrelevant evolutionary analogies, because comparing human gastric acidity (pH 1.5–2.0) to the highly specialized, often "battery acid" pH of obligate scavengers like true vultures, which evolved massive acid secretion as a defense mechanism against the high pathogen load of carrion, is a profoundly flawed argument that misinterprets the evolution of host defense. The low infectious dose of these genetically virulent pathogens simply overwhelms the evolutionary filter of the human stomach, making the thermal intervention non-negotiable for public safety. You are wrong. End of story.
 
i just had my first ever raw meat it was tartar 200 grams just raw out of the package i wanna start eating other raw animal food like liver for example, is there anyone that can guide me, and tell me how they went about it, im 16 years old.
raw liver mogs everything
 
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Reactions: WaterDog
The burden of proof, you idiot, is not on the person stating that high-consequence pathogens are dangerous; it is on the one claiming that consuming food laced with them is healthier than not. The global health apparatus already confirms the risk: the WHO estimates that foodborne diseases cause 600 million illnesses and 420,000 deaths annually worldwide, resulting in 33 million Disability-Adjusted Life Years (DALYs) lost. You want to argue that this massive, persistent biological catastrophe is less significant than preserving some vitamin C? That's not a healthy choice; that's suicidal recklessness backed by profound scientific illiteracy. The fact that animal-source foods account for roughly one-third of the total global foodborne disease burden just slams the door on your raw meat fantasy. In the US, meat and poultry are linked to 29% of all foodborne deaths, primarily due to the specific organisms you are minimizing.
Let's dissect the molecular warfare these pathogens employ, proving they are not just context-dependent commensals, but genetically armed killers that necessitate a thermal "kill step."
Take Shiga toxin-producing Escherichia coli (STEC), particularly O157:H7, which is frequently linked to raw ground beef dishes like steak tartare or cannibal sandwiches.[1, 2] You think this is just benign bacteria? STEC's lethality comes from its virulence factors, notably the Shiga toxin (Stx) and the Locus of Enterocyte Effacement (LEE).[3] The LEE pathogenicity island allows the bacteria to colonize the bowel by creating attaching and effacing (A/E) lesions on the enterocytes.[4] The real terror, though, is the Stx. Once the bacterial cells are lysed (often triggered by certain environmental factors), the Stx is released into the gut lumen.[4] This toxin is composed of an enzymatically active A subunit and a pentameric B subunit.[4] The B subunit specifically binds to the glycosphingolipid receptor globotriaosylceramide (\text{Gb}_3), which is highly expressed on the surface of human vascular endothelial cells, particularly in the kidneys.[5, 4] The toxin is internalized via endocytosis and then undergoes retrograde transport to the endoplasmic reticulum, completely avoiding the normal lysosomal degradation pathway.[5, 6] Once the active A1 fragment translocates into the cytoplasm, it functions as an RNA N-glycosidase, cleaving one specific adenine residue from the 28S rRNA of the 60S ribosomal subunit.[5, 4, 6] This molecular sabotage irreversibly inhibits host protein synthesis, triggering the ribotoxic and endoplasmic reticulum stress responses, which directly leads to endothelial cell apoptosis and subsequent activation of complement pathways.[5, 6] This systemic damage to the kidney's microvasculature is the exact mechanism that causes Hemolytic Uremic Syndrome (HUS), leading to acute renal failure, which occurs in 15–20% of infected children and has a 3% mortality rate in young children and 20% in older adults. This is a direct, quantifiable poison, operating at an estimated infectious dose as low as 2 to less than 9 Colony-Forming Units (CFU), according to outbreak data. Your "healthy gut" cannot handle a pathogenic dose that low, and cooking is the required step to eliminate it.[7]
Now, let's talk about Listeria monocytogenes, the pathogen you condescendingly admitted is irrelevant to "baseline human physiology." Listeriosis is the third leading cause of death from foodborne illness, with a terrifying case-fatality rate of about 20%. Why is it so lethal? Because it is a facultative intracellular pathogen, meaning it has evolved an intricate molecular arsenal to invade and spread inside host cells, bypassing humoral immunity.[8, 9] The invasion is orchestrated by surface proteins, primarily Internalin A (InlA) and Internalin B (InlB), which force the host cell to engulf the bacteria.[8, 10] Once internalized, it relies on the secreted pore-forming toxin, Listeriolysin O (LLO), which disrupts the phagosome membrane, allowing the pathogen to escape directly into the host cell cytoplasm. Inside the cytoplasm, the bacteria hijacks the host's cytoskeleton using the ActA protein to generate an actin comet tail. This ActA-mediated motility is a biological jet propulsion system that allows the Listeria to push itself into adjacent cells, forming a double-membraned secondary vacuole which is then lysed by LLO and phospholipases, completing the cell-to-cell spread without ever exposing itself to the immune system.[9] This molecular stealth is why Listeria is neuroinvasive, causing meningitis, which carries a staggering consequence: a French national prospective study showed that 44% of patients who survive neurolisteriosis suffer long-term neurological sequelae. Furthermore, nearly 25% of pregnancy-associated cases result in fetal loss or the death of the newborn. To call this a non-significant, "edge case" risk when it causes irreversible neurological damage and massive infant mortality is ethically indefensible and scientifically negligent.
And finally, Campylobacter jejuni, another ubiquitous contaminant of raw poultry and meat that you think is just building up the immune system. Campylobacter infection is the leading cause of Guillain-Barré Syndrome (GBS), the most common cause of acute flaccid paralysis worldwide. This isn't a direct bacterial attack; it’s an autoimmune catastrophe caused by molecular mimicry. The lipopolysaccharides (LPS) on the surface of neuropathy-associated C. jejuni strains share specific ganglioside-like epitopes with the myelin sheath and axons of human peripheral nerves. When the body mounts an immune response to the bacteria ingested from raw meat, the resulting antibodies cross-react with and attack the host's own nerve tissues, leading to demyelination and paralysis. This is a profound, chronic, and severe sequela, with approximately 1 in every 1,000 people infected with Campylobacter developing GBS. This is a direct, causal link between a raw meat pathogen and life-altering paralysis, and it is a major contributor to the DALY burden from foodborne disease.
The entire argument that a loss of a minimal amount of vitamins during a Kill Step—which regulatory bodies like the USDA and FDA mandate to achieve a verified reduction of pathogens to acceptable levels—is offset by the risk of HUS, paralysis, or fetal death is idiotic. Cooking is the only validated control that stops these organisms from unleashing their molecular weapons on the host. Stop talking about irrelevant evolutionary analogies, because comparing human gastric acidity (pH 1.5–2.0) to the highly specialized, often "battery acid" pH of obligate scavengers like true vultures, which evolved massive acid secretion as a defense mechanism against the high pathogen load of carrion, is a profoundly flawed argument that misinterprets the evolution of host defense. The low infectious dose of these genetically virulent pathogens simply overwhelms the evolutionary filter of the human stomach, making the thermal intervention non-negotiable for public safety. You are wrong. End of story.
Your first paragraph is pointless and I hope you receive 0 credit for this entire answer as you clearly did not go from 5. grade writing to this level overnight, nothing wrong in using chatbots as they themselves cannot find proof of anything that is false they can however act as an eco chamber for the users ideas and reinforce them heavily with their rhetorical writing. So look out you might be brainwashing yourself and others and I hope you receive 0 credit for "high iq post":soy:.

And why did your even mention WHO as a source of evidence, it just proves that the majority of cases come from vegetables out of every other type of type, Including what I assume more cooked than raw meat. Also raw meat is not isolated at all, if anything you are proving the DALYs of cooked meat because of your statement of people generally eating more cooked than raw. Not stating that more cases = more risk just stating that your paragraph is irrelevant to this discussion and even proves according to your logic that more cases come from the type of food you are advocating.


What you describe is an accurate and detailed explanation of what can happen if someone gets infected with certain pathogens — such as Shiga toxin-producing E. coli, Listeria monocytogenes, and Campylobacter jejuni. You document thoroughly how these bacteria operate and how severe the consequences of infection can be. All of this is true. But what you do not do — and what you cannot do — is prove that raw meat in itself is dangerous or unhealthy.

Let’s analyze your argument logically:

You confuse cause and context​

You describe the consequences of infection with certain pathogens and use that as an argument against eating raw meat. But this is a logical fallacy to assume that because contaminated raw meat can cause illness, all raw meat is dangerous.
It’s like saying: “A salad can cause STEC infection — therefore, salad itself is dangerous.”
No — the salad is only dangerous if it has been contaminated. And contamination is not an inherent property of the product, but a matter of handling.

These bacteria are not natural components of meat biology​

You argue as if these pathogens are a fixed part of raw meat. This is incorrect:

  • STEC primarily exists in the intestines of some cattle, not in muscle tissue.
  • Listeria often comes from environmental contamination (soil, processing equipment).
  • Campylobacter is typically found in the intestines and on the surface of poultry, but not inside the actual meat.
This means a piece of raw meat — under hygienic conditions — Is completely free of these bacteria. In fact, this is exactly how dishes like tartare, carpaccio, and sashimi can be safely served. It’s not because one “accepts the risk” — it’s because the meat is not inherently contaminated.

If raw meat was dangerous, these dishes would never exist safely. But they do — every day, worldwide.

Your argument implies contamination but understand that raw meat is not inherently contaminated, there fore you do not prove that "cooked meat is healthier than raw meat" you do prove that "cooked meat is healthier than contaminated raw meat". Since the contamination is not an inherent property of raw meat I could also use that argument, imagine you have a normal piece of raw meat (inherently no risk of illness) and then you cook it, by cooking it the meat is introduced to a greater number of possibly contaminated surfaces, raw meat is ready to eat without any preparation whatsoever, 0, but cooking needs >0, making cooked meat more risky.

Your answer is only a criticism of contamination.

STEC (Shiga toxin-producing E. coli)

  • Spinach outbreak (USA, 2006): Linked to bagged fresh spinach, not meat. Source traced to contaminated irrigation water or animal feces.
    (CDC, 2006)
  • Rocket salad outbreak (Finland, 2016): STEC isolated from arugula/rocket salad served at multiple events.
    (PubMed ID: 30180926)
  • FDA Leafy Greens Action Plan: Recognizes repeated E. coli outbreaks linked to leafy greens like romaine and spinach.
    (FDA, 2020)

Listeria monocytogenes

  • Cantaloupe outbreak (USA, 2011): Caused 33 deaths, traced to contaminated melons, not meat or dairy.
    (CDC Final Update, 2012)
  • Frozen vegetables (Europe, 2015–2018): Multi-country outbreak linked to frozen corn and vegetables.
    (EFSA & ECDC joint report)

Campylobacter jejuni

  • Kindergarten outbreak (Norway, 2012): Linked to a contaminated cleaning cloth, not food.
    (Euro Surveill. 2013;18(25):20520)
See? you only explained the mechanistic action of these infections but did not prove that they are inherent to raw meat. So far you are mentioning these things but have not proven that they are inherent to raw meat. You have only argued that contaminated food is more risky than uncontaminated food, this is unrelated to your point of cooked meat being healthier than raw, as cooked meat can also become contaminated. This argument fundamentally implies that raw meat is contaminated but that is not an inherent property to raw meat. The only argument you can derive from your explanation is that contaminated meat is risky. Now you have to understand what raw meat is and not make any hypothetical situations where it has become contaminated, if that was a logically allowed argument anything could be unhealthy, even using as washcloth as I in your logic just proved. Literally according to your logic using a washcloth is unhealthy. Please understand how to actually prove something, like fundamental use of logic and argumentation it would save you a lot of time.

“If you look at peer-reviewed microbiology, muscle tissue of healthy animals is typically sterile. The bacteria you speak of are introduced afterward — from hide, intestines, handling, environment — not because raw meat inherently harbors pathogens. To argue that raw meat is ‘inherently dangerous’ is to ignore what microbiology says about meat in its natural, uncontaminated state.” https://pubmed.ncbi.nlm.nih.gov/29564/

Here are some points I would like to make:

1. Raw meat is not inherently contaminated and therefore not risky to consume. You cannot make an argument using your earlier mentioned pathogens as that would imply contamination, something that could also apply to cooked food. At the very best making them equal.

2. Cooking does not remove the risk. I proved that according to your logic it would increase the risk from an increase in the risk of contamination by increasing the possibility of potential contamination e.g by increasing number of contacted contaminated surfaces. It is called post-cooking contamination. This makes cooking more risky.

3. Cooking always increases harmfull health-damaging compounds, including carcinogens, mutagens, and pro-inflammatory agents like AGEs and heterocyclic amines but it does not always reduce any harmfull bacteria (which was your argument) because as I proved they are not always present. This means 100% of the time you make the meat unhealthier with damaging compounds in order to compensate for a <100% chance the meat containing harmfull bacteria:feelshah:. This makes cooking more risky

In short, raw meat is not contaminated, cooking increases risk of contamination by number of surfaces and reduces all the earlier mentioned harmfull compounds and I could also mention the things we have agreed on in the beginning with nutrition and all.


Do you understand that you keep shooting yourself in the foot with every answer.

New nickname achieved: hasty generalization fallacy master.


by this point I can see you have reached chekmate, you have no arguements left. I you wanted to keep going with the bacteria thing you would additionally have to prove that the risk cannot be mitigated by any other means than cooking, because remember your point "cooking is healthier than raw", therefore if you want to even use the cooking reduce harmful pathogens I could just make you prove that cooking is the superior pathogen reducing mitigation technique as I have proven that cooking increases harmfull compounds.

LMAO after this you are genuinely never going to win this

Here is the full list of your logical failures, so you can stop pretending this is science:

  1. Burden of proof misplacement
    What you do: You demand I prove bacteria are not harmful.
    Why it fails: You claim cooked > raw, so the burden is on you to show positive human outcomes.
    Callout: The burden is yours. You claim cooked is healthier, you must prove it.
  2. Appeal to authority
    What you do: You wave WHO numbers without causal evidence for raw vs cooked.
    Why it fails: Authority totals are not proof of your specific claim.
    Callout: WHO totals ≠ evidence that cooked > raw. Show relative risks per portion or drop it.
  3. Non sequitur
    What you do: From rare pathogenic strains you conclude all raw meat is unsafe.
    Why it fails: The conclusion does not follow from the premise.
    Callout: A rare strain ≠ proof that raw meat itself is inherently unsafe.
  4. Cherry picking
    What you do: You highlight HUS, neurolisteriosis and GBS as if they are typical.
    Why it fails: Extreme outliers are not general rules.
    Callout: You cherry-pick edge cases. Show baseline incidence per portion in healthy people.
  5. Base rate neglect
    What you do: You ignore how rare severe outcomes are compared to total exposures.
    Why it fails: Risk requires a base rate.
    Callout: Where is your per-portion rate in healthy populations? Without it, your fear-mongering is statistics-free.
  6. Confusing hazard and risk
    What you do: You list molecular hazards and call it risk.
    Why it fails: Hazard ≠ risk without dose and exposure.
    Callout: Hazard is not risk. Show real dose-response under normal raw intake.
  7. Necessary vs sufficient
    What you do: You treat bacterial presence as proof of illness.
    Why it fails: Presence is not sufficient without host state, dose, strain.
    Callout: Presence ≠ causation. Prove sufficiency in humans.
  8. Ecological fallacy
    What you do: You use population totals to claim truth about individuals eating raw meat.
    Why it fails: Group stats don’t automatically apply to individual cases.
    Callout: Totals from many foods ≠ proof about raw vs cooked meat in healthy individuals.
  9. Category error
    What you do: You treat contamination as if it is an inherent property of raw meat.
    Why it fails: Contamination is handling and environment, not essence.
    Callout: You only prove contaminated food is risky, not raw meat itself.
  10. Ignoring post-cooking contamination
    What you do: You act as if heat ends the problem.
    Why it fails: Listeria and others thrive post-cooking in chilled foods.
    Callout: If heat solved it, there would be no outbreaks in cooked ready-meals.
  11. Straw man
    What you do: You pretend I claim all pathogens are beneficial.
    Why it fails: I say outcomes are context-dependent, not that all bacteria are good.
    Callout: You’re fighting a straw man. I never made that claim.
  12. False dichotomy
    What you do: You present it as raw=poison vs cooked=safe.
    Why it fails: Many mitigation strategies exist besides heat, and cooked food can also be contaminated.
    Callout: It’s not raw poison vs cooked safe. Show net health benefit, not just hazard reduction.
  13. Appeal to fear
    What you do: You drown in scary words like “molecular warfare.”
    Why it fails: Fear rhetoric ≠ data.
    Callout: Fear is not evidence. Show controlled human comparisons.
  14. Moving the goalposts
    What you do: You jump between WHO, models, toxins, infants to dodge the core claim.
    Why it fails: The criterion is simple: outcomes per portion, raw vs cooked.
    Callout: Stay on target. Show direct comparisons in humans or retract.
  15. Model reification
    What you do: You treat quantitative risk models as truth.
    Why it fails: Models = assumptions, not observed outcomes.
    Callout: A model is not proof. Show real human data, not math.
  16. Texas sharpshooter
    What you do: You match outbreaks to your story after the fact.
    Why it fails: Selective matching is not evidence.
    Callout: You pick cases that fit and ignore those that contradict. Show the full dataset.
  17. Composition/division fallacy
    What you do: You assume if some strains are harmful, all are harmful.
    Why it fails: Subset traits ≠ universal truth.
    Callout: A few virulent strains ≠ all raw meat, all hosts, all contexts.
  18. Begging the question
    What you do: You assume heat is necessary and conclude heat is necessary.
    Why it fails: Circular reasoning.
    Callout: You assume your conclusion. Show net health benefits beyond alternatives.
  19. Isolated demand for rigor
    What you do: You demand impossible RCTs from me but rely on weaker evidence yourself.
    Why it fails: Double standard.
    Callout: If ethics block RCTs, that applies to you too. Don’t demand what you can’t provide.
  20. False analogy with vultures
    What you do: You dismiss human stomach acid comparisons by jumping to extreme carrion eaters.
    Why it fails: Irrelevant species ≠ proof for humans.
    Callout: Vulture analogies don’t prove cooked > raw in healthy humans.
  21. Equivocation on “pathogen”
    What you do: You treat “pathogen” as if it always means guaranteed disease.
    Why it fails: Pathogen = potential, not inevitability.
    Callout: Potential ≠ outcome. Context decides.
  22. No True Scotsman
    What you do: Every time data contradicts you, you redefine what counts as “relevant risk.”
    Why it fails: Unfalsifiable.
    Callout: Stop shifting definitions. Fix clear criteria or concede.
  23. Anecdote & availability bias
    What you do: You rely on dramatic outbreaks instead of base rates.
    Why it fails: Memorable ≠ frequent.
    Callout: Anecdotes are not statistics. Show per-portion incidence.
  24. Moralistic/naturalistic misuse
    What you do: You call infection trials “unethical” and treat that as proof.
    Why it fails: Ethics block methods, not truth.
    Callout: Saying “it’s unethical” doesn’t prove your claim.


:feelsuhh::feelsuhh::feelsuhh:
 
i just had my first ever raw meat it was tartar 200 grams just raw out of the package i wanna start eating other raw animal food like liver for example, is there anyone that can guide me, and tell me how they went about it, im 16 years old.
Just eat
 
Your first paragraph is pointless and I hope you receive 0 credit for this entire answer as you clearly did not go from 5. grade writing to this level overnight, nothing wrong in using chatbots as they themselves cannot find proof of anything that is false they can however act as an eco chamber for the users ideas and reinforce them heavily with their rhetorical writing. So look out you might be brainwashing yourself and others and I hope you receive 0 credit for "high iq post":soy:.

And why did your even mention WHO as a source of evidence, it just proves that the majority of cases come from vegetables out of every other type of type, Including what I assume more cooked than raw meat. Also raw meat is not isolated at all, if anything you are proving the DALYs of cooked meat because of your statement of people generally eating more cooked than raw. Not stating that more cases = more risk just stating that your paragraph is irrelevant to this discussion and even proves according to your logic that more cases come from the type of food you are advocating.


What you describe is an accurate and detailed explanation of what can happen if someone gets infected with certain pathogens — such as Shiga toxin-producing E. coli, Listeria monocytogenes, and Campylobacter jejuni. You document thoroughly how these bacteria operate and how severe the consequences of infection can be. All of this is true. But what you do not do — and what you cannot do — is prove that raw meat in itself is dangerous or unhealthy.

Let’s analyze your argument logically:

You confuse cause and context​

You describe the consequences of infection with certain pathogens and use that as an argument against eating raw meat. But this is a logical fallacy to assume that because contaminated raw meat can cause illness, all raw meat is dangerous.
It’s like saying: “A salad can cause STEC infection — therefore, salad itself is dangerous.”
No — the salad is only dangerous if it has been contaminated. And contamination is not an inherent property of the product, but a matter of handling.

These bacteria are not natural components of meat biology​

You argue as if these pathogens are a fixed part of raw meat. This is incorrect:

  • STEC primarily exists in the intestines of some cattle, not in muscle tissue.
  • Listeria often comes from environmental contamination (soil, processing equipment).
  • Campylobacter is typically found in the intestines and on the surface of poultry, but not inside the actual meat.
This means a piece of raw meat — under hygienic conditions — Is completely free of these bacteria. In fact, this is exactly how dishes like tartare, carpaccio, and sashimi can be safely served. It’s not because one “accepts the risk” — it’s because the meat is not inherently contaminated.

If raw meat was dangerous, these dishes would never exist safely. But they do — every day, worldwide.

Your argument implies contamination but understand that raw meat is not inherently contaminated, there fore you do not prove that "cooked meat is healthier than raw meat" you do prove that "cooked meat is healthier than contaminated raw meat". Since the contamination is not an inherent property of raw meat I could also use that argument, imagine you have a normal piece of raw meat (inherently no risk of illness) and then you cook it, by cooking it the meat is introduced to a greater number of possibly contaminated surfaces, raw meat is ready to eat without any preparation whatsoever, 0, but cooking needs >0, making cooked meat more risky.

Your answer is only a criticism of contamination.

