Whats Better For Height, Physiological or Supraphysiological levels of testosterone.

[Ezraoh]

Soon Ill taper up to 8ius of hgh. With aromasin.
And I know test levels are crucial during growth periods ( Im 16)
So main question or topic is what would be better for maximum final height , physiological or supraphysiological levels of T
I know that With more testosterone means more aromatase but we can just up the aromasin dose.
But what I don't know fully is that I heard, higher T levels effect chondrocyte maturation more. (Less maximum final height)
I dont know how true this claim is.

 

[Ezraoh]

bump

 

[LTLightskin]

Testosterone/TRT doesn't contribute to growth nor any growth hormones. Estrogen is what's responsible, focus on minimizing estrogen and taking peptides.

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citeseerx.ist.psu.edu

In contrast, patients with complete andro-gen insensitivity syndrome have a nearly normal pubertal growth spurt (the amplitude of the spurt is intermediate between male and female) and undergo epiphyseal fusion during pubertal development. Androgens are thus not necessary for the pubertal growth acceleration or for the cessation of linear growth that occurs in late puberty.

The role of estrogen in bone growth and maturation during childhood and adolescence

Twenty years ago it was believed that pubertal growth was stimulated by testicular androgen in boys and by adrenal androgen in girls. Estrogen, which …

www.sciencedirect.com

We concluded that the pubertal growth spurt of both sexes is driven primarily by estrogen, and that the more rapid epiphyseal maturation of prepubertal girls (vs boys) may be explained by their higher estradiol levels.

The growth hormone (GH)– insulin-like growth factor axis during testosterone replacement therapy in GH-treated hypopituitary males

Several studies suggest a direct effect of sex steroids on insulin-like growth factor-I (IGF-I) production. Oestrogen has been hypothesized directly t…

www.sciencedirect.com

We conclude that major changes in total IGF-I are not induced during conventional intramuscular testosterone replacement in GH-treated hypopituitary males, suggesting that testosterone effects on IGF-I are likely to be secondary to a stimulation of endogenous GH release.