Why do sped kids always look like (that)

foreveriqletsigma

foreveriqletsigma

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Is it development or their actual disorder (Down syndrome, severe autism, etc.) that causes them to look like that? I want to know the specifics why they always look so horrendously subhuman.
 
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almost all conditions impact your entire body, autism isn't something that only affects parts of your brain, your entire central nervous system and digestive system are affected, so also your face.

same goes for adhd.
 
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  • Hmm...
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Is it development or their actual disorder (Down syndrome, severe autism, etc.) that causes them to look like that? I want to know the specifics why they always look so horrendously subhuman.
they dont always there is this kid on instagram who is non verbal htn+ and has teen girsl in his comments saying how handsome he is and that theyd like to marry him




look at the comments
 
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Off the top of my head “Jayden your bus is here” and “Hey bartender you assume I cannot have a margarita” Sorry cant send photos rn.
1761579458049

1761579482482

These?
 
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same goes for schizophrenia by the way.

>long face, prominent nose with bulbous tip, and narrow orbital fissures
explain the kid i just sent in. most people are just sub 5 anywhere paired with shit expression control and body movements and usually being overwieght they look like freaks
 
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same goes for schizophrenia by the way.

>long face, prominent nose with bulbous tip, and narrow orbital fissures
wtf:hnghn::hnghn:almost literally described me,but i broke my nose so thats doesnt count
 
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they dont always there is this kid on instagram who is non verbal htn+ and has teen girsl in his comments saying how handsome he is and that theyd like to marry him




look at the comments

Since he’s non verbal he could’ve retained proper tongue posture during craniofacial development, leading him to look the way he is. Im talking about other disorders though, this is an exception.
 
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Since he’s non verbal he could’ve retained proper tongue posture during craniofacial development, leading him to look the way he is. Im talking about other disorders though, this is an exception.
in 2025 you still believe that. teen girls want him while incels mew their hardest having suboptimal craniofacial development
 
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in 2025 you still believe that. teen girls want him while incels mew their hardest having suboptimal craniofacial development
Craniofacial development during early YOUTH, im not talking about him mewing during adolescent years.
 
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same goes for schizophrenia by the way.

>long face, prominent nose with bulbous tip, and narrow orbital fissures
could also be bc of the drugs they give you, i stopped growing ever since they gave me those (for example im 21cm shorter than my father) 😢
 
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could also be bc of the drugs they give you, i stopped growing ever since they gave me those (for example im 21cm shorter than my father) 😢
explain the kid i sent in this thread then he has girls simping for him despite beong non verbal
 
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Environmental factors, also the reason why mouth breathers look awful
i was a mouth breather due to autism during development and have top tier mandible. 4.5 inch ramus and 5 inch mandible plane length
 
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they dont always there is this kid on instagram who is non verbal htn+ and has teen girsl in his comments saying how handsome he is and that theyd like to marry him




look at the comments

Nooo brutal bp :feelsrope::feelsrope::feelsrope:
 
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explain the kid i just sent in. most people are just sub 5 anywhere paired with shit expression control and body movements and usually being overwieght they look like freaks
nothing prevents you from being goodlooking if you have all your other features going for you.
could also be bc of the drugs they give you, i stopped growing ever since they gave me those (for example im 21cm shorter than my father) 😢
most of your skull is nearly done developing by the time you're diagnosed.
 
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It's just epigenetics and developmental traits, like the other guy said if someone is non verbal they won't use their jaw or facial muscles besides eating, causing bad craniofacial development.
There are thousands of reasons but mainly developmental traits
 
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explain the kid i sent in this thread then he has girls simping for him despite beong non verbal
idk probably just genetics but also could be because woman have this kind of protector kinda feeling towards kids right. idk i used to get along well with girls in my childhood aswell so maybe thats the case but now im kinda isolated
 
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It's just epigenetics and developmental traits, like the other guy said if someone is non verbal they won't use their jaw or facial muscles besides eating, causing bad craniofacial development.
There are thousands of reasons but mainly developmental traits
Interesting
 
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almost all conditions impact your entire body, autism isn't something that only affects parts of your brain, your entire central nervous system and digestive system are affected, so also your face.

same goes for adhd.
What wpuld be common facial features of adhd
 
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nothing prevents you from being goodlooking if you have all your other features going for you.

most of your skull is nearly done developing by the time you're diagnosed.
its kinda mad he gets that much attention and proposals and hes probably never gonna have an interest back in them ore be married to some mtb some day who dresses and washes him like a kid and probably holds him still to have kids with him
 
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What wpuld be common facial features of adhd
there are so many moggers with adhd you cant even start to act like it would have cause on your looks
 
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Interesting
This is GPT but very informative.