STEC (Shiga toxin-producing E. coli)

  • Spinach outbreak (USA, 2006): Linked to bagged fresh spinach, not meat. Source traced to contaminated irrigation water or animal feces.
    (CDC, 2006)
  • Rocket salad outbreak (Finland, 2016): STEC isolated from arugula/rocket salad served at multiple events.
    (PubMed ID: 30180926)
  • FDA Leafy Greens Action Plan: Recognizes repeated E. coli outbreaks linked to leafy greens like romaine and spinach.
    (FDA, 2020)

Listeria monocytogenes

  • Cantaloupe outbreak (USA, 2011): Caused 33 deaths, traced to contaminated melons, not meat or dairy.
    (CDC Final Update, 2012)
  • Frozen vegetables (Europe, 2015–2018): Multi-country outbreak linked to frozen corn and vegetables.
    (EFSA & ECDC joint report)

Campylobacter jejuni

  • Kindergarten outbreak (Norway, 2012): Linked to a contaminated cleaning cloth, not food.
    (Euro Surveill. 2013;18(25):20520)
See? you only explained the mechanistic action of these infections but did not prove that they are inherent to raw meat. So far you are mentioning these things but have not proven that they are inherent to raw meat. You have only argued that contaminated food is more risky than uncontaminated food, this is unrelated to your point of cooked meat being healthier than raw, as cooked meat can also become contaminated. This argument fundamentally implies that raw meat is contaminated but that is not an inherent property to raw meat. The only argument you can derive from your explanation is that contaminated meat is risky. Now you have to understand what raw meat is and not make any hypothetical situations where it has become contaminated, if that was a logically allowed argument anything could be unhealthy, even using as washcloth as I in your logic just proved. Literally according to your logic using a washcloth is unhealthy. Please understand how to actually prove something, like fundamental use of logic and argumentation it would save you a lot of time.

“If you look at peer-reviewed microbiology, muscle tissue of healthy animals is typically sterile. The bacteria you speak of are introduced afterward — from hide, intestines, handling, environment — not because raw meat inherently harbors pathogens. To argue that raw meat is ‘inherently dangerous’ is to ignore what microbiology says about meat in its natural, uncontaminated state.” https://pubmed.ncbi.nlm.nih.gov/29564/

Here are some points I would like to make:

1. Raw meat is not inherently contaminated and therefore not risky to consume. You cannot make an argument using your earlier mentioned pathogens as that would imply contamination, something that could also apply to cooked food. At the very best making them equal.

2. Cooking does not remove the risk. I proved that according to your logic it would increase the risk from an increase in the risk of contamination by increasing the possibility of potential contamination e.g by increasing number of contacted contaminated surfaces. It is called post-cooking contamination. This makes cooking more risky.

3. Cooking always increases harmfull health-damaging compounds, including carcinogens, mutagens, and pro-inflammatory agents like AGEs and heterocyclic amines but it does not always reduce any harmfull bacteria (which was your argument) because as I proved they are not always present. This means 100% of the time you make the meat unhealthier with damaging compounds in order to compensate for a <100% chance the meat containing harmfull bacteria:feelshah:. This makes cooking more risky

In short, raw meat is not contaminated, cooking increases risk of contamination by number of surfaces and reduces all the earlier mentioned harmfull compounds and I could also mention the things we have agreed on in the beginning with nutrition and all.


Do you understand that you keep shooting yourself in the foot with every answer.

New nickname achieved: hasty generalization fallacy master.


by this point I can see you have reached chekmate, you have no arguements left. I you wanted to keep going with the bacteria thing you would additionally have to prove that the risk cannot be mitigated by any other means than cooking, because remember your point "cooking is healthier than raw", therefore if you want to even use the cooking reduce harmful pathogens I could just make you prove that cooking is the superior pathogen reducing mitigation technique as I have proven that cooking increases harmfull compounds.

LMAO after this you are genuinely never going to win this

Here is the full list of your logical failures, so you can stop pretending this is science:

  1. Burden of proof misplacement
    What you do: You demand I prove bacteria are not harmful.
    Why it fails: You claim cooked > raw, so the burden is on you to show positive human outcomes.
    Callout: The burden is yours. You claim cooked is healthier, you must prove it.
  2. Appeal to authority
    What you do: You wave WHO numbers without causal evidence for raw vs cooked.
    Why it fails: Authority totals are not proof of your specific claim.
    Callout: WHO totals ≠ evidence that cooked > raw. Show relative risks per portion or drop it.
  3. Non sequitur
    What you do: From rare pathogenic strains you conclude all raw meat is unsafe.
    Why it fails: The conclusion does not follow from the premise.
    Callout: A rare strain ≠ proof that raw meat itself is inherently unsafe.
  4. Cherry picking
    What you do: You highlight HUS, neurolisteriosis and GBS as if they are typical.
    Why it fails: Extreme outliers are not general rules.
    Callout: You cherry-pick edge cases. Show baseline incidence per portion in healthy people.
  5. Base rate neglect
    What you do: You ignore how rare severe outcomes are compared to total exposures.
    Why it fails: Risk requires a base rate.
    Callout: Where is your per-portion rate in healthy populations? Without it, your fear-mongering is statistics-free.
  6. Confusing hazard and risk
    What you do: You list molecular hazards and call it risk.
    Why it fails: Hazard ≠ risk without dose and exposure.
    Callout: Hazard is not risk. Show real dose-response under normal raw intake.
  7. Necessary vs sufficient
    What you do: You treat bacterial presence as proof of illness.
    Why it fails: Presence is not sufficient without host state, dose, strain.
    Callout: Presence ≠ causation. Prove sufficiency in humans.
  8. Ecological fallacy
    What you do: You use population totals to claim truth about individuals eating raw meat.
    Why it fails: Group stats don’t automatically apply to individual cases.
    Callout: Totals from many foods ≠ proof about raw vs cooked meat in healthy individuals.
  9. Category error
    What you do: You treat contamination as if it is an inherent property of raw meat.
    Why it fails: Contamination is handling and environment, not essence.
    Callout: You only prove contaminated food is risky, not raw meat itself.
  10. Ignoring post-cooking contamination
    What you do: You act as if heat ends the problem.
    Why it fails: Listeria and others thrive post-cooking in chilled foods.
    Callout: If heat solved it, there would be no outbreaks in cooked ready-meals.
  11. Straw man
    What you do: You pretend I claim all pathogens are beneficial.
    Why it fails: I say outcomes are context-dependent, not that all bacteria are good.
    Callout: You’re fighting a straw man. I never made that claim.
  12. False dichotomy
    What you do: You present it as raw=poison vs cooked=safe.
    Why it fails: Many mitigation strategies exist besides heat, and cooked food can also be contaminated.
    Callout: It’s not raw poison vs cooked safe. Show net health benefit, not just hazard reduction.
  13. Appeal to fear
    What you do: You drown in scary words like “molecular warfare.”
    Why it fails: Fear rhetoric ≠ data.
    Callout: Fear is not evidence. Show controlled human comparisons.
  14. Moving the goalposts
    What you do: You jump between WHO, models, toxins, infants to dodge the core claim.
    Why it fails: The criterion is simple: outcomes per portion, raw vs cooked.
    Callout: Stay on target. Show direct comparisons in humans or retract.
  15. Model reification
    What you do: You treat quantitative risk models as truth.
    Why it fails: Models = assumptions, not observed outcomes.
    Callout: A model is not proof. Show real human data, not math.
  16. Texas sharpshooter
    What you do: You match outbreaks to your story after the fact.
    Why it fails: Selective matching is not evidence.
    Callout: You pick cases that fit and ignore those that contradict. Show the full dataset.
  17. Composition/division fallacy
    What you do: You assume if some strains are harmful, all are harmful.
    Why it fails: Subset traits ≠ universal truth.
    Callout: A few virulent strains ≠ all raw meat, all hosts, all contexts.
  18. Begging the question
    What you do: You assume heat is necessary and conclude heat is necessary.
    Why it fails: Circular reasoning.
    Callout: You assume your conclusion. Show net health benefits beyond alternatives.
  19. Isolated demand for rigor
    What you do: You demand impossible RCTs from me but rely on weaker evidence yourself.
    Why it fails: Double standard.
    Callout: If ethics block RCTs, that applies to you too. Don’t demand what you can’t provide.
  20. False analogy with vultures
    What you do: You dismiss human stomach acid comparisons by jumping to extreme carrion eaters.
    Why it fails: Irrelevant species ≠ proof for humans.
    Callout: Vulture analogies don’t prove cooked > raw in healthy humans.
  21. Equivocation on “pathogen”
    What you do: You treat “pathogen” as if it always means guaranteed disease.
    Why it fails: Pathogen = potential, not inevitability.
    Callout: Potential ≠ outcome. Context decides.
  22. No True Scotsman
    What you do: Every time data contradicts you, you redefine what counts as “relevant risk.”
    Why it fails: Unfalsifiable.
    Callout: Stop shifting definitions. Fix clear criteria or concede.
  23. Anecdote & availability bias
    What you do: You rely on dramatic outbreaks instead of base rates.
    Why it fails: Memorable ≠ frequent.
    Callout: Anecdotes are not statistics. Show per-portion incidence.
  24. Moralistic/naturalistic misuse
    What you do: You call infection trials “unethical” and treat that as proof.
    Why it fails: Ethics block methods, not truth.
    Callout: Saying “it’s unethical” doesn’t prove your claim.
View attachment 4154697

:feelsuhh::feelsuhh::feelsuhh:
I only started writing more analyticaly to try and get this information into your thick skull. Hopefully you get it this time but I doubt it. This is beyond ridiculous. You're trying to use a semantic argument about the word "contaminated" to deny the entire reality of the industrialized food chain, simultaneously committing almost every logical fallacy listed in your pathetic little recap. You haven't reached checkmate; you've reached "I don't understand microbial ecology or risk management." Let me dismantle your entire, baseless defense brick by agonizing brick.
I. The WHO Data, Context, and the Fatal Irrelevance of Produce
First, you try to cherry-pick the WHO data, arguing that because produce accounts for more illness cases overall, meat is somehow safer or irrelevant. You are fundamentally confusing incidence (the number of people who get sick) with severity and lethality (the number of people who die or are permanently disabled). Get this straight: the CDC explicitly states that while produce contributes 46% of all foodborne illnesses, meat and poultry account for 29% of all deaths.[1] This isn't my logic; it’s the analysis of the US food system—where meat is cooked—proving that when the pathogens associated with raw meat do cause infection, the outcome is disproportionately lethal due to the specific virulence factors I already detailed.
The economic cost reflects this severity: the costliest food-pathogen pairs in the US are Campylobacter in poultry ($6.9 billion), and Salmonella in chicken and pork ($2.8 and $1.9 billion, respectively).[2] You are advocating for removing the control step—cooking—for the food categories that are the primary drivers of lethal and chronic disease, regardless of what happens with spinach. Your attempt to pivot to salad contamination is a monumental non sequitur because food safety standards demand that both lettuce and meat have controls. The control for meat is thermal inactivation; the control for lettuce is washing and sourcing. Claiming one failure invalidates the necessity of the other is idiotic.
II. The Categorical Error: Raw Meat Is Contaminated by Definition
Your entire argument hinges on the Category Error that raw meat is not inherently contaminated and that contamination is only a matter of handling. This is scientifically false in the context of industrial food production, which is where 99% of your meat comes from, and it’s why regulatory bodies exist.
Muscle Tissue vs. Product: While the muscle tissue of a healthy animal may be sterile at the moment of slaughter, the raw meat product you buy is not. The moment the animal is dressed, commensal and pathogenic flora from the hide, feces, and viscera contaminate the surface.[3] For ground meat (which includes products used for steak tartare or cannibal sandwiches you cited), surface contamination is mixed throughout the product during grinding, ensuring that low-dose pathogens are no longer confined to the surface, significantly increasing the risk.[4, 5]
Ubiquitous Pathogen Prevalence: This isn't a hypothetical situation; it's the base rate. Numerous studies document high baseline contamination: E. coli was detected in over half (53.6%) of total retail meat samples in one analysis, with 87.5% of poultry and 48.2% of ground beef being contaminated with E. coli. Similarly, Salmonella prevalence in various raw pork and beef products ranges from 4.4% to almost 14%, depending on the retail source. Raw poultry and pork, specifically, show contamination rates for Salmonella species at 39.6% and 42.9%, respectively, in certain markets.[6] These are not "rare, hypothetical contamination events"; these are documented, inherent microbial loads in the raw, non-sterile products of the industrialized food chain.
Regulatory Definition of Risk: Raw meat is officially designated a "potentially hazardous food" that naturally contains disease-causing microorganisms by regulatory bodies because of this predictable baseline contamination.[9, 10] Therefore, the safety argument is not about "cooked meat vs. uncontaminated raw meat," but cooked meat vs. inherently non-sterile raw meat.
You citing a 1974 study on microbiology isn't going to cut it against modern, mandatory, HACCP-based controls.[11, 12] Regulators mandate cooking because they recognize that contamination is reasonably likely to occur at the farm, slaughter, and processing stages.[10]
III. Post-Cooking Contamination vs. Microbial Inactivation: An Inexcusable False Dichotomy
Your central failure—the core of your checkmate fantasy—is the claim that post-cooking contamination makes raw meat less risky. This is a fundamental misrepresentation of food safety control hierarchy.
The Kill Step is Necessary: Cooking to a verified internal temperature (a "kill step") is the only method to achieve a predictable logarithmic reduction of pathogens (e.g., Salmonella or STEC) to an acceptable level. This is mandated precisely because the contamination load before cooking is high and virulent. Without this step, you retain the full, life-threatening load of organisms like Listeria (case-fatality rate ~20% ) and STEC (HUS risk up to 20% in children ).
Post-Cooking Contamination (PCC) is Preventable: PCC, where cooked food is re-contaminated by raw juices or contaminated surfaces, is a known and manageable hazard.[6] But the difference is critical: PCC introduces a new, secondary risk, whereas consuming raw meat means you are retaining the primary, known risk at the source. Public health advice for cooked meat explicitly addresses this by mandating strict separation of raw and cooked items (e.g., using separate cutting boards and utensils).[6, 7] The CDC, FDA, and EFSA universally recommend cooking and preventing cross-contamination.[6, 7]
The Trade-Off is Catastrophic: Your logic is that because cooking adds a preventable secondary risk (PCC), it is riskier than retaining the guaranteed primary risk of high-dose, genetically virulent pathogens. This is a deliberate misapplication of risk analysis. You sacrifice a near-certain survival benefit (pathogen elimination by heat) for the preservation of marginal nutrients, accepting a quantifiable risk of irreversible paralysis (GBS from Campylobacter - 1 in 1,000 infected ) or death.
IV. HCAs vs. DALYs: Trading a Carcinogen Risk for Near-Immediate Organ Failure
Finally, you try to use the formation of harmful compounds like Heterocyclic Amines (HCAs) during cooking as a definitive loss, claiming 100% harm (HCAs) outweighs <100% chance of bacterial harm. This is a catastrophic failure of risk comparison:
HCA Risk is Chronic and Dose-Dependent: The risk associated with HCAs and Advanced Glycation End products (AGEs) is chronic, accumulated over decades, and requires specific dose-response models often linked to high-temperature cooking.
Pathogen Risk is Acute and Lethal: The risk associated with STEC, Listeria, and Salmonella is acute, severe, and measured not in cancer probability fifty years from now, but in immediate hospitalization (nearly 100% for listeriosis ) and DALYs (e.g., 32,900 DALYs for Salmonella [8]). You are trading a long-term, statistical cancer risk for an acute, high-lethality risk of kidney failure, brain damage (44% neurological sequelae in listeriosis survivors ), or death.
Your argument that "raw meat is not contaminated" is refuted by microbial baseline data, and your argument that "cooking increases risk" is a ridiculous category error that confuses a preventable handling failure with the elimination of the primary, lethal biological hazard. You have not reached checkmate; you've merely proven you don't understand the science of microbial virulence or public health risk assessment.
 
don't u get parasites from raw meat? Forgive me if im wrong but im pretty sure some bacteria can actually kill you with 100% lethality rate, woudlnt risk if I were you
fucking slave
 
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Reactions: WaterDog
I only started writing more analyticaly to try and get this information into your thick skull. Hopefully you get it this time but I doubt it. This is beyond ridiculous. You're trying to use a semantic argument about the word "contaminated" to deny the entire reality of the industrialized food chain, simultaneously committing almost every logical fallacy listed in your pathetic little recap. You haven't reached checkmate; you've reached "I don't understand microbial ecology or risk management." Let me dismantle your entire, baseless defense brick by agonizing brick.
I. The WHO Data, Context, and the Fatal Irrelevance of Produce
First, you try to cherry-pick the WHO data, arguing that because produce accounts for more illness cases overall, meat is somehow safer or irrelevant. You are fundamentally confusing incidence (the number of people who get sick) with severity and lethality (the number of people who die or are permanently disabled). Get this straight: the CDC explicitly states that while produce contributes 46% of all foodborne illnesses, meat and poultry account for 29% of all deaths.[1] This isn't my logic; it’s the analysis of the US food system—where meat is cooked—proving that when the pathogens associated with raw meat do cause infection, the outcome is disproportionately lethal due to the specific virulence factors I already detailed.
The economic cost reflects this severity: the costliest food-pathogen pairs in the US are Campylobacter in poultry ($6.9 billion), and Salmonella in chicken and pork ($2.8 and $1.9 billion, respectively).[2] You are advocating for removing the control step—cooking—for the food categories that are the primary drivers of lethal and chronic disease, regardless of what happens with spinach. Your attempt to pivot to salad contamination is a monumental non sequitur because food safety standards demand that both lettuce and meat have controls. The control for meat is thermal inactivation; the control for lettuce is washing and sourcing. Claiming one failure invalidates the necessity of the other is idiotic.
II. The Categorical Error: Raw Meat Is Contaminated by Definition
Your entire argument hinges on the Category Error that raw meat is not inherently contaminated and that contamination is only a matter of handling. This is scientifically false in the context of industrial food production, which is where 99% of your meat comes from, and it’s why regulatory bodies exist.
Muscle Tissue vs. Product: While the muscle tissue of a healthy animal may be sterile at the moment of slaughter, the raw meat product you buy is not. The moment the animal is dressed, commensal and pathogenic flora from the hide, feces, and viscera contaminate the surface.[3] For ground meat (which includes products used for steak tartare or cannibal sandwiches you cited), surface contamination is mixed throughout the product during grinding, ensuring that low-dose pathogens are no longer confined to the surface, significantly increasing the risk.[4, 5]
Ubiquitous Pathogen Prevalence: This isn't a hypothetical situation; it's the base rate. Numerous studies document high baseline contamination: E. coli was detected in over half (53.6%) of total retail meat samples in one analysis, with 87.5% of poultry and 48.2% of ground beef being contaminated with E. coli. Similarly, Salmonella prevalence in various raw pork and beef products ranges from 4.4% to almost 14%, depending on the retail source. Raw poultry and pork, specifically, show contamination rates for Salmonella species at 39.6% and 42.9%, respectively, in certain markets.[6] These are not "rare, hypothetical contamination events"; these are documented, inherent microbial loads in the raw, non-sterile products of the industrialized food chain.
Regulatory Definition of Risk: Raw meat is officially designated a "potentially hazardous food" that naturally contains disease-causing microorganisms by regulatory bodies because of this predictable baseline contamination.[9, 10] Therefore, the safety argument is not about "cooked meat vs. uncontaminated raw meat," but cooked meat vs. inherently non-sterile raw meat.
You citing a 1974 study on microbiology isn't going to cut it against modern, mandatory, HACCP-based controls.[11, 12] Regulators mandate cooking because they recognize that contamination is reasonably likely to occur at the farm, slaughter, and processing stages.[10]
III. Post-Cooking Contamination vs. Microbial Inactivation: An Inexcusable False Dichotomy
Your central failure—the core of your checkmate fantasy—is the claim that post-cooking contamination makes raw meat less risky. This is a fundamental misrepresentation of food safety control hierarchy.
The Kill Step is Necessary: Cooking to a verified internal temperature (a "kill step") is the only method to achieve a predictable logarithmic reduction of pathogens (e.g., Salmonella or STEC) to an acceptable level. This is mandated precisely because the contamination load before cooking is high and virulent. Without this step, you retain the full, life-threatening load of organisms like Listeria (case-fatality rate ~20% ) and STEC (HUS risk up to 20% in children ).
Post-Cooking Contamination (PCC) is Preventable: PCC, where cooked food is re-contaminated by raw juices or contaminated surfaces, is a known and manageable hazard.[6] But the difference is critical: PCC introduces a new, secondary risk, whereas consuming raw meat means you are retaining the primary, known risk at the source. Public health advice for cooked meat explicitly addresses this by mandating strict separation of raw and cooked items (e.g., using separate cutting boards and utensils).[6, 7] The CDC, FDA, and EFSA universally recommend cooking and preventing cross-contamination.[6, 7]
The Trade-Off is Catastrophic: Your logic is that because cooking adds a preventable secondary risk (PCC), it is riskier than retaining the guaranteed primary risk of high-dose, genetically virulent pathogens. This is a deliberate misapplication of risk analysis. You sacrifice a near-certain survival benefit (pathogen elimination by heat) for the preservation of marginal nutrients, accepting a quantifiable risk of irreversible paralysis (GBS from Campylobacter - 1 in 1,000 infected ) or death.
IV. HCAs vs. DALYs: Trading a Carcinogen Risk for Near-Immediate Organ Failure
Finally, you try to use the formation of harmful compounds like Heterocyclic Amines (HCAs) during cooking as a definitive loss, claiming 100% harm (HCAs) outweighs <100% chance of bacterial harm. This is a catastrophic failure of risk comparison:
HCA Risk is Chronic and Dose-Dependent: The risk associated with HCAs and Advanced Glycation End products (AGEs) is chronic, accumulated over decades, and requires specific dose-response models often linked to high-temperature cooking.
Pathogen Risk is Acute and Lethal: The risk associated with STEC, Listeria, and Salmonella is acute, severe, and measured not in cancer probability fifty years from now, but in immediate hospitalization (nearly 100% for listeriosis ) and DALYs (e.g., 32,900 DALYs for Salmonella [8]). You are trading a long-term, statistical cancer risk for an acute, high-lethality risk of kidney failure, brain damage (44% neurological sequelae in listeriosis survivors ), or death.
Your argument that "raw meat is not contaminated" is refuted by microbial baseline data, and your argument that "cooking increases risk" is a ridiculous category error that confuses a preventable handling failure with the elimination of the primary, lethal biological hazard. You have not reached checkmate; you've merely proven you don't understand the science of microbial virulence or public health risk assessment.
My point is that everything you mention in this chapter is completely irrelevant to this debate:
I. The WHO Data, Context, and the Fatal Irrelevance of Produce


II. The Categorical Error: Raw Meat Is Contaminated by Definition
"This is scientifically false in the context of industrial food production"
We are not talking about variables we are talking about cooked vs raw meat.

I do not advocate eating industrial food and it is not inherent to raw meat, raw meat is not industrial so invalid point.
We are not talking about "product" as your categorise it, just the raw meat muscle tissue which is not inherently contaminated by your mentioned processes, just eaten as is.

