Are roids gonna do jack shit for my bones at the big 18?

[samthechud]

Title

 

[itsoverforme333]

Title

Give up

 

[hezaymoglet]

Title

yes

 

[WilhelmThyWizard]

Clavicles and Dimo

 

[nathan V]

Bones in general nigger?

 

[samthechud]

Give up

no

 

[samthechud]

Bones in general nigger?

yes bones in general nigger

 

[itsoverforme333]

yes bones in general nigger

Roids won’t do shit to your ass its over

 

[160cmcurry]

no, they don’t. juicing doesn’t give bone growth, it gives the illusion it does by pumping up muscle mass around the frame so the bones look thicker and more pronounced under the skin. once your growth plates fuse thats it, no more lengthening. what you get is water retention, denser muscle fibers pulling harder on attachment points, and sometimes minor increases in bone mineral density from the extra stress and hormones.

 

[nathan V]

yes bones in general nigger

No nigger

 

[Greenguy]

Probably not, you can try but wouldnt recommend it as u would require some stuff you prob are way to afraid to take and u might not even get results from them..

 

[samthechud]

Probably not, you can try but wouldnt recommend it as u would require some stuff you prob are way to afraid to take and u might not even get results from them..

I'm not scared to do anything as long as I'm not lonely anymore

 

[samthechud]

I'm not scared to do anything as long as I'm not lonely anymore

genuinely don't give a fuck if I die at 30

 

[infra.nizar]

Title

growth plates at the ends of long bones fuse by age 18. this fusion stops bones from growing longer. hormonal substances will not restart this process. These substances increase muscle mass. they do not increase height after fusion. misusing such substances accelerates growth plate closure in teenagers. early closure permanently shortens adult stature. these substances disrupt natural hormone levels. disruption weakens the existing skeleton over time. long term use increases risks for bone fractures.
focus on weight bearing exercise and nutrition to maintain current bone density.

 

[Greenguy]

I'm not scared to do anything as long as I'm not lonely anymore

Alright. Injectable mtren, halo, tren, test, winstrol is prob a good stack. Since u dont give a fuck we will go for whats optimal. Use low test high tren so like 400tren 100test. Winstrol 10-20mg is enough. Halo 5-10mg, mtren I would go 250-750mcg. Go in cycles and do it smartly without fucking ur health and killing urself in the process. If dont correct its relatively safe.

 

[samthechud]

Alright. Injectable mtren, halo, tren, test, winstrol is prob a good stack. Since u dont give a fuck we will go for whats optimal. Use low test high tren so like 400tren 100test. Winstrol 10-20mg is enough. Halo 5-10mg, mtren I would go 250-750mcg. Go in cycles and do it smartly without fucking ur health and killing urself in the process. If dont correct its relatively safe.

aight thanks

 

[Greenguy]

growth plates at the ends of long bones fuse by age 18. this fusion stops bones from growing longer. hormonal substances will not restart this process. These substances increase muscle mass. they do not increase height after fusion. misusing such substances accelerates growth plate closure in teenagers. early closure permanently shortens adult stature. these substances disrupt natural hormone levels. disruption weakens the existing skeleton over time. long term use increases risks for bone fractures.
focus on weight bearing exercise and nutrition to maintain current bone density.


solution

Yo bro you are just saying rn that u dont know how to use roids correctly. Roids will be benefcial for density and final height if done correctly instead and if u did ur research instead of lurking on .org

 

[peterk1287]

Alright. Injectable mtren, halo, tren, test, winstrol is prob a good stack. Since u dont give a fuck we will go for whats optimal. Use low test high tren so like 400tren 100test. Winstrol 10-20mg is enough. Halo 5-10mg, mtren I would go 250-750mcg. Go in cycles and do it smartly without fucking ur health and killing urself in the process. If dont correct its relatively safe.

You can’t run halo, or Mtren long enough for any facial benefit lol

 

[Greenguy]

You can’t run halo, or Mtren long enough for any facial benefit lol

Who said so lmao

 

[Greenguy]

You can’t run halo, or Mtren long enough for any facial benefit lol

Use correct ancillaries and track ur bloodwork. From people ik who have taken it and myself bloodwork has been pretty clean lmao.

 

[RickDirer]

1g of tren and 100 mg of mk67 will do the job. pray to gandy and youll be okay.