Factors Influencing Craniofacial Development in Autism and Down Syndrome​


1. Genetic and Epigenetic Influences


  • Down Syndrome (Trisomy 21): Down syndrome is caused by an extra copy of chromosome 21, which affects gene expression across the body, including genes involved in craniofacial development. For example, genes like DYRK1A and DSCAM influence skull and facial structure, potentially leading to characteristic features such as a flatter midface, smaller jaw, or upslanting palpebral fissures. Epigenetic modifications, like DNA methylation patterns, may further modulate how these genes are expressed, affecting bone and tissue development.
  • Autism Spectrum Disorder (ASD): Autism is genetically heterogeneous, with hundreds of associated genes (e.g., SHANK3, CHD8). Some of these genes are involved in neural crest cell migration, which is critical for forming facial bones and cartilage. Altered expression due to epigenetic changes (e.g., histone modification or DNA methylation) could subtly affect craniofacial structure, though this varies widely among individuals.
  • Epigenetic Stressors: Prenatal exposure to environmental factors (e.g., maternal stress, toxins, or nutritional deficiencies) can alter epigenetic markers, potentially affecting craniofacial development in both conditions. For example, folate deficiency during pregnancy has been linked to altered methylation patterns, which may influence facial symmetry or jaw development.

2. Developmental Muscle Use and Neuromuscular Factors


  • Reduced Muscle Engagement in ASD: As suggested in the query, some individuals with autism, particularly those who are non-verbal, may use facial and jaw muscles less for communication (e.g., speaking or expressive gestures). This reduced muscle activity could theoretically influence jaw and facial bone development, as mechanical stress from muscle use helps shape bone structure (a process called mechanotransduction). For example, less frequent chewing or speaking might lead to weaker jaw muscle development, potentially affecting mandibular growth.
  • Motor Challenges in Down Syndrome: Individuals with Down syndrome often have hypotonia (low muscle tone), which can affect the muscles of the face and mouth. Reduced muscle tone may lead to less robust stimulation of craniofacial bones during development, potentially contributing to features like a smaller midface or underdeveloped jaw.
  • Orofacial Motor Patterns: Both conditions may involve atypical orofacial motor patterns (e.g., atypical chewing, swallowing, or breathing patterns). For instance, mouth breathing, which is common in some individuals with Down syndrome due to airway issues, can alter tongue posture and lead to changes in dental arches or jaw alignment over time.

3. Prenatal and Early Developmental Factors


  • Embryonic Tissue Development: Craniofacial features are largely determined by the neural crest cells during embryonic development. In Down syndrome, the overexpression of genes on chromosome 21 can disrupt the migration or differentiation of these cells, leading to distinct facial characteristics. In autism, mutations in genes regulating neural crest development (e.g., PAX6 or HOX genes) might subtly affect facial symmetry or proportions in some individuals.
  • Intrauterine Environment: Factors like maternal inflammation, infection, or metabolic conditions (e.g., gestational diabetes) during pregnancy can influence fetal craniofacial development. These factors may disproportionately affect individuals with genetic predispositions, such as those with Down syndrome or autism-linked mutations.
  • Growth Hormone Dysregulation: In Down syndrome, growth hormone pathways may be disrupted, leading to slower or altered growth of facial bones. In autism, hormonal imbalances (e.g., oxytocin or serotonin signaling) could indirectly affect tissue development, though evidence is less clear.