We are not talking about ground meat, Raw meat is not inherently ground. I only mentioned the popular dishes as you earlier mentioned that nobody is stupid enough to eat raw meat. Estimated 50 million sushi dishes are served daily, other european and middle esatern raw dishes 5-10 million a day, Afrika, South America and asia 5-15 million a day, total of 40 to 65 million a day.
- not a point or base of argumentation whatsoever just kind of invalidating your earlier statements about nobody doing it, it being not allowed or whatever you said.

Again you refer to industrial meat (raw meat is not inherently industrial) to prove that completely normal bacteria are present, you are just repeating a point from earlier which I already debunked? Quoting percentages across assorted markets and species without strain typing, enumeration and serving size is classic base rate neglect. To argue cooked is safer than raw you must show incidence per portion in humans when handling is controlled. You have not.
  • You say retail raw meat often shows “contamination” (e.g., E. coli on ~50% of samples; Salmonella on a fraction of pork/beef; higher on poultry). That’s a hazard statement, not risk.
  • Now add exposure. Conservatively, 40–65 million people eat raw animal foods every day worldwide (sushi/sashimi ~30–40M; raw dishes like tartare/carpaccio/kitfo/kibbeh ~5–10M; traditional/raw-fermented in Africa/Asia/SA ~5–15M; niche diets <1M). That’s tens of millions of daily portions.
  • If your “baseline contamination” automatically translated to clinically meaningful disease in healthy hosts, the observed illness burden from raw foods should be astronomical. Back-of-envelope: at just 1 symptomatic case per 100,000 raw servings, 40M servings/day would yield ~400 clinical cases every single day (≈146,000/year) from raw alone. Severe outcomes (HUS/GBS/neurolisteriosis) would be visible at scale. They aren’t—because…
  • Dose, strain, viability, and handling determine risk.
    • Most E. coli strains on meat are commensal/non-STEC; presence ≠ O157:H7.
    Dose matters: low counts rarely overcome a healthy host.
    Viability drops with time/temp/salt/acid; many servings are frozen (sushi-grade fish), salted, marinated, or promptly consumed.
    Matrix matters: intact muscle ≠ ground meat; risk rises when you grind (hazard mixed throughout), which is a processvariable—not an inherent property of “raw.”
- Refering to post #111, you might want to read It again since you didn't understand it the first time and I won't bother explaining it again. Most E coli are commensals. Presence alone does not equal disease.

"Regulatory Definition of Risk: Raw meat is officially designated a "potentially hazardous food" that naturally contains disease-causing microorganisms by regulatory bodies because of this predictable baseline contamination.[9, 10] Therefore, the safety argument is not about "cooked meat vs. uncontaminated raw meat," but cooked meat vs. inherently non-sterile raw meat.
You citing a 1974 study on microbiology isn't going to cut it against modern, mandatory, HACCP-based controls.[11, 12] Regulators mandate cooking because they recognize that contamination is reasonably likely to occur at the farm, slaughter, and processing stages.[10]"
- This chapter proves nothing? how is definition and official labels and regulations proof of anything. And raw meat is still inherently not contaminated with poison or disease causing organisms (and don't bring up variables such as "what about if the animal is sick", not an inherent property so don't bother). How the fuck does HACCP have anything to do with the study? This whole chapter is just you refering to guidelines and trying to use definitions but it doesn't prove anything it is not science. Appealing to a policy label does not establish your scientific conclusion.
recap:

  1. Essence vs. process
    You call raw meat “contaminated by definition.” That’s a category error. Intact muscle tissue from a healthy animal is initially low in bacteria; contamination arises through process (hide/gut contact, equipment, water, hands, air) during slaughter and handling. If something depends on handling, it’s not an inherent property.
  2. Hazard ≠ risk
    Your prevalence numbers show detection of bacteria (hazard). Risk requires strain, viable dose per portion, and host factors. “E. coli found in X%” means nothing without serotype and dose. Presence ≠ disease.
  3. Whole cuts ≠ ground products
    You’re conflating cases. Grinding mixes surface flora throughout the product, raising risk—that applies to ground meat. It cannot be generalized to all raw preparations made from freshly trimmed surfaces under hygienic handling. Using a high-risk subset as a universal rule is a non sequitur.
  4. Regulations are policy, not proof
    HACCP/“potentially hazardous food” are management frameworks for the industrial chain, designed for worst-case. They are not biological proof that raw meat is inherently unsafe for healthy humans under controlled sourcing and handling. Appealing to a policy label is not causal evidence.
  5. Base rate & dose neglected
    Quoting percentages across species/markets without strain typing, CFU counts, portion size, and host condition is base-rate neglect. To prove “cooked > raw,” you must show incidence per portion in humans, with raw status isolated from handling/supply chain factors. You haven’t.
  6. Begging the question
    Your hidden premise is: “Retail raw products usually contain dangerous doses of virulent pathogens, therefore cooking is necessary.” That assumes your conclusion. You first have to demonstrate that dangerous dose/virulence is the norm at point of consumption for properly handled raw foods. “Contamination can occur” is not the same.
  7. Post-cooking contamination ignored
    Heat only reduces what’s present at the moment of cooking. Re-contamination after the “kill step” is a well-documented outbreak source. If heat alone solved the problem, ready-to-eat cooked foods would never cause outbreaks—but they do. The decisive factor is hygiene and time/temperature throughout the chain, not raw status.
Conclusion
Presence is not cause. Process is not essence. Policy is not proof. All you’ve shown is that contaminated food is risky—that’s trivial. You have not shown “cooked > raw” in human per-portion outcomes with controlled handling. Bring that evidence, or drop the claim.

Basically you claim category error but "in the context of industrial food production" which is not inherent to raw meat either, so raw meat remains inherently uncontaminated at least with poison or the other disease causing things you are arguing fore which is what I have been saying this whole time. "Ubiquitous Pathogen Prevalence" the evidence to show the amount of bacteria (which as earlier mentioned are not even a problem) again inherently imply industrial handling and packaging etc. not inherent to raw meat.



III. Post-Cooking Contamination vs. Microbial Inactivation: An Inexcusable False Dichotomy

again In this chapter you mention "Salmonella or STEC", why? it is like bringing up nutrients again, go back and read my answers so you understand. From now on stop referring to guidelines absolutely proves nothing.

Again you are just repeating the fact that you did not understand anything from previous writing.

Debate Summary: Raw vs Cooked Meat​

  1. Nutrients
  • Cooking always reduces vitamins (B12, folate, vitamin C, taurine, etc).
  • Raw meat preserves these.
  • You admitted this, so we agree raw > cooked nutritionally.
  1. Harmful Compounds
  • Cooking creates toxins every time (HCAs, PAHs, AGEs, oxidized fats).
  • Raw meat never creates new toxins.
  • 0 harmful compounds = less healthy, period.
  1. Bacteria & Pathogens
  • Your entire case rests on saying “raw meat is inherently contaminated and therefore dangerous.”
  • But:
    • Healthy muscle tissue is sterile. Pathogens come from handling, environment, or processing — not the raw state itself.
    • Presence ≠ disease. Most E. coli are commensals, Salmonella and Campylobacter are carried asymptomatically by millions.
    • Koch’s postulates: presence of bacteria does not prove causation.
    • Pathogen = hazard, not guaranteed risk. Risk = hazard × dose × host context. You conflate the two.
    • Outbreaks prove contamination events, not that raw meat itself is inherently dangerous.
  1. Cooking and Safety
  • You argue cooking is the “kill step” that solves risk. But:
    • Cooked foods still cause outbreaks (Listeria in ready meals, Salmonella in peanut butter, E. coli in frozen veg).
    • Cooking adds toxic compounds 100% of the time, while bacterial contamination is <100%.
    • Cooking also increases risk of post-cooking contamination.
  1. Logical Errors in Your Case
  • Misplaced burden of proof: you claim cooked > raw, so you must provide positive human outcome data.
  • Appeal to authority: WHO/CDC data on global outbreaks is not direct evidence of cooked > raw per portion.
  • Cherry picking: highlighting rare, extreme outcomes (HUS, neurolisteriosis, GBS) as if they are baseline.
  • Base rate neglect: ignoring how rare these cases are compared to the total raw servings consumed daily (tens of millions worldwide).
  • Category error: treating industrial processing contamination as if it is inherent to raw meat.
  • False dichotomy: presenting the choice as “cook or die,” ignoring handling, sourcing, and hygiene.
  • Straw man: acting like I claimed all pathogens are “good,” when I said they are context-dependent.
  • Hazard ≠ risk: describing scary molecular mechanisms but not showing per-portion risk in healthy humans.
  • Policy ≠ science: HACCP rules and “hazardous food” labels are regulations, not scientific proof.
  1. Bottom Line
  • Raw meat is not inherently contaminated or poisonous.
  • Cooking always reduces nutrients and creates harmful compounds.
  • Pathogens are context-dependent, not automatic disease.
  • You have not shown a single controlled human study proving cooked meat is healthier than raw meat.
So the current status:

  • Nutrient loss → raw wins.
  • Harmful compounds → raw wins.
  • Pathogens → you rely on fear, policies, and hazard descriptions, but you cannot show direct per-portion human outcome data proving cooked > raw.
Therefore, your position that “cooked meat is healthier than raw meat” is unproven.

Why you still haven’t proven bacterial danger in raw meat​

  1. Presence ≠ Proof of Danger
    You keep repeating names of bacteria (E. coli, Salmonella, Listeria, Campylobacter) as if detection = disease. But microbiology 101 says otherwise:
  • Most E. coli strains are commensal and live in every human from birth (literally essential for vitamin K synthesis and gut balance). Only a few subtypes like O157:H7 are virulent.
  • Salmonella and Campylobacter are carried asymptomatically by tens of millions worldwide without illness.
  • Listeria is found everywhere in soil, water, and plants — yet healthy people rarely get sick.
    This is Koch’s postulates in action: you cannot jump from “present” to “causal disease.” Until you isolate strain, dose, and host context, your claim is just hazard listing, not proof of risk.
  1. Hazard ≠ Risk
    You describe scary mechanisms (Shiga toxins, LLO, mimicry in GBS) — but those are hazards, not risks.
  • Risk requires dose + exposure + host susceptibility.
  • Raw meat is not automatically contaminated, and even when pathogens are present, dose often isn’t sufficient for disease in healthy hosts.
  • Otherwise, every single sushi, tartare, carpaccio, sashimi consumer would end up in hospital. That’s clearly not reality.
  1. Contamination ≠ Inherent Property
    Raw muscle tissue in a healthy animal is sterile at slaughter (see Gill, 1979, PMID: 29564). Contamination happens during handling or environment, not because “raw meat is poison.”
    That means your entire “raw = dangerous” framing is a category error. You’re really proving “contaminated food can be dangerous.” Congratulations, that applies equally to cooked food.
  2. Cooked Meat Also Causes Outbreaks
    If heat was the magical solution, there would be no outbreaks from cooked foods. But there are:
  • Listeria in frozen cooked meals (CDC outbreak reports, 2018).
  • Salmonella in cooked chicken products (CDC, 2019 outbreak linked to cooked, breaded chicken).
  • E. coli in pasteurized foods (documented by EFSA, 2020).
    Cooking doesn’t eliminate risk — it just shifts it to post-cooking contamination and adds toxic compounds (HCAs, PAHs, AGEs) every single time.


    Please only mention relevant things I am wasting time on your public safety guidelines and things I have already proven and debunked. And do not claim that it is your own writing, pathetic. Provide per-portion human incidence (healthy adults) comparing raw vs cooked with handling controlled, or a QRA that matches real-world case counts for raw servings. Hazards and policy labels are not proof of cooked > raw; or use PURE logic and argumentation you write way too much bullshit
 
My point is that everything you mention in this chapter is completely irrelevant to this debate:
I. The WHO Data, Context, and the Fatal Irrelevance of Produce


II. The Categorical Error: Raw Meat Is Contaminated by Definition
"This is scientifically false in the context of industrial food production"
We are not talking about variables we are talking about cooked vs raw meat.

I do not advocate eating industrial food and it is not inherent to raw meat, raw meat is not industrial so invalid point.
We are not talking about "product" as your categorise it, just the raw meat muscle tissue which is not inherently contaminated by your mentioned processes, just eaten as is.

We are not talking about ground meat, Raw meat is not inherently ground. I only mentioned the popular dishes as you earlier mentioned that nobody is stupid enough to eat raw meat. Estimated 50 million sushi dishes are served daily, other european and middle esatern raw dishes 5-10 million a day, Afrika, South America and asia 5-15 million a day, total of 40 to 65 million a day.
- not a point or base of argumentation whatsoever just kind of invalidating your earlier statements about nobody doing it, it being not allowed or whatever you said.

Again you refer to industrial meat (raw meat is not inherently industrial) to prove that completely normal bacteria are present, you are just repeating a point from earlier which I already debunked? Quoting percentages across assorted markets and species without strain typing, enumeration and serving size is classic base rate neglect. To argue cooked is safer than raw you must show incidence per portion in humans when handling is controlled. You have not.
  • You say retail raw meat often shows “contamination” (e.g., E. coli on ~50% of samples; Salmonella on a fraction of pork/beef; higher on poultry). That’s a hazard statement, not risk.
  • Now add exposure. Conservatively, 40–65 million people eat raw animal foods every day worldwide (sushi/sashimi ~30–40M; raw dishes like tartare/carpaccio/kitfo/kibbeh ~5–10M; traditional/raw-fermented in Africa/Asia/SA ~5–15M; niche diets <1M). That’s tens of millions of daily portions.
  • If your “baseline contamination” automatically translated to clinically meaningful disease in healthy hosts, the observed illness burden from raw foods should be astronomical. Back-of-envelope: at just 1 symptomatic case per 100,000 raw servings, 40M servings/day would yield ~400 clinical cases every single day (≈146,000/year) from raw alone. Severe outcomes (HUS/GBS/neurolisteriosis) would be visible at scale. They aren’t—because…
  • Dose, strain, viability, and handling determine risk.
    • Most E. coli strains on meat are commensal/non-STEC; presence ≠ O157:H7.
    Dose matters: low counts rarely overcome a healthy host.
    Viability drops with time/temp/salt/acid; many servings are frozen (sushi-grade fish), salted, marinated, or promptly consumed.
    Matrix matters: intact muscle ≠ ground meat; risk rises when you grind (hazard mixed throughout), which is a processvariable—not an inherent property of “raw.”
- Refering to post #111, you might want to read It again since you didn't understand it the first time and I won't bother explaining it again. Most E coli are commensals. Presence alone does not equal disease.

"Regulatory Definition of Risk: Raw meat is officially designated a "potentially hazardous food" that naturally contains disease-causing microorganisms by regulatory bodies because of this predictable baseline contamination.[9, 10] Therefore, the safety argument is not about "cooked meat vs. uncontaminated raw meat," but cooked meat vs. inherently non-sterile raw meat.
You citing a 1974 study on microbiology isn't going to cut it against modern, mandatory, HACCP-based controls.[11, 12] Regulators mandate cooking because they recognize that contamination is reasonably likely to occur at the farm, slaughter, and processing stages.[10]"
- This chapter proves nothing? how is definition and official labels and regulations proof of anything. And raw meat is still inherently not contaminated with poison or disease causing organisms (and don't bring up variables such as "what about if the animal is sick", not an inherent property so don't bother). How the fuck does HACCP have anything to do with the study? This whole chapter is just you refering to guidelines and trying to use definitions but it doesn't prove anything it is not science. Appealing to a policy label does not establish your scientific conclusion.
recap:

  1. Essence vs. process
    You call raw meat “contaminated by definition.” That’s a category error. Intact muscle tissue from a healthy animal is initially low in bacteria; contamination arises through process (hide/gut contact, equipment, water, hands, air) during slaughter and handling. If something depends on handling, it’s not an inherent property.
  2. Hazard ≠ risk
    Your prevalence numbers show detection of bacteria (hazard). Risk requires strain, viable dose per portion, and host factors. “E. coli found in X%” means nothing without serotype and dose. Presence ≠ disease.
  3. Whole cuts ≠ ground products
    You’re conflating cases. Grinding mixes surface flora throughout the product, raising risk—that applies to ground meat. It cannot be generalized to all raw preparations made from freshly trimmed surfaces under hygienic handling. Using a high-risk subset as a universal rule is a non sequitur.
  4. Regulations are policy, not proof
    HACCP/“potentially hazardous food” are management frameworks for the industrial chain, designed for worst-case. They are not biological proof that raw meat is inherently unsafe for healthy humans under controlled sourcing and handling. Appealing to a policy label is not causal evidence.
  5. Base rate & dose neglected
    Quoting percentages across species/markets without strain typing, CFU counts, portion size, and host condition is base-rate neglect. To prove “cooked > raw,” you must show incidence per portion in humans, with raw status isolated from handling/supply chain factors. You haven’t.
  6. Begging the question
    Your hidden premise is: “Retail raw products usually contain dangerous doses of virulent pathogens, therefore cooking is necessary.” That assumes your conclusion. You first have to demonstrate that dangerous dose/virulence is the norm at point of consumption for properly handled raw foods. “Contamination can occur” is not the same.
  7. Post-cooking contamination ignored
    Heat only reduces what’s present at the moment of cooking. Re-contamination after the “kill step” is a well-documented outbreak source. If heat alone solved the problem, ready-to-eat cooked foods would never cause outbreaks—but they do. The decisive factor is hygiene and time/temperature throughout the chain, not raw status.
Conclusion
Presence is not cause. Process is not essence. Policy is not proof. All you’ve shown is that contaminated food is risky—that’s trivial. You have not shown “cooked > raw” in human per-portion outcomes with controlled handling. Bring that evidence, or drop the claim.

Basically you claim category error but "in the context of industrial food production" which is not inherent to raw meat either, so raw meat remains inherently uncontaminated at least with poison or the other disease causing things you are arguing fore which is what I have been saying this whole time. "Ubiquitous Pathogen Prevalence" the evidence to show the amount of bacteria (which as earlier mentioned are not even a problem) again inherently imply industrial handling and packaging etc. not inherent to raw meat.



III. Post-Cooking Contamination vs. Microbial Inactivation: An Inexcusable False Dichotomy

again In this chapter you mention "Salmonella or STEC", why? it is like bringing up nutrients again, go back and read my answers so you understand. From now on stop referring to guidelines absolutely proves nothing.

Again you are just repeating the fact that you did not understand anything from previous writing.

Debate Summary: Raw vs Cooked Meat​

  1. Nutrients
  • Cooking always reduces vitamins (B12, folate, vitamin C, taurine, etc).
  • Raw meat preserves these.
  • You admitted this, so we agree raw > cooked nutritionally.
  1. Harmful Compounds
  • Cooking creates toxins every time (HCAs, PAHs, AGEs, oxidized fats).
  • Raw meat never creates new toxins.
  1. Bacteria & Pathogens
  • Your entire case rests on saying “raw meat is inherently contaminated and therefore dangerous.”
  • But:
    • Healthy muscle tissue is sterile. Pathogens come from handling, environment, or processing — not the raw state itself.
    • Presence ≠ disease. Most E. coli are commensals, Salmonella and Campylobacter are carried asymptomatically by millions.
    • Koch’s postulates: presence of bacteria does not prove causation.
    • Pathogen = hazard, not guaranteed risk. Risk = hazard × dose × host context. You conflate the two.
    • Outbreaks prove contamination events, not that raw meat itself is inherently dangerous.
  1. Cooking and Safety
  • You argue cooking is the “kill step” that solves risk. But:
    • Cooked foods still cause outbreaks (Listeria in ready meals, Salmonella in peanut butter, E. coli in frozen veg).
    • Cooking adds toxic compounds 100% of the time, while bacterial contamination is <100%.
    • Cooking also increases risk of post-cooking contamination.
  1. Logical Errors in Your Case
  • Misplaced burden of proof: you claim cooked > raw, so you must provide positive human outcome data.
  • Appeal to authority: WHO/CDC data on global outbreaks is not direct evidence of cooked > raw per portion.
  • Cherry picking: highlighting rare, extreme outcomes (HUS, neurolisteriosis, GBS) as if they are baseline.
  • Base rate neglect: ignoring how rare these cases are compared to the total raw servings consumed daily (tens of millions worldwide).
  • Category error: treating industrial processing contamination as if it is inherent to raw meat.
  • False dichotomy: presenting the choice as “cook or die,” ignoring handling, sourcing, and hygiene.
  • Straw man: acting like I claimed all pathogens are “good,” when I said they are context-dependent.
  • Hazard ≠ risk: describing scary molecular mechanisms but not showing per-portion risk in healthy humans.
  • Policy ≠ science: HACCP rules and “hazardous food” labels are regulations, not scientific proof.
  1. Bottom Line
  • Raw meat is not inherently contaminated or poisonous.
  • Cooking always reduces nutrients and creates harmful compounds.
  • Pathogens are context-dependent, not automatic disease.
  • You have not shown a single controlled human study proving cooked meat is healthier than raw meat.
So the current status:

  • Nutrient loss → raw wins.
  • Harmful compounds → raw wins.
  • Pathogens → you rely on fear, policies, and hazard descriptions, but you cannot show direct per-portion human outcome data proving cooked > raw.
Therefore, your position that “cooked meat is healthier than raw meat” is unproven.

Why you still haven’t proven bacterial danger in raw meat​

  1. Presence ≠ Proof of Danger
    You keep repeating names of bacteria (E. coli, Salmonella, Listeria, Campylobacter) as if detection = disease. But microbiology 101 says otherwise:
  • Most E. coli strains are commensal and live in every human from birth (literally essential for vitamin K synthesis and gut balance). Only a few subtypes like O157:H7 are virulent.
  • Salmonella and Campylobacter are carried asymptomatically by tens of millions worldwide without illness.
  • Listeria is found everywhere in soil, water, and plants — yet healthy people rarely get sick.
    This is Koch’s postulates in action: you cannot jump from “present” to “causal disease.” Until you isolate strain, dose, and host context, your claim is just hazard listing, not proof of risk.
  1. Hazard ≠ Risk
    You describe scary mechanisms (Shiga toxins, LLO, mimicry in GBS) — but those are hazards, not risks.
  • Risk requires dose + exposure + host susceptibility.
  • Raw meat is not automatically contaminated, and even when pathogens are present, dose often isn’t sufficient for disease in healthy hosts.
  • Otherwise, every single sushi, tartare, carpaccio, sashimi consumer would end up in hospital. That’s clearly not reality.
  1. Contamination ≠ Inherent Property
    Raw muscle tissue in a healthy animal is sterile at slaughter (see Gill, 1979, PMID: 29564). Contamination happens during handling or environment, not because “raw meat is poison.”
    That means your entire “raw = dangerous” framing is a category error. You’re really proving “contaminated food can be dangerous.” Congratulations, that applies equally to cooked food.
  2. Cooked Meat Also Causes Outbreaks
    If heat was the magical solution, there would be no outbreaks from cooked foods. But there are:
  • Listeria in frozen cooked meals (CDC outbreak reports, 2018).
  • Salmonella in cooked chicken products (CDC, 2019 outbreak linked to cooked, breaded chicken).
  • E. coli in pasteurized foods (documented by EFSA, 2020).
    Cooking doesn’t eliminate risk — it just shifts it to post-cooking contamination and adds toxic compounds (HCAs, PAHs, AGEs) every single time.


