test without fusing growth plates

[darodiddler]

so we all know anyone whos not retarded knows that ai works systemcaly in the blood and doesnt affect the growth plates bon tissue reciptors

and to pervent that i was thinking about a SERM( Selective Estrogen Receptor Modulato)

can directly on estrogen receptors and pervent fusing from the growth plates while taking test and not fusing the growth plates

am i correct

 

[Zeylet]

so we all know anyone whos not retarded knows that ai works systemcaly in the blood and doesnt affect the growth plates bon tissue reciptors

and to pervent that i was thinking about a SERM( Selective Estrogen Receptor Modulato)

can directly on estrogen receptors and pervent fusing from the growth plates while taking test and not fusing the growth plates

am i correct

prob are correct

but what is ur height and what height u tryna get

 

[pingukraz]

serds mog serms, some serms could pottentially lower igf-1 levels
fulvestrant or camizestrant prefferable camizestrant because it specifically targets ERa

 

[polarpop]

you do realize a serm is an agonist in bone tissue right?

endocrinologists use a serm purposefully to accelerate bone maturation and fuse the plates of children suffering from gigantism lmao

 

[goatislove691]

so we all know anyone whos not retarded knows that ai works systemcaly in the blood and doesnt affect the growth plates bon tissue reciptors

Yeah it doesnt affect any receptors it lowers estrogen, which is what causes your plates to fuse. Less estrogen = Less bone plates fusing, SERMS wont decrease estrogen just do AIs

 

[darodiddler]

prob are correct

but what is ur height and what height u tryna get

im ghd and like 167-170

if my plates stay for longer i would get alot taller since im ghd

 

[darodiddler]

d

you do realize a serm is an agonist in bone tissue right?

endocrinologists use a serm purposefully to accelerate bone maturation and fuse the plates of children suffering from gigantism lmao

deadass?
SO IT WOUDLNT WORK? i thought it like targeted the reciptors like th breast for example to reverse gyno idk tho so it wouldnt work?

 

[darodiddler]

Yeah it doesnt affect any receptors it lowers estrogen, which is what causes your plates to fuse. Less estrogen = Less bone plates fusing, SERMS wont decrease estrogen just do AIs

idk this guy said it iwll fuse them

 

[darodiddler]

you do realize a serm is an agonist in bone tissue right?

endocrinologists use a serm purposefully to accelerate bone maturation and fuse the plates of children suffering from gigantism lmao

@Zagro

 

[darodiddler]

hallooooo

 

[polarpop]

d

deadass?
SO IT WOUDLNT WORK? i thought it like targeted the reciptors like th breast for example to reverse gyno idk tho so it wouldnt work?

dont wanna go into the science, but a serm in breast tissue acts like an antagonist and in bone tissue as an agonist

also rep me faggot

 

[darodiddler]

idk dont wanna go into the science, but a serm in breast tissue acts like an antagonist and in bone tissue as an agonist

also rep me faggot

idk what that means but its like activates the estrogen receiptors? which will fuse them?

 

[polarpop]

idk what that means but its like activates the estrogen receiptors? which will fuse them?

in the breast tissue example you gave, a serm acts like a shield, so it blocks real estrogen from attaching which ultimately saves you from actually getting gyno

but in bones it turns into fake estrogen, so it physically attaches to the growth plates and activates them

because a serm acts exactly like estrogen when it touches the bone it sends a signal to the bone "close growth plates now"

so bottom line, If you take a serm it will stop gyno, but your bones will think they are being flooded with pure estrogen. It will permanently fuse your plates. That is why you have to use an ai an ai just stops the body from making estrogen in the first place, so there is nothing there to fuse the plates.