 

[Greenguy]

1g of tren and 100 mg of mk67 will do the job. pray to gandy and youll be okay.

mk67 for boneessssssssss

 

[RickDirer]

mk67 for boneessssssssss

also opens growth plates btw

 

[infra.nizar]

Yo bro you are just saying rn that u dont know how to use roids correctly. Roids will be benefcial for density and final height if done correctly instead and if u did ur research instead of lurking on .org

u confuse bone density with linear height. steroids increase bone mineral density. they do not increase height after age 18.
doctors prescribe steroids to osteoporosis patients to thicken existing bone. increased density makes your skeleton heavier. Increased density fails to make a fused bone grow longer.
your final height depends on cartilage in your growth plates. excess testosterone from steroids converts into estrogen. estrogen triggers growth plates to calcify and close permanently.
once cartilage turns into solid bone, the window for linear growth closes forever. no protocol or compound recreates cartilage after fusion. you gain bone density. you do not change your height.

 

[Greenguy]

also opens growth plates btw

Ghrelin for sox9 and increased igf btw. Please buy my enclomiphene and mk67 from swisschems. Dont forget the rad140 for penis withdrawal

 

[samthechud]

u confuse bone density with linear height. steroids increase bone mineral density. they do not increase height after age 18.
doctors prescribe steroids to osteoporosis patients to thicken existing bone. increased density makes your skeleton heavier. Increased density fails to make a fused bone grow longer.
your final height depends on cartilage in your growth plates. excess testosterone from steroids converts into estrogen. estrogen triggers growth plates to calcify and close permanently.
once cartilage turns into solid bone, the window for linear growth closes forever. no protocol or compound recreates cartilage after fusion. you gain bone density. you do not change your height.

what about aromatase inhibitors for the estrogen part

 

[Greenguy]

u confuse bone density with linear height. steroids increase bone mineral density. they do not increase height after age 18.
doctors prescribe steroids to osteoporosis patients to thicken existing bone. increased density makes your skeleton heavier. Increased density fails to make a fused bone grow longer.
your final height depends on cartilage in your growth plates. excess testosterone from steroids converts into estrogen. estrogen triggers growth plates to calcify and close permanently.
once cartilage turns into solid bone, the window for linear growth closes forever. no protocol or compound recreates cartilage after fusion. you gain bone density. you do not change your height.

Now idk why u are setting ages. It depends on human to human, ofc if sutures are closed and growth plates are closed u wont find any benefit. There is no set age tho on when it closes, I have also heard not so sure but that dkk1 inhibitors with sclerostin inhibitors can theoritcally increase Appositional Growth, and that this has been seen in studies on apes not so sure tho.

 

[Greenguy]

what about aromatase inhibitors for the estrogen part

Depends each one has its specfic reasons and when on higher test doses u can also combine aromasin + letro/arimidex depending on ur situation.

 

[infra.nizar]

what about aromatase inhibitors for the estrogen part

growth plates determine your final height. these plates consist of cartilage near the ends of your long bones. bones stop growing once these plates fuse into solid bone. fusion typically finishes by age 18. no known substance restarts this biological process.
aromatase inhibitors stop the conversion of testosterone into estrogen. estrogen signals the body to close growth plates. inhibitors delay this closure only while plates remain open. they provide zero height benefits for adults

 

[Greenguy]

u confuse bone density with linear height. steroids increase bone mineral density. they do not increase height after age 18.
doctors prescribe steroids to osteoporosis patients to thicken existing bone. increased density makes your skeleton heavier. Increased density fails to make a fused bone grow longer.
your final height depends on cartilage in your growth plates. excess testosterone from steroids converts into estrogen. estrogen triggers growth plates to calcify and close permanently.
once cartilage turns into solid bone, the window for linear growth closes forever. no protocol or compound recreates cartilage after fusion. you gain bone density. you do not change your height.

OH also I want to add that if ur growth plates are open and u use roids correctly ull grow. This has been shown in studies countless times and it should work theoritically. I am not talking about sum test+anavar cycle btw.

 

[infra.nizar]

Now idk why u are setting ages. It depends on human to human, ofc if sutures are closed and growth plates are closed u wont find any benefit. There is no set age tho on when it closes, I have also heard not so sure but that dkk1 inhibitors with sclerostin inhibitors can theoritcally increase Appositional Growth, and that this has been seen in studies on apes not so sure tho.

an X ray confirms if your growth plates closed. age 18 is the biological average for fusion.
u correctly named appositional growth. appositional growth means bones grow in thickness it does not mean bones grow in length.
Dkk1 inhibitors and sclerostin inhibitors stimulate bone formation. tomosozumab is a sclerostin inhibitor used for osteoporosis. this drug activates the WNT signaling pathway.
studies show these inhibitors increase bone mass and bone thickness. they widen the bone shaft. they do not regenerate the epiphyseal cartilage needed for linear height.
thicker bones make you wider. thicker bones do not make you taller. no study shows these inhibitors restart longitudinal growth after fusion