    Please only mention relevant things I am wasting time on your public safety guidelines and things I have already proven and debunked. And do not claim that it is your own writing, pathetic. Provide per-portion human incidence (healthy adults) comparing raw vs cooked with handling controlled, or a QRA that matches real-world case counts for raw servings. Hazards and policy labels are not proof of cooked > raw; or use PURE logic and argumentation you write way too much bullshit
You claim "raw meat is not inherently contaminated" because the muscle tissue is initially sterile. This is a deliberate, moronic Category Error that ignores the process, and the process is the product. The meat you consume is not excised in an aseptic lab; it comes from a slaughter line where pathogens from the animal's hide, feet, and viscera—the reservoirs of virulent E. coli and Salmonella—contaminate the surface the moment the carcass is dressed.[1]
We don't need hypothetical scenarios. We have real-world microbial baselines:
Retail meat surveys show E. coli contamination in 53.6% of samples, including 48.2% of ground beef, and 25% of whole beef cuts.[2]
Salmonella prevalence in raw pork and beef ranges from 4.4% to almost 14% in retail markets, and contamination is even higher in poultry (around 40%).
Raw beef preparations in markets have been found to harbor high microbial loads, with Total Aerobic Counts ranging from 10^8 to 10^{10}\ \text{CFU/g}, and 82.5% of E. coli isolates being multi-drug resistant.[3, 4]
This isn't contamination by chance; it is contamination by definition of the non-sterile commodity. Your argument is literally, "If we ignore all the pathogens, raw meat is safe." Congratulations, if we ignore all gravity, flying is safe.
You then commit Base Rate Neglect by arguing the massive number of daily raw servings (sushi, tartare) means the risk is negligible. This ignores the ultra-low infectious dose (ID) of the real killers:
STEC O157:H7 outbreaks confirm a minimal ID as low as 10–100 CFU is sufficient to cause infection, with some surveillance data suggesting \sim 50 CFU.
Campylobacter jejuni can cause illness upon ingestion of as little as \sim 500 cells.
When retail meat carries microbial loads of 10^8 \text{CFU/g} [4], even if only 0.001% of that load is a virulent strain, you are still ingesting thousands of times the infectious dose. Your healthy gut, while having excellent colonization resistance , cannot nullify a concentrated, low-ID, virulent dose of a specific toxin-producing organism.
Now let's ram the lethality data down your throat, because HCAs are a statistical pinprick compared to this:
Listeria Monocytogenes: The Ultimate Fetal and Neuro Killer.
This is the third leading cause of foodborne death [5], with a case-fatality rate of 20%.
The pathogen uses a complex molecular arsenal, controlled by the PrfA master regulon, which activates LLO (Listeriolysin O) to rupture the phagocytic vacuole and ActA to hijack the host's actin system, turning the bacterium into an intracellular rocket traveling at 0.3\ \mu\text{m/s} to spread cell-to-cell, completely bypassing the immune system.
This direct neuro-invasion causes catastrophic long-term damage: a major French study found that 44% of patients surviving neurolisteriosis suffered long-term neurological sequelae , and CNS infection generally confers a three-fold higher risk of developing dementia over time.[6]
For pregnant individuals, the risk is unconscionable: nearly 25% of pregnancy-associated cases result in fetal loss or death of the newborn. This is not an "edge case"; this is a documented, high-probability disaster caused by a pathogen you are defending.
Campylobacter: Molecular Mimicry and Paralysis.
Campylobacter infection is a leading cause of the severe autoimmune paralysis, Guillain-Barré Syndrome (GBS).
This occurs because the pathogen's lipooligosaccharides (LOS) share structural homology with human gangliosides in peripheral nerves—a phenomenon called molecular mimicry. The antibodies your body generates to fight the bacteria from the raw meat turn around and attack your own nervous system, causing axonal injury and paralysis.
The risk is quantified: approximately 1 in every 1,000 people infected with C. jejuni develops GBS, and evidence of recent Campylobacter infection is found in as many as 40% of GBS cases. Treating the initial infection with antibiotics has not been definitively proven to prevent the neurological complications once the autoimmune cascade has started, meaning only prevention of infection works.
STEC: HUS and Kidney Failure.
STEC's Shiga toxin binds to the \text{Gb}_3 receptor on vascular endothelial cells, particularly in the kidney [7, 8], where the active A1 subunit cleaves a specific adenine residue from the 28S rRNA of the 60S ribosomal subunit, which irreversibly inhibits host protein synthesis. This molecular sabotage causes the microvascular damage that leads directly to Hemolytic Uremic Syndrome (HUS) and acute renal failure, which affects 15-20% of infected children and carries a mortality rate up to 20% in older adults.
The Final Scientific Verdict: Cooking is the Non-Negotiable Control
You argue that Post-Cooking Contamination (PCC) makes cooking riskier. This is the ultimate False Dichotomy. The regulatory system—which is built on HACCP principles —requires that the thermal process achieve a robust 7-log reduction of Salmonella in poultry and 6.5-log reduction in beef [9, 10], thereby eliminating the primary, high-dose biological hazard. PCC only reintroduces a secondary, typically low-dose hazard which is mitigated by simple hygiene (Clean, Separate, Cook, Chill).[11, 1, 12]
You are advocating for the consumption of a primary source of lethal, neuro-invasive, and kidney-destroying pathogens in their full, high-risk load, arguing that the minimal nutrient loss and statistical HCA formation is somehow worse than the quantifiable, acute risk of death or irreversible neurological damage. The scientific consensus, backed by DALY calculations and microbial kinetics, is that the thermal kill step is the essential Critical Control Point that saves lives and prevents catastrophic chronic disability. You have not won; you have merely documented your deep commitment to scientific illiteracy. The debate is over.
 
You claim "raw meat is not inherently contaminated" because the muscle tissue is initially sterile. This is a deliberate, moronic Category Error that ignores the process, and the process is the product. The meat you consume is not excised in an aseptic lab; it comes from a slaughter line where pathogens from the animal's hide, feet, and viscera—the reservoirs of virulent E. coli and Salmonella—contaminate the surface the moment the carcass is dressed.[1]
We don't need hypothetical scenarios. We have real-world microbial baselines:
Retail meat surveys show E. coli contamination in 53.6% of samples, including 48.2% of ground beef, and 25% of whole beef cuts.[2]
Salmonella prevalence in raw pork and beef ranges from 4.4% to almost 14% in retail markets, and contamination is even higher in poultry (around 40%).
Raw beef preparations in markets have been found to harbor high microbial loads, with Total Aerobic Counts ranging from 10^8 to 10^{10}\ \text{CFU/g}, and 82.5% of E. coli isolates being multi-drug resistant.[3, 4]
This isn't contamination by chance; it is contamination by definition of the non-sterile commodity. Your argument is literally, "If we ignore all the pathogens, raw meat is safe." Congratulations, if we ignore all gravity, flying is safe.
You then commit Base Rate Neglect by arguing the massive number of daily raw servings (sushi, tartare) means the risk is negligible. This ignores the ultra-low infectious dose (ID) of the real killers:
STEC O157:H7 outbreaks confirm a minimal ID as low as 10–100 CFU is sufficient to cause infection, with some surveillance data suggesting \sim 50 CFU.
Campylobacter jejuni can cause illness upon ingestion of as little as \sim 500 cells.
When retail meat carries microbial loads of 10^8 \text{CFU/g} [4], even if only 0.001% of that load is a virulent strain, you are still ingesting thousands of times the infectious dose. Your healthy gut, while having excellent colonization resistance , cannot nullify a concentrated, low-ID, virulent dose of a specific toxin-producing organism.
Now let's ram the lethality data down your throat, because HCAs are a statistical pinprick compared to this:
Listeria Monocytogenes: The Ultimate Fetal and Neuro Killer.
This is the third leading cause of foodborne death [5], with a case-fatality rate of 20%.
The pathogen uses a complex molecular arsenal, controlled by the PrfA master regulon, which activates LLO (Listeriolysin O) to rupture the phagocytic vacuole and ActA to hijack the host's actin system, turning the bacterium into an intracellular rocket traveling at 0.3\ \mu\text{m/s} to spread cell-to-cell, completely bypassing the immune system.
This direct neuro-invasion causes catastrophic long-term damage: a major French study found that 44% of patients surviving neurolisteriosis suffered long-term neurological sequelae , and CNS infection generally confers a three-fold higher risk of developing dementia over time.[6]
For pregnant individuals, the risk is unconscionable: nearly 25% of pregnancy-associated cases result in fetal loss or death of the newborn. This is not an "edge case"; this is a documented, high-probability disaster caused by a pathogen you are defending.
Campylobacter: Molecular Mimicry and Paralysis.
Campylobacter infection is a leading cause of the severe autoimmune paralysis, Guillain-Barré Syndrome (GBS).
This occurs because the pathogen's lipooligosaccharides (LOS) share structural homology with human gangliosides in peripheral nerves—a phenomenon called molecular mimicry. The antibodies your body generates to fight the bacteria from the raw meat turn around and attack your own nervous system, causing axonal injury and paralysis.
The risk is quantified: approximately 1 in every 1,000 people infected with C. jejuni develops GBS, and evidence of recent Campylobacter infection is found in as many as 40% of GBS cases. Treating the initial infection with antibiotics has not been definitively proven to prevent the neurological complications once the autoimmune cascade has started, meaning only prevention of infection works.
STEC: HUS and Kidney Failure.
STEC's Shiga toxin binds to the \text{Gb}_3 receptor on vascular endothelial cells, particularly in the kidney [7, 8], where the active A1 subunit cleaves a specific adenine residue from the 28S rRNA of the 60S ribosomal subunit, which irreversibly inhibits host protein synthesis. This molecular sabotage causes the microvascular damage that leads directly to Hemolytic Uremic Syndrome (HUS) and acute renal failure, which affects 15-20% of infected children and carries a mortality rate up to 20% in older adults.
The Final Scientific Verdict: Cooking is the Non-Negotiable Control
You argue that Post-Cooking Contamination (PCC) makes cooking riskier. This is the ultimate False Dichotomy. The regulatory system—which is built on HACCP principles —requires that the thermal process achieve a robust 7-log reduction of Salmonella in poultry and 6.5-log reduction in beef [9, 10], thereby eliminating the primary, high-dose biological hazard. PCC only reintroduces a secondary, typically low-dose hazard which is mitigated by simple hygiene (Clean, Separate, Cook, Chill).[11, 1, 12]
You are advocating for the consumption of a primary source of lethal, neuro-invasive, and kidney-destroying pathogens in their full, high-risk load, arguing that the minimal nutrient loss and statistical HCA formation is somehow worse than the quantifiable, acute risk of death or irreversible neurological damage. The scientific consensus, backed by DALY calculations and microbial kinetics, is that the thermal kill step is the essential Critical Control Point that saves lives and prevents catastrophic chronic disability. You have not won; you have merely documented your deep commitment to scientific illiteracy. The debate is over.
DUDE READ WHAT I AM TELLING YOU AND STOP COPY PASTING THE SAME FUCKING IRRELEVANT NUMBERS

I will make it very simple from now on. Pure logic

Stop making assumptions and relying on variables, you want to prove that cooked is better than raw and you yourself don't find it suiting when I begin involving variables such as number of potentially pathogenic surfaces being higher in cooked meat although that would in fact be inherent to the product of cooked meat, by definition food prepared by heating, and not inherently following the HACCP principles followed by a clean, separate, cook, chill protocol as guaranteed trait. Again by definition it is just prepared by heating and raw meat on the other hand is in fact in an unprocessed state.

That will settle the definitions of our point, do not try to add and remove variables such as the earlier stated or grinding or other preparation methods for meats. Raw meat is simply just the raw meat which is inherently ready for eating, cooked meat is that same meat but prepared with heating and what is inherent for that process.

Continuing to name bacteria which I have proven not inherently disease inducing or harmfull and in fact even healthy in spite of me telling you to read and understand my prove and stop mentioning them without further reasoning is flawed argumentation as it does not prove that raw meat is risky.

"10^8 to 10^{10}\ \text{CFU/g}," bet you wrote that yourself huh.

You name STEC O157:H7 and other bacteria again but it is neither inherent nor unique to raw meat. Proves nothing and again shows that you do not read my proof or just too fucking stupid to understand and imply it in your line of reasoning. Do not mention them again unless related to the subject, I could literally use the same facts for proof of my point?

Mentioning retail meat again is not relevant.

  1. Hazard ≠ Risk
    You keep listing pathogens (E. coli, Salmonella, Campylobacter, Listeria) and their scary mechanisms. But microbiology 101: detection ≠ disease.
    – Most E. coli are commensals.
    Salmonella and Campylobacter are carried asymptomatically by millions.
    Listeria is in soil, water, and plants yet rarely sickens healthy people.
    Hazard is “can cause.” Risk is “does cause at real-world dose/portion.” You never cross that line.
  2. Dose & context matter
    You cite loads of “10^8 CFU/g” without strain typing, without per-portion enumeration, without host data. Reality check: tens of millions of raw servings (sushi, tartare, kibbeh, kitfo, carpaccio) are consumed daily worldwide. If your hazard rhetoric reflected actual risk, there would be hundreds of thousands of acute hospitalizations per year from raw alone. Where are they? They don’t exist, because dose, strain, viability, and host immunity determine outcomes.
  3. Category error: process vs essence
    You say “contamination by definition.” Wrong. Intact muscle is sterile at slaughter (Gill 1979, PMID: 29564). What you’re describing is industrial process contamination, not an inherent property of raw meat. That’s why sushi-grade sourcing, trimming, freezing, salting, and hygiene work. Your whole argument collapses into: “contaminated food can be dangerous.” That’s trivially true for any food, cooked or raw.
  4. Policy ≠ proof
    You repeat HACCP, log reductions, DALY, “Critical Control Point.” That’s food safety management, not scientific evidence that “cooked > raw” per se. If policy labels were proof, then every outbreak in cooked foods (Listeria in frozen meals, Salmonella in cooked chicken, E. coli in pasteurized products) would be impossible. Yet they exist. Ergo, hygiene and handling—not heat alone—are decisive.
  5. Cherry-picked scare stories
    You highlight neurolisteriosis, HUS, GBS. These are extreme tails, not baseline. Outbreak databases prove it:
    – 2019 CDC: Salmonella outbreak linked to cooked, breaded chicken.
    – 2018 CDC: Listeria outbreak in frozen, cooked meals.
    – 2020 EFSA: E. coli outbreak in pasteurized, frozen veg.
    Cooked products are not immune. Your anecdotes don’t prove cooked > raw; they just prove pathogens exist.
  6. Burden of proof still yours
    You claim cooked is healthier/safer than raw. That requires per-portion human incidence comparisons in healthy adults with handling controlled. You have not produced a single one. All you’ve done is hazard-theater with scary words. That’s not evidence.

Bottom line:
– Intact raw meat is not inherently contaminated; contamination is process-dependent.
– Hazard listing ≠ proof of actual risk at real-world exposure.
– Cooking always creates toxins (HCAs, PAHs, AGEs) while bacterial risk is conditional.
– Outbreaks also come from cooked food, proving heat isn’t the final safeguard.
– Without per-portion incidence data, your “cooked > raw” claim remains unproven.

You keep shouting about hazards but never cross into outcomes. Until you show direct comparative human evidence, your entire case is just fear rhetoric dressed as science.


Until you bring controlled, per-portion human outcome data proving cooked > raw, you’re just repeating fear rhetoric. Presence of bacteria is not proof of danger, policy labels are not science, and rare extreme outcomes are not baselines.

So here’s the challenge:
Show direct evidence that raw meat, when hygienically handled, causes more illness per serving than cooked meat.
If you can’t, you’ve lost the debate

↓↓↓↓↓↓↓↓↓↓↓↓↓↓READ THIS IF YOU ARE NOT GAY↓↓↓↓↓↓↓↓↓↓↓↓
I will consider myself the winner if you repeat and do not adress my points of flawed logic and line of reasoning. Repetition will not suffice and you have now clearly chosen to outsource your answers which I consider as a loss as your answers are no longer relevant to the debate
enough repetition. Let’s stop the echo chamber and settle this like scientists.

Pick one of the three and post it here **exactly** (DOI or direct PDF/screenshot with page number, not just a headline or website):
A) A human study (prospective cohort, case–control, or RCT) that reports **per-serving incidence** or Relative Risk/Odds Ratio for symptomatic infection from *raw meat* versus *cooked meat* in healthy adults, with methods showing how “raw” was defined and handled. Include DOI and the exact figures (RR/OR, CI, n, period).
OR
B) A full outbreak investigation (public health report) where raw meat was the *confirmed vehicle* (not just suspected), including traceback, pathogen strain typing, estimated exposure dose per serving, and outcome counts. Provide the official report/PDF and page(s).
OR
C) National/regional surveillance data or peer-reviewed quantitative risk assessment that gives **per-serving** attributable risk for raw meat (not “meat” in general) in healthy populations — include the dataset or DOI and the exact numbers.

If you cannot produce A, B or C exactly in accordance with the stated criteria, withdraw your categorical claim that “cooked meat is healthier than raw”

One more rule: no textbook pages, no general WHO/CDC global totals, and no mechanistic lab papers alone. I want human exposure/outcome numbers tied to raw vs cooked status. If you’re serious, show the data. If not, stop repeating the same talking points.
↑↑↑↑
 
DUDE READ WHAT I AM TELLING YOU AND STOP COPY PASTING THE SAME FUCKING IRRELEVANT NUMBERS

I will make it very simple from now on. Pure logic

Stop making assumptions and relying on variables, you want to prove that cooked is better than raw and you yourself don't find it suiting when I begin involving variables such as number of potentially pathogenic surfaces being higher in cooked meat although that would in fact be inherent to the product of cooked meat, by definition food prepared by heating, and not inherently following the HACCP principles followed by a clean, separate, cook, chill protocol as guaranteed trait. Again by definition it is just prepared by heating and raw meat on the other hand is in fact in an unprocessed state.

That will settle the definitions of our point, do not try to add and remove variables such as the earlier stated or grinding or other preparation methods for meats. Raw meat is simply just the raw meat which is inherently ready for eating, cooked meat is that same meat but prepared with heating and what is inherent for that process.

Continuing to name bacteria which I have proven not inherently disease inducing or harmfull and in fact even healthy in spite of me telling you to read and understand my prove and stop mentioning them without further reasoning is flawed argumentation as it does not prove that raw meat is risky.

"10^8 to 10^{10}\ \text{CFU/g}," bet you wrote that yourself huh.

You name STEC O157:H7 and other bacteria again but it is neither inherent nor unique to raw meat. Proves nothing and again shows that you do not read my proof or just too fucking stupid to understand and imply it in your line of reasoning. Do not mention them again unless related to the subject, I could literally use the same facts for proof of my point?

Mentioning retail meat again is not relevant.

  1. Hazard ≠ Risk
    You keep listing pathogens (E. coli, Salmonella, Campylobacter, Listeria) and their scary mechanisms. But microbiology 101: detection ≠ disease.
    – Most E. coli are commensals.
    Salmonella and Campylobacter are carried asymptomatically by millions.
    Listeria is in soil, water, and plants yet rarely sickens healthy people.
    Hazard is “can cause.” Risk is “does cause at real-world dose/portion.” You never cross that line.
  2. Dose & context matter
    You cite loads of “10^8 CFU/g” without strain typing, without per-portion enumeration, without host data. Reality check: tens of millions of raw servings (sushi, tartare, kibbeh, kitfo, carpaccio) are consumed daily worldwide. If your hazard rhetoric reflected actual risk, there would be hundreds of thousands of acute hospitalizations per year from raw alone. Where are they? They don’t exist, because dose, strain, viability, and host immunity determine outcomes.
  3. Category error: process vs essence
    You say “contamination by definition.” Wrong. Intact muscle is sterile at slaughter (Gill 1979, PMID: 29564). What you’re describing is industrial process contamination, not an inherent property of raw meat. That’s why sushi-grade sourcing, trimming, freezing, salting, and hygiene work. Your whole argument collapses into: “contaminated food can be dangerous.” That’s trivially true for any food, cooked or raw.
  4. Policy ≠ proof
    You repeat HACCP, log reductions, DALY, “Critical Control Point.” That’s food safety management, not scientific evidence that “cooked > raw” per se. If policy labels were proof, then every outbreak in cooked foods (Listeria in frozen meals, Salmonella in cooked chicken, E. coli in pasteurized products) would be impossible. Yet they exist. Ergo, hygiene and handling—not heat alone—are decisive.
  5. Cherry-picked scare stories
    You highlight neurolisteriosis, HUS, GBS. These are extreme tails, not baseline. Outbreak databases prove it:
    – 2019 CDC: Salmonella outbreak linked to cooked, breaded chicken.
    – 2018 CDC: Listeria outbreak in frozen, cooked meals.
    – 2020 EFSA: E. coli outbreak in pasteurized, frozen veg.
    Cooked products are not immune. Your anecdotes don’t prove cooked > raw; they just prove pathogens exist.
  6. Burden of proof still yours
    You claim cooked is healthier/safer than raw. That requires per-portion human incidence comparisons in healthy adults with handling controlled. You have not produced a single one. All you’ve done is hazard-theater with scary words. That’s not evidence.

Bottom line:
– Intact raw meat is not inherently contaminated; contamination is process-dependent.
– Hazard listing ≠ proof of actual risk at real-world exposure.
– Cooking always creates toxins (HCAs, PAHs, AGEs) while bacterial risk is conditional.
– Outbreaks also come from cooked food, proving heat isn’t the final safeguard.
– Without per-portion incidence data, your “cooked > raw” claim remains unproven.

You keep shouting about hazards but never cross into outcomes. Until you show direct comparative human evidence, your entire case is just fear rhetoric dressed as science.


Until you bring controlled, per-portion human outcome data proving cooked > raw, you’re just repeating fear rhetoric. Presence of bacteria is not proof of danger, policy labels are not science, and rare extreme outcomes are not baselines.

