justmaxing
Iron
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- Feb 12, 2026
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I want to talk about my experience with using Serratiopeptidase for the purposes of bonesmashing for easier hematoma formation and the ossification of it.
About a week ago, I went to the doctor to get my leg stitched up, after a deep cut (luckily the Achilles tendon was only lightly grazed, and didn’t require surgery). After the doctor stitched me up, he told me to take this medication called Serratiopeptidase, which I’ve been taking for the past 8 days.
When you take Serratiopeptidase, you are introducing an enzyme that actively breaks down fibrin networks in the body. When you take hits during bonesmashing, your capillaries rupture and your body quickly forms a fibrin plug to seal the microtears in the vessels. Serratiopeptidase disrupts that initial clotting process, allowing the hematomas to form more easily, therefore getting more results and avoiding the need to go hard enough to damage the skin.
In my experience while bonesmashing this week, taking Serratiopeptidase made deep hematomas form far easier and much faster than usual, even from pretty weak hits, because the damaged vessels kept leaking blood into the subperiosteal tissue layer without being clogged by fibrin.
The theory I believe bonesmashing follows is the hematoma ossification theory, not the Wolff’s law. So the plan I now have is to keep combining daily bonesmashing with blood thinners even after finishing my 10 day Serratiopeptidase course, something like Aspirin could be more sustainable. I’ll do 81 mg Aspirin for the next couple of weeks while I’m healing my leg at home to maximize the volume of trapped blood under the periosteum after hitting the bone, forcing a massive subperiosteal hematoma that will eventually calcify and ossify into permanent bonelike tissue.
About a week ago, I went to the doctor to get my leg stitched up, after a deep cut (luckily the Achilles tendon was only lightly grazed, and didn’t require surgery). After the doctor stitched me up, he told me to take this medication called Serratiopeptidase, which I’ve been taking for the past 8 days.
When you take Serratiopeptidase, you are introducing an enzyme that actively breaks down fibrin networks in the body. When you take hits during bonesmashing, your capillaries rupture and your body quickly forms a fibrin plug to seal the microtears in the vessels. Serratiopeptidase disrupts that initial clotting process, allowing the hematomas to form more easily, therefore getting more results and avoiding the need to go hard enough to damage the skin.
In my experience while bonesmashing this week, taking Serratiopeptidase made deep hematomas form far easier and much faster than usual, even from pretty weak hits, because the damaged vessels kept leaking blood into the subperiosteal tissue layer without being clogged by fibrin.
The theory I believe bonesmashing follows is the hematoma ossification theory, not the Wolff’s law. So the plan I now have is to keep combining daily bonesmashing with blood thinners even after finishing my 10 day Serratiopeptidase course, something like Aspirin could be more sustainable. I’ll do 81 mg Aspirin for the next couple of weeks while I’m healing my leg at home to maximize the volume of trapped blood under the periosteum after hitting the bone, forcing a massive subperiosteal hematoma that will eventually calcify and ossify into permanent bonelike tissue.
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