4. Oral and Dental Development


  • Dental Anomalies in Down Syndrome: Individuals with Down syndrome often have delayed tooth eruption, smaller teeth, or missing teeth, which can affect jaw alignment and facial aesthetics. These traits stem from genetic disruptions and altered bone remodeling processes.
  • Bruxism in Autism: Some individuals with autism exhibit bruxism (teeth grinding), which can alter dental occlusion and jaw structure over time. Chronic bruxism may lead to changes in the temporomandibular joint (TMJ) or facial muscle strain, potentially affecting appearance.
  • Orthodontic Issues: Both conditions may be associated with malocclusion (misaligned teeth) due to atypical jaw growth or muscle tone. For example, a high-arched palate in Down syndrome or atypical tongue thrusting in autism can influence dental and facial structure.

5. Environmental and Behavioral Influences


  • Diet and Nutrition: In both autism and Down syndrome, dietary preferences or restrictions (e.g., sensory-based food aversions in autism or gastrointestinal issues in Down syndrome) may affect nutritional intake, which is critical for bone and tissue growth. For instance, deficiencies in calcium or vitamin D could impair craniofacial bone development.
  • Sensory Processing and Habits: In autism, sensory sensitivities may lead to habits like repetitive facial touching or atypical chewing patterns, which could influence muscle and bone development over time. In Down syndrome, repetitive behaviors or oral habits (e.g., tongue protrusion) may similarly affect orofacial structure.
  • Sleep and Airway Issues: Obstructive sleep apnea, more common in Down syndrome due to anatomical airway differences, can lead to mouth breathing, which alters tongue position and may contribute to a narrower palate or elongated face. Some individuals with autism may also have sleep-disordered breathing, with similar effects.

6. Syndromic and Comorbid Conditions


  • Associated Syndromes in Autism: Some individuals with autism have co-occurring genetic syndromes (e.g., Fragile X syndrome), which can include distinct craniofacial features like a long face or prominent ears. These are driven by specific genetic mutations affecting developmental pathways.
  • Midface Hypoplasia in Down Syndrome: The characteristic flat midface in Down syndrome is linked to underdevelopment of the maxilla, driven by genetic and cellular mechanisms. This can alter facial proportions and is a hallmark of the condition.
  • Connective Tissue Differences: In Down syndrome, connective tissue abnormalities (e.g., due to collagen gene dysregulation) can affect skin elasticity and facial structure, contributing to softer or less defined facial contours.

Notes and Considerations​


  • Variability: Both autism and Down syndrome are highly variable. Not all individuals with these conditions exhibit noticeable craniofacial differences, and many have typical or aesthetically pleasing features. The notion of "inoptimal lookism traits" is subjective and not universally applicable.
  • Avoiding Stigmatization: Framing physical differences as "bad" or "inoptimal" can perpetuate harmful stereotypes. Craniofacial features in these conditions are part of natural variation and do not inherently reflect health or value.
  • Research Gaps: While some studies link genetic and epigenetic factors to craniofacial development in Down syndrome, the evidence for autism is less consistent due to its heterogeneity. More research is needed to understand subtle developmental influences.
  • Ethical Framing: Discussions about appearance should focus on function (e.g., speech, chewing, breathing) and health rather than aesthetic judgments, as the latter can reinforce harmful societal biases.
 
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idk probably just genetics but also could be because woman have this kind of protector kinda feeling towards kids right. idk i used to get along well with girls in my childhood aswell so maybe thats the case but now im kinda isolated
"hes so pretty" "hes so handsome" "i want him" some protector traits that is. we know womens nature, hypergamous. they are making their intentions clear and he doesnt look like a little kid. he cant even talk so they cant cope that oh hes so interesting or makes me laugh they want to fuck his mute ass cus his skull
 
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This is GPT but very informative.