 

[darodiddler]

in the breast tissue example you gave, a serm acts like a shield, so it blocks real estrogen from attaching which ultimately saves you from actually getting gyno

but in bones it turns into fake estrogen, so it physically attaches to the growth plates and activates them

because a serm acts exactly like estrogen when it touches the bone it sends a signal to the bone "close growth plates now"

so bottom line, If you take a serm it will stop gyno, but your bones will think they are being flooded with pure estrogen. It will permanently fuse your plates. That is why you have to use an ai an ai just stops the body from making estrogen in the first place, so there is nothing there to fuse the plates.

damm idk so theres noway to run test without fusing the growth plates

 

[polarpop]

damm idk so theres noway to run test without fusing the growth plates

nga you take an aromatase inhibtor

 

[darodiddler]

nga you take an aromatase inhibtor

but doesnt an ai work systimcaly in the blood and doesnt target the bone tissue?

??? idk what u mean so like taking ai serm together works?

 

[polarpop]

but doesnt an ai work systimcaly in the blood and doesnt target the bone tissue?

??? idk what u mean so like taking ai serm together works?

no bro, your growth plates make their own estrogen in their cartilage meaning an ai will also go into your bones aswell, so this means itll also shut off the growth plates ability to make estrogen locally.

also dont take an ai with serm, thats the worst thing you can do lmao i told you why above

 

[darodiddler]

no bro, your growth plates make their own estrogen in their cartilage meaning an ai will also go into your bones aswell, so this means itll also shut off the growth plates ability to make estrogen locally.

also dont take an ai with serm, thats the worst thing you can do lmao i told you why above

i thought ai doesnt affect the growth plates and local estrogen would fuse it even if u have low e2?

idk

 

[polarpop]

i thought ai doesnt affect the growth plates and local estrogen would fuse it even if u have low e2?

idk

local estrogen is the estrogen your growth plates make, but you keep thinking an ai only stays in your blood, an ai doesnt just float in your veins it gets absoprbed by organs, fat and your bones.

and the whole point of an ai is to shut down the ability of the growth plate to produce its own local estrogen.

 

[darodiddler]

local estrogen is the estrogen your growth plates make, but you keep thinking an ai only stays in your blood, an ai doesnt just float in your veins it gets absoprbed by organs, fat and your bones.

and the whole point of an ai is to shut down the ability of the growth plate to produce its own local estrogen.

but if u take high test it will supress that and aromtize right? idk

 

[polarpop]

but if u take high test it will supress that and aromtize right? idk

yes, but were talking like 1000mg of test a week, the remaining 1% of unblocked aromatase inside the bone might eventually convert enough of that testinto estrogen to slowly fuse the plates.

 

[darodiddler]

yes, but were talking like 1000mg of test a week, the remaining 1% of unblocked aromatase inside the bone might eventually convert enough of that testinto estrogen to slowly fuse the plates.

i was thinking of 100mg weekly brah so its safe?

 

[polarpop]

i was thinking of 100mg weekly brah so its safe?

your good but 100mg is a waste lmao thats just a trt dose, your gonna shutdown your natural production for the same amount you produce naturally lmao. its a miserable protocol

but you do you bro

 

[darodiddler]

your good but 100mg is a waste lmao thats just a trt dose, your gonna shutdown your natural production for the same amount you produce naturally lmao. its a miserable protocol

but you do you bro

i was thinking of adding anavar with that low dose test? idk brah i dont want to fuse my growth plates

 

[polarpop]

i was thinking of adding anavar with that low dose test? idk brah i dont want to fuse my growth plates

i have no knowledge about anavar.

but you wont fuse your plates bro

i just told you why lmao

if you think im lying or some shit go DYOR on actual science then come back and youll see im right lmao

 

[bluebandz]

so we all know anyone whos not retarded knows that ai works systemcaly in the blood and doesnt affect the growth plates bon tissue reciptors

and to pervent that i was thinking about a SERM( Selective Estrogen Receptor Modulato)

can directly on estrogen receptors and pervent fusing from the growth plates while taking test and not fusing the growth plates

am i correct

there’s also androgenic fusion

 