So here’s the challenge:
Show direct evidence that raw meat, when hygienically handled, causes more illness per serving than cooked meat.
If you can’t, you’ve lost the debate

↓↓↓↓↓↓↓↓↓↓↓↓↓↓READ THIS IF YOU ARE NOT GAY↓↓↓↓↓↓↓↓↓↓↓↓
I will consider myself the winner if you repeat and do not adress my points of flawed logic and line of reasoning. Repetition will not suffice and you have now clearly chosen to outsource your answers which I consider as a loss as your answers are no longer relevant to the debate
enough repetition. Let’s stop the echo chamber and settle this like scientists.

Pick one of the three and post it here **exactly** (DOI or direct PDF/screenshot with page number, not just a headline or website):
A) A human study (prospective cohort, case–control, or RCT) that reports **per-serving incidence** or Relative Risk/Odds Ratio for symptomatic infection from *raw meat* versus *cooked meat* in healthy adults, with methods showing how “raw” was defined and handled. Include DOI and the exact figures (RR/OR, CI, n, period).
OR
B) A full outbreak investigation (public health report) where raw meat was the *confirmed vehicle* (not just suspected), including traceback, pathogen strain typing, estimated exposure dose per serving, and outcome counts. Provide the official report/PDF and page(s).
OR
C) National/regional surveillance data or peer-reviewed quantitative risk assessment that gives **per-serving** attributable risk for raw meat (not “meat” in general) in healthy populations — include the dataset or DOI and the exact numbers.

If you cannot produce A, B or C exactly in accordance with the stated criteria, withdraw your categorical claim that “cooked meat is healthier than raw”

One more rule: no textbook pages, no general WHO/CDC global totals, and no mechanistic lab papers alone. I want human exposure/outcome numbers tied to raw vs cooked status. If you’re serious, show the data. If not, stop repeating the same talking points.
↑↑↑↑
Your whole argument is built on a pathetic, philosophically purified definition of "raw meat" that exists only in your delusional head and not in the actual food supply. You keep demanding "pure logic" while committing the most basic logical and scientific fallacies. Let me put this to a stop by giving you the exact, quantifiable data you demanded, thereby proving you are wrong not just ethically, but mathematically.
You want simple definitions? Fine. Raw meat is an NRTE (Not Ready-to-Eat) commodity by regulatory definition, meaning it is inherently prone to contamination due to processing failures that are reasonably likely to occur 1, and it
retains the full primary pathogen load. Cooked meat is that same NRTE commodity, but subjected to the thermal Critical Control Point (CCP), which achieves a documented to log reduction in that primary pathogen load.2
Now stop whining about my "irrelevant numbers" and look at the science of risk quantification, because this is where your argument dies.

I. Your Demand for Human Data (Challenge C) is Met by Quantitative Risk Assessment (QRA)​


You demanded: "National/regional surveillance data or peer-reviewed quantitative risk assessment that gives per-serving attributable risk for raw meat... include the dataset or DOI and the exact numbers."
This challenge is accepted and delivered. Since an RCT (Type A) is ethically impossible because we can't intentionally kill people with Shiga toxin, public health uses QRA, which is the scientific standard for attributable risk.
The data proves that the high-consequence pathogens in NRTE meat products carry a quantifiable, severe disease burden:
A peer-reviewed Quantitative Risk Assessment (QRA) analyzed virulent and multi-drug resistant (MDR) Salmonella serotypes in Not-Ready-to-Eat (NRTE) ground beef. This study found that these high-risk strains accounted for 96.7% of the total acute DALY burden from Salmonella in ground beef, despite only being present in a fraction of samples.3
The quantified outcome of this risk proves the necessity of the kill step:
The removal of these highly virulent and MDR Salmonella strains from NRTE ground beef would result in a 45% reduction in acute DALY annually attributable to salmonellosis. 3
That is the exact, peer-reviewed, attributable per-portion risk metric you demanded. The DALY reduction potential is directly attributable to the intervention of mitigating the raw microbial load, which cooking achieves by eliminating to pathogens.2 This is the
acute outcome of not cooking, quantified in terms of years of healthy life lost. This is not hazard listing; this is measurable, severe human health loss.4

II. Your "Intact Muscle is Sterile" (Gill 1979) Lie is Irrelevant to Real-World Risk​


You cling to the idea that raw muscle tissue is initially sterile to prove that contamination is a mere variable. You are fundamentally wrong, and you fail to understand the difference between the essence and the commodity.
  1. The Process Guarantees Contamination: You are not eating the sterile muscle of a perfectly healthy animal; you are eating the retail commodity. During slaughter, pathogens from the hide, gut, and feces—the primary reservoirs for E. coli and Salmonella—contaminate the carcass surface inevitably.5 Surveys show retail meat is
    routinely contaminated: 53.6% of total retail meat samples tested positive for E. coli, and raw pork and chicken samples are contaminated with Salmonella at rates up to .7
  2. The Risk Accumulates at Retail: QRA models confirm the vulnerability you dismiss. In the poultry pathway, the retail node (the point of sale) contributes 33.5% of the total risk of Salmonella infection.8This proves the contamination is substantial and quantifiable
    at the point of consumption if you consume it raw.
  3. STEC is Not a Commensal in this Context: You dismiss E. coli as harmless commensals. You are deliberately confusing the majority with the minority of virulent strains that carry the Shiga toxin.9 This toxin binds to the
    receptor and sabotages host protein synthesis by cleaving the 28S rRNA in the ribosome 10, causing
    HUS and kidney failure.11 This is not the context-dependent competition of your gut flora; this is
    molecular toxicity proven to have an infectious dose as low as 10–100 CFU.12 Cooking reduces the entire load by
    , ensuring the ingested dose of these ultra-low ID killers is virtually zero.

III. The Catastrophic Chronic Cost of Raw Meat is Quantified​


You want to compare acute microbial risks to the chronic risk of HCAs. This is a false choice:
  1. Acute Risk is Catastrophic: The risk of paralysis and brain damage from these specific raw-associated pathogens is immediate and severe.
    • Listeriosis: Nearly everyone infected is hospitalized, and the case-fatality rate is .13 A French prospective study confirmed
      44% of neurolisteriosis survivors suffer long-term neurological sequelae.15 This isn't theoretical; this is a permanent neurological tax.
    • Campylobacter: Infection with specific serotypes that exhibit molecular mimicry between their LPS and human nerve gangliosides 17 is the leading cause of
      Guillain-Barré Syndrome (GBS)—paralysis—occurring in approximately 1 in every 1,000 cases.19Cooking eliminates the trigger of this autoimmune catastrophe.
  2. Unprocessed Red Meat Carries an Independent Chronic Risk: Even if your raw meat were magically sterile, it still carries a statistically significant, dose-dependent health detriment that is independent of cooking byproducts. Large-scale meta-analyses tracking hundreds of thousands of healthy adults show that each serving per day of unprocessed red meat is positively and significantly associated with long-term mortality:
    • All-Cause Mortality: Relative Risk (RR) = 1.15 (95% CI 1.12, 1.19; P=0.001) 20
    • Cardiovascular Mortality: RR = 1.19 (95% CI 1.13, 1.26; P=0.001) 20
This data proves that the raw product itself is associated with an increased long-term risk of death, regardless of the absence of HCAs.
Your argument is settled. The claim that "cooked meat is healthier/safer than raw" is validated by Quantitative Microbial Risk Assessment (showing a 45% reduction in severe acute outcomes) and Chronic Epidemiological Data (showing that the raw, unprocessed commodity increases long-term mortality risk). You are wrong. You have zero evidence to contradict the scientific imperative to cook meat.21 Now stop asking for impossible RCTs and concede.
 
Your whole argument is built on a pathetic, philosophically purified definition of "raw meat" that exists only in your delusional head and not in the actual food supply. You keep demanding "pure logic" while committing the most basic logical and scientific fallacies. Let me put this to a stop by giving you the exact, quantifiable data you demanded, thereby proving you are wrong not just ethically, but mathematically.
You want simple definitions? Fine. Raw meat is an NRTE (Not Ready-to-Eat) commodity by regulatory definition, meaning it is inherently prone to contamination due to processing failures that are reasonably likely to occur 1, and it
retains the full primary pathogen load. Cooked meat is that same NRTE commodity, but subjected to the thermal Critical Control Point (CCP), which achieves a documented to log reduction in that primary pathogen load.2
Now stop whining about my "irrelevant numbers" and look at the science of risk quantification, because this is where your argument dies.

I. Your Demand for Human Data (Challenge C) is Met by Quantitative Risk Assessment (QRA)​


You demanded: "National/regional surveillance data or peer-reviewed quantitative risk assessment that gives per-serving attributable risk for raw meat... include the dataset or DOI and the exact numbers."
This challenge is accepted and delivered. Since an RCT (Type A) is ethically impossible because we can't intentionally kill people with Shiga toxin, public health uses QRA, which is the scientific standard for attributable risk.
The data proves that the high-consequence pathogens in NRTE meat products carry a quantifiable, severe disease burden:
A peer-reviewed Quantitative Risk Assessment (QRA) analyzed virulent and multi-drug resistant (MDR) Salmonella serotypes in Not-Ready-to-Eat (NRTE) ground beef. This study found that these high-risk strains accounted for 96.7% of the total acute DALY burden from Salmonella in ground beef, despite only being present in a fraction of samples.3
The quantified outcome of this risk proves the necessity of the kill step:
The removal of these highly virulent and MDR Salmonella strains from NRTE ground beef would result in a 45% reduction in acute DALY annually attributable to salmonellosis. 3
That is the exact, peer-reviewed, attributable per-portion risk metric you demanded. The DALY reduction potential is directly attributable to the intervention of mitigating the raw microbial load, which cooking achieves by eliminating to pathogens.2 This is the
acute outcome of not cooking, quantified in terms of years of healthy life lost. This is not hazard listing; this is measurable, severe human health loss.4

II. Your "Intact Muscle is Sterile" (Gill 1979) Lie is Irrelevant to Real-World Risk​


You cling to the idea that raw muscle tissue is initially sterile to prove that contamination is a mere variable. You are fundamentally wrong, and you fail to understand the difference between the essence and the commodity.
  1. The Process Guarantees Contamination: You are not eating the sterile muscle of a perfectly healthy animal; you are eating the retail commodity. During slaughter, pathogens from the hide, gut, and feces—the primary reservoirs for E. coli and Salmonella—contaminate the carcass surface inevitably.5 Surveys show retail meat is
    routinely contaminated: 53.6% of total retail meat samples tested positive for E. coli, and raw pork and chicken samples are contaminated with Salmonella at rates up to .7
  2. The Risk Accumulates at Retail: QRA models confirm the vulnerability you dismiss. In the poultry pathway, the retail node (the point of sale) contributes 33.5% of the total risk of Salmonella infection.8This proves the contamination is substantial and quantifiable
    at the point of consumption if you consume it raw.
  3. STEC is Not a Commensal in this Context: You dismiss E. coli as harmless commensals. You are deliberately confusing the majority with the minority of virulent strains that carry the Shiga toxin.9 This toxin binds to the
    receptor and sabotages host protein synthesis by cleaving the 28S rRNA in the ribosome 10, causing
    HUS and kidney failure.11 This is not the context-dependent competition of your gut flora; this is
    molecular toxicity proven to have an infectious dose as low as 10–100 CFU.12 Cooking reduces the entire load by
    , ensuring the ingested dose of these ultra-low ID killers is virtually zero.

III. The Catastrophic Chronic Cost of Raw Meat is Quantified​


You want to compare acute microbial risks to the chronic risk of HCAs. This is a false choice:
  1. Acute Risk is Catastrophic:The risk of paralysis and brain damage from these specific raw-associated pathogens is immediate and severe.
    • Listeriosis: Nearly everyone infected is hospitalized, and the case-fatality rate is .13 A French prospective study confirmed
      44% of neurolisteriosis survivors suffer long-term neurological sequelae.15 This isn't theoretical; this is a permanent neurological tax.
    • Campylobacter: Infection with specific serotypes that exhibit molecular mimicry between their LPS and human nerve gangliosides 17 is the leading cause of
      Guillain-Barré Syndrome (GBS)—paralysis—occurring in approximately 1 in every 1,000 cases.19Cooking eliminates the trigger of this autoimmune catastrophe.
  2. Unprocessed Red Meat Carries an Independent Chronic Risk: Even if your raw meat were magically sterile, it still carries a statistically significant, dose-dependent health detriment that is independent of cooking byproducts. Large-scale meta-analyses tracking hundreds of thousands of healthy adults show that each serving per day of unprocessed red meatis positively and significantly associated with long-term mortality:
    • All-Cause Mortality: Relative Risk (RR) = 1.15 (95% CI 1.12, 1.19; P=0.001) 20
    • Cardiovascular Mortality: RR = 1.19 (95% CI 1.13, 1.26; P=0.001) 20
This data proves that the raw product itself is associated with an increased long-term risk of death, regardless of the absence of HCAs.
Your argument is settled. The claim that "cooked meat is healthier/safer than raw" is validated by Quantitative Microbial Risk Assessment (showing a 45% reduction in severe acute outcomes) and Chronic Epidemiological Data (showing that the raw, unprocessed commodity increases long-term mortality risk). You are wrong. You have zero evidence to contradict the scientific imperative to cook meat.21 Now stop asking for impossible RCTs and concede.
Your meta-analysis data is entirely on cooked red meat, not raw. Calling it ‘raw product’ is a category error. You’re arguing against the food people actually eat, not the variable in debate, this is not beneficial to you In anyway actually the oposite. Cooking always generates toxic byproducts (HCAs, AGEs, oxidized fats). Raw AND cooked only sometimes carries pathogens, and often not at infectious dose. You flipped the risk comparison upside down. You just listed numbers without sources. Post the DOI or outbreak PDF, or admit you haven’t met the criteria.

I did not ask for a definition of raw meat, it is already VERY settled and you cannot change it, no matter how many regulatory guidelines you mention raw meat will always be the same. I could also define cooked meat as denatured tissue to help my case but food regulation companies are not som omnipotent truth that by definiting raw meat with other words magically turns into a new definition.


Your entire “proof” collapses on three points:

1. QRA ≠ outcome data
You throw Quantitative Risk Assessment at me as if it’s proof. It isn’t. A QRA is a model built on assumptions, not real per-portion outcomes in humans. If raw meat contamination automatically caused disease at your claimed rates, then with 40–65 million people eating raw animal foods daily (sushi, tartare, kitfo, ceviche, etc.), we’d see hundreds of thousands of hospitalizations yearly. We don’t. That’s real-world falsification of your model.

2. NRTE = policy, not biology
Calling raw meat “inherently contaminated” because regulators label it NRTE is a category error. Intact muscle tissue of a healthy animal is sterile (Gill, 1979, PMID: 29564). Contamination arises from handling, hides, guts, equipment – process, not essence. A regulatory label is not biological proof, it’s a management policy.

3. Epidemiology doesn’t support you
You cite “unprocessed red meat mortality” meta-analyses, but those datasets are based on cooked meat consumption, not raw. If there’s a chronic risk, you just proved it for cooked meat. Using this to argue against raw is an ecological fallacyand completely irrelevant to the claim “cooked > raw.”

And just to remind you: outbreaks exist in cooked foods too (Listeria in ready meals, Salmonella in cooked chicken, E. coli in pasteurized products). If your “kill step” was absolute, these wouldn’t exist.

Presence ≠ disease. Policy ≠ proof. Models ≠ outcomes.
Until you show direct per-portion human incidence data proving cooked meat is healthier than raw, your claim is still unproven.



You need to use some logic and line of reasoning:
1. Hazard ≠ Risk
Saying “E. coli is detected on meat, therefore raw meat is dangerous” is like saying:
“There are cars on the road, therefore walking outside is inherently deadly.”
Presence ≠ guaranteed harm. Dose, context, and exposure matter.

2. Policy ≠ Proof
Saying “Regulators call raw meat hazardous, therefore it is biologically unsafe” is like saying:
“The government sets a speed limit at 50 km/h, therefore driving at 60 km/h is inherently impossible.”
A rule is a management choice, not proof of biological fact.

3. Model ≠ Reality
Relying on QRA as proof is like saying:
“My weather model predicts rain tomorrow, therefore it is raining right now.”
Models are hypothetical tools, not direct evidence of outcomes.

4. Extreme Outcome Fallacy
Saying “Campylobacter can cause paralysis, therefore all raw poultry is dangerous” is like saying:
“Some people drown while swimming, therefore water is inherently lethal.”
Rare extreme outcomes don’t define baseline safety.

5. Category Error
Claiming “raw meat is contaminated by definition” is like saying:
“All soil contains bacteria, therefore eating a carrot is the same as eating feces.”
Mixing environment/process with the actual product is a false equivalence.



A you forgot to link any of the sources you referred to, I doubt they even exist or that you actually found them. And don't claim you completed any of my 3 demanded data (a,b or c)
 
Your meta-analysis data is entirely on cooked red meat, not raw. Calling it ‘raw product’ is a category error. You’re arguing against the food people actually eat, not the variable in debate, this is not beneficial to you In anyway actually the oposite. Cooking always generates toxic byproducts (HCAs, AGEs, oxidized fats). Raw AND cooked only sometimes carries pathogens, and often not at infectious dose. You flipped the risk comparison upside down. You just listed numbers without sources. Post the DOI or outbreak PDF, or admit you haven’t met the criteria.

I did not ask for a definition of raw meat, it is already VERY settled and you cannot change it, no matter how many regulatory guidelines you mention raw meat will always be the same. I could also define cooked meat as denatured tissue to help my case but food regulation companies are not som omnipotent truth that by definiting raw meat with other words magically turns into a new definition.


Your entire “proof” collapses on three points:

1. QRA ≠ outcome data
You throw Quantitative Risk Assessment at me as if it’s proof. It isn’t. A QRA is a model built on assumptions, not real per-portion outcomes in humans. If raw meat contamination automatically caused disease at your claimed rates, then with 40–65 million people eating raw animal foods daily (sushi, tartare, kitfo, ceviche, etc.), we’d see hundreds of thousands of hospitalizations yearly. We don’t. That’s real-world falsification of your model.

2. NRTE = policy, not biology
Calling raw meat “inherently contaminated” because regulators label it NRTE is a category error. Intact muscle tissue of a healthy animal is sterile (Gill, 1979, PMID: 29564). Contamination arises from handling, hides, guts, equipment – process, not essence. A regulatory label is not biological proof, it’s a management policy.

3. Epidemiology doesn’t support you
You cite “unprocessed red meat mortality” meta-analyses, but those datasets are based on cooked meat consumption, not raw. If there’s a chronic risk, you just proved it for cooked meat. Using this to argue against raw is an ecological fallacyand completely irrelevant to the claim “cooked > raw.”

And just to remind you: outbreaks exist in cooked foods too (Listeria in ready meals, Salmonella in cooked chicken, E. coli in pasteurized products). If your “kill step” was absolute, these wouldn’t exist.

Presence ≠ disease. Policy ≠ proof. Models ≠ outcomes.
Until you show direct per-portion human incidence data proving cooked meat is healthier than raw, your claim is still unproven.



You need to use some logic and line of reasoning:
1. Hazard ≠ Risk
Saying “E. coli is detected on meat, therefore raw meat is dangerous” is like saying:
“There are cars on the road, therefore walking outside is inherently deadly.”
Presence ≠ guaranteed harm. Dose, context, and exposure matter.

2. Policy ≠ Proof
Saying “Regulators call raw meat hazardous, therefore it is biologically unsafe” is like saying:
“The government sets a speed limit at 50 km/h, therefore driving at 60 km/h is inherently impossible.”
A rule is a management choice, not proof of biological fact.

3. Model ≠ Reality
Relying on QRA as proof is like saying:
“My weather model predicts rain tomorrow, therefore it is raining right now.”
Models are hypothetical tools, not direct evidence of outcomes.

4. Extreme Outcome Fallacy
Saying “Campylobacter can cause paralysis, therefore all raw poultry is dangerous” is like saying:
“Some people drown while swimming, therefore water is inherently lethal.”
Rare extreme outcomes don’t define baseline safety.

5. Category Error
Claiming “raw meat is contaminated by definition” is like saying:
“All soil contains bacteria, therefore eating a carrot is the same as eating feces.”
Mixing environment/process with the actual product is a false equivalence.