Factors Influencing Craniofacial Development in Autism and Down Syndrome​


1. Genetic and Epigenetic Influences


  • Down Syndrome (Trisomy 21): Down syndrome is caused by an extra copy of chromosome 21, which affects gene expression across the body, including genes involved in craniofacial development. For example, genes like DYRK1A and DSCAM influence skull and facial structure, potentially leading to characteristic features such as a flatter midface, smaller jaw, or upslanting palpebral fissures. Epigenetic modifications, like DNA methylation patterns, may further modulate how these genes are expressed, affecting bone and tissue development.
  • Autism Spectrum Disorder (ASD): Autism is genetically heterogeneous, with hundreds of associated genes (e.g., SHANK3, CHD8). Some of these genes are involved in neural crest cell migration, which is critical for forming facial bones and cartilage. Altered expression due to epigenetic changes (e.g., histone modification or DNA methylation) could subtly affect craniofacial structure, though this varies widely among individuals.
  • Epigenetic Stressors: Prenatal exposure to environmental factors (e.g., maternal stress, toxins, or nutritional deficiencies) can alter epigenetic markers, potentially affecting craniofacial development in both conditions. For example, folate deficiency during pregnancy has been linked to altered methylation patterns, which may influence facial symmetry or jaw development.

2. Developmental Muscle Use and Neuromuscular Factors


  • Reduced Muscle Engagement in ASD: As suggested in the query, some individuals with autism, particularly those who are non-verbal, may use facial and jaw muscles less for communication (e.g., speaking or expressive gestures). This reduced muscle activity could theoretically influence jaw and facial bone development, as mechanical stress from muscle use helps shape bone structure (a process called mechanotransduction). For example, less frequent chewing or speaking might lead to weaker jaw muscle development, potentially affecting mandibular growth.
  • Motor Challenges in Down Syndrome: Individuals with Down syndrome often have hypotonia (low muscle tone), which can affect the muscles of the face and mouth. Reduced muscle tone may lead to less robust stimulation of craniofacial bones during development, potentially contributing to features like a smaller midface or underdeveloped jaw.
  • Orofacial Motor Patterns: Both conditions may involve atypical orofacial motor patterns (e.g., atypical chewing, swallowing, or breathing patterns). For instance, mouth breathing, which is common in some individuals with Down syndrome due to airway issues, can alter tongue posture and lead to changes in dental arches or jaw alignment over time.

3. Prenatal and Early Developmental Factors


  • Embryonic Tissue Development: Craniofacial features are largely determined by the neural crest cells during embryonic development. In Down syndrome, the overexpression of genes on chromosome 21 can disrupt the migration or differentiation of these cells, leading to distinct facial characteristics. In autism, mutations in genes regulating neural crest development (e.g., PAX6 or HOX genes) might subtly affect facial symmetry or proportions in some individuals.
  • Intrauterine Environment: Factors like maternal inflammation, infection, or metabolic conditions (e.g., gestational diabetes) during pregnancy can influence fetal craniofacial development. These factors may disproportionately affect individuals with genetic predispositions, such as those with Down syndrome or autism-linked mutations.
  • Growth Hormone Dysregulation: In Down syndrome, growth hormone pathways may be disrupted, leading to slower or altered growth of facial bones. In autism, hormonal imbalances (e.g., oxytocin or serotonin signaling) could indirectly affect tissue development, though evidence is less clear.

4. Oral and Dental Development


  • Dental Anomalies in Down Syndrome: Individuals with Down syndrome often have delayed tooth eruption, smaller teeth, or missing teeth, which can affect jaw alignment and facial aesthetics. These traits stem from genetic disruptions and altered bone remodeling processes.
  • Bruxism in Autism: Some individuals with autism exhibit bruxism (teeth grinding), which can alter dental occlusion and jaw structure over time. Chronic bruxism may lead to changes in the temporomandibular joint (TMJ) or facial muscle strain, potentially affecting appearance.
  • Orthodontic Issues: Both conditions may be associated with malocclusion (misaligned teeth) due to atypical jaw growth or muscle tone. For example, a high-arched palate in Down syndrome or atypical tongue thrusting in autism can influence dental and facial structure.

