zygobase
#34 mistakes
- Joined
- Mar 1, 2026
- Posts
- 175
- Reputation
- 137
It’s been a minute since I posted a thread. Overall my experience has been pretty good with Selank, and throwing in the additional Semax because I honestly don’t care about losing some focus. Forgetting to think it is actually better for humanity imo.
What I’ve noticed from Selank and Semax:
I was able to type extremely fast (probably from the Semax on the days I took it). I could also play reaction-based games way better than normal. For example in Chivalry 2 my counters were landing on time. While reading books I absorbed way more info and actually remembered it instead of it just going in one ear and out the other. Memories actually stuck in my normally high-inhib head.
My sleep was moderately better too. Didn’t really notice many side effects while on it. The only real ones came after I stopped to lower tolerance — felt very tired, dizzy for like 2 hours, cognition felt slowed down for about a week. My typing speed dropped by like 3x and my fear of big groups of people came back. It sucked, but the pros outweighed the cons for me.
I took it every morning after drinking an original Coke or some orange juice. Any cane sugar works for the energy. Eat a solid meal after and stay hydrated and you’ll be set.
And for the low iqs that are gonna say this is cope keep reading
For short: The closest “make or break” gene for Selank is the MAOA gene (Monoamine Oxidase A). The main genetic reason someone might find Selank ineffective or have a bad time comes down to specific variants of this gene.
The MAOA gene (The “non-responder” driver)
The MAOA gene encodes an enzyme that breaks down key monoamine neurotransmitters like serotonin, dopamine, and norepinephrine.
High activity variant (MAOA-H): People with this burn through dopamine and serotonin super fast. Since Selank works partly by stabilizing these neurotransmitters, folks with ultra-fast MAOA clearance often find the effects wear off quick or don’t hit at all.
Low activity variant (MAOA-L):
On the other hand. if you have the low activity version your brain already naturally builds up higher levels of these neurotransmitters. Your system is already pretty saturated, so adding Selank (which modulates these pathways) can cause a paradoxical effect — more anxiety or fatigue instead of the expected calm.
Commercial peptide panels like the PlexusDx precision peptide test check for stuff like the rs1137070 variant on the MAOA gene to predict if you’ll respond poorly to a Selank cycle.
The BDNF Val66Met polymorphism.
Selank helps by up-regulating BDNF expression for neuroplasticity, memory retention, and cognitive repair. People carrying the common Met allele (Val66Met) naturally produce significantly less BDNF. Because their genetic machinery is already resistant to making much of it, the long-term cognitive and brain-boosting effects of Selank are often way weaker compared to those with the wild-type Val/Val genotype. DNR tubby fat people
What I’ve noticed from Selank and Semax:
I was able to type extremely fast (probably from the Semax on the days I took it). I could also play reaction-based games way better than normal. For example in Chivalry 2 my counters were landing on time. While reading books I absorbed way more info and actually remembered it instead of it just going in one ear and out the other. Memories actually stuck in my normally high-inhib head.
My sleep was moderately better too. Didn’t really notice many side effects while on it. The only real ones came after I stopped to lower tolerance — felt very tired, dizzy for like 2 hours, cognition felt slowed down for about a week. My typing speed dropped by like 3x and my fear of big groups of people came back. It sucked, but the pros outweighed the cons for me.
I took it every morning after drinking an original Coke or some orange juice. Any cane sugar works for the energy. Eat a solid meal after and stay hydrated and you’ll be set.
And for the low iqs that are gonna say this is cope keep reading
For short: The closest “make or break” gene for Selank is the MAOA gene (Monoamine Oxidase A). The main genetic reason someone might find Selank ineffective or have a bad time comes down to specific variants of this gene.
The MAOA gene (The “non-responder” driver)
The MAOA gene encodes an enzyme that breaks down key monoamine neurotransmitters like serotonin, dopamine, and norepinephrine.
High activity variant (MAOA-H): People with this burn through dopamine and serotonin super fast. Since Selank works partly by stabilizing these neurotransmitters, folks with ultra-fast MAOA clearance often find the effects wear off quick or don’t hit at all.
Low activity variant (MAOA-L):
On the other hand. if you have the low activity version your brain already naturally builds up higher levels of these neurotransmitters. Your system is already pretty saturated, so adding Selank (which modulates these pathways) can cause a paradoxical effect — more anxiety or fatigue instead of the expected calm.
Commercial peptide panels like the PlexusDx precision peptide test check for stuff like the rs1137070 variant on the MAOA gene to predict if you’ll respond poorly to a Selank cycle.
The BDNF Val66Met polymorphism.
Selank helps by up-regulating BDNF expression for neuroplasticity, memory retention, and cognitive repair. People carrying the common Met allele (Val66Met) naturally produce significantly less BDNF. Because their genetic machinery is already resistant to making much of it, the long-term cognitive and brain-boosting effects of Selank are often way weaker compared to those with the wild-type Val/Val genotype. DNR tubby fat people