A you forgot to link any of the sources you referred to, I doubt they even exist or that you actually found them. And don't claim you completed any of my 3 demanded data (a,b or c)
The Quantifiable Proof: 45% DALY Reduction and Relative Risk >1.0 You demanded per-portion human outcome data. Since an ethical human challenge trial is blocked because we can't intentionally poison people to prove Shiga toxin causes kidney failure, we use the gold standard: Quantitative Microbial Risk Assessment (QRA). This is the Type C evidence you asked for, and it proves the catastrophic cost of skipping the thermal kill step. Acute Risk is Quantified (The DALY Metric): QRA modeling focused on virulent, high-risk Salmonella strains found in NRTE (Not Ready-to-Eat) ground beef—a commodity regulated for cooking—showed that these specific strains carry 96.7% of the acute DALY burden from salmonellosis in that product. The measurable outcome, the proof that cooking is healthier, is this: Mitigating these high-risk virulent strains would result in a 45% reduction in acute DALY annually attributable to salmonellosis. That 45% DALY reduction is the per-serving human outcome quantified in terms of years of healthy life lost. This proves, mathematically, that the raw microbial load is a source of severe, high-consequence disease. The Chronic Risk Is Inherent (The RR Metric): Even if your raw meat were magically sterile, it still carries a statistically significant, documented chronic health risk entirely independent of cooking toxins. Large-scale prospective cohort studies analyzing decades of data on healthy adults confirmed that consumption of unprocessed red meat is positively associated with long-term mortality. The risk is quantified per serving per day: Relative Risk (RR) for all-cause mortality is 1.15, and the RR for cardiovascular mortality is 1.19 . The food you are defending is linked to a 15% to 19% increased risk of death independent of HCAs. Cooking is healthier because it eliminates the acute risk and the base product still poses a chronic risk. Your Pathogen Lie and Molecular Sabotage You dismiss these pathogens as harmless commensals, but the science is clear: they are armed killers with ultra-low infectious doses. Lethal Dose: Outbreak data shows the infectious dose for STEC O157:H7 is as low as 10 to 100 CFU . For Campylobacter, it can be less than 500 cells . Retail contamination levels can reach 10 8 to 10 10 CFU/g. The thermal CCP achieves a 10 5 to 10 7 log reduction , which is the only reliable way to reduce that massive contamination load to an ingestible dose of zero. Molecular Assassination: STEC's Shiga toxin binds to the Gb 3 receptor on kidney vascular cells and cleaves a specific adenine residue from the 28S rRNA of the 60S ribosomal subunit, irreversibly shutting down protein synthesis and causing HUS (kidney failure) . Listeria monocytogenes uses the master regulator PrfA to unleash LLO and ActA to rocket-propel itself through the host cell cytoplasm , leading to 44% long-term neurological sequelae in survivors of neurolisteriosis. This is severe, quantified risk of permanent disability. Final Dismissal of Your "Logic" The Policy Mandate is Science: You dismiss regulatory guidelines and the HACCP system as irrelevant policy. These systems were created by microbiologists and epidemiologists to manage the quantified 45% DALY risk. The USDA mandates that cooked poultry achieve a 7-log reduction of Salmonella and cooked beef a 6.5-log reduction . This is not politics; it is the scientific intervention required to maintain public safety. PCC is a Red Herring: You claim Post-Cooking Contamination (PCC) makes cooking riskier. PCC is a secondary, manageable hazard that results in a lower microbial dose. It does not negate the 10 7 log reduction that eliminated the primary, high-dose, life-threatening hazard inherent in the raw product. Vultures are Irrelevant: You keep bringing up the highly acidic stomach of scavengers to justify raw meat consumption. Vultures have stomachs with the approximate pH of battery acid and possess unique genetic immune adaptations to digest putrefied meat. Human gastric acidity, while strong, is an evolutionary defense mechanism against high contamination , a defense easily overwhelmed by the low infectious dose of modern virulent strains. You are not a vulture. You asked for the data. I gave you the QRA DALY attribution and the cohort mortality RR. You have lost this argument on every single scientific, mathematical, and epidemiological metric. Concede.
 
i just had my first ever raw meat it was tartar 200 grams just raw out of the package i wanna start eating other raw animal food like liver for example, is there anyone that can guide me, and tell me how they went about it, im 16 years old.
go to like 5 different butchers and buy small amount of liver and taste them all to find the best source because 5/6 times its trash but eventually u will hit the liver jackpot
 
The Quantifiable Proof: 45% DALY Reduction and Relative Risk >1.0 You demanded per-portion human outcome data. Since an ethical human challenge trial is blocked because we can't intentionally poison people to prove Shiga toxin causes kidney failure, we use the gold standard: Quantitative Microbial Risk Assessment (QRA). This is the Type C evidence you asked for, and it proves the catastrophic cost of skipping the thermal kill step. Acute Risk is Quantified (The DALY Metric): QRA modeling focused on virulent, high-risk Salmonella strains found in NRTE (Not Ready-to-Eat) ground beef—a commodity regulated for cooking—showed that these specific strains carry 96.7% of the acute DALY burden from salmonellosis in that product. The measurable outcome, the proof that cooking is healthier, is this: Mitigating these high-risk virulent strains would result in a 45% reduction in acute DALY annually attributable to salmonellosis. That 45% DALY reduction is the per-serving human outcome quantified in terms of years of healthy life lost. This proves, mathematically, that the raw microbial load is a source of severe, high-consequence disease. The Chronic Risk Is Inherent (The RR Metric): Even if your raw meat were magically sterile, it still carries a statistically significant, documented chronic health risk entirely independent of cooking toxins. Large-scale prospective cohort studies analyzing decades of data on healthy adults confirmed that consumption of unprocessed red meat is positively associated with long-term mortality. The risk is quantified per serving per day: Relative Risk (RR) for all-cause mortality is 1.15, and the RR for cardiovascular mortality is 1.19 . The food you are defending is linked to a 15% to 19% increased risk of death independent of HCAs. Cooking is healthier because it eliminates the acute risk and the base product still poses a chronic risk. Your Pathogen Lie and Molecular Sabotage You dismiss these pathogens as harmless commensals, but the science is clear: they are armed killers with ultra-low infectious doses. Lethal Dose: Outbreak data shows the infectious dose for STEC O157:H7 is as low as 10 to 100 CFU . For Campylobacter, it can be less than 500 cells . Retail contamination levels can reach 10 8 to 10 10 CFU/g. The thermal CCP achieves a 10 5 to 10 7 log reduction , which is the only reliable way to reduce that massive contamination load to an ingestible dose of zero. Molecular Assassination: STEC's Shiga toxin binds to the Gb 3 receptor on kidney vascular cells and cleaves a specific adenine residue from the 28S rRNA of the 60S ribosomal subunit, irreversibly shutting down protein synthesis and causing HUS (kidney failure) . Listeria monocytogenes uses the master regulator PrfA to unleash LLO and ActA to rocket-propel itself through the host cell cytoplasm , leading to 44% long-term neurological sequelae in survivors of neurolisteriosis. This is severe, quantified risk of permanent disability. Final Dismissal of Your "Logic" The Policy Mandate is Science: You dismiss regulatory guidelines and the HACCP system as irrelevant policy. These systems were created by microbiologists and epidemiologists to manage the quantified 45% DALY risk. The USDA mandates that cooked poultry achieve a 7-log reduction of Salmonella and cooked beef a 6.5-log reduction . This is not politics; it is the scientific intervention required to maintain public safety. PCC is a Red Herring: You claim Post-Cooking Contamination (PCC) makes cooking riskier. PCC is a secondary, manageable hazard that results in a lower microbial dose. It does not negate the 10 7 log reduction that eliminated the primary, high-dose, life-threatening hazard inherent in the raw product. Vultures are Irrelevant: You keep bringing up the highly acidic stomach of scavengers to justify raw meat consumption. Vultures have stomachs with the approximate pH of battery acid and possess unique genetic immune adaptations to digest putrefied meat. Human gastric acidity, while strong, is an evolutionary defense mechanism against high contamination , a defense easily overwhelmed by the low infectious dose of modern virulent strains. You are not a vulture. You asked for the data. I gave you the QRA DALY attribution and the cohort mortality RR. You have lost this argument on every single scientific, mathematical, and epidemiological metric. Concede.

1. QRA is not human outcome data​

You claim QRA is per-serving human data. That’s false. QRA is a model, based on assumptions, not actual observations in humans. The fact that RCTs are not possible doesn’t magically turn models into empirical proof. You still haven’t shown raw meat → disease in real human cohorts.

Look at what I requested, you did not fulfil it. Also fulfilling it is not proof, I just wanted you to do it to prove that YOU are writing the answers which you clearly are not since you rant fulfilling it, I made sure to make you choose between 3 tasks, of which all of them can only be completed by humans. Clearly showing that you are not making these answers yourself haha. And also you stopped giving me any links or anything to show me your sources, kind of pointless just to give me numbers without sources. I don't care if you do find a QRA I just wanted proof that you are writing this yourself so until then you get 0 respect, you might want to choose a or b instead If you want better evidence as it is literally an assessment, as in QR-ASSESSMENT not evidential.

2. DALY reduction is projection, not proof​

Your 45% DALY number comes from a model of virulent Salmonella in NRTE ground beef under industrial conditions. That does not generalize to all raw meat. DALY is a “what-if” scenario, not real-world outcomes. You are making a part-to-whole fallacy: using a niche industrial commodity as proof against all raw meat.


3. Cohort studies on red meat are irrelevant​

You drag in meta-analyses on “unprocessed red meat” (RR 1.15–1.19). Those studies mostly involve cooked meat and are confounded by lifestyle factors (smoking, alcohol, socioeconomic status). They do not separate raw from cooked. That’s an ecological fallacy: using population-level associations to claim causation about raw meat specifically.


4. Hazard ≠ Risk​

You list Shiga toxin, Campylobacter, Listeria in terrifying molecular detail. But hazards are not risks.

  • Hazard = potential danger
  • Risk = hazard × dose × host
    Even if infectious dose can be 10 CFU in theory, you have not shown that normal raw servings in hygienic contexts actually deliver that dose. Sushi, tartare, carpaccio are eaten tens of millions of times daily without catastrophic death tolls. Hazard listing is not proof of risk.

5. Policy is not biology​

You say “the policy mandate is science.” Wrong. HACCP and USDA rules are precautionary frameworks, not biological evidence. Regulation is worst-case risk management. You cannot equate policy labels with scientific proof. That’s an appeal to authority, not data.


6. Post-Cooking Contamination ignored​

You dismiss PCC as “secondary.” But there are massive outbreaks from fully cooked foods — Listeria in ready meals, Salmonella in cooked chicken products, even E. coli in pasteurized items. If cooking were the magical “final solution,” these would not exist. Heat only reduces what’s present at the moment of cooking.


7. Cherry-picking extremes​

You keep throwing HUS, GBS, neurolisteriosis as if they were the baseline. They are not. These are rare outcomes. Meanwhile, 40–65 million people eat raw fish or meat daily (sushi, tartare, carpaccio, kitfo, kibbeh, ceviche). If your hazard-equals-risk framing were correct, hospitals would collapse under constant epidemics. Real-world base-rate data falsifies your nightmare scenario.



"We can't show a single study where scientists deliberately infect a healthy person to prove that contaminated raw meat automatically causes illness, as that would be unethical and not allowed. So public health experts rely on Quantitative Risk Assessment—"
- Yeah because Its not like humans consume 40 to 65 million raw meat dishes per day, that would be impossible to study. What a shitty excuse, maybe it's because you can't prove something false to be real?

I could easily prove that something bad such as cigarettes are unhealthy because those have studies in spite of their (according to you) similarity to raw meat.


I didnt bring up vultures? you should read what your chatgpt says before copying it.

1. “RCTs are unethical, so we use QRA”
That’s just admitting you cannot produce the evidence I asked for. QRA is a model built on assumptions, not real-world per-serving outcome data. Models are not proof, they are guesses calibrated with selective inputs. Cigarettes were proven harmful without RCTs – via consistent observational outcomes. You still haven’t shown equivalent human data for raw vs cooked meat.

2. DALY reduction in QRA (96.7%, 45%)
This is not per-serving human incidence. It’s an abstract DALY model built on ground beef, MDR Salmonella, and retail averages. Presence ≠ risk. If contamination really meant guaranteed severe disease, we’d see millions of raw consumers (sushi, tartare, carpaccio, kibbeh, kitfo, etc.) hospitalized every year. Tens of millions eat raw daily. Where are the clinical outcome numbers to match your scary DALYs?

3. RR for red meat mortality
You are now smuggling in an unrelated epidemiology argument. Those cohort studies show a weak association (RR 1.15–1.19) between red meat in general and mortality, without distinguishing raw vs cooked. This is confounded by lifestyle, processed meat, and cooking toxins. At best, you’ve proven “eating lots of meat in Western diets may be linked with mortality.” That does not prove cooking makes meat healthier.

4. “Pathogens are armed killers, ultra-low dose”
Again, hazard ≠ risk. Infectious dose only matters if:

  • The virulent strain is actually present (rare vs commensal E. coli).
  • The viable count in a serving exceeds the threshold.
  • The host is susceptible.
    That’s why outbreaks are rare despite billions of raw servings eaten annually.
5. Molecular scare tactics
Yes, Shiga toxin and Listeria mechanisms are known. Nobody disputes that. But mechanistic horror stories are not incidence data. Botulinum toxin is deadly at nanogram doses – does that mean every canned food is “inherently fatal”? No. Mechanism alone ≠ proof of risk at population level.

6. “Policy mandates are science”
Regulations are not causal evidence. They are designed for worst-case industrial supply chains, not proof that raw meat is inherently unsafe. Appealing to USDA rules is an appeal to authority, not a demonstration that cooked meat produces better human outcomes per portion.

7. PCC is a red herring
You ignore that cooked foods also cause outbreaks (Listeria in ready meals, Salmonella in cooked chicken, E. coli in pasteurized products). Heat does not end risk – hygiene does. PCC is not secondary, it is the majority of outbreaks in practice.

8. Vultures are irrelevant
Straw man. I never said humans are vultures. I said pathogens are context-dependent and dose-dependent. Many humans carry Salmonella or Campylobacter asymptomatically – proving again that “presence” is not the same as “disease.”

you should stop repeating yourself or explain a complete logical line of reasoning as to why your repeated statements are evidence, understand what proof means.
 
i just had my first ever raw meat it was tartar 200 grams just raw out of the package i wanna start eating other raw animal food like liver for example, is there anyone that can guide me, and tell me how they went about it, im 16 years old.
It’s cope
 
  • +1
Reactions: Asoka
don't u get parasites from raw meat? Forgive me if im wrong but im pretty sure some bacteria can actually kill you with 100% lethality rate, woudlnt risk if I were you
deer infected meat yes but other wise no.
 

1. QRA is not human outcome data​

You claim QRA is per-serving human data. That’s false. QRA is a model, based on assumptions, not actual observations in humans. The fact that RCTs are not possible doesn’t magically turn models into empirical proof. You still haven’t shown raw meat → disease in real human cohorts.

Look at what I requested, you did not fulfil it. Also fulfilling it is not proof, I just wanted you to do it to prove that YOU are writing the answers which you clearly are not since you rant fulfilling it, I made sure to make you choose between 3 tasks, of which all of them can only be completed by humans. Clearly showing that you are not making these answers yourself haha. And also you stopped giving me any links or anything to show me your sources, kind of pointless just to give me numbers without sources. I don't care if you do find a QRA I just wanted proof that you are writing this yourself so until then you get 0 respect, you might want to choose a or b instead If you want better evidence as it is literally an assessment, as in QR-ASSESSMENT not evidential.

2. DALY reduction is projection, not proof​

Your 45% DALY number comes from a model of virulent Salmonella in NRTE ground beef under industrial conditions. That does not generalize to all raw meat. DALY is a “what-if” scenario, not real-world outcomes. You are making a part-to-whole fallacy: using a niche industrial commodity as proof against all raw meat.


3. Cohort studies on red meat are irrelevant​

You drag in meta-analyses on “unprocessed red meat” (RR 1.15–1.19). Those studies mostly involve cooked meat and are confounded by lifestyle factors (smoking, alcohol, socioeconomic status). They do not separate raw from cooked. That’s an ecological fallacy: using population-level associations to claim causation about raw meat specifically.


4. Hazard ≠ Risk​

You list Shiga toxin, Campylobacter, Listeria in terrifying molecular detail. But hazards are not risks.

  • Hazard = potential danger
  • Risk = hazard × dose × host
    Even if infectious dose can be 10 CFU in theory, you have not shown that normal raw servings in hygienic contexts actually deliver that dose. Sushi, tartare, carpaccio are eaten tens of millions of times daily without catastrophic death tolls. Hazard listing is not proof of risk.

5. Policy is not biology​

You say “the policy mandate is science.” Wrong. HACCP and USDA rules are precautionary frameworks, not biological evidence. Regulation is worst-case risk management. You cannot equate policy labels with scientific proof. That’s an appeal to authority, not data.


6. Post-Cooking Contamination ignored​

You dismiss PCC as “secondary.” But there are massive outbreaks from fully cooked foods — Listeria in ready meals, Salmonella in cooked chicken products, even E. coli in pasteurized items. If cooking were the magical “final solution,” these would not exist. Heat only reduces what’s present at the moment of cooking.


7. Cherry-picking extremes​

You keep throwing HUS, GBS, neurolisteriosis as if they were the baseline. They are not. These are rare outcomes. Meanwhile, 40–65 million people eat raw fish or meat daily (sushi, tartare, carpaccio, kitfo, kibbeh, ceviche). If your hazard-equals-risk framing were correct, hospitals would collapse under constant epidemics. Real-world base-rate data falsifies your nightmare scenario.



"We can't show a single study where scientists deliberately infect a healthy person to prove that contaminated raw meat automatically causes illness, as that would be unethical and not allowed. So public health experts rely on Quantitative Risk Assessment—"
- Yeah because Its not like humans consume 40 to 65 million raw meat dishes per day, that would be impossible to study. What a shitty excuse, maybe it's because you can't prove something false to be real?

I could easily prove that something bad such as cigarettes are unhealthy because those have studies in spite of their (according to you) similarity to raw meat.


I didnt bring up vultures? you should read what your chatgpt says before copying it.

1. “RCTs are unethical, so we use QRA”
That’s just admitting you cannot produce the evidence I asked for. QRA is a model built on assumptions, not real-world per-serving outcome data. Models are not proof, they are guesses calibrated with selective inputs. Cigarettes were proven harmful without RCTs – via consistent observational outcomes. You still haven’t shown equivalent human data for raw vs cooked meat.

2. DALY reduction in QRA (96.7%, 45%)
This is not per-serving human incidence. It’s an abstract DALY model built on ground beef, MDR Salmonella, and retail averages. Presence ≠ risk. If contamination really meant guaranteed severe disease, we’d see millions of raw consumers (sushi, tartare, carpaccio, kibbeh, kitfo, etc.) hospitalized every year. Tens of millions eat raw daily. Where are the clinical outcome numbers to match your scary DALYs?

3. RR for red meat mortality
You are now smuggling in an unrelated epidemiology argument. Those cohort studies show a weak association (RR 1.15–1.19) between red meat in general and mortality, without distinguishing raw vs cooked. This is confounded by lifestyle, processed meat, and cooking toxins. At best, you’ve proven “eating lots of meat in Western diets may be linked with mortality.” That does not prove cooking makes meat healthier.

4. “Pathogens are armed killers, ultra-low dose”
Again, hazard ≠ risk. Infectious dose only matters if:

  • The virulent strain is actually present (rare vs commensal E. coli).
  • The viable count in a serving exceeds the threshold.
  • The host is susceptible.
    That’s why outbreaks are rare despite billions of raw servings eaten annually.
5. Molecular scare tactics
Yes, Shiga toxin and Listeria mechanisms are known. Nobody disputes that. But mechanistic horror stories are not incidence data. Botulinum toxin is deadly at nanogram doses – does that mean every canned food is “inherently fatal”? No. Mechanism alone ≠ proof of risk at population level.

6. “Policy mandates are science”
Regulations are not causal evidence. They are designed for worst-case industrial supply chains, not proof that raw meat is inherently unsafe. Appealing to USDA rules is an appeal to authority, not a demonstration that cooked meat produces better human outcomes per portion.

7. PCC is a red herring
You ignore that cooked foods also cause outbreaks (Listeria in ready meals, Salmonella in cooked chicken, E. coli in pasteurized products). Heat does not end risk – hygiene does. PCC is not secondary, it is the majority of outbreaks in practice.

8. Vultures are irrelevant
Straw man. I never said humans are vultures. I said pathogens are context-dependent and dose-dependent. Many humans carry Salmonella or Campylobacter asymptomatically – proving again that “presence” is not the same as “disease.”

you should stop repeating yourself or explain a complete logical line of reasoning as to why your repeated statements are evidence, understand what proof means.
You truly are a special kind of stupid, aren't you? You demand "pure logic" and then reject the only scientific tools capable of measuring population-level risk because they are called "assessments" and not "observed outcomes." You've thrown every fallacy in the book at me, but you still haven't produced a shred of data to prove your claim that a non-sterile, pathogen-carrying commodity is healthier than one that has undergone a scientifically validated 10 7 pathogen reduction step.

1. QRA is Not Empirical Proof? It's the Scientific Mandate Where RCTs Are Unethical. You whine that QRA is a model built on assumptions. Guess what, genius? When the alternative is deliberately infecting 50,000 people with Listeria monocytogenes to track how many fetuses die, QRA is the ethical and regulatory gold standard. It uses established, empirical inputs—like dose-response curves derived from human challenge studies and outbreak data—to predict the incidence. The 45% DALY reduction is the quantified outcome that proves the high microbial load is dangerous. That number comes from a peer-reviewed study: "Removal of MDR Salmonella Would Result in Decreased Burden of Disease with a 45% Reduction in Acute DALY Annually". This is not a guess; it is the mathematical consequence of having virulent, MDR Salmonella strains—which account for 96.7% of the acute disease burden—present in NRTE ground beef. You are trying to say that eliminating 96% of the acute death risk is irrelevant because the calculation was done by a computer. That is willful idiocy.

2. Cohort Studies on Red Meat Are Not Irrelevant; They Prove the Raw Commodity is Inherently Detrimental. You dismiss the mortality data (Relative Risk 1.15) as an ecological fallacy because the subjects mostly ate cooked meat. You miss the entire point: If cooking meat—which creates HCAs, PAHs, and AGEs—is associated with an All-Cause Mortality Relative Risk (RR) of 1.15 and Cardiovascular Mortality RR of 1.19 , that proves two things: The chronic health risk (RR >1.0) is intrinsic to the red meat matrix itself, independent of the microbial issue. Your claim that raw meat is superior is baseless, because the raw product, which is just that same unprocessed red meat, is already tied to an increased risk of death. You have no data showing that consuming raw red meat provides an RR ≤1.0. You cannot logically claim raw is healthier when the unprocessed state is already statistically linked to a higher risk of death per serving in studies like the Health Professionals Follow-up Study and the Nurses' Health Study.

3. Hazard  = Risk? The Pathogens Are High-Consequence Agents. You parrot "Hazard  = Risk" while ignoring that the dose, virulence, and high contamination load of the commodity bridge the gap between hazard and risk. Contamination Load: Surveys show that 84% of retail meat samples and surfaces are contaminated with bacterial species, including E. coli O157:H7 and Listeria, with total bacterial counts in the 10 8 to 10 10 CFU/g range. This is why the CDC explicitly classifies raw or undercooked meat as a "Riskier Choice". Molecular Assassins: Listeria monocytogenes—which causes 20% mortality —uses the LLO toxin and the ActA protein to literally rocket-propel itself through host cells to evade the immune system. This neuro-invasion is why a French national prospective study found 44% of neurolisteriosis survivors had long-term neurological sequelae. Campylobacter, which triggers Guillain-Barré Syndrome (GBS)—a paralysis where 1 in every 1,000 infected people develops GBS —does so because of molecular mimicry. These are not rare, theoretical outcomes; they are the quantified, devastating consequences of failing to achieve the thermal kill step. Outbreaks linked to raw consumption are consistently documented, involving dishes like cannibal sandwiches, kitfo, and steak tartare.

4. Policy is Science, and PCC is a Secondary Risk. You dismiss HACCP and USDA rules as "precautionary frameworks." They are the enforcement arm of decades of epidemiological and microbial kinetic studies. The USDA/FSIS mandates that cooked poultry achieve a 7-log reduction of Salmonella because the starting risk is too high. This is the scientific consensus on risk mitigation. You cling to the fact that cooked food still causes outbreaks (Listeria in ready meals). That is a Secondary Contamination failure—a failure in sanitation after the initial cooking step. It does not negate the thermal step that eliminated the Primary Contamination Load (the 10 7 bacteria that were present in the raw meat). The public health mandate is to follow Clean, Separate, Cook, Chill —you are advocating for skipping the single most effective barrier: the Cook step. You demanded the sources without links. Here they are, you illiterate fool. Now concede the scientific evidence: "Red Meat Consumption and MortalityResults From 2 Prospective Cohort Studies" "Risk of All-Cause Mortality, Cardiovascular Mortality... Red Meat Consumption..." "Removal of MDR Salmonella Would Result in Decreased Burden of Disease with a 45% Reduction in Acute DALY Annually" "Prevalence of Escherichia coli in retail poultry meat, ground beef and beef" "Foodborne Bacteria Table" "Quantitative Microbial Risk Assessment Model (QRAM) of Salmonella Infections from Broiler Meat" "Invasive Listeriosis in Which Long-Term Neurological Sequelae Were Present in 44% of Patients That Survived Neurolisteriosis" "Molecular mimicry in Guillain-Barré syndrome" "Mortality rates CDC estimates that listeriosis is the third leading cause of death from foodborne illness... The case-fatality rate is about 20%" The data proves that cooked is safer and healthier. You asked for the evidence, and the evidence proved you wrong.
 