[darodiddler]

there’s also androgenic fusion

theres no androgenic fusion i was the one who made that post

i made that up

 

[bluebandz]

theres no androgenic fusion i was the one who made that post

i made that up

fuck
i was trying to sound smart
your a genius

 

[darodiddler]

L

fuck
i was trying to sound smart
your a genius

LMAOAOAOOA

 

[darodiddler]

i have no knowledge about anavar.

but you wont fuse your plates bro

i just told you why lmao

if you think im lying or some shit go DYOR on actual science then come back and youll see im right lmao

idk

what dose should i run then without fusing my shit?

ah idk how to do that

 

[Alexanderr]

Any androgens will hasten growth plate closure (senescence), period. Aromatase might be a primary driver but it’s not the only. This was well known here in 2020 already…
If you’re even remotely interested in heightmaxxing you should stay far away from pinning test.

 

[Alexanderr]

theres no androgenic fusion i was the one who made that post

i made that up

No, exposure to androgens will by itself hasten growth plate senescence indepedent of aromatase.

 

[darodiddler]

No, exposure to androgens will by itself hasten growth plate senescence indepedent of aromatase.

What do I do brah should I take var with no test base?like 6 daily var in also ongg

So no ass is good for growing taller?

 

[darodiddler]

Any androgens will hasten growth plate closure (senescence), period. Aromatase might be a primary driver but it’s not the only. This was well known here in 2020 already…
If you’re even remotely interested in heightmaxxing you should stay far away from pinning test.

I don't know they said direct androgenic isn't a thing and I was wrong
Where was it known since 2020

 

[doidfestroyer]

so we all know anyone whos not retarded knows that ai works systemcaly in the blood and doesnt affect the growth plates bon tissue reciptors

and to pervent that i was thinking about a SERM( Selective Estrogen Receptor Modulato)

can directly on estrogen receptors and pervent fusing from the growth plates while taking test and not fusing the growth plates

am i correct

bebeh js learned about SERMS

 

[Zagro]

@Zagro

He’s bullshitting dude do not listen when retards provide zero studies

 

[Zagro]

No, exposure to androgens will by itself hasten growth plate senescence indepedent of aromatase.

It’s fully dependent on aromatase

 

[darodiddler]

He’s bullshitting dude do not listen when retards provide zero studies

i dont know tho cuz google said serm is a anogist or something

 

[darodiddler]

It’s fully dependent on aromatase

bro i made the post the direct androgenic fusion niggas was comin
g sayin that and i said i made that up

 

[darodiddler]

Any androgens will hasten growth plate closure (senescence), period. Aromatase might be a primary driver but it’s not the only. This was well known here in 2020 already…
If you’re even remotely interested in heightmaxxing you should stay far away from pinning test.

couldu ellaborate more

 

[goatislove691]

idk this guy said it iwll fuse them

Nah i was talking about AIs, they'll lower estrogen just get bloods done before id say

Yeah be careful with SERMS just do an ai at that point

 

[goatislove691]

What do I do brah should I take var with no test base?like 6 daily var in also ongg

So no ass is good for growing taller?

Do not take a high dose of anavar without a test base, max 10 mg daily
Compounds like anavar are real for height growth though, and taking a lose dose is fine, because it will cause less test -> less aromatization -> less estrogen all around.
Do HGH too though, and you can always do higher dose var + small test base (100mg) to keep e2 levels low, maybe AI if necessary

 

[JporkFoid]

so we all know anyone whos not retarded knows that ai works systemcaly in the blood and doesnt affect the growth plates bon tissue reciptors

and to pervent that i was thinking about a SERM( Selective Estrogen Receptor Modulato)

can directly on estrogen receptors and pervent fusing from the growth plates while taking test and not fusing the growth plates

am i correct

chatgpt or get a rat and figure out

 

[Alexanderr]

couldu ellaborate more

Once I’m back home and on my computer I’ll take the time to explain why @Zagro is retarded, yes.

