bimatoprost mogs minoxicope. (Iris Heterochromia)

[KLEORB]

never in my life have ive seen someone get actual good results off minoxidil. its str8 bullshit.​

YES HALF OF THIS IS CHAT GPT IM TO CHUD TO GIVE U A FULL STUDY OFF HOMEMADE RESEARCH.
WILL I BE TRYING BITMATOPROST? yes.

Eyelashes: Bimatoprost vs No Treatment​

https://onlinelibrary.wiley.com/doi/10.1111/j.1346-8138.2012.01579.x
https://pmc.ncbi.nlm.nih.gov/articles/PMC4499467/ ( bimatoprost can cause iris color changes, which can result in heterochromia (different-colored eyes) if only one eye is exposed to the medication.)
Bimatoprost was originally developed as a glaucoma medication. When applied in or near the eye, it can increase brown pigment (melanin) in the iris over time. This effect:

• Is more likely in people with mixed-color irises (e.g., green-brown, hazel-brown, blue-brown).
• Usually develops gradually over months to years.
• May be permanent, even if the medication is stopped.
• Can lead to noticeable heterochromia if one eye is treated and the other is not.

For people using bimatoprost for eyelash growth (e.g., applied to the upper eyelid margin), the risk appears to be much lower than when the drops are placed directly into the eye, but it is still listed as a potential adverse effect because some medication can reach the ocular surface.


Other eye-area changes can include:

• Darkening of the eyelid skin.
• Increased pigmentation around the eye.
• Longer, thicker eyelashes.
• Rarely, deepening of the upper eyelid sulcus ("sunken-eye" appearance), sometimes called prostaglandin-associated periorbitopathy. (RARE)

Clinical trials of Bimatoprost showed approximately:

• +25% eyelash length
• +106% eyelash thickness/fullness
• +18% darkness
• 78% of users achieved a clinically meaningful improvement by 16 weeks.

Bimatoprost eyelash improvements at 16 weeks
Average changes reported in clinical studies.

Eyebrows: Bimatoprost vs Minoxidil​

A randomized study comparing 0.03% bimatoprost with 2% minoxidil for eyebrow hypotrichosis found:

Outcome at 4 months	Bimatoprost 0.03%	Minoxidil 2%
Patients with ≥1 grade improvement	100%	80%
Hair count increase	Higher numerically	Lower numerically
Hair diameter increase	Higher numerically	Lower numerically
Statistical superiority	No	No

human data testing?:

Treatment	Evidence Strength
Minoxidil	Strong, FDA-approved, decades of data
Bimatoprost	Experimental, limited human data. THIS TREAD WILL BE UPDATED

 

[aids]

Anybody that takes Bimatoprost doesn't value their orbital fat.
Just take the oral minoxidil and stop coping.

 

[KLEORB]

Anybody that takes Bimatoprost doesn't value their orbital fat.
Just take the oral minoxidil and stop coping.

JFL if u think minoxidil actually works u need to delete ur account iqlet

 

[KLEORB]

Anybody that takes Bimatoprost doesn't value their orbital fat.
Just take the oral minoxidil and stop coping.

also its ur fault if u dont have good enough orbital fat it rarely decreases it. do ur research kid.

 

[Gudliferr]

Bro uses chatgpt

 

[aids]

also its ur fault if u dont have good enough orbital fat it rarely decreases it. do ur research kid.

Speak coherent English so that people know what you're talking about enough to give you a proper reply.

JFL if u think minoxidil actually works u need to delete ur account iqlet

Feel free to show me literature or credible anecdotes where sustained oral minoxidil use was proven ineffective.

 

[KLEORB]

Bro uses chatgpt

yeah so autist like u will actually read it rather than just DNR.

 

[KLEORB]

Speak coherent English so that people know what you're talking about enough to give you a proper reply.


Feel free to show me literature or credible anecdotes where sustained oral minoxidil use was proven ineffective.

Compliance to Topical Minoxidil and Reasons for Discontinuation among Patients with Androgenetic Alopecia - PMC

This study assessed the levels of compliance to topical minoxidil (TM) among male and female patients with androgenetic alopecia (AGA) and analyzed the factors associated with minoxidil discontinuation. A retrospective study was conducted among 400 ...

pmc.ncbi.nlm.nih.gov

copy and paste kiddo.
Minoxidil isn't universally ineffective, but it fails for roughly 30-50% of users. It doesn't work for everyone because it requires a specific scalp enzyme (sulfotransferase) to convert the drug into its active form. Additionally, it only addresses the hair cycle, meaning any gains disappear if you stop using it.