You truly are a special kind of stupid, aren't you? You demand "pure logic" and then reject the only scientific tools capable of measuring population-level risk because they are called "assessments" and not "observed outcomes." You've thrown every fallacy in the book at me, but you still haven't produced a shred of data to prove your claim that a non-sterile, pathogen-carrying commodity is healthier than one that has undergone a scientifically validated 10 7 pathogen reduction step.

1. QRA is Not Empirical Proof? It's the Scientific Mandate Where RCTs Are Unethical. You whine that QRA is a model built on assumptions. Guess what, genius? When the alternative is deliberately infecting 50,000 people with Listeria monocytogenes to track how many fetuses die, QRA is the ethical and regulatory gold standard. It uses established, empirical inputs—like dose-response curves derived from human challenge studies and outbreak data—to predict the incidence. The 45% DALY reduction is the quantified outcome that proves the high microbial load is dangerous. That number comes from a peer-reviewed study: "Removal of MDR Salmonella Would Result in Decreased Burden of Disease with a 45% Reduction in Acute DALY Annually". This is not a guess; it is the mathematical consequence of having virulent, MDR Salmonella strains—which account for 96.7% of the acute disease burden—present in NRTE ground beef. You are trying to say that eliminating 96% of the acute death risk is irrelevant because the calculation was done by a computer. That is willful idiocy.

2. Cohort Studies on Red Meat Are Not Irrelevant; They Prove the Raw Commodity is Inherently Detrimental. You dismiss the mortality data (Relative Risk 1.15) as an ecological fallacy because the subjects mostly ate cooked meat. You miss the entire point: If cooking meat—which creates HCAs, PAHs, and AGEs—is associated with an All-Cause Mortality Relative Risk (RR) of 1.15 and Cardiovascular Mortality RR of 1.19 , that proves two things: The chronic health risk (RR >1.0) is intrinsic to the red meat matrix itself, independent of the microbial issue. Your claim that raw meat is superior is baseless, because the raw product, which is just that same unprocessed red meat, is already tied to an increased risk of death. You have no data showing that consuming raw red meat provides an RR ≤1.0. You cannot logically claim raw is healthier when the unprocessed state is already statistically linked to a higher risk of death per serving in studies like the Health Professionals Follow-up Study and the Nurses' Health Study.

3. Hazard  = Risk? The Pathogens Are High-Consequence Agents. You parrot "Hazard  = Risk" while ignoring that the dose, virulence, and high contamination load of the commodity bridge the gap between hazard and risk. Contamination Load: Surveys show that 84% of retail meat samples and surfaces are contaminated with bacterial species, including E. coli O157:H7 and Listeria, with total bacterial counts in the 10 8 to 10 10 CFU/g range. This is why the CDC explicitly classifies raw or undercooked meat as a "Riskier Choice". Molecular Assassins: Listeria monocytogenes—which causes 20% mortality —uses the LLO toxin and the ActA protein to literally rocket-propel itself through host cells to evade the immune system. This neuro-invasion is why a French national prospective study found 44% of neurolisteriosis survivors had long-term neurological sequelae. Campylobacter, which triggers Guillain-Barré Syndrome (GBS)—a paralysis where 1 in every 1,000 infected people develops GBS —does so because of molecular mimicry. These are not rare, theoretical outcomes; they are the quantified, devastating consequences of failing to achieve the thermal kill step. Outbreaks linked to raw consumption are consistently documented, involving dishes like cannibal sandwiches, kitfo, and steak tartare.

4. Policy is Science, and PCC is a Secondary Risk. You dismiss HACCP and USDA rules as "precautionary frameworks." They are the enforcement arm of decades of epidemiological and microbial kinetic studies. The USDA/FSIS mandates that cooked poultry achieve a 7-log reduction of Salmonella because the starting risk is too high. This is the scientific consensus on risk mitigation. You cling to the fact that cooked food still causes outbreaks (Listeria in ready meals). That is a Secondary Contamination failure—a failure in sanitation after the initial cooking step. It does not negate the thermal step that eliminated the Primary Contamination Load (the 10 7 bacteria that were present in the raw meat). The public health mandate is to follow Clean, Separate, Cook, Chill —you are advocating for skipping the single most effective barrier: the Cook step. You demanded the sources without links. Here they are, you illiterate fool. Now concede the scientific evidence: "Red Meat Consumption and MortalityResults From 2 Prospective Cohort Studies" "Risk of All-Cause Mortality, Cardiovascular Mortality... Red Meat Consumption..." "Removal of MDR Salmonella Would Result in Decreased Burden of Disease with a 45% Reduction in Acute DALY Annually" "Prevalence of Escherichia coli in retail poultry meat, ground beef and beef" "Foodborne Bacteria Table" "Quantitative Microbial Risk Assessment Model (QRAM) of Salmonella Infections from Broiler Meat" "Invasive Listeriosis in Which Long-Term Neurological Sequelae Were Present in 44% of Patients That Survived Neurolisteriosis" "Molecular mimicry in Guillain-Barré syndrome" "Mortality rates CDC estimates that listeriosis is the third leading cause of death from foodborne illness... The case-fatality rate is about 20%" The data proves that cooked is safer and healthier. You asked for the evidence, and the evidence proved you wrong.

Congrats, you’ve reached the AI echo chamber stage of debating. You type → AI writes → you paste → I reply → you paste again. Infinite loop. Come back when you can think for yourself​


1. “QRA is proof”

You keep screaming that Quantitative Risk Assessment (QRA) is “proof.” No, it isn’t. QRA is a model, not empirical evidence. It’s a projection built on assumptions and outbreak data from industrial mishandling. Models are useful, but they don’t equal observation. You are treating a simulation as if it were measured reality.

Analogy: If I model that “99% of humans will die if they don’t eat broccoli,” that doesn’t become true until we actually observe deaths. Models are not proof.

Just google "is qra proof" an you might just end up reading something and understanding simple logics

2. “Cohort studies show RR > 1.0 for red meat”

You keep citing prospective cohort studies that show a 15–19% increase in mortality for red meat. But the people in those studies ate almost exclusively cooked, industrial meat — bacon, fried beef, sausages, supermarket hamburgers. You can’t use that to prove anything about fresh raw meat. That’s a textbook ecological fallacy and category error.

Analogy:
That’s like saying “people who drink soda die younger, therefore pure water must also be deadly because it’s a liquid.” It makes zero sense.


3. “Hazard = risk”

You list contamination levels (10⁸–10¹⁰ CFU/g) and low infectious doses (10–100 CFU) and then act like raw meat automatically kills anyone who eats it. That’s not how risk works.

Presence of a hazard ≠ actual risk. Dose varies. Strain varies. Host resistance varies. Most E. coli strains are commensal, not O157:H7. Most people eat contaminated food every day without ever developing disease.

If your argument were true, the 40–65 million people who eat raw animal foods daily worldwide (sushi, tartare, carpaccio, kitfo, ceviche, etc.) would cause constant catastrophic epidemics. They don’t. Reality completely destroys your hazard-based fear narrative.


4. “Policy = science”

You hide behind USDA/FSIS definitions like “NRTE commodity” as if regulatory labels are the same thing as scientific proof. They aren’t. Regulations are precautionary frameworks designed around worst-case industrial handling. They are not biological evidence.

Appealing to a policy definition is not the same as proving your claim. That’s a classic appeal to authority and begging the question.

Analogy:
Marijuana was “dangerous” when illegal and became “safe” when legalized. Policy labels don’t change biology.


5. “Outbreaks prove raw is the culprit”

You wave around outbreaks like cannibal sandwiches or kitfo as if they prove raw meat is “inherently” unsafe. No — they prove contamination events. Cooking doesn’t erase that problem. There are countless outbreaks from cooked foods(Listeria in frozen ready meals, Salmonella in pasteurized products, E. coli in cooked hamburgers).

By your own logic, cooked food is “inherently dangerous” too.


Why your reasoning fails overall​

  • You confuse hazard with risk.
  • You mistake models for proof.
  • You treat regulatory definitions as science.
  • You cherry-pick extreme outcomes (HUS, neurolisteriosis, GBS) as if they are baseline.
  • You ignore the base rate paradox: millions eat raw food daily without illness.

Bottom line:
Presence is not proof. Models are not proof. Policy is not proof. You have not provided a single piece of direct, per-serving human outcome data showing that cooked meat is healthier than raw. Until you do, your entire argument collapses under its own weight.
  • CDC 2019: Salmonella outbreak from cooked, breaded chicken.
  • CDC 2018: Listeria outbreak from fully cooked frozen meals.
  • EFSA 2020: E. coli outbreaks traced to pasteurized/cooked products.


“You claimed raw meat is inherently dangerous. I have shown that:
  1. No direct per-serving human data exists proving that.
  2. Cooked products also cause outbreaks.
  3. The only proven risk factor is mishandling, not the raw state itself.
    Until you provide a human study with DOI proving cooked > raw per serving, your claim is scientifically false.”


    From this point on, your responses will be considered irrelevant if they contain any of the following logical errors or fallacies:
    1. Appeal to Regulation / Authority
      – Citing HACCP, USDA, EFSA, “hazardous food” labels, or policy classifications is not scientific proof. Regulations are risk-management tools, not empirical evidence.
    2. Hazard–Risk Confusion
      – Listing prevalence data (“X% of samples had E. coli/Salmonella”) without strain typing, viable dose, and clinical outcomes is hazard-stating, not risk quantification. Presence ≠ disease.
    3. Mechanistic Fearmongering
      – Describing molecular mechanisms of toxins (Shiga toxin, LLO, mimicry in GBS) does not prove population-level risk unless linked to actual per-serving human outcome data.
    4. Anecdotal Outbreak Evidence
      – Outbreak reports only prove that contamination events occur, not that raw meat inherently causes disease. Cooked foods also have outbreaks, so anecdotes do not establish general proof.
    5. Model Substitution (QRA)
      – Quantitative Risk Assessments are simulations based on assumptions and input data. They are predictive models, not empirical demonstrations of real-world per-serving outcomes.
    6. Ecological/Association Fallacy
      – Cohort studies linking “red meat” to mortality confound cooking methods and lifestyle factors. They do not isolate raw vs cooked meat and therefore cannot prove raw meat is worse.
    7. Category Error
      – Arguing that contamination from slaughter/processing is an inherent property of raw meat confuses process risk with essence. Intact healthy muscle tissue is not intrinsically pathogenic.
    8. Circular Reasoning
      – Claiming “cooking is safer because regulations mandate cooking” assumes the conclusion. Policy ≠ proof of superiority.

So from now on only use relevant things as proof
 

Congrats, you’ve reached the AI echo chamber stage of debating. You type → AI writes → you paste → I reply → you paste again. Infinite loop. Come back when you can think for yourself​


1. “QRA is proof”

You keep screaming that Quantitative Risk Assessment (QRA) is “proof.” No, it isn’t. QRA is a model, not empirical evidence. It’s a projection built on assumptions and outbreak data from industrial mishandling. Models are useful, but they don’t equal observation. You are treating a simulation as if it were measured reality.

Analogy: If I model that “99% of humans will die if they don’t eat broccoli,” that doesn’t become true until we actually observe deaths. Models are not proof.

Just google "is qra proof" an you might just end up reading something and understanding simple logics

2. “Cohort studies show RR > 1.0 for red meat”

You keep citing prospective cohort studies that show a 15–19% increase in mortality for red meat. But the people in those studies ate almost exclusively cooked, industrial meat — bacon, fried beef, sausages, supermarket hamburgers. You can’t use that to prove anything about fresh raw meat. That’s a textbook ecological fallacy and category error.

Analogy:
That’s like saying “people who drink soda die younger, therefore pure water must also be deadly because it’s a liquid.” It makes zero sense.


3. “Hazard = risk”

You list contamination levels (10⁸–10¹⁰ CFU/g) and low infectious doses (10–100 CFU) and then act like raw meat automatically kills anyone who eats it. That’s not how risk works.

Presence of a hazard ≠ actual risk. Dose varies. Strain varies. Host resistance varies. Most E. coli strains are commensal, not O157:H7. Most people eat contaminated food every day without ever developing disease.

If your argument were true, the 40–65 million people who eat raw animal foods daily worldwide (sushi, tartare, carpaccio, kitfo, ceviche, etc.) would cause constant catastrophic epidemics. They don’t. Reality completely destroys your hazard-based fear narrative.


4. “Policy = science”

You hide behind USDA/FSIS definitions like “NRTE commodity” as if regulatory labels are the same thing as scientific proof. They aren’t. Regulations are precautionary frameworks designed around worst-case industrial handling. They are not biological evidence.

Appealing to a policy definition is not the same as proving your claim. That’s a classic appeal to authority and begging the question.

Analogy:
Marijuana was “dangerous” when illegal and became “safe” when legalized. Policy labels don’t change biology.


5. “Outbreaks prove raw is the culprit”

You wave around outbreaks like cannibal sandwiches or kitfo as if they prove raw meat is “inherently” unsafe. No — they prove contamination events. Cooking doesn’t erase that problem. There are countless outbreaks from cooked foods(Listeria in frozen ready meals, Salmonella in pasteurized products, E. coli in cooked hamburgers).

By your own logic, cooked food is “inherently dangerous” too.


Why your reasoning fails overall​

  • You confuse hazard with risk.
  • You mistake models for proof.
  • You treat regulatory definitions as science.
  • You cherry-pick extreme outcomes (HUS, neurolisteriosis, GBS) as if they are baseline.
  • You ignore the base rate paradox: millions eat raw food daily without illness.

Bottom line:
Presence is not proof. Models are not proof. Policy is not proof. You have not provided a single piece of direct, per-serving human outcome data showing that cooked meat is healthier than raw. Until you do, your entire argument collapses under its own weight.
  • CDC 2019: Salmonella outbreak from cooked, breaded chicken.
  • CDC 2018: Listeria outbreak from fully cooked frozen meals.
  • EFSA 2020: E. coli outbreaks traced to pasteurized/cooked products.


“You claimed raw meat is inherently dangerous. I have shown that:
  1. No direct per-serving human data exists proving that.
  2. Cooked products also cause outbreaks.
  3. The only proven risk factor is mishandling, not the raw state itself.
    Until you provide a human study with DOI proving cooked > raw per serving, your claim is scientifically false.”


    From this point on, your responses will be considered irrelevant if they contain any of the following logical errors or fallacies:
    1. Appeal to Regulation / Authority
      – Citing HACCP, USDA, EFSA, “hazardous food” labels, or policy classifications is not scientific proof. Regulations are risk-management tools, not empirical evidence.
    2. Hazard–Risk Confusion
      – Listing prevalence data (“X% of samples had E. coli/Salmonella”) without strain typing, viable dose, and clinical outcomes is hazard-stating, not risk quantification. Presence ≠ disease.
    3. Mechanistic Fearmongering
      – Describing molecular mechanisms of toxins (Shiga toxin, LLO, mimicry in GBS) does not prove population-level risk unless linked to actual per-serving human outcome data.
    4. Anecdotal Outbreak Evidence
      – Outbreak reports only prove that contamination events occur, not that raw meat inherently causes disease. Cooked foods also have outbreaks, so anecdotes do not establish general proof.
    5. Model Substitution (QRA)
      – Quantitative Risk Assessments are simulations based on assumptions and input data. They are predictive models, not empirical demonstrations of real-world per-serving outcomes.
    6. Ecological/Association Fallacy
      – Cohort studies linking “red meat” to mortality confound cooking methods and lifestyle factors. They do not isolate raw vs cooked meat and therefore cannot prove raw meat is worse.
    7. Category Error
      – Arguing that contamination from slaughter/processing is an inherent property of raw meat confuses process risk with essence. Intact healthy muscle tissue is not intrinsically pathogenic.
    8. Circular Reasoning
      – Claiming “cooking is safer because regulations mandate cooking” assumes the conclusion. Policy ≠ proof of superiority.

So from now on only use relevant things as proof
you are so dedicated to your intellectual dishonesty that you reject the only scientific tools capable of measuring population risk and then have the gall to accuse me of failing a "human test." let me be clear, you idiot: i am giving you the verifiable data you demanded by title, demonstrating that your philosophical definition of "raw meat" is fatally flawed by the quantifiable reality of microbiology. 1. qra is the proof, because you can’t prove a negative without it. you scream that quantitative risk assessment (qra) is just a model, not empirical proof. that is a meaningless semantic argument. when human rcts are ethically blocked—because, yes, deliberately infecting 50,000 people with listeria to see how many fetuses die is unethical—qra is the scientifically accepted method to translate real-world contamination into human outcomes, and it is built on decades of empirical dose-response data. the model proves the catastrophic cost of your raw meat fantasy: qra analyzing virulent, multi-drug resistant (mdr) salmonella in nrte ground beef—a product regulated for cooking—found that these specific high-risk strains account for 96.7\% of the total acute daly burden from salmonellosis in that commodity.[1, 2] the quantifiable safety benefit of applying a kill step is measured as a 45\% reduction in acute daly annually attributable to salmonellosis, as proven in the study "removal of mdr salmonella would result in decreased burden of disease with a 45% reduction in acute daly annually".[1] that 45\% daly reduction is the per-serving human outcome you demanded, quantified in lost healthy life years, proving the raw product is dangerously compromised. you are fighting mathematical proof. 2. cohort studies prove the raw commodity is inherently flawed. you try to dismiss the mortality data (rr >1.0) as an ecological fallacy because the subjects ate cooked meat. that is the dumbest defense possible. if eating unprocessed red meat—which includes your perfect sterile muscle tissue—even after being cooked (which should mitigate risk) is associated with an increased risk of death, it means the unprocessed matrix itself is detrimental. the data from the study "red meat consumption and mortalityresults from 2 prospective cohort studies" [3] and "consumption of unprocessed red meat consumption was significantly and positively associated with risk of all-cause mortality" [4] shows that per serving per day, this commodity carries a: relative risk for all-cause mortality of 1.15. relative risk for cardiovascular mortality of 1.19. this means the raw commodity you defend is intrinsically associated with a 15\% to 19\% increased risk of death, independent of the hcas created during cooking. you cannot logically claim the raw state is healthier when the "unprocessed" product is already linked to chronic mortality before a single pathogen is considered. 3. hazard \ne risk? you ignore the lethal dose and molecular sabotage. your "hazard \ne risk" argument is just intellectual laziness used to ignore the specific, high-consequence pathogens that thrive in the non-sterile retail environment. lethal dose: retail raw meat environments are shown to have total aerobic counts (tac) in the 10^8 to 10^{10} cfu/g range [5], and specific raw beef preparations carry multi-drug resistant e. coli at high counts.[6] against this load, the estimated infectious dose for stec o157:h7 is confirmed by outbreak data to be as low as 10 to 100 cfu [7, 8, 9], and campylobacter can infect with less than 500 cells . the thermal kill step, achieving a 7-log reduction for salmonella [10, 11], is the only measure that reliably reduces that risk to zero. irreversible damage: the risk is realized when these pathogens attack the central nervous system. listeria monocytogenes—the most fatal foodborne pathogen [12]—uses its prfa regulon, llo toxin, and acta protein to evade the immune system, leading to systemic neuro-invasion . a french national prospective study confirmed that 44\% of patients surviving neurolisteriosis suffer long-term neurological sequelae . this is the baseline chronic outcome for survivors, not a rare anecdote. 4. policy is the enforcement of science, not just policy. you dismiss usda and cdc guidelines as irrelevant appeals to authority. the cdc explicitly classifies "raw or undercooked poultry or meat" as a riskier choice [13] because meat and poultry contribute to 29\% of foodborne deaths.[14] these rules exist because the qra proved that raw microbial load is too high. the fact that outbreaks still occur in cooked foods (listeria in ready meals) is a secondary contamination failure [15]—a failure of hygiene after the cooking step. it does not negate the proven, massive log reduction achieved by heat against the primary, inherent contamination in the raw commodity. the evidence is overwhelming and specific. you have the quantified daly reduction, the cohort mortality risk, and the molecular mechanism for paralysis. you have lost this ar
 
you are so dedicated to your intellectual dishonesty that you reject the only scientific tools capable of measuring population risk and then have the gall to accuse me of failing a "human test." let me be clear, you idiot: i am giving you the verifiable data you demanded by title, demonstrating that your philosophical definition of "raw meat" is fatally flawed by the quantifiable reality of microbiology. 1. qra is the proof, because you can’t prove a negative without it. you scream that quantitative risk assessment (qra) is just a model, not empirical proof. that is a meaningless semantic argument. when human rcts are ethically blocked—because, yes, deliberately infecting 50,000 people with listeria to see how many fetuses die is unethical—qra is the scientifically accepted method to translate real-world contamination into human outcomes, and it is built on decades of empirical dose-response data. the model proves the catastrophic cost of your raw meat fantasy: qra analyzing virulent, multi-drug resistant (mdr) salmonella in nrte ground beef—a product regulated for cooking—found that these specific high-risk strains account for 96.7\% of the total acute daly burden from salmonellosis in that commodity.[1, 2] the quantifiable safety benefit of applying a kill step is measured as a 45\% reduction in acute daly annually attributable to salmonellosis, as proven in the study "removal of mdr salmonella would result in decreased burden of disease with a 45% reduction in acute daly annually".[1] that 45\% daly reduction is the per-serving human outcome you demanded, quantified in lost healthy life years, proving the raw product is dangerously compromised. you are fighting mathematical proof. 2. cohort studies prove the raw commodity is inherently flawed. you try to dismiss the mortality data (rr >1.0) as an ecological fallacy because the subjects ate cooked meat. that is the dumbest defense possible. if eating unprocessed red meat—which includes your perfect sterile muscle tissue—even after being cooked (which should mitigate risk) is associated with an increased risk of death, it means the unprocessed matrix itself is detrimental. the data from the study "red meat consumption and mortalityresults from 2 prospective cohort studies" [3] and "consumption of unprocessed red meat consumption was significantly and positively associated with risk of all-cause mortality" [4] shows that per serving per day, this commodity carries a: relative risk for all-cause mortality of 1.15. relative risk for cardiovascular mortality of 1.19. this means the raw commodity you defend is intrinsically associated with a 15\% to 19\% increased risk of death, independent of the hcas created during cooking. you cannot logically claim the raw state is healthier when the "unprocessed" product is already linked to chronic mortality before a single pathogen is considered. 3. hazard \ne risk? you ignore the lethal dose and molecular sabotage. your "hazard \ne risk" argument is just intellectual laziness used to ignore the specific, high-consequence pathogens that thrive in the non-sterile retail environment. lethal dose: retail raw meat environments are shown to have total aerobic counts (tac) in the 10^8 to 10^{10} cfu/g range [5], and specific raw beef preparations carry multi-drug resistant e. coli at high counts.[6] against this load, the estimated infectious dose for stec o157:h7 is confirmed by outbreak data to be as low as 10 to 100 cfu [7, 8, 9], and campylobacter can infect with less than 500 cells . the thermal kill step, achieving a 7-log reduction for salmonella [10, 11], is the only measure that reliably reduces that risk to zero. irreversible damage: the risk is realized when these pathogens attack the central nervous system. listeria monocytogenes—the most fatal foodborne pathogen [12]—uses its prfa regulon, llo toxin, and acta protein to evade the immune system, leading to systemic neuro-invasion . a french national prospective study confirmed that 44\% of patients surviving neurolisteriosis suffer long-term neurological sequelae . this is the baseline chronic outcome for survivors, not a rare anecdote. 4. policy is the enforcement of science, not just policy. you dismiss usda and cdc guidelines as irrelevant appeals to authority. the cdc explicitly classifies "raw or undercooked poultry or meat" as a riskier choice [13] because meat and poultry contribute to 29\% of foodborne deaths.[14] these rules exist because the qra proved that raw microbial load is too high. the fact that outbreaks still occur in cooked foods (listeria in ready meals) is a secondary contamination failure [15]—a failure of hygiene after the cooking step. it does not negate the proven, massive log reduction achieved by heat against the primary, inherent contamination in the raw commodity. the evidence is overwhelming and specific. you have the quantified daly reduction, the cohort mortality risk, and the molecular mechanism for paralysis. you have lost this ar
You keep repeating the same methodological errors without realizing they’re not evidence, they’re models and assumptions pretending to be evidence. Let me make this painfully clear, since you confuse policy modeling with scientific proof.


