lettucemale
Njul
- Joined
- Apr 1, 2026
- Posts
- 5
- Reputation
- 2
I've been designing a 30 week protocol that I tried to make it for linear growth and after its done I will do a nice recomp cycle, any thoughts?
Background / Stats:
Age: 16 (turning 17 on Dec 24)
Current Height: 5'9
Parents: Mom is 5'7–5'8, Dad is 5'9–5'10
Growth History: Haven't grown at all in the past year; have basically been stuck at 5'9 since I was 13 or 14.
Here is what I came up with:
Weeks 0–22: Height
150mg Test E (weekly base)
2.5mg Letrozole EOD
10mg Tamoxifen daily
8mg Erdafitinib daily (5 weeks on, 2 weeks off)
Weeks 22–30: Body Recomp Phase
350mg Test E weekly
25mg Anavar daily
300mg Masteron weekly
2.5mg Letrozole EOD
10mg Tamoxifen daily
My reasoning for each compound:
Erdafitinib (Balversa): It’s a pan-FGFR inhibitor. FGFR3 basically acts as the biological brake on bone growth in cartilage. My thinking is that blocking FGFR3 takes off the natural speed limiter on chondrocytes, triggering a rapid linear growth spurt independently of standard GH/IGF-1 signaling. I set the 5-on/2-off pulse to manage systemic toxicity.
Letrozole: Estrogen is what actually fuses and closes epiphyseal growth plates. Running 2.5mg EOD is meant to crush aromatization down to zero so the growth plates stay open indefinitely while Erda elongates the long bones.
Tamoxifen: Added as extra insurance to block estrogen receptors directly at the bone tissue level in case any residual E2 slips through.
Test E (150mg): Just a baseline TRT dose to keep normal physiological functions running while endogenous production is shut down.
Phase 2 (350mg Test / Anavar / Masteron): Standard dry anabolic stack to rapidly build lean tissue, harden up, and drop body fat once the height phase ends.
A few potential issues / questions I want feedback on:
Letro + Tamox interaction: Does Tamoxifen induce hepatic clearance of Letrozole enough to make running them together counterproductive compared to just using an AI solo?
Crashed E2 Risks: Is 2.5mg Letro EOD going to crash E2 so hard that joint pain, ruined lipids, and cartilage degradation offset the growth benefits?
Erdafitinib Toxicity: For anyone who has looked into pan-FGFR clinical data, how significant are the risks of Central Serous Retinopathy (retinal fluid buildup) and hyperphosphatemia (phosphate spikes), and is there a reliable way to mitigate them off-label?
Steroids vs. Plate Fusion: Will blasting Test, Var, and Masto in Phase 2 accelerate skeletal maturation and fuse whatever plate cartilage is left, even with heavy AI usage?
Background / Stats:
Age: 16 (turning 17 on Dec 24)
Current Height: 5'9
Parents: Mom is 5'7–5'8, Dad is 5'9–5'10
Growth History: Haven't grown at all in the past year; have basically been stuck at 5'9 since I was 13 or 14.
Here is what I came up with:
Weeks 0–22: Height
150mg Test E (weekly base)
2.5mg Letrozole EOD
10mg Tamoxifen daily
8mg Erdafitinib daily (5 weeks on, 2 weeks off)
Weeks 22–30: Body Recomp Phase
350mg Test E weekly
25mg Anavar daily
300mg Masteron weekly
2.5mg Letrozole EOD
10mg Tamoxifen daily
My reasoning for each compound:
Erdafitinib (Balversa): It’s a pan-FGFR inhibitor. FGFR3 basically acts as the biological brake on bone growth in cartilage. My thinking is that blocking FGFR3 takes off the natural speed limiter on chondrocytes, triggering a rapid linear growth spurt independently of standard GH/IGF-1 signaling. I set the 5-on/2-off pulse to manage systemic toxicity.
Letrozole: Estrogen is what actually fuses and closes epiphyseal growth plates. Running 2.5mg EOD is meant to crush aromatization down to zero so the growth plates stay open indefinitely while Erda elongates the long bones.
Tamoxifen: Added as extra insurance to block estrogen receptors directly at the bone tissue level in case any residual E2 slips through.
Test E (150mg): Just a baseline TRT dose to keep normal physiological functions running while endogenous production is shut down.
Phase 2 (350mg Test / Anavar / Masteron): Standard dry anabolic stack to rapidly build lean tissue, harden up, and drop body fat once the height phase ends.
A few potential issues / questions I want feedback on:
Letro + Tamox interaction: Does Tamoxifen induce hepatic clearance of Letrozole enough to make running them together counterproductive compared to just using an AI solo?
Crashed E2 Risks: Is 2.5mg Letro EOD going to crash E2 so hard that joint pain, ruined lipids, and cartilage degradation offset the growth benefits?
Erdafitinib Toxicity: For anyone who has looked into pan-FGFR clinical data, how significant are the risks of Central Serous Retinopathy (retinal fluid buildup) and hyperphosphatemia (phosphate spikes), and is there a reliable way to mitigate them off-label?
Steroids vs. Plate Fusion: Will blasting Test, Var, and Masto in Phase 2 accelerate skeletal maturation and fuse whatever plate cartilage is left, even with heavy AI usage?
