Should i use KY19382 and an fgfr3 inhib instead of gh

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Itd probably cost more but should i just skip the gh and allat and use ky19382 and fgfr3 inhibs instead
 
Solution
19NorCel
whats wrong with nuking e2? also igf-1 is the most important pathway for bone growth, do you have any knowledge on endocrine physiology? ky is good because it inhibits cxxc5 pathway you absolute retard
Genuine iqlet, don’t you get sarcasm. Nuking e2 lowers igf-1 conversion and makes you feel like shit while making your bones very fragile. And where did I say KY is bad ? I just said that if you think I fgfr3 inhibitors are too niche/hard to find then why advise KY, but yes both are very good I never said the opposite. And the reason fgfr3 is so glazed is because it’s the one with the most proven efficacy when it comes to these compounds. Genuinely never give advice again it’s clear you don’t know shit
No you shouldn't because:
  • KY19382 has no human height data and is unproven as of right now(risk)
  • FGFR3 inhibitors mainly treat achondroplasia with good growth gains in those patients but it lacks evidence as a general height booster in healthy people and carry risks
  • GH remains the established, clinically monitored option for short stature with known results

These alternatives are riskier and not ready to replace it

Mark as solution by clicking the checkmark on the right :feelsautistic:
 
Itd probably cost more but should i just skip the gh and allat and use ky19382 and fgfr3 inhibs instead
what is this obsession with fgfr3 inhibitors? just do rhGh, letro and maybe ky with abalo
 
No you shouldn't because:
  • KY19382 has no human height data and is unproven as of right now(risk)
  • FGFR3 inhibitors mainly treat achondroplasia with good growth gains in those patients but it lacks evidence as a general height booster in healthy people and carry risks
  • GH remains the established, clinically monitored option for short stature with known results

These alternatives are riskier and not ready to replace it

Mark as solution by clicking the checkmark on the right :feelsautistic:
Please shut up you know nothing, fgfr3 inhibitors barely have sides and the fact that it’s mainly studied in achondroplasia DOES NOT mean it only works on achondroplasia
 
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what is this obsession with fgfr3 inhibitors? just do rhGh, letro and maybe ky with abalo
Yeah dude just nuke your E2 and igf-1 so you can feel like shit instead of taking an objectively superior compound. And how can you just say stick to HGH and then advise ky which is just as hard to source as fgfr3 inhibitors
 
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Reactions: Deleted member 180385
Yeah dude just nuke your E2 and igf-1 so you can feel like shit instead of taking an objectively superior compound. And how can you just say stick to HGH and then advise ky which is just as hard to source as fgfr3 inhibitors
whats wrong with nuking e2? also igf-1 is the most important pathway for bone growth, do you have any knowledge on endocrine physiology? ky is good because it inhibits cxxc5 pathway you absolute retard
 
whats wrong with nuking e2? also igf-1 is the most important pathway for bone growth, do you have any knowledge on endocrine physiology? ky is good because it inhibits cxxc5 pathway you absolute retard
Genuine iqlet, don’t you get sarcasm. Nuking e2 lowers igf-1 conversion and makes you feel like shit while making your bones very fragile. And where did I say KY is bad ? I just said that if you think I fgfr3 inhibitors are too niche/hard to find then why advise KY, but yes both are very good I never said the opposite. And the reason fgfr3 is so glazed is because it’s the one with the most proven efficacy when it comes to these compounds. Genuinely never give advice again it’s clear you don’t know shit
 
Solution
Genuine iqlet, don’t you get sarcasm. Nuking e2 lowers igf-1 conversion
:lul::lul: nobody cares about lower igf-1 "conversion" when the effect is literally negligible and avoidable via rhGH
and makes you feel like shit while making your bones very fragile
so? the goal here is height you dumbass nigger. nobody gives a shit about mood. imagine putting mood over few inches of height :lul: and fragile bones is avoidable as well, not that its important compared to lenght but still
I just said that if you think I fgfr3 inhibitors are too niche/hard to find then why advise KY, but yes both are very good I never said the opposite. And the reason fgfr3 is so glazed is because it’s the one with the most proven efficacy when it comes to these compounds. Genuinely never give advice again it’s clear you don’t know shit
they're not "hard to find." they're easily sourcable. the problem is money and fragility of the compound. also most fgfr3 inhibitors are actually bad for height because they inhibit fgfr2 as well. also the chondrocytes are safe from fgfr3 fate via rhGH usage

there are way more "niche" compounds than these. and ky isnt expensive nor hard to source

you don't have achondroplasia so you won't have the same effects from fgfr3 inhibitors as someone with achondroplasia would
 
:lul::lul: nobody cares about lower igf-1 "conversion" when the effect is literally negligible and avoidable via rhGH

so? the goal here is height you dumbass nigger. nobody gives a shit about mood. imagine putting mood over few inches of height :lul: and fragile bones is avoidable as well, not that its important compared to lenght but still

they're not "hard to find." they're easily sourcable. the problem is money and fragility of the compound. also most fgfr3 inhibitors are actually bad for height because they inhibit fgfr2 as well. also the chondrocytes are safe from fgfr3 fate via rhGH usage

there are way more "niche" compounds than these. and ky isnt expensive nor hard to source

you don't have achondroplasia so you won't have the same effects from fgfr3 inhibitors as someone with achondroplasia would
You promote GH and then say "who cares about igf-1 conversion" ? Yeah I give you clearly have deep mental retardation
 
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You promote GH and then say "who cares about igf-1 conversion" ? Yeah I give you clearly have deep mental retardation
yeah, obviously you fucking retard, that lower igf1 conversion effect is negligible if you use high dosages of rhGH.
 

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