 

[Zagro]

Once I’m back home and on my computer I’ll take the time to explain why @Zagro is retarded, yes.

Unc you don’t even know shit about heightmaxxing do you wanna get your ass flamed or sum

Blud thinks his shit from 2020 holds truth, “yes bro run 500mg test 6.25mg aromasin to not crash E2 for height” ahh times

 

[Alexanderr]

Unc you don’t even know shit about heightmaxxing do you wanna get your ass flamed or sum

Blud thinks his shit from 2020 holds truth, “yes bro run 500mg test 6.25mg aromasin to not crash E2 for height” ahh times

No one back then was advocating to run test to heightmax— it’s just retarded.

Heightmaxxing knowledge on this forum has actually regressed.

I don’t even need to get my computer, I’ll just do it now:

Androgens accelerate growth plate closure through multiple pathways, not just aromatase/estrogen conversion.

While estrogen (via aromatase) is a major driver, androgen receptors in growth plate cartilage can independently promote senescence.

This is literally well-established in endocrinology.

See, growth plate chondrocytes have their own androgen receptors. Testosterone and DHT can bind these directly. This then triggers IGF 1 and GH related signaling that independently promotes closure. Literally zero aromatase conversion needed.

Last point: if aromatase/estrogen were the only pathway controlling plate closure, people with aromatase deficiency would never fuse their plates but clinically, they just fuse them much later, not never. This proves androgens contribute independently.

 

[Boshitoshi]

No one back then was advocating to run test to heightmax— it’s just retarded.

Heightmaxxing knowledge on this forum has actually regressed.

I don’t even need to get my computer, I’ll just do it now:

Androgens accelerate growth plate closure through multiple pathways, not just aromatase/estrogen conversion.

While estrogen (via aromatase) is a major driver, androgen receptors in growth plate cartilage can independently promote senescence.

This is literally well-established in endocrinology.

See, growth plate chondrocytes have their own androgen receptors. Testosterone and DHT can bind these directly. This then triggers IGF 1 and GH related signaling that independently promotes closure. Literally zero aromatase conversion needed.

Last point: if aromatase/estrogen were the only pathway controlling plate closure, people with aromatase deficiency would never fuse their plates but clinically, they just fuse them much later, not never. This proves androgens contribute independently.

so theres no fix

 

[darodiddler]

chatgpt or get a rat and figure out

or i gotta be the rat

 

[darodiddler]

No one back then was advocating to run test to heightmax— it’s just retarded.

Heightmaxxing knowledge on this forum has actually regressed.

I don’t even need to get my computer, I’ll just do it now:

Androgens accelerate growth plate closure through multiple pathways, not just aromatase/estrogen conversion.

While estrogen (via aromatase) is a major driver, androgen receptors in growth plate cartilage can independently promote senescence.

This is literally well-established in endocrinology.

See, growth plate chondrocytes have their own androgen receptors. Testosterone and DHT can bind these directly. This then triggers IGF 1 and GH related signaling that independently promotes closure. Literally zero aromatase conversion needed.

Last point: if aromatase/estrogen were the only pathway controlling plate closure, people with aromatase deficiency would never fuse their plates but clinically, they just fuse them much later, not never. This proves androgens contribute independently.

SO DIRECT ANDROGENIC FUSION IS REAL I WASNT RETARDED??

 

[Alexanderr]

so theres no fix

For what? If you mean there’s no way to permanently halt growth plate closure, then yes, there’s no fix. Even if you nuked androgens and aromatase they’re still closing— just very late.

I didn’t even mention the other half a dozen factors that contribute to growth plate senescence like DNA methylation, finite proliferation capacity of the chrondocytes (growth plates), etc.

Fact is we cannot fully halt cellular aging and as a consequence they will fuse eventually.

This doesn’t mean you can’t keep them open for a long time, just that it’s retarded to speed up the aging process by pinning androgens like test.