Cycle help

reidm10Z!

reidm10Z!

Iron
Joined
Aug 2, 2025
Posts
161
Reputation
84
I’m going to run 10-20mg anavar and maybe 6-8iu of growth added on with basic add ons for liver nac and Tudca what should I add for height?
 
Last edited:
Please don’t be retarted. You can’t run var without a test base. GH is literally 4x the price of test and you still gotta inject it. On top of that you don’t even gotta refrigerate test. Get a test base of like 200 and then use your var. TBH you should just be running a test cycle for your first cycle. I would recommend low dose test for your first cycle, test out how your body reacts. Then run test with low dose var. your gonna make way more gains on a test only cycle than var anyways. Btw if you run anavar only, your test will crash and once you hop of var you will feel like shit and turn into a skeleton, losing all your gains.
High IQ greycel
 
They quite literally do grow endlessly LOL there's documented growth and xrays that show even at 24 they have open plates.

Also iss kids are normal, they don't have receptor issues otherwise they wouldn't be idiopathically short.. you know we can literally test receptor sensitivity..:lul::lul:

Also rhGH alone won't do much, it's a package with ai's.
Those kids don’t grow endlessly dumbass. Some don’t even end up tall, open plates doesn’t mean height growth in those cases. Even if you put rhGH and AI’s together you still aren’t getting significant height growth. You have no idea about the complexity of bone plate maturation. It’s not only got to do with e2. Those kids with iss are not normal, it’s a diagnosis of exclusion. If that were the case then normal kids would also grow significantly with this stack. That’s not the case. In the end it’s all about genes, if you are a normal kid (like op) you’re not getting any significant reliable height growth from your theoretical stack. You’re just putting your plates at risk and getting at risk of stunting your growth. Those kids in those trials also are heavily supervised, their e2 remains in healthy ranges. Your “height stack” has no real definitive proof. If it did, big pharma would be pushing the shit out of it anyways. Look at all these desperate 5’7 kids doing anything they can to even grow an inch.
 
Those kids don’t grow endlessly dumbass. Some don’t even end up tall, open plates doesn’t mean height growth in those cases. Even if you put rhGH and AI’s together you still aren’t getting significant height growth. You have no idea about the complexity of bone plate maturation. It’s not only got to do with e2. Those kids with iss are not normal, it’s a diagnosis of exclusion. If that were the case then normal kids would also grow significantly with this stack. That’s not the case. In the end it’s all about genes, if you are a normal kid (like op) you’re not getting any significant reliable height growth from your theoretical stack. You’re just putting your plates at risk and getting at risk of stunting your growth. Those kids in those trials also are heavily supervised, their e2 remains in healthy ranges. Your “height stack” has no real definitive proof. If it did, big pharma would be pushing the shit out of it anyways. Look at all these desperate 5’7 kids doing anything they can to even grow an inch.
More broscience that you haven't backed just claims. Lets make a list of your unjustifiable claims.

"You have no idea about the complexity of bone plate maturation" (this isn't an argument)
"Those kids with iss are not normal" (zero reasoning)
"if that were the case then normal kids would also grow significant with this. That's not the case." (zero evidence for this)
"You're just putting your plates at risk and getting at risk of stunting your growth." (zero evidence for this)
"Your "height stack" has no real definitive proof. If it did, big pharma would be pushing the shit out of it anyways."

(more proof you have ZERO knowledge in this topic at all. That's an abundance of studies of them using aromatase inhibitors for people with FMPP and decrease bone age advancement. The issue is they didn't use rhGH to add to the stem cell pool and induce proliferation. It's like delaying closure without the nutrients needed to actually grow. This is where rhGH comes in. https://pubmed.ncbi.nlm.nih.gov/20489637/)

"in the end it's all about genes, if you are a normal kid (like op) you're not getting any significant reliable height growth from your theoretical stack" (it's not all about genes, it's actually mainly about hormones and gene expression/activation. Height is majorly a hormone-driven process as people can have shitty height genes and history and still end up tall. It just depends on individuality of genes and hormones which can directly be influenced by taking unnatural amounts of hormones that induce growth, whilst also messing with hormones that end growth. i.e. igf-1/estradiol)


I'm not responding until you link a study for anything you say, DNR retard you know nothing.
 
More broscience that you haven't backed just claims. Lets make a list of your unjustifiable claims.

"You have no idea about the complexity of bone plate maturation" (this isn't an argument)
"Those kids with iss are not normal" (zero reasoning)
"if that were the case then normal kids would also grow significant with this. That's not the case." (zero evidence for this)
"You're just putting your plates at risk and getting at risk of stunting your growth." (zero evidence for this)
"Your "height stack" has no real definitive proof. If it did, big pharma would be pushing the shit out of it anyways."

(more proof you have ZERO knowledge in this topic at all. That's an abundance of studies of them using aromatase inhibitors for people with FMPP and decrease bone age advancement. The issue is they didn't use rhGH to add to the stem cell pool and induce proliferation. It's like delaying closure without the nutrients needed to actually grow. This is where rhGH comes in. https://pubmed.ncbi.nlm.nih.gov/20489637/)

"in the end it's all about genes, if you are a normal kid (like op) you're not getting any significant reliable height growth from your theoretical stack" (it's not all about genes, it's actually mainly about hormones and gene expression/activation. Height is majorly a hormone-driven process as people can have shitty height genes and history and still end up tall. It just depends on individuality of genes and hormones which can directly be influenced by taking unnatural amounts of hormones that induce growth, whilst also messing with hormones that end growth. i.e. igf-1/estradiol)


I'm not responding until you link a study for anything you say, DNR retard you know nothing.
You’re mixing up how it works in theory vs what actually happens in studies.


Yeah, GH boosts IGF-1 and growth rate, and AIs can slow bone age. That part is true. But that doesn’t automatically mean you get a big increase in final adult height.


Here’s what the actual data shows:






Main thing you’re missing:


You’re treating “faster growth + delayed bone age” like it automatically = big extra adult height. The long-term data doesn’t really support that.


And if it actually gave big reliable gains in normal kids, it would already be standard practice everywhere. It isn’t.


So yeah, it can tweak growth patterns, but it’s not the reliable “height stack” you’re making it out to be.
 

Similar threads

A
Replies
7
Views
37
someonesomeday
someonesomeday
foiddestoryer
Replies
3
Views
43
Aenterion2
A
someonesomeday
Replies
17
Views
71
Aenterion2
A
A
Replies
10
Views
38
alex.135
A
A
Replies
4
Views
25
alex.135
A

Users who are viewing this thread

Back
Top