 

[samthechud]

OH also I want to add that if ur growth plates are open and u use roids correctly ull grow. This has been shown in studies countless times and it should work theoritically. I am not talking about sum test+anavar cycle btw.

goddamn that's alot of shit to buy and to run but thanks for the info

 

[samthechud]

goddamn that's alot of shit to buy and to run but thanks for the info

gonna research and find sources in canada (specifically quebec)

 

[infra.nizar]

OH also I want to add that if ur growth plates are open and u use roids correctly ull grow. This has been shown in studies countless times and it should work theoritically. I am not talking about sum test+anavar cycle btw.

steroids compromise final height even when growth plates are open.
anabolic steroids accelerate the maturation of skeletal tissue. pediatric studies show these compounds cause premature growth plate fusion. medical pros avoid prescribing anabolic steroids to growing children due to this specific risk.
anabolic compounds trigger rapid skeletal maturation. this process forces growth plates to close early. u experience a brief burst of growth. This burst ends the natural growing period. final adult stature remains lower than your natural potential.
growth plates require cartilage to function. Steroids turn this cartilage into solid bone. this transformation is permanent. no known protocol reverses this fusion. using these substances on open growth plates causes irreversible height loss.
you prioritize short term muscle gains over your permanent skeleton.

 

[Greenguy]

an X ray confirms if your growth plates closed. age 18 is the biological average for fusion.
u correctly named appositional growth. appositional growth means bones grow in thickness it does not mean bones grow in length.
Dkk1 inhibitors and sclerostin inhibitors stimulate bone formation. tomosozumab is a sclerostin inhibitor used for osteoporosis. this drug activates the WNT signaling pathway.
studies show these inhibitors increase bone mass and bone thickness. they widen the bone shaft. they do not regenerate the epiphyseal cartilage needed for linear height.
thicker bones make you wider. thicker bones do not make you taller. no study shows these inhibitors restart longitudinal growth after fusion

Yes I do know that I mentioned it talking more about craniofacial ppositional growth. I believe it could be positivly influensed by this combo. https://pubmed.ncbi.nlm.nih.gov/38052372/

 

[Greenguy]

steroids compromise final height even when growth plates are open.
anabolic steroids accelerate the maturation of skeletal tissue. pediatric studies show these compounds cause premature growth plate fusion. medical pros avoid prescribing anabolic steroids to growing children due to this specific risk.
anabolic compounds trigger rapid skeletal maturation. this process forces growth plates to close early. u experience a brief burst of growth. This burst ends the natural growing period. final adult stature remains lower than your natural potential.
growth plates require cartilage to function. Steroids turn this cartilage into solid bone. this transformation is permanent. no known protocol reverses this fusion. using these substances on open growth plates causes irreversible height loss.
you prioritize short term muscle gains over your permanent skeleton.

Multiple studies show increased final height gng. If u are refering to stuff to anavar or hgh or test without ai that is indeed seen.

 

[Greenguy]

gonna research and find sources in canada (specifically quebec)

I gotta a source that ships worldwide if u want, add me on dc willysilly.00

 

[infra.nizar]

Yes I do know that I mentioned it talking more about craniofacial ppositional growth. I believe it could be positivly influensed by this combo. https://pubmed.ncbi.nlm.nih.gov/38052372/

Multiple studies show increased final height gng. If u are refering to stuff to anavar or hgh or test without ai that is indeed seen.

ur study proves my point. the title confirms blocking sclerostin and DKK1 causes skull bone thickening. this process is appositional growth. this growth widens your skull and jaw. appositional growth fails to make your long bones grow longer. study

regarding open plates, u misunderstand the medical data. anabolic steroids still compromise final height even when combined with aromatase inhibitors. estrogen closure is only one path. androgens directly accelerate growth plate senescence. this direct action burns through your remaining cartilage faster. study 1, study 2

pediatric doctors use high doses of growth hormone for specific medical deficiencies. they do not use standard bodybuilder steroid cycles. tdding an aromatase inhibitor protects your growth plates from estrogen conversion. the inhibitor fails to protect your cartilage from direct androgenic burnout. u sacrifice total height potential for a brief growth spurt. study 1, study 2

 

[peterk1287]

Who said so lmao

your retarted

 