 

[Gudliferr]

yeah so autist like u will actually read it rather than just DNR.

I read until u said chatgpt

 

[KLEORB]

Speak coherent English so that people know what you're talking about enough to give you a proper reply.


Feel free to show me literature or credible anecdotes where sustained oral minoxidil use was proven ineffective.

also ur stupid for saying i claimed it to be entirely ineffective. thats impossible its a SULT1A1 activator.

 

[KLEORB]

I read until u said chatgpt

good boy.

 

[aids]

Compliance to Topical Minoxidil and Reasons for Discontinuation among Patients with Androgenetic Alopecia - PMC

This study assessed the levels of compliance to topical minoxidil (TM) among male and female patients with androgenetic alopecia (AGA) and analyzed the factors associated with minoxidil discontinuation. A retrospective study was conducted among 400 ...

pmc.ncbi.nlm.nih.gov

copy and paste kiddo.
Minoxidil isn't universally ineffective, but it fails for roughly 30-50% of users. It doesn't work for everyone because it requires a specific scalp enzyme (sulfotransferase) to convert the drug into its active form. Additionally, it only addresses the hair cycle, meaning any gains disappear if you stop using it.

Congratulations for completely disregarding the highly relevant adjective "oral" in my reply, dipshit.
There are ways to increase efficacy of topical minoxidil that these pieces of literature disregard, regardless.
@Shobek

 

[KLEORB]

Congratulations for completely disregarding the highly relevant adjective "oral" in my reply, dipshit.
There are ways to increase efficacy of topical minoxidil that these pieces of literature disregard, regardless.
@Shobek

lol this dude did not even read it holy retard. too illiterate. ur too retarded to understand that its impossible for it to be ineffective. ur just putting words in my mouth LOL

 

[Gudliferr]

good boy.

Fuck you i bet your dad and uncle 2mand u when u were a child and thats ehy youre such a fucking fag kys

 

[KLEORB]

Fuck you i bet your dad and uncle 2mand u when u were a child and thats ehy youre such a fucking fag kys

DNR 190CMCEL

 

[aids]

lol this dude did not even read it holy retard. too illiterate. ur too retarded to understand that its impossible for it to be ineffective. ur just putting words in my mouth LOL

Feel free to show me literature or credible anecdotes where sustained oral minoxidil use was proven ineffective.

Compliance to Topical Minoxidil and Reasons for Discontinuation among Patients with Androgenetic Alopecia - PMC

This study assessed the levels of compliance to topical minoxidil (TM) among male and female patients with androgenetic alopecia (AGA) and analyzed the factors associated with minoxidil discontinuation. A retrospective study was conducted among 400 ...

pmc.ncbi.nlm.nih.gov

copy and paste kiddo.
Minoxidil isn't universally ineffective, but it fails for roughly 30-50% of users. It doesn't work for everyone because it requires a specific scalp enzyme (sulfotransferase) to convert the drug into its active form. Additionally, it only addresses the hair cycle, meaning any gains disappear if you stop using it.

It's implicit from the context of your ChatGPT-pasted reply that you're referring to topical minoxidil, not oral, as I mentioned, given enzymatic activity at the scalp is completely irrelevant when it comes to oral minoxidil use. You are an utter brainlet.
*In addition to the study literally being about topical minoxidil.

Further, this study, that you also obviously acquired from ChatGPT lacks relevance given it is in context of androgenetic alopecia (male pattern baldness), not eyebrow/eyelash density, which Bimatoprost addresses and is the reason for your thread.

No shit you genuine spastic, minoxidil does not address the root cause of androgenetic alopecia which is the presence of DHT in the body. Hence why I never recommended you use it for hair loss because that is not what this thread is regarding!

Please stop posting threads. Cheers.

 

[KLEORB]

It's implicit from the context of your ChatGPT-pasted reply that you're referring to topical minoxidil, not oral, as I mentioned, given enzymatic activity at the scalp is completely irrelevant when it comes to oral minoxidil use. You are an utter brainlet.

