All endogenous and exogenous hormones and where theyre structurally derived from - AI COMPILATION

eology

eology

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DHT — naturally occurring metabolite of testosterone. Formula: C19H30O2. Structurally, it is testosterone with the Δ4–5 double bond reduced to the 5α configuration.

Testosterone — naturally occurring androgen, produced mainly by the gonads. Formula: C19H28O2. It is a C19 steroid with a 17β-hydroxyl group and a ketone at C3.

Nandrolone — synthetic derivative of testosterone. Formula: C18H26O2. Its defining structural modification is the 19-nor configuration, meaning the C19 methyl group is absent.

Trenbolone — synthetic derivative of nandrolone. Formula: C18H22O2. It retains the 19-nor structure and contains three carbon-carbon double bonds.

Boldenone — synthetic derivative of testosterone. Formula: C19H28O2. It differs from testosterone mainly by having an additional double bond at Δ1.

Methenolone (Primobolan) — synthetic testosterone derivative. Formula: C22H32O2 for methenolone; pharmaceutical preparations are commonly administered as an ester. It contains a Δ1 double bond and a 1-methyl modification.

Drostanolone — synthetic DHT derivative. Formula: C20H32O2. It is essentially a 2α-methylated DHT derivative.

Mesterolone (Proviron) — synthetic DHT derivative. Formula: C20H32O2. It contains a 1α-methyl modification relative to DHT.

Stanozolol — synthetic DHT derivative. Formula: C21H32N2O. Its structure is substantially modified and contains a pyrazole ring.

Oxandrolone (Anavar) — synthetic DHT derivative. Formula: C19H30O3. It contains an additional oxygen in the steroid framework, replacing a carbon in the A-ring structure.

Methandienone (Dianabol) — synthetic testosterone derivative. Formula: C20H28O2. It has a Δ1 double bond and a 17α-methyl group.

Methandrostenolone — another name for methandienone; they are the same compound.

Oxymetholone (Anadrol) — synthetic DHT-related derivative. Formula: C21H32O3. It contains a 17α-methyl group and a 2-hydroxymethylene modification.

Turinabol — derivative of methandienone. Formula: C20H28ClO2. Its characteristic modifications include a 4-chloro group and a 17α-methyl group.

Fluoxymesterone (Halotestin) — synthetic testosterone derivative. Formula: C20H29FO3. It contains a 9α-fluoro group, a 17α-methyl group, and additional structural modifications around C11.

Methyltestosterone — methylated testosterone derivative. Formula: C20H30O2. Its defining modification is a 17α-methyl group.

Danazol — synthetic derivative related to ethisterone. Formula: C22H27NO2. It is substantially modified compared with the classical androgens and contains nitrogen.


The biosynthetic pathway is roughly:
Cholesterol → pregnenolone → progesterone → androstenedione → testosterone
From testosterone:
Testosterone → DHT through 5α-reductase
Testosterone → boldenone through introduction of a Δ1 double bond
Testosterone → methandienone through a Δ1 double bond + 17α-methylation


Another important branch is:
Testosterone → nandrolone (19-nor)
Nandrolone → trenbolone

Another family is derived structurally from DHT:
DHT → mesterolone
DHT → drostanolone
DHT → oxandrolone
DHT → stanozolol
DHT-related structures → oxymetholone

“19-nor” is one of the most important structural modifications.
Testosterone has 19 carbon atoms, while nandrolone has 18:
Testosterone: C19H28O2
Nandrolone: C18H26O2
The missing C19 methyl group substantially changes the molecule’s interaction with the androgen receptor and its metabolism.
 
Last edited:
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mirin the effort
DNR+will not read later
 
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AGA impact tierlist with 5ar inhibitors(fin/dut) intake as a parameter:


Lower intrinsic androgenic/hair risk:
Nandrolone
Methenolone
Oxandrolone
Turinabol
Boldenone

Intermediate:
Testosterone
Methandienone
Oxymetholone
Trenbolone

Higher:
Mesterolone
Drostanolone
Stanozolol
Fluoxymesterone

Very high:
DHT
 
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Why the fuck would I run down through ai slop?
 
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Why the fuck would I run down through ai slop?
its more of a very compact and precise log for me so i can go back and forth

if you alr know this data feel free to criticize it
 
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About how "DHT derivative ≠ DHT" in terms of hairloss aggravation


Being a “DHT derivative” does not mean a compound will have the same effect on hair as DHT itself.
DHT is a specific molecule with a particular three-dimensional structure and a very strong interaction with the androgen receptor (AR). A DHT-derived AAS can have structural modifications that change its AR binding, receptor activation, metabolism, and tissue selectivity.

For example, mesterolone, drostanolone, oxandrolone, and stanozolol are all structurally related to DHT, but they are not simply “DHT in another form.” Adding or removing even a relatively small functional group can substantially change how the molecule interacts with the AR.

For AGA, the important factor is the androgenic signal reaching susceptible hair follicles. The fact that a molecule originated from DHT does not tell you by itself how strong that signal will be.

So:
DHT derivative ≠ DHT
DHT-like structure ≠ automatically the same hair-loss potential

What matters more is the compound's intrinsic AR agonism in scalp follicles, its local metabolism, and the individual's genetic susceptibility to AGA.
This distinction is especially relevant when ignoring 5α-reductase. If 5AR conversion is removed from the equation, you're comparing the direct androgenic activity of the parent compounds themselves rather than asking how much testosterone gets converted into DHT.
 

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