jotunnr
Jinn
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I'm too tired to format this, and I've been researching this subject for god knows how long. I've probably made at least 3 threads on this prior
TABLE OF CONTENTS
INTRODUCTION
OSTEOGENESIS
PERIOSTEAL HEMATOMAS
SENESCENT MECHANOTRANSDUCTION
TABLE OF CONTENTS
INTRODUCTION
OSTEOGENESIS
PERIOSTEAL HEMATOMAS
SENESCENT MECHANOTRANSDUCTION
First off, there're 2 ways to achieve "bone growth"
1- osteogenesis
2- periosteal hematomas
Osteogenesis is utilized using controlled, cyclic pressure. Not mindless hammering. This is real bone, and the mechanism turns past 18 to retaining mineral density.
Periosteal hematomas are coagulations of blood pooling in due to rapid, high impact force. This is what most of you are experiencing. Bad news is, if you don't stay consistent and on point, these blood coagulations drain.
I've already posted this a few times but people trust you more when you have an anecdote for obvious reasons
To start off, if you've ever hammered, you were NOT CREATING BONE OR SCAR TISSUE!
Bone osteogenesis occurs under controlled conditions that most of you have never came close to achieving.
Bone responds cellularly at the sweet spot of 0.5-0.9MPa's, this is roughly the exact same force of a typical massage gun if you put a little strength to it yourself.

To put that into comparison, the average force you're producing with your knuckles OR hammer is around 3-5MPa's, which fits EXACTLY into the periosteal hematoma category (will talk about this later)


There's a thin line between perfect bone ostoeblast differentiation and bone RESORPTION, showing losses in both BMD and periosteal formation
Not to mention, you don't need to fucking bonesmash for 100 hours a day and 725 hours a week, it genuinely takes MINUTES to activate bone osteoblast differentiation
You also don't need to fist your skull to the point of bleeding either.
Unlike hematomas, bone formation stops and osteoblasts desensitize after a while of repeat pressure, EVEN if it's dynamic. If you want GENUINE growth, you're going to have to cycle it on and off. 8 weeks on, 2 weeks off, 8 weeks on, 2 weeks off.
CONCLUSION
1- massage gun with a bit of steady force from your hand for a few minutes everyday (1-2 times a day)
2- 8 weeks smashing, 2 weeks off, 8 weeks smashing, 2 weeks off. Cycle it.
3- More, prolonged pressure =/ more appositional growth
Periosteal hematomas are what ALL of you are experiencing, and im not saying it's bad, but it's not real bone.
There are two phases of periosteal hematomas:
1- Firmness
You just got done hammering, it's completely red, swollen and soft to the touch. This is histamines and macrophages pulling up to the scene, nothing more
2- Calcification
Your body recognizes that the area is bruised, and fills the bruising with pools of blood. The blood coagulates, hardens and locks. This mimics bone, but it is NOT bone.
The pattern on these is unpredictable, and it explains why some might end up with worse looking morphological changes than others.
These pools of blood usually take a few years to drain IF left untouched. Most of you will not leave it untouched.
Unlike osteogenesis, this mechanism does not disappear. Sure, bone cells get desensitized on a cellular level, but it's not even about bone. As long as you're causing physical trauma, blood is going to keep pooling in.
However, you obviously risk nerve damage to an extent.
Asymmetry's extremely rare and not as common as retards think, most people feel like they're asymmetrical because more than likely they have a skeletal cant and/or some sort of muscle imbalance that doesn't show in the beginning because they have ZERO angularity.
To stabilize this injured pocket, the periosteum immediately begins throwing down disorganized, woven bone. This hardens into lumpy, bony bumps.
CONCLUSION:
- hammer twice a day until the area swells, use the swelling as an indicator but also use your brain
- fluctuate the strength of your hits evenly on both sides of your face
- risk nerve damage and deep bruising that leaves you in permanent chronic pain.
The reason adult bones are less responsive to mechanical stimulation and slowly lose density as we get older is due to cellular senescence, your cytoskeletons stiffen, making it way harder for fluid to move due to pressure
Not to mention, they also enhance the secretion of SASPs, which greatly encourage osteoclast activity (bone-breaking cells)
If you've been using low pressure and your sutures are fused, this is ONE HUNDRED percent the issue as to why you're not experiencing any growth. At that point, your only option is hematomas.
SOURCES:
- https://journals.sagepub.com/doi/pdf/10.1177/20417314231172573
- https://pmc.ncbi.nlm.nih.gov/articles/PMC10214107/
- https://pmc.ncbi.nlm.nih.gov/articles/PMC9306018/
- https://peerj.com/articles/19310.pdf
https://pubmed.ncbi.nlm.nih.gov/28447744/
HONORABLE RETARD LTN MENTIONS @Idontknow- @LongForgotten