Further, this study, that you also obviously acquired from ChatGPT lacks relevance given it is in context of androgenetic alopecia (male pattern baldness), not eyebrow/eyelash density, which Bimatoprost addresses and is the reason for your thread.

No shit you genuine spastic, minoxidil does not address the root cause of androgenetic alopecia which is the presence of DHT in the body.

Please stop posting threads. Cheers.

lol this study wasnt from chat gpt it takes simple studies and quick research to realize its top prioritized. please delete your account.
its hypocritical also. You're conflating two issues. Whether the study is about androgenetic alopecia is a valid limitation when discussing eyebrows or eyelashes. But that doesn't mean scalp follicle sulfotransferase activity is irrelevant to oral minoxidil. Recent research has found associations between follicular sulfotransferase activity and response to oral minoxidil, suggesting local follicular enzyme activity may still play a role.

 

[KLEORB]

It's implicit from the context of your ChatGPT-pasted reply that you're referring to topical minoxidil, not oral, as I mentioned, given enzymatic activity at the scalp is completely irrelevant when it comes to oral minoxidil use. You are an utter brainlet.
*In addition to the study literally being about topical minoxidil.

Further, this study, that you also obviously acquired from ChatGPT lacks relevance given it is in context of androgenetic alopecia (male pattern baldness), not eyebrow/eyelash density, which Bimatoprost addresses and is the reason for your thread.

No shit you genuine spastic, minoxidil does not address the root cause of androgenetic alopecia which is the presence of DHT in the body. Hence why I never recommended you use it for hair loss because that is not what this thread is regarding!

Please stop posting threads. Cheers.

stick to basic studying rather than making two retarded separate claims. ur putting criticism on yourself.

 

[aids]

lol this study wasnt from chat gpt it takes simple studies and quick research to realize its top prioritized. please delete your account.
its hypocritical also. You're conflating two issues. Whether the study is about androgenetic alopecia is a valid limitation when discussing eyebrows or eyelashes. But that doesn't mean scalp follicle sulfotransferase activity is irrelevant to oral minoxidil. Recent research has found associations between follicular sulfotransferase activity and response to oral minoxidil, suggesting local follicular enzyme activity may still play a role.

I don't care for your recital of ChatGPT to argue with me.

If you actually had an IQ above 130, you would understand how easy it is to recognise patterns in AI-generated text and you would realise that it's blatantly obvious that you very quickly paraphrase your arguments after producing them with ChatGPT to respond to me.

Given you argue with AI rather than actually by yourself, you will never understand what I'm saying and always try to add some irrelevant nuance for you to be correct, since ChatGPT is very obviously going to support your entirely biased perspective because AI is not designed to disagree with you.

I am not responding further because it's implicit you're an absolute degenerate.

 

[KLEORB]

I don't care for your recital of ChatGPT to argue with me.

If you actually had an IQ above 130, you would understand how easy it is to recognise patterns in AI-generated text and you would realise that it's blatantly obvious that you very quickly paraphrase your arguments after producing them with ChatGPT to respond to me.

Given you argue with AI rather than actually by yourself, you will never understand what I'm saying and always try to add some irrelevant nuance for you to be correct, since ChatGPT is very obviously going to support your entirely biased perspective because AI is not designed to disagree with you.

I am not responding further because it's implicit you're an absolute degenerate.

LOL. i actually cant with you. you know nothing. I mog you regardless of youre claims DNR

 

[aids]

LOL. i actually cant with you. you know nothing. I mog you regardless of youre claims DNR

Yet you don't because I read your threads and you were being called a philtrumcel in your ratings.
Brutal mate. Maybe break HMTN before you develop an ego.
Enjoy your eventual LeFort with impaction or otherwise doomed philtrumcel future.

 

[KLEORB]

Yet you don't because I read your threads and you were being called a philtrumcel in your ratings.
Brutal mate. Maybe break HMTN before you develop an ego.
Enjoy your eventual LeFort with impaction or otherwise doomed philtrumcel future.

DNR my CTP is 2.23 (IDEAL)
that was 2 years back

 

[aids]

DNR my CTP is 2.23 (IDEAL)
that was 2 years back

All I hear are excuses.
Not replying any further to your attacks on character because you make it overtly implicit how you are incapable of engaging in genuinely productive discourse.

 

[zennn]

Nice avi