5. Environmental and Behavioral Influences


  • Diet and Nutrition: In both autism and Down syndrome, dietary preferences or restrictions (e.g., sensory-based food aversions in autism or gastrointestinal issues in Down syndrome) may affect nutritional intake, which is critical for bone and tissue growth. For instance, deficiencies in calcium or vitamin D could impair craniofacial bone development.
  • Sensory Processing and Habits: In autism, sensory sensitivities may lead to habits like repetitive facial touching or atypical chewing patterns, which could influence muscle and bone development over time. In Down syndrome, repetitive behaviors or oral habits (e.g., tongue protrusion) may similarly affect orofacial structure.
  • Sleep and Airway Issues: Obstructive sleep apnea, more common in Down syndrome due to anatomical airway differences, can lead to mouth breathing, which alters tongue position and may contribute to a narrower palate or elongated face. Some individuals with autism may also have sleep-disordered breathing, with similar effects.

6. Syndromic and Comorbid Conditions


  • Associated Syndromes in Autism: Some individuals with autism have co-occurring genetic syndromes (e.g., Fragile X syndrome), which can include distinct craniofacial features like a long face or prominent ears. These are driven by specific genetic mutations affecting developmental pathways.
  • Midface Hypoplasia in Down Syndrome: The characteristic flat midface in Down syndrome is linked to underdevelopment of the maxilla, driven by genetic and cellular mechanisms. This can alter facial proportions and is a hallmark of the condition.
  • Connective Tissue Differences: In Down syndrome, connective tissue abnormalities (e.g., due to collagen gene dysregulation) can affect skin elasticity and facial structure, contributing to softer or less defined facial contours.

Notes and Considerations​


  • Variability: Both autism and Down syndrome are highly variable. Not all individuals with these conditions exhibit noticeable craniofacial differences, and many have typical or aesthetically pleasing features. The notion of "inoptimal lookism traits" is subjective and not universally applicable.
  • Avoiding Stigmatization: Framing physical differences as "bad" or "inoptimal" can perpetuate harmful stereotypes. Craniofacial features in these conditions are part of natural variation and do not inherently reflect health or value.
  • Research Gaps: While some studies link genetic and epigenetic factors to craniofacial development in Down syndrome, the evidence for autism is less consistent due to its heterogeneity. More research is needed to understand subtle developmental influences.
  • Ethical Framing: Discussions about appearance should focus on function (e.g., speech, chewing, breathing) and health rather than aesthetic judgments, as the latter can reinforce harmful societal biases.
down syndrome and autism are not comprable. all downies look the same due to chromosomes. autists have a range of appearances
 
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they dont always there is this kid on instagram who is non verbal htn+ and has teen girsl in his comments saying how handsome he is and that theyd like to marry him




look at the comments

averange anglo
 
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more.

they've got longer mid-face with more facial projection.
So I have adhd

I dont have a long midface, its infact short
I have a bulbous nose
I have alot of facial projection (high facial depth)
Not sure on ears

So almost every sign Hmm

Maybe this facial thing has high accuracy tbh
 
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as a celt ik so many subhuman celts and anglos. its just a lottery
i mean compared to like slavs anglos pretty gl ngl.

Its all an average, u cant tell me essex boys arent literally the top percentile worldwide or dutch guys, if u can slay in those lobbies u can slay everywhere
 
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Variability: Both autism and Down syndrome are highly variable. Not all individuals with these conditions exhibit noticeable craniofacial differences, and many have typical or aesthetically pleasing features. The notion of "inoptimal lookism traits" is subjective and not universally applicable.
Can the typical red Gatorade lips and unibrow be explained by this or is it just poor hygiene?
 
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Can the typical red Gatorade lips and unibrow be explained by this or is it just poor hygiene?
I'm assuming behavioural traits like licking lips every .2 seconds, sensory food preference like loving red40.
It's common in downsyndrome to have drier skin or mucous membranes due to connective tissue differences or thyroid dysfunction
 
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i mean compared to like slavs anglos pretty gl ngl.

Its all an average, u cant tell me essex boys arent literally the top percentile worldwide or dutch guys, if u can slay in those lobbies u can slay everywhere
only htns are. on average slavs are more dimorphic eyes wise, more forward grown, sloped forehead with nw0
 
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only htns are. on average slavs are more dimorphic eyes wise, more forward grown, sloped forehead with nw0
dude i made a thread on how average slavic guys look. Nah bro ive been in both lobbies, essex , netherlands and that whole small circle in west europe is like where 90% of chads come from.
 
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