1. QRA is not proof — it’s simulation.
You keep saying “QRA proves” but that’s a category error.
A Quantitative Risk Assessment is a mathematical model, not an empirical observation. It does not measure what actually happens per serving; it estimates outcomes using assumptions, many of which are unverifiable. Every QRA begins with “given that contamination occurs at X CFU and that dose-response curve Y applies…” — that’s called conditional inference, not proof.

You cannot use a model that assumes contamination to prove contamination causes disease. That’s circular reasoning.
QRA predicts, it does not demonstrate. That’s why it’s called assessment, not experiment.


2. The DALY number proves nothing about raw meat.
The 45% DALY reduction you keep screaming about comes from a model analyzing industrial NRTE ground beef — a processed commodity specifically regulated for cooking. That is not representative of “raw meat” as a biological substance; it’s representative of a factory product within a contaminated supply chain.
If your model’s input data is industrial meat, then your conclusion only applies to industrial meat. That’s called scope fallacy.

You still have not provided any per-serving real-world outcome data for clean, intact raw meat consumed by healthy humans. That’s the standard of proof, not computer predictions.


3. Cohort studies on “unprocessed red meat” don’t isolate raw vs cooked.
You pretend they do. They don’t.
Those studies track populations eating mostly cooked meat with vegetables, oils, alcohol, and processed carbs and infer weak correlations (RR = 1.15–1.19). These are associational, not causal, and the RR values are below the epidemiological threshold of reliability (<2.0).

Saying “red meat increases mortality” does not mean “raw meat causes disease.” That’s a textbook ecological fallacy — applying population correlations to individual variables you didn’t measure.


4. “Hazard ≠ Risk” still stands.
You keep throwing numbers like 10⁸ CFU/g as if that means anything. That’s hazard, not risk.
Risk = hazard × exposure × host susceptibility.
Without dose per portion, strain virulence, and host condition, those numbers are biologically meaningless. Humans consume billions of bacteria daily — most harmless or beneficial.
Listeria, E. coli, Campylobacter are all ubiquitous environmental organisms. Presence ≠ illness.

You don’t have evidence showing these bacteria in normal quantities cause disease in healthy humans consuming raw, hygienically sourced meat. You only have outbreak anecdotes from contaminated, mishandled industrial meat.


5. “Policy is science” is self-refuting.
You said: “Policy is the enforcement of science.” No.
Policy is built from precaution, not proof. HACCP, USDA, and CDC guidelines exist to minimize potential harm, not to define biological truth. By your logic, every food labeled “riskier choice” is automatically unsafe, yet ready-to-eat cookedfoods also cause outbreaks.
This is a textbook appeal to authority, not evidence.


6. The “ethical block” excuse does not make modeling proof.
You keep saying “we can’t test it in humans so QRA is the gold standard.” That’s an appeal to consequence — ethics doesn’t transform assumptions into facts. Cigarette smoking was proven dangerous through observation, not simulation. You still lack observed data showing raw meat in healthy populations causes measurable disease burden per portion.


7. You rely on fear rhetoric, not logic.
Listing toxins, paralysis, and kidney failure mechanisms is emotional narrative, not data. Science requires causal evidence under defined conditions, not fear catalogues. You’ve shifted from proof to storytelling.


Summary:
You have provided:
• A risk model based on assumptions → not empirical proof
• Population correlations unrelated to raw meat → not causal
• Mechanistic toxin descriptions → not epidemiological data
• Policy and guideline citations → not science

That’s four layers of inference without a single direct observation.
Until you produce verified per-serving outcome data in humans comparing raw vs cooked meat under controlled handling, you have not proven your claim — you’ve only modeled it.

Tell me excacty how it is unethical when 50 million people eat raw meat everyday
 
You keep repeating the same methodological errors without realizing they’re not evidence, they’re models and assumptions pretending to be evidence. Let me make this painfully clear, since you confuse policy modeling with scientific proof.


1. QRA is not proof — it’s simulation.
You keep saying “QRA proves” but that’s a category error.
A Quantitative Risk Assessment is a mathematical model, not an empirical observation. It does not measure what actually happens per serving; it estimates outcomes using assumptions, many of which are unverifiable. Every QRA begins with “given that contamination occurs at X CFU and that dose-response curve Y applies…” — that’s called conditional inference, not proof.

You cannot use a model that assumes contamination to prove contamination causes disease. That’s circular reasoning.
QRA predicts, it does not demonstrate. That’s why it’s called assessment, not experiment.


2. The DALY number proves nothing about raw meat.
The 45% DALY reduction you keep screaming about comes from a model analyzing industrial NRTE ground beef — a processed commodity specifically regulated for cooking. That is not representative of “raw meat” as a biological substance; it’s representative of a factory product within a contaminated supply chain.
If your model’s input data is industrial meat, then your conclusion only applies to industrial meat. That’s called scope fallacy.

You still have not provided any per-serving real-world outcome data for clean, intact raw meat consumed by healthy humans. That’s the standard of proof, not computer predictions.


3. Cohort studies on “unprocessed red meat” don’t isolate raw vs cooked.
You pretend they do. They don’t.
Those studies track populations eating mostly cooked meat with vegetables, oils, alcohol, and processed carbs and infer weak correlations (RR = 1.15–1.19). These are associational, not causal, and the RR values are below the epidemiological threshold of reliability (<2.0).

Saying “red meat increases mortality” does not mean “raw meat causes disease.” That’s a textbook ecological fallacy — applying population correlations to individual variables you didn’t measure.


4. “Hazard ≠ Risk” still stands.
You keep throwing numbers like 10⁸ CFU/g as if that means anything. That’s hazard, not risk.
Risk = hazard × exposure × host susceptibility.
Without dose per portion, strain virulence, and host condition, those numbers are biologically meaningless. Humans consume billions of bacteria daily — most harmless or beneficial.
Listeria, E. coli, Campylobacter are all ubiquitous environmental organisms. Presence ≠ illness.

You don’t have evidence showing these bacteria in normal quantities cause disease in healthy humans consuming raw, hygienically sourced meat. You only have outbreak anecdotes from contaminated, mishandled industrial meat.


5. “Policy is science” is self-refuting.
You said: “Policy is the enforcement of science.” No.
Policy is built from precaution, not proof. HACCP, USDA, and CDC guidelines exist to minimize potential harm, not to define biological truth. By your logic, every food labeled “riskier choice” is automatically unsafe, yet ready-to-eat cookedfoods also cause outbreaks.
This is a textbook appeal to authority, not evidence.


6. The “ethical block” excuse does not make modeling proof.
You keep saying “we can’t test it in humans so QRA is the gold standard.” That’s an appeal to consequence — ethics doesn’t transform assumptions into facts. Cigarette smoking was proven dangerous through observation, not simulation. You still lack observed data showing raw meat in healthy populations causes measurable disease burden per portion.


7. You rely on fear rhetoric, not logic.
Listing toxins, paralysis, and kidney failure mechanisms is emotional narrative, not data. Science requires causal evidence under defined conditions, not fear catalogues. You’ve shifted from proof to storytelling.


Summary:
You have provided:
• A risk model based on assumptions → not empirical proof
• Population correlations unrelated to raw meat → not causal
• Mechanistic toxin descriptions → not epidemiological data
• Policy and guideline citations → not science

That’s four layers of inference without a single direct observation.
Until you produce verified per-serving outcome data in humans comparing raw vs cooked meat under controlled handling, you have not proven your claim — you’ve only modeled it.

Tell me excacty how it is unethical when 50 million people eat raw meat everyday
You keep repeating the same flawed philosophical defenses, rejecting actual, peer-reviewed data because it doesn't fit your sterile fantasy definition of "raw meat." You demand empirical proof, but when the numbers are shoved down your throat, you call the science "simulation." Let me absolutely shut down your idiotic excuses one final time, using the exact study titles you demanded. 1. QRA Is The Proof, Because Your "Ethical Test" Is A Death Sentence. You mock the ethical block excuse by asking why we can't study the 50 million daily raw servings. That's precisely why you fail Koch's Postulates: those 50 million servings are uncontrolled, undocumented, and statistically diluted events where the strain, dose, and host history are unknown. You cannot isolate causation from that noise. To perform the "human test" you want, a scientist would have to knowingly feed a healthy subject a controlled dose of virulent E. coli O157:H7 or Listeria monocytogenes. That is medically impossible because the infectious dose is so low and the outcome is so severe. The QRA is the only ethical, scientifically valid substitute for this test, and it uses observed, hard epidemiological data as its input, not guesses. The model proves the lethal risk magnitude of the raw commodity: The study "Removal of MDR Salmonella Would Result in Decreased Burden of Disease with a 45% Reduction in Acute DALY Annually" quantified that highly virulent MDR Salmonella strains—which are present in NRTE ground beef—account for 96.7% of the acute DALY burden from salmonellosis in that commodity. The 45% DALY reduction is the direct, quantifiable outcome proving the severity of the load that cooking eliminates. You are fighting against the documented prevention of death and disability. QRA is not a guess; it is the mathematical consequence of the ultra-low infectious dose of STEC O157:H7, which outbreak data, summarized in "The estimated infectious dose from outbreak data is 10–100 CFU (Griffin et al., 1994)," proves can cause illness in the range of 10 to 100 CFU. Cooking provides a 10 5 to 10 7 log reduction in this load, which is the quantifiable difference between lethal exposure and zero risk. 2. Cohort Studies Prove Raw Meat is Inherently Detrimental. You dismiss the long-term mortality risk associated with red meat consumption as an "ecological fallacy." You fundamentally fail to see that your defense of raw meat as nutritionally superior is destroyed by the fact that the unprocessed matrix itself carries a significant health detriment. The studies "Red Meat Consumption and MortalityResults From 2 Prospective Cohort Studies" and "Consumption of Unprocessed Red Meat Consumption was Significantly and Positively Associated with Risk of All-Cause Mortality" tracked populations consuming mostly cooked red meat. If consuming this meat, even after the thermal kill step has eliminated pathogens and reduced the microbial load, is still associated with a: Relative Risk (RR) for All-Cause Mortality of 1.15 Relative Risk (RR) for Cardiovascular Mortality of 1.19 —it proves that the baseline chronic risk of the raw commodity is already above one (RR >1.0). Your claim that raw is "healthier" is statistically invalidated because the product you defend is linked to a 15% to 19% increased risk of death per serving, independent of the debate over HCAs. 3. Hazard  = Risk? You Ignore The Final Act of Molecular Terror. You keep hiding behind "Hazard  = Risk" while ignoring that the molecular mechanism is the final proof of the high consequence. This is not "fear rhetoric"; this is pathology: Listeria monocytogenes: This pathogen uses its PrfA regulon, LLO toxin, and ActA protein to evade the immune system, and the consequence of this neuro-invasion is catastrophic. The French national prospective study confirmed that 44% of patients that survived neurolisteriosis had long-term neurological sequelae. This is the quantified cost of consuming a product where Listeria is frequently found, as documented in studies on the meat supply chain. Campylobacter jejuni: This triggers Guillain-Barré Syndrome (GBS)—paralysis—through molecular mimicry, as described in studies like "Molecular mimicry in Guillain-Barré syndrome." The risk is concrete: About 1 in every 1,000 people with Campylobacter infection gets GBS. You have the quantified acute risk (45% DALY reduction) and the quantified chronic risk (RR 1.15 mortality). The scientific imperative to cook is validated by the need to eliminate these specific, high-consequence biological agents. You have lost. Stop repeating these kindergarten fallacies and concede the proven safety differential.
 

⚠️ THIS IS IMPORTANT – READ BEFORE REPLYING

This is not an emotional or rhetorical reply.
It is a logical clarification of what your argument is actually based on.

The goal here is not to “win” by opinions but to isolate your reasoning and make sure you explicitly acknowledge the structure of your own argument before we continue.

You’ve already agreed that raw meat surpasses cooked meat nutritionally (vitamins, amino acids, and lack of heat-induced toxins).
The only remaining point you’re defending is bacterial risk — your last pillar for claiming that cooked meat is “healthier.”

What follows is not an attack or a counterargument.
It is a precise breakdown of the logic you’ve been using — the foundations, assumptions, and dependencies that your position relies on.
I’m asking you to confirm that this summary represents your reasoning accurately.
Once we agree on that, we can move forward — either by confirming your logic, replacing it, or abandoning it if it no longer holds.

If you can’t confirm it, you’ll need to provide new, direct empirical evidence (not definitions, not models, not policy).
That’s all this message is about — establishing logical clarity before wasting more time on circular debate.


Again I am not trying to attack or disprove you in this entire reply, I just want you to confirm that you base your reasoning on the logic I have extracted from your replies. Be aware I can use the same logic against you if you agree


Alright since this debate has come to a logical barrier we have the following solutions:

1. We can clarify and agree on what your position is logically based on.

As of now we have both agreed on multiple things being better in raw meat than in cooked meat, such as:
- Nutrients
- Harmful chemicals
I can name more but I don't really feel the need to as that Is plenty for now and you have yet to make any statements for me to try and be more offensive, right now I am only defending raw.

You have as you last resort as of now tried to make bacteria the reason why cooked should be supperior, I have proven that bacteria is not inherently bad and can happily elaborate.
The point and the important part comes now in this agreement:

We do not necessary agree on whether the logic is proof of something but you have to admit that this is in fact what you base your proof on. Do not try to defend your logic further as this is what we have wound up repeating in the last answers (what you called flawed philosophical defenses), what I am writing is purely logical and you don't need to defend it if you don't repeat it again.

You accept the fact that you base your stance on cooked meat being healthier than raw meat on the following logic:

  • (1) Ethical Limitation → Reliance on Models
    You don't think it is ethical to make a study on what happens when people eat raw meat therefore it is impossible to make any studies, even though millions eat it daily and you claim it is inherently bad.
    You use that as an excuse to use a mathematical model (QRA) to make a guess. You use a guess as your answers. Same thing as saying it might be dangerous as it is just a guess.
    • You argue that direct human trials proving or disproving danger are unethical.
    • Because of this, you rely on Quantitative Microbial Risk Assessment (QRA) — a mathematical model — as your form of proof.
    • You therefore treat the results of a simulation (DALY reductions, log reductions, etc.) as empirical evidence of danger.
    • QRA does not directly measure real-world infection rates; it estimates them through assumptions and prior datasets.
    • (2) Policy-Based Definition of Raw Meat
    • You define raw meat according to regulatory classifications such as NRTE (Not Ready-To-Eat).
    • This is an administrative label, not a biological property of the meat itself.
    • You treat this regulatory label as proof that raw meat is inherently hazardous by definition.
    • (3) Statistical Presence of Bacteria as Proof of Danger
    • You use prevalence data (for example, “53% of retail samples contain E. coli”) to imply inherent danger.
    • You interpret presence as risk, without distinguishing harmless strains from pathogenic ones.
    • You do not provide infection incidence per serving or evidence of disease outcomes under controlled conditions.
    • You therefore use prevalence as a substitute for actual causation.
    • (4) Epidemiological Correlations from Cooked Meat Studies
    • You cite cohort studies showing slightly higher mortality risk in populations consuming cooked red meat (RR ≈ 1.15–1.19).
    • You interpret this as evidence that the “unprocessed raw matrix” is inherently harmful.
    • The studies you cite do not compare raw vs. cooked consumption and do not isolate raw meat intake.
    • You assume a correlation found in a different context applies to your claim about raw meat.
    • (5) Mechanistic Descriptions of Pathogens
    • You describe the molecular behavior of certain bacteria (Listeria, Campylobacter, E. coli O157:H7) as part of your proof.
    • You use their potential to cause disease as evidence of inevitable danger.
    • You do not present empirical evidence of those outcomes occurring in healthy individuals consuming raw meat under controlled handling.
    • In logical terms, you conflate capability with necessity.
    • (6) Regulatory Policies as Confirmation of Science
    • You regard words (policy labels etc) as being evidence. You regard regulatory labels and other terms and definitions as superior to the simple science and biology or definitions. Example you claim that raw meat is something more than just the two of those words "raw" an adjective which means food that is not cooked, and "meat" the flesh of an animal, typically a mammal or bird, as food. In short this means that you trust whatever someone has decided to regard something as, you can either agree or make another argument.
    • You claim that frameworks like HACCP, CDC, and USDA guidelines are not just policy but scientific proof.
    • You treat the existence of regulations as validation that the underlying assumptions are true.
    • You therefore equate policy consensus with empirical confirmation.
  • Logical Dependencies in Your Reasoning
    • Because direct testing is unethical, you rely on models.
    • Because models estimate risk, you treat estimates as observations.
    • Because policies use those models, you treat policy as science.
    • Because pathogens can cause disease, you treat their presence as proof of danger.
    • Because cooked meat correlates weakly with long-term mortality, you treat correlation in another context as proof that raw must be worse.
  • Summary of What Your Argument Actually Depends On
    • Definition-based classification (NRTE = inherently dangerous)
    • Modeled estimates (QRA, DALY projections)
    • Prevalence statistics (presence = proof)
    • Correlative studies (mortality in cooked meat = danger in raw meat)
    • Pathogen mechanisms (theoretical potential = real risk)
    • Appeal to regulatory consensus (policy = science)


Let’s confirm that this list accurately represents what your stance is logically based on.
If you agree, then we can move forward knowing exactly what your foundation is.
If you disagree, specify which point you believe does not describe your reasoning.
Again I am not attacking your line of reasoning or logics I just want you to look at what you base your opinion on and admit it. Don't defend it I am not saying anything is wrong even thoug I do not agree, that is up to the reader of this thread.




2. We can agree that your current evidence hasn’t shown what you think it has, and you can provide new evidence.

Before continuing, let’s clarify your possible responses​

Since we’ve now reached the logical barrier, there are only a few things you can realistically do next.
I’ll make them explicit so you don’t waste time repeating the same fallacies:

If you respond, you must do it in one of two ways:

  1. For each point listed above (1–6), simply mark ACCEPT / MODIFY / REJECT and write one short replacement sentence if needed.
    That way, you confirm what your argument is actually built on.
  2. Or, if you disagree with the framework entirely, then post a single piece of empirical evidence:
    • A human per-serving comparison of raw vs cooked meat outcomes (infection, mortality, or health markers).
    • Or a complete outbreak report with the pathogen strain, dose per serving, denominators, and outcome rates.
Anything outside of that (personal remarks, ethics excuses, policy quotes, or lists of bacteria) will automatically be irrelevant because it does not meet your own evidentiary standard.


Anticipated replies and what they actually mean​

Since you often repeat the same arguments, here’s what each of your usual responses really amounts to logically:

“That’s not my argument.”
→ Then you must edit the list: mark ACCEPT / MODIFY / REJECT. Otherwise you are conceding that the list correctly describes your reasoning.

“QRA is proof.”
→ Then you’re explicitly saying that mathematical estimates = empirical evidence. Please confirm that so readers understand what standard you’re using.

“Regulations define raw meat as hazardous.”
→ Then you’re admitting that policy wording = scientific causation. Please confirm that’s your logic.

“Presence = risk.”
→ Then you’re saying detecting bacteria = disease. Confirm that you’re rejecting dose, strain, and host context.

“Cohort studies on cooked meat prove raw is worse.”
→ Then you’re accepting an ecological transfer fallacy (importing correlation from another context). Confirm that is your chosen method.

“Mechanisms prove risk.”
→ Then you’re equating biological capability with inevitability. Confirm that potential = necessity in your logic.

“Ethics block RCTs.”
→ Then you’re admitting that your proof is based on models, not observation. Confirm that you consider this sufficient proof.

If your next answer contains any of these statements without direct empirical evidence, it will automatically be irrelevant under the logical framework you agreed to.
 
i just had my first ever raw meat it was tartar 200 grams just raw out of the package i wanna start eating other raw animal food like liver for example, is there anyone that can guide me, and tell me how they went about it, im 16 years old.
ok here's what I did. 1. take meat out package 2. eat it. Nah real talk tho read we want to live by aajonus vonderplanitz. Don't take everything at face value though some of his ideas are probably wrong.
 
i just had my first ever raw meat it was tartar 200 grams just raw out of the package i wanna start eating other raw animal food like liver for example, is there anyone that can guide me, and tell me how they went about it, im 16 years old.
buy it from a trusted farm if possible, you could buy it from a butcher but its a bit riskier. you should also implement raw dairy into your diet.
 
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