[Greenguy]

ur study proves my point. the title confirms blocking sclerostin and DKK1 causes skull bone thickening. this process is appositional growth. this growth widens your skull and jaw. appositional growth fails to make your long bones grow longer. study

regarding open plates, u misunderstand the medical data. anabolic steroids still compromise final height even when combined with aromatase inhibitors. estrogen closure is only one path. androgens directly accelerate growth plate senescence. this direct action burns through your remaining cartilage faster. study 1, study 2

pediatric doctors use high doses of growth hormone for specific medical deficiencies. they do not use standard bodybuilder steroid cycles. tdding an aromatase inhibitor protects your growth plates from estrogen conversion. the inhibitor fails to protect your cartilage from direct androgenic burnout. u sacrifice total height potential for a brief growth spurt. study 1, study 2

I mean the sclerostin and DKK1 arent for height. I just mentioned it regarding potential skull results. Regarding roids there are humans with conditions where we have seen great results. They do have conditions but I believe based on my results and other anecdotal results along with mice studies that roids are the way in puberty.

Therapeutic effects of growth hormone combined with low-dose stanozolol on growth velocity and final height of girls with Turner syndrome - PubMed

In girls with TS, 9-12 years of age, rhGH combined with low-dose stanozolol may effectively increase growth. At least a 2-year course of this treatment may effectively improve FAH with proper delay of oestrogen-induced development.

pubmed.ncbi.nlm.nih.gov

Long-term results of treatment with low-dose fluoxymesterone in constitutional delay of growth and puberty and in genetic short stature - PubMed

This prospective study was designed to assess growth response, side effects, other possible long-term effects, and final adult height in boys treated with the oral androgen, fluoxymesterone. From 1973 to 1984, eighty-two short boys (71 with constitutional delay of growth and puberty [CDGP] and...

pubmed.ncbi.nlm.nih.gov

 

[ASM5]

Title

They did for me

 

[Greenguy]

I mean the sclerostin and DKK1 arent for height. I just mentioned it regarding potential skull results. Regarding roids there are humans with conditions where we have seen great results. They do have conditions but I believe based on my results and other anecdotal results along with mice studies that roids are the way in puberty.

Therapeutic effects of growth hormone combined with low-dose stanozolol on growth velocity and final height of girls with Turner syndrome - PubMed

In girls with TS, 9-12 years of age, rhGH combined with low-dose stanozolol may effectively increase growth. At least a 2-year course of this treatment may effectively improve FAH with proper delay of oestrogen-induced development.

pubmed.ncbi.nlm.nih.gov

Long-term results of treatment with low-dose fluoxymesterone in constitutional delay of growth and puberty and in genetic short stature - PubMed

This prospective study was designed to assess growth response, side effects, other possible long-term effects, and final adult height in boys treated with the oral androgen, fluoxymesterone. From 1973 to 1984, eighty-two short boys (71 with constitutional delay of growth and puberty [CDGP] and...

pubmed.ncbi.nlm.nih.gov

Stanozolol regulates proliferation of growth plate chondrocytes via activation of ERalpha in GnRHa-treated adolescent rats - PubMed

Improving the final adult height is one of the most important aims for treatment of central precocious puberty. Stanozolol (ST) is a synthetic derivative of androgen. In this study, we investigated the effects and the mechanisms of ST on the proliferation of growth plate chondrocytes isolated...

pubmed.ncbi.nlm.nih.gov

GnRHa/Stanozolol Combined Therapy Maintains Normal Bone Growth in Central Precocious Puberty - PMC

Gonadotropin-releasing hormone agonist (GnRHa) is the gold standard in the treatment of Central Precocious Puberty (CPP) with progressive puberty and accelerative growth. However, GnRHa treatment is reported to result in growth deceleration and ...

pmc.ncbi.nlm.nih.gov

THE EFFECT OF STANOZOLOL ON FINAL HEIGHT AND SKELETAL MATURATION IN TURNER'S SYNDROME - Pediatric Research

The effect of stanozolol on the final height and the skeletal maturation was studied in 35 patients with Turner's syndrome without spontaneous genital bleeding. All patients received stanozolol (1mg/day), estrogen and progesterone therapy. Bone age was evaluated by TW2 RUS method. Height SD...

www.nature.com

These are only 2 compounds I am using to show u some examples. Mechanisticly there are other roids that will benefit you, just that no good studies are present regarding them.

 

[Faustum]

Title

Your clavicles are influenced by IGF-1, not so much Testosterone. Same with your maxilla.

Testosterone drives growth of your mandible and brow ridge, although all growth requires adequate IGF-1.