Eduardo DOV
Fuchsia
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All this talk about telegony has arisen in recent years due to a study with flies.
It's not real for humans, and even for the flies, the DNA of the offspring was either from the first male or the second male—there is no mixing of DNA. This idea of DNA mixing is false even with the flies! What happened was that the seminal fluid from the first male interacted with the fly's still immature ovaries, causing a chemical change in how the ovaries absorb nutrients during development. This led the eggs to form a much more nutritious vitello (it's the yolk in chicken eggs), which caused larvae with stronger yolk to become larger, and those with weaker yolk to become smaller. And this final size of the larva's development influences the final size of the fly (I don't know exactly why, but it has to do with energy, obtaining resources easier or harder—whatever).
In other words, the seminal fluid from the first male influenced vitellogenesis (yolk/vitello formation), increasing or decreasing it depending on the male's size. But vitellogenesis doesn't exist in humans! We aren't born from eggs; our nutrition is determined by our mother's nutrition, and the little bit of "vitello" (it's not even called vitello for humans) is only for the first cell divisions.
These female flies store sperm for later use, and the seminal fluid of flies diffuses into the ovaries (that's what causes the telegony)—it's a very different mechanism from humans/mammals in general. These female flies have a specific organ for storing sperm, called spermatheca, present in polyandrous species (which mate with multiple males). The spermatheca is anatomically close to the ovarioles (the tubes in the ovary where eggs develop). This facilitates the diffusion of molecules from the seminal fluid to the ovary.
The effect observed was in the size of the offspring: the size was determined by the first male that ejaculated when the ovaries were immature. But the DNA was 100% from the sperm that fertilized the egg (either the first or the second one, because, like I said, flies store sperm—in 13% of cases the sperm that fertilized was from the first, 87% from the second).
THE EFFECT HAS NOTHING TO DO WITH DNA CHANGE. IT IS SIMPLY A MORE NUTRITIVE FORMATION OF THE YOLK (SOME MOLECULES IN THE SEMINAL FLUID INTERACT WITH RECEPTORS IN THE OVARY). THE FATHER'S DNA WAS 100% THERE.
BUT WHAT IF SEMINAL FLUID HAD SOME INFLUENCE IN HUMAN IMMATURE OVARIES?? From what I've researched, human seminal fluid doesn't come into contact with immature ovaries. It's already difficult for sperm to reach a mature egg, imagine the fluid. Now imagine the immature ovaries that are even further away. And even if they did eventually reach it, the design seems made to discourage this from happening, whereas in flies it seems made to encourage it—that is, probably no effect like this would happen, and in fact it probably would be detrimental to ovarian health. The design suggests that immature human ovaries are protecting themselves from the external environment.
source: grok. i talked a lot with grok and